Search results for "Acellular"

showing 10 items of 1986 documents

Analysis of liver-specific protein LSP using murine monoclonal antibodies.

1987

. We describe twenty murine monoclonal antibodies directed against different antigenic determinants of human and rabbit liver-specific protein LSP. Among them, nine were directed against liver-specific epitopes as judged from immunohistological studies. Immunoelectronmicroscopy revealed that seven of these monoclonals recognized membrane determinants differing in staining of distinct areas of the hepatocellular surface. Eleven antibodies were directed against intracellular structures. Western blot analysis showed that the epitopes detected were displayed on either single or multiple protein bands with apparent molecular weights between 24 000 and 60 000. Further differences were observed wi…

medicine.drug_classClinical BiochemistryMonoclonal antibodyBiochemistryEpitopeEpitopesMiceWestern blotAntigenmedicineAnimalsHumansbiologymedicine.diagnostic_testMolecular massAntibodies MonoclonalMembrane ProteinsProteinsGeneral MedicineMolecular biologyImmunohistochemistryStainingLiverAntigens Surfacebiology.proteinRabbitsAntibodyIntracellularEuropean journal of clinical investigation
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Stimulation with carbachol alters endomembrane distribution and plasma membrane expression of intracellular proteins in lacrimal acinar cells.

2000

The events that lead to Sjogren's autoimmune processes in the lacrimal gland remain poorly understood. The acinar cell's responses to acute cholinergic stimulation include release of secretory products across the apical plasma membrane (apm) and a number of processes related to traffic between endomembrane compartments and the basal-lateral plasma membranes (blm), such as recruitment of Na, K-ATPase, accelerated recycling, and accelerated transcytosis of secretory IgA. We tested the hypothesis that stimulation-induced acceleration of endomembrane traffic is accompanied by changes in compartmentation and increased blm expression of proteins that are normally sequestered in endomembrane compa…

medicine.medical_specialtyAcid PhosphataseImmunoblottingGolgi ApparatusStimulationBiologyCholinergic AgonistsCathepsin BCathepsin BCellular and Molecular Neurosciencesymbols.namesakeInternal medicinemedicineAcinar cellAnimalsEndomembrane systemCells Culturedrab5 GTP-Binding ProteinsDifferential centrifugationEnzyme PrecursorsCell MembraneHistocompatibility Antigens Class IIMembrane Proteinsalpha-GlucosidasesGolgi apparatusGalactosyltransferasesCathepsinsSensory SystemsStimulation Chemicalbeta-N-AcetylhexosaminidasesCell biologyOphthalmologyEndocrinologySjogren's SyndromeTranscytosisrab GTP-Binding ProteinssymbolsCarbacholElectrophoresis Polyacrylamide GelFemaleRabbitsSodium-Potassium-Exchanging ATPaseIntracellularExperimental eye research
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Inhibition of Delta-like 4 mediated signaling induces abortion in mice due to deregulation of decidual angiogenesis.

2013

Objective: To explore whether the Dll4/Notch1 pathway plays a key role in regulating the vascular endothelial growth factor (VEGF)/VEGF receptor 2 (VEGFR2) driven decidual angiogenesis and related pregnancy through induction of a tip/stalk phenotype. Methods: Progesterone-replaced ovariectomized pregnant mice received a single injection of YW152F (Dll4 blocking antibody, BAb) or placebo at embryonic day (E) 4.5. Animals were sacrificed at different time points; blood and uterus were collected for further analysis. Number of embryos and implantation site, uteri weight, and serum progesterone levels were assessed. Alterations in the tip/stalk phenotype were determined by quantitative immunofl…

medicine.medical_specialtyAngiogenesisNotch pathwayNotch signaling pathwayUterusEmbryonic DevelopmentNeovascularization PhysiologicApoptosisGestational AgeDll4BiologyPregnancy disruptionAndrologychemistry.chemical_compoundMicePregnancyInternal medicinemedicineDeciduaAnimalsAntibodies BlockingAdaptor Proteins Signal TransducingCell ProliferationCell growthDeciduaCalcium-Binding ProteinsIntracellular Signaling Peptides and ProteinsObstetrics and GynecologyMembrane ProteinsEmbryoVEGFVascular Endothelial Growth Factor Receptor-2Vascular endothelial growth factorDisease Models Animalmedicine.anatomical_structureEndocrinologyReproductive MedicinechemistryApoptosiscardiovascular systemEmbryo LossFemaleAngiogenesisDevelopmental BiologySignal TransductionPlacenta
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Exercise as a Model to Study Oxidative Stress

