Search results for "Acetyltransferases"

showing 10 items of 69 documents

The effects of a number of short-term exogenous stimuli on pineal serotonin-N-acetyltransferase activity in rats

1984

The present study deals with the question as to what extent the sympathetically innervated rat pineal gland is affected by a number of short-term exogenous stimuli given during day-time, as assessed by measuring pineal serotonin-N-acetyltransferase activity (NAT) which is directly proportional to melatonin formation. In male Sprague-Dawley rats kept under LD 12:12 pineal NAT was statistically significantly depressed by physical immobilization for 2 hours, swimming for 15 min in water of 10 and 30 degrees C, exposure for 2 hours to cold (5 degrees C) or heat (40 degrees C), noise (90 db) for 2 hours and hunger for 17 hours. An increase in NAT was noted after swimming for 15 min in water of 2…

Maleendocrine systemmedicine.medical_specialtyFood deprivationHot TemperatureArylamine N-AcetyltransferaseHypoglycemiaBiologyPineal GlandThirstRat Pineal GlandImmobilizationAcetyltransferasesStress PhysiologicalInternal medicinemedicineAnimalsInsulinSwimmingBiological PsychiatryNormal rangeAnestheticsWater DeprivationRats Inbred StrainsPhysical Immobilizationmedicine.diseaseHypoglycemiaRatsCold TemperaturePsychiatry and Mental healthEndocrinologyMelatonin formationNeurologyNeurology (clinical)medicine.symptomSerotonin N-acetyltransferase activityFood DeprivationNoiseJournal of Neural Transmission
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The effects of short pulses of light at night on numbers of pineal "synaptic" ribbons and serotonin N-acetyltransferase activity in male Sprague-Dawl…

1986

To characterize further the functionally enigmatic "synaptic" ribbons (SR) of the mammalian pineal gland and to study possible relationships to melatonin synthesis, in the present investigation rats were exposed to short pulses of light at night when both SR numbers and serotonin N-acetyltransferase (NAT) activity are high in comparison to day-time values. Male Sprague-Dawley rats were killed at 13:00 and 01:00 h, respectively, and at 01:10 and 02:00 h after exposure to light for 10 and 60 min, respectively. The pineals were rapidly taken out and cut sagittally in half. One half was processed for electron-microscopic quantitation of SR numbers and the other half for NAT determinations. It w…

Maleendocrine systemmedicine.medical_specialtyHistologyLightArylamine N-AcetyltransferaseBiologyNAT activityPositive correlationPineal GlandPathology and Forensic MedicineLight at nightPineal glandAcetyltransferasesInternal medicinemedicineSprague dawley ratsAnimalsCircadian rhythmRats Inbred StrainsCell BiologyDarknessCircadian RhythmRatsMicroscopy Electronmedicine.anatomical_structureEndocrinologySynapsesSerotoninSerotonin N-acetyltransferase activityCell and tissue research
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Atypical 24-hour rhythms of serotonin N-acetyltransferase activity in the rat pineal gland.

1988

Previous long-term studies have shown that in the pineal gland of rats melatonin synthesis is subject to infradian rhythms with periods between 4 and 7 days. Since in these studies melatonin-related parameters were measured at one timepoint of a 24-hr cycle only, the aim of the present investigation was to extend these experiments by more frequent sampling, to characterize the infradian rhythmicity in more detail. Male Sprague-Dawley rats kept under a light schedule of LD 12:12 (lights on at 0700) were killed at 6-hr intervals on 8 consecutive days. After decapitation the pineal gland was rapidly dissected out, followed by measurements of one of the melatonin-forming enzymes, serotonin N-ac…

Maleendocrine systemmedicine.medical_specialtyPhysiologyArylamine N-AcetyltransferaseRats Inbred StrainsBiologyPineal GlandCircadian RhythmRatsRat Pineal GlandPineal glandmedicine.anatomical_structureRhythmEndocrinologyInfradian rhythmAcetyltransferasesPhysiology (medical)Internal medicinemedicineAnimalsSerotoninMelatonin synthesisCircadian rhythmSerotonin N-acetyltransferase activityChronobiology international
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Synaptic ribbons of the rat pineal gland: responses to in-vivo and in-vitro treatment with inhibitors of protein synthesis.

