Search results for "Activation"

showing 10 items of 2079 documents

Dynamic regulation of CD8 T cell tolerance induction by liver sinusoidal endothelial cells.

2010

Abstract Cross-presentation of soluble Ag on MHC class I molecules to naive CD8 T cells by liver sinusoidal endothelial cells (LSECs) leads to induction of T cell tolerance that requires interaction between coinhibitory B7-H1 on LSECs and programmed cell death-1 on CD8 T cells. In this study, we investigate whether cross-presentation of high as well as low Ag concentrations allowed for LSEC-induced tolerance. Ag concentration directly correlated with the cross-presentation capacity of murine LSECs and thus strength of TCR stimulation. Although LSEC cross-presentation at low-Ag concentrations resulted in tolerance, they induced differentiation into effector T cells (CTL) at high-Ag concentra…

OvalbuminT cellImmunologychemical and pharmacologic phenomenaMice TransgenicCD8-Positive T-LymphocytesLymphocyte ActivationResting Phase Cell CycleMiceCross-PrimingAntigenMHC class ImedicineImmune ToleranceImmunology and AllergyCytotoxic T cellAnimalsCells CulturedMice KnockoutAntigen PresentationbiologyT-cell receptorEndothelial CellsCytotoxicity Tests ImmunologicCoculture TechniquesCell biologyMice Inbred C57BLTolerance inductionCTL*medicine.anatomical_structureLiverbiology.proteinCD80Journal of immunology (Baltimore, Md. : 1950)
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Stem cell-secreted factor therapy regenerates the ovarian niche and rescues follicles.

2021

Background Ovarian senescence is a normal age-associated phenomenon, but increasingly younger women are affected by diminished ovarian reserves or premature ovarian insufficiency. There is an urgent need for developing therapies to improve ovarian function in these patients. In this context, previous studies suggest that stem cell–secreted factors could have regenerative properties in the ovaries. Objective This study aimed to test the ability of various human plasma sources, enriched in stem cell–secreted factors, and the mechanisms behind their regenerative properties, to repair ovarian damage and to promote follicular development. Study Design In the first phase, the effects of human pla…

Ovarian CortexOvaryBone Marrow CellsPrimary Ovarian InsufficiencyPremature ovarian insufficiencyHematopoietic Cell Growth Factors03 medical and health sciencesMicePlasma0302 clinical medicineOvarian FollicleMice Inbred NODGranulocyte Colony-Stimulating FactorMedicineAnimalsHumans030212 general & internal medicinePlatelet activationOvarian ReserveStem Cell Factor030219 obstetrics & reproductive medicinebusiness.industryCell growthStem CellsOvaryInfant NewbornObstetrics and GynecologyBone Marrow Stem CellCell cycleFetal BloodDisease Models Animalmedicine.anatomical_structureCancer researchHeterograftsFemaleStem cellbusinessAmerican journal of obstetrics and gynecology
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Oxidant stress and endothelial activation in hypertensive patients with and without left ventricular hypertrophy

2006

Oxidant stress endothelial activation hypertension left ventricular hypertrophy
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In Vitro Evaluation of the Squaramide-Conjugated Fibroblast Activation Protein Inhibitor-Based Agents AAZTA5.SA.FAPi and DOTA.SA.FAPi

2021

Recently, the first squaramide-(SA) containing FAP inhibitor-derived radiotracers were introduced. DATA5m.SA.FAPi and DOTA.SA.FAPi with their non-radioactive complexes showed high affinity and selectivity for FAP. After a successful preclinical study with [68Ga]Ga-DOTA.SA.FAPi, the first patient studies were realized for both compounds. Here, we present a new squaramide-containing compound targeting FAP, based on the AAZTA5 chelator 1,4-bis-(carboxylmethyl)-6-[bis-(carboxymethyl)-amino-6-pentanoic-acid]-perhydro-1,4-diazepine. For this molecule (AAZTA5.SA.FAPi), complexation with radionuclides such as gallium-68, scandium-44, and lutetium-177 was investigated, and the in vitro properties of…