2011

Physical exercise generates free radicals. The major source of radicals in exercise appears to be extracellular. Our experiments show that xanthine oxidase is a key player in the generation of superoxide during exercise. Mitochondrial contribution appears to be less important: during high oxygen utilization by mitochondria in state 3, the proportion of oxygen that is converted to superoxide is on an order of magnitude lower than in resting, state 4 conditions. Exercise-induced radicals constitute a double-edged sword: high intensity ­exercise causes the generation of relatively high concentrations of radicals that cause oxidative stress and eventually damage. On the other hand, low intensit…

medicine.medical_specialtyAntioxidantChemistrySuperoxideRadicalmedicine.medical_treatmentPhysical exerciseMitochondrionmedicine.disease_causechemistry.chemical_compoundEndocrinologyInternal medicinemedicineExtracellularXanthine oxidaseOxidative stress
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Signal transduction in isolated fat body from the cockroach Blaptica dubia exposed to hypertrehalosaemic neuropeptide

1998

Hypertrehalosaemic hormones stimulate trehalogenesis while inhibiting glycolysis in cockroach fat body. Signal transduction of the hypertrehalosaemic peptide Bld HrTH was examined in isolated fat body of the Argentine cockroach Blaptica dubia with respect to its effects on the increase in trehalose production and decrease in the content of the glycolytic activator fructose 2,6-bisphosphate in the tissue. Cyclic AMP does not seem to be involved in these processes as the cAMP analogue cpt-cAMP and the phosphodiesterase inhibitor IBMX, which both permeate cell membranes, had no effect on either parameter. Octopamine at physiological concentrations (10−7 mol · l−1) was also ineffective, but at …

medicine.medical_specialtyBlaptica dubiaIBMXbiologyPhysiologychemistry.chemical_elementFructoseCalciumbiology.organism_classificationBiochemistryTrehaloseCytosolchemistry.chemical_compoundEndocrinologyEndocrinologyBiochemistrychemistryInternal medicinemedicineExtracellularAnimal Science and ZoologyGlycolysisEcology Evolution Behavior and SystematicsJournal of Comparative Physiology B: Biochemical, Systemic, and Environmental Physiology
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Decidual endothelial cells express surface-bound C1q as a molecular bridge between endovascular trophoblast and decidual endothelium.

2008

This study was prompted by the observation that decidual endothelial cells (DECs), unlike endothelial cells (ECs) of blood vessels in normal skin, kidney glomeruli and brain, express surface-bound C1q in physiologic pregnancy. This finding was unexpected, because deposits of C1q are usually observed in pathologic conditions and are associated with complement activation. In the case of DECs, we failed to detect immunoglobulins and C4 co-localized with C1q on the cell surface. Surprisingly, DECs expressed mRNA for the three chains of C1q and secreted detectable level of this component in serum-free medium. The ability to synthesize C1q is acquired by DECs during pregnancy and is not shared by…

medicine.medical_specialtyC1q; Trophoblast; Endothelial cells; GlycosaminoglycansEndotheliumBlood VesselEndothelial cellsCellImmunologychemical and pharmacologic phenomenaBiologyurologic and male genital diseasesArticleEndothelial cellimmune system diseasesPregnancyInternal medicineparasitic diseasesmedicineCell AdhesionDeciduaHumansReceptorCell adhesionskin and connective tissue diseasesMolecular BiologyC1qGlycosaminoglycansC1q; Endothelial cells; Glycosaminoglycans; Trophoblast; Blood Vessels; Cell Adhesion; Complement C1q; Decidua; Endothelial Cells; Female; Humans; Membrane Glycoproteins; Pregnancy; Receptors Complement; Trophoblasts; Molecular Biology; ImmunologyEndothelial CellMembrane GlycoproteinsComplement C1qDeciduaTrophoblastTrophoblastComplement systemCell biologyTrophoblastsReceptors Complementmedicine.anatomical_structureEndocrinologyGlycosaminoglycanBlood VesselsFemaleMembrane GlycoproteinIntracellularHuman
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Antiapoptotic effect of calcitonin gene-related peptide on oxidative stress-induced injury in H9c2 cardiomyocytes via the RAMP1/CRLR complex.