1990

To elucidate the role of protein synthesis in the nocturnal increase of synaptic ribbons in the rat pineal gland, actinomycin-D, which inhibits transcription, and cycloheximide, an inhibitor of translation, were used. To assure that the drugs were effective and to relate morphological changes to pineal biosynthetic phenomena, the activity of N-acetyltransferase and levels of pineal indoleamine were measured. Results of in-vivo, short-term and long-term treatment with either drug suggest that transcription of proteins related to synaptic ribbon formation occurs during the first half of the light phase, whereas translation takes place during the first few hours of the dark phase. In contrast,…

Maleendocrine systemmedicine.medical_specialtySerotoninHistologyTranscription GeneticStimulationCycloheximideBiologyPineal GlandPathology and Forensic MedicineMelatoninchemistry.chemical_compoundPineal glandIn vivoTranscription (biology)AcetyltransferasesInternal medicinemedicineProtein biosynthesisAnimalsCycloheximideMelatoninSynaptic ribbonRats Inbred StrainsCell BiologyCell biologyRatsMicroscopy ElectronEndocrinologymedicine.anatomical_structurenervous systemchemistryProtein BiosynthesisSynapsesDactinomycinsense organshormones hormone substitutes and hormone antagonistsmedicine.drugCell and tissue research
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Fos-related antigen 2 (Fra-2) memorizes photoperiod in the rat pineal gland

2004

As the physiological role of fos-related antigen-2 (Fra-2) is largely unknown and since the pineal plays an important role in the photoperiodic control of the body, we have tested the hypothesis that Fra-2 expression is photoperiod-dependent and may be involved in imprinting photoperiod on the pineal gland and the body as a whole. To this end, we have investigated Fra-2 mRNA expression and Fra-2 protein expression under various light/dark (LD) cycles. A clear nocturnal increase occurs for both monitored parameters under all photoperiodic conditions studied. The level of Fra-2 protein expression clearly depends on photoperiod, because the amount of protein at dark onset and during the night …

Maleendocrine systemmedicine.medical_specialtyTime FactorsPhotoperiodBlotting WesternDeiodinaseFos-Related Antigen-2CREBPineal GlandRats Sprague-DawleyPhenylephrinePineal glandOrgan Culture TechniquesAcetyltransferasesInternal medicineCyclic AMPmedicineAnimalsDrug InteractionsProtein phosphorylationRNA MessengerCircadian rhythmCyclic GMPHeat-Shock ProteinsbiologyReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceIsoproterenolAdrenergic beta-AgonistsAdaptation PhysiologicalPeptide FragmentsRatsDNA-Binding ProteinsEndocrinologymedicine.anatomical_structureGene Expression RegulationIodothyronine deiodinasebiology.proteinArylalkylamineFemaleAdrenergic alpha-Agonistshormones hormone substitutes and hormone antagonistsTranscription FactorsEndocrine glandNeuroscience
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Crystal structure of vinorine synthase, the first representative of the BAHD superfamily.

2005

Vinorine synthase is an acetyltransferase that occupies a central role in the biosynthesis of the antiarrhythmic monoterpenoid indole alkaloid ajmaline in the plant Rauvolfia. Vinorine synthase belongs to the benzylalcohol acetyl-, anthocyanin-O-hydroxy-cinnamoyl-, anthranilate-N-hydroxy-cinnamoyl/benzoyl-, deacetylvindoline acetyltransferase (BAHD) enzyme superfamily, members of which are involved in the biosynthesis of several important drugs, such as morphine, Taxol, or vindoline, a precursor of the anti-cancer drugs vincaleucoblastine and vincristine. The x-ray structure of vinorine synthase is described at 2.6-angstrom resolution. Despite low sequence identity, the two-domain structure…

Models MolecularStereochemistryMolecular Sequence DataSequence alignmentBiologyCrystallography X-RayBiochemistryIndole AlkaloidsProtein structureAcetyltransferasesTransferaseCoenzyme AAmino Acid SequenceDihydrolipoyl transacetylaseMolecular BiologyPlant ProteinsAjmalineATP synthaseMolecular StructureActive siteCell BiologyProtein Structure TertiaryBiochemistryAcyltransferasesAcetyltransferasebiology.proteinAnti-Arrhythmia AgentsSequence AlignmentThe Journal of biological chemistry
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MOZ/TIF2-induced acute myeloid leukaemia in transgenic fish.