2005

Calcitonin gene-related peptide (CGRP) plays an important role in the mediation of protective effects observed in situations such as ischemic preconditioning in rat hearts. In this study, we investigated in H9c2 rat cardiomyoblasts if the protective effect of CGRP could be linked to an inhibitory effect on the apoptotic pathway. We also determined the specificity of observed effects by treatment with adrenomedullin (ADM) in stress conditions generated by 100 microM hydrogen peroxide. Using MTT assays, we demonstrate that a pretreatment with CGRP decreases by half the loss of cell viability induced by H(2)O(2). CGRP inhibits phosphatidylserine externalization, caspase 3 activation and DNA fr…

medicine.medical_specialtyCalcitonin Gene-Related PeptideCaspase 3DNA FragmentationCalcitonin gene-related peptideReceptor Activity-Modifying Protein 2Receptor Activity-Modifying Protein 3Receptor Activity-Modifying ProteinsCell LineReceptor Activity-Modifying Protein 1Internal medicinemedicineAnimalsMyocytes CardiacViability assayMolecular BiologyReceptor activity-modifying proteinintegumentary systemChemistryCalcitonin Receptor-Like ProteinIntracellular Signaling Peptides and ProteinsMembrane ProteinsReceptors CalcitoninPeptide FragmentsRatsAdrenomedullinOxidative StressEndocrinologyGene Expression RegulationRAMP2ApoptosisRAMP1Multiprotein ComplexesIschemic Preconditioning MyocardialCardiology and Cardiovascular MedicineMioticsSignal TransductionJournal of molecular and cellular cardiology
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Endothelial Cell Swelling and Brain Perfusion

1997

Background: Whereas the contribution of glial swelling to no-reflow conditions in the ischemic penumbra or during reperfusion after global ischemia is widely discussed, little is known about cell volume control of endothelial cells under reperfusion conditions. Methods: The effect of extracellular acidosis-a key mediator of secondary brain damage-on cell volume was studied in the GM7373 endothelial cell line. Experiments were performed at pH = 6.0 in the presence or absence of bicarbonate, and during exposure to inhibitors of specific transport systems such as ethyl isopropyl amiloride or 4,4'-diisothiocyanatostilbene-2,2'-disulfonic acid. Results: Endothelial swelling to 111.1 ± 3.4% was f…

medicine.medical_specialtyCell Membrane PermeabilityBicarbonateIschemiaPharmacologyBrain IschemiaCell Linechemistry.chemical_compoundmedicineExtracellularAnimalsCell Sizebusiness.industryMicrocirculationPenumbraHydrogen-Ion Concentrationmedicine.diseaseAmilorideSurgeryEndothelial stem cellchemistryCerebrovascular CirculationReperfusion InjuryCattleEndothelium VascularSwellingmedicine.symptomAcidosisbusinessIsopropylmedicine.drugThe Journal of Trauma: Injury, Infection, and Critical Care
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Adaptation of neuronal cells to chronic oxidative stress is associated with altered cholesterol and sphingolipid homeostasis and lysosomal function

2009

Chronic oxidative stress has been causally linked to several neurodegenerative disorders. As sensitivity for oxidative stress greatly differs between brain regions and neuronal cell types, specific cellular mechanisms of adaptation to chronic oxidative stress should exist. Our objective was to identify molecular mechanisms of adaptation of neuronal cells after applying chronic sublethal oxidative stress. We demonstrate that cells resistant to oxidative stress exhibit altered cholesterol and sphingomyelin metabolisms. Stress-resistant cells showed reduced levels of molecules involved in cholesterol trafficking and intracellular accumulation of cholesterol, cholesterol precursors, and metabol…

medicine.medical_specialtyCell typeCerebellumLipid metabolismBiologymedicine.disease_causeBiochemistrySphingolipidCellular and Molecular Neurosciencemedicine.anatomical_structureEndocrinologyCell cultureInternal medicinemedicineIntracellularOxidative stressHomeostasisJournal of Neurochemistry
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The Homeostasis of Brain Choline

1993

The interest in the homeostasis of brain choline is reinforced by the role of choline as immediate precursor of acetylcholine, phosphatidylcholine and other phospholipids in the brain. In order to obtain a comprehensive view of the mochanisms of homeostasis it appeared necessary to elucidate the negative arteriovenous difference of choline across the brain (net release), a phenomenon that has been known for 20 years and is present in mammals and in man. This finding prompted an intense search for a de novo synthesis of choline in the brain. We detected in anaesthetized rats a reversal of the net release into a net uptake (positive arterio-venous difference), when the plasma level of choline…

medicine.medical_specialtyChemistryDe novo synthesischemistry.chemical_compoundEndocrinologyPhosphatidylcholineInternal medicinemedicineExtracellularPhosphorylationCholineAcetylcholineHomeostasismedicine.drugPhosphocholine
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