2008

The inv(8)(p11q13) chromosomal abnormality, described in acute myeloid leukaemias (AML), fuses the histone acetyl-transferase (HAT) MYST3 (MOZ) gene with another HAT gene, NCOA2 (TIF2). We generated a transgenic zebrafish in which the MYST3/NCOA2 fusion gene was expressed under control of the spi1 promoter. An AML developed in 2 of 180 MYST3/NCOA2-EGFP-expressing embryos, 14 and 26 months after injection of the fusion gene in a one-cell embryo, respectively. This leukaemia was characterised by an extensive invasion of kidneys by myeloid blast cells. This model, which is the first zebrafish model of AML, demonstrates the oncogenic potency of MYST3/NCOA2 fusion gene.

MyeloidMicroinjectionsOncogene Proteins FusionTransgeneBiologyKidneyMYST3Fusion geneAnimals Genetically ModifiedNuclear Receptor Coactivator 2hemic and lymphatic diseasesmedicineAnimalsZebrafishGeneZebrafishHistone AcetyltransferasesSPI1Reverse Transcriptase Polymerase Chain ReactionHematologymedicine.diseasebiology.organism_classificationMolecular biologyLeukemiaDisease Models AnimalLeukemia Myeloid Acutemedicine.anatomical_structureembryonic structuresCancer researchGene FusionBritish journal of haematology
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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Role of Redox Signaling, Protein Phosphatases and Histone Acetylation in the Inflammatory Cascade in Acute Pancreatitis: Therapeutic Implications

2010

Acute pancreatitis starts as a local inflammation of the pancreatic tissue but often leads to the systemic inflammatory response syndrome and death by multiple organ failure. Pro-inflammatory cytokines, particularly TNF-alpha and Il-1beta, play a pivotal role together with oxidative stress and glutathione depletion in the inflammatory response in this disease. Most inflammatory mediators act through mitogen activated protein kinases and nuclear factor kB. Nevertheless, elucidation of the precise mechanisms involved in activation and attenuation phases of the inflammatory cascade is still underway. Redox signaling mediated by inactivation of protein phosphatases and histone acetylation trigg…

Phosphodiesterase InhibitorsImmunologyPhosphataseBiologyHistonesDual-specificity phosphatasePhosphoprotein PhosphatasesHumansImmunology and AllergyPancreasHistone AcetyltransferasesInflammationPharmacologyHistone AcetyltransferasesKinaseAcetylationGeneral MedicineProtein phosphatase 2ChromatinCell biologyHistone Deacetylase InhibitorsHistonePancreatitisBiochemistryAcetylationAcute Diseasebiology.proteinSignal transductionOxidation-ReductionSignal TransductionInflammation & Allergy - Drug Targets
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Regulation of NADPH oxidase-mediated superoxide production by acetylation and deacetylation

2021

Oral treatment of apolipoprotein E-knockout (ApoE-KO) mice with the putative sirtuin 1 (SIRT1) activator resveratrol led to a reduction of nicotinamide adenine dinucleotide phosphate (NADPH) oxidase activity in the heart. In contrast, the SIRT1 inhibitor EX527 enhanced the superoxide production in isolated human polymorphonuclear granulocytes. In human monocytic THP-1 cells, phorbol ester-stimulated superoxide production was enhanced by inhibitors of histone deacetylases (HDACs; including quisinostat, trichostatin A (TSA), PCI34051, and tubastatin A) and decreased by inhibitors of histone acetyltransferases [such as garcinol, curcumin, and histone acetyltransferase (HAT) Inhibitor II]. Thes…

Physiologyresveratrolsirtuin 1histone acetyltransferaseSirtuinechemistry.chemical_compoundHistone deacetylasesSirtuin 1Physiology (medical)NADPH-OxidasemedicineQP1-981ddc:610Original ResearchacetylationAcetyltransferasenNADPH oxidasebiologyNADPH oxidaseSirtuin 1SuperoxideAcetylationHistone acetyltransferaseTrichostatin AchemistryBiochemistryHistone acetyltransferasesAcetylationResveratrolNADPH oxidaseshistone deacetylasebiology.proteinHistone deacetylaseDDC 610 / Medicine & healthNicotinamide adenine dinucleotide phosphateRac1medicine.drug
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