PREPPharmaceutical ScienceAcetatesLutetiumLigands030218 nuclear medicine & medical imagingAnalytical ChemistrySerinechemistry.chemical_compoundQD241-4410302 clinical medicineFibroblast activation protein alphaPositron Emission Tomography Computed TomographyDrug Discoverylutetium-177AAZTA; scandium-44; lutetium-177; FAP; SA; DPP; PREPQuinineChemistrySerine EndopeptidasesAzepinesscandium-44ChemistryChemistry (miscellaneous)030220 oncology & carcinogenesisMolecular MedicineSelectivityDPPGallium RadioisotopesConjugated systemArticleHeterocyclic Compounds 1-Ring03 medical and health sciencesSAEndopeptidasesHumansDOTAChelationPhysical and Theoretical ChemistryBiologyAAZTARadioisotopesOrganic ChemistrySquaramideMembrane ProteinsFAPFibroblastsCombinatorial chemistryIn vitroRadiopharmaceuticalsScandiumMolecules
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Analysis of phosphorylation-dependent modulation of Kv1.1 potassium channels.

2003

The voltage-gated potassium channel Kv1.1 contains phosphorylation sites for protein kinase A (PKA) and protein kinase C (PKC). To study Kv1.1 protein expression and cellular distribution in regard to its level of phosphorylation, the effects of PKA and PKC activation on Kv1.1 were investigated in HEK 293 cells stably transfected with Kv1.1 (HEK 293/1). Without kinase activation, HEK 293/1 cells carry unphosphorylated Kv1.1 protein in the plasma membranes, whereas large amounts of phosphorylated and unphosphorylated Kv1.1 protein were located intracellularly. Activation of PKA resulted in phosphorylation of intracellular Kv1.1 protein, followed by a rapid translocation of Kv1.1 into the pla…

Patch-Clamp TechniquesPotassium Channelscomplex mixturesCell LineCellular and Molecular NeuroscienceHumansnatural sciencesProtein phosphorylationPatch clampPhosphorylationProtein kinase AProtein kinase CProtein Kinase CPharmacologyurogenital systemKinaseChemistryHEK 293 cellsAntibodies MonoclonalCyclic AMP-Dependent Protein KinasesPotassium channelCell biologyEnzyme ActivationKineticsProtein Transportnervous systemBiochemistryPotassium Channels Voltage-GatedPhosphorylationbiological phenomena cell phenomena and immunityKv1.1 Potassium ChannelIon Channel GatingNeuropharmacology
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Expression of Transketolase-like 1 and Activation of Akt in Grade IV Glioblastomas Compared With Grades II and III Astrocytic Gliomas

2008

Transketolases link the Embden-Meyerhof pathway to the pentose phosphate pathway. An influence of p-Akt on this metabolism was described. This study was performed to compare the expression of transketolase-like 1 (TKTL1) and p-Akt in glioblastoma multiforme (GBM) and other astrocytic gliomas (AGs, grades II and III). We analyzed 15 GBMs, 15 AGs (grade II), and 3 normal brain samples for TKTL1 expression by semiquantitative reverse transcription–polymerase chain reaction and Western blotting and 23 GBMs, 9 grade III AGs, and 7 grade II AGs immunohistochemically (TKTL1 and p-Akt). On the protein level, TKTL1 was significantly overexpressed in tumors. Immunohistochemically, the tumor grade sig…

Pathologymedicine.medical_specialtyAstrocytomaGeneral MedicineMetabolismAstrocytomaPentose phosphate pathwayTransketolaseBiologymedicine.diseaseEnzyme assayUp-RegulationEnzyme ActivationGene Expression Regulation NeoplasticBlotmedicinebiology.proteinCancer researchHumansGlycolysisRNA MessengerTransketolaseGlioblastomaProto-Oncogene Proteins c-aktProtein kinase BAmerican Journal of Clinical Pathology
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Collagenase-3 (MMP-13) Enhances Remodeling of Three-Dimensional Collagen and Promotes Survival of Human Skin Fibroblasts

2006

Collagenase-3 (MMP-13) is a matrix metalloproteinase capable of cleaving a multitude of extracellular matrix proteins in addition to fibrillar collagens. Human MMP-13 is expressed by fibroblasts in chronic cutaneous ulcers, but not in normally healing adult skin wounds. However, MMP-13 is produced by fibroblasts in adult gingival and in fetal skin wounds characterized by rapid collagen remodeling and scarless healing. Here, we have examined the role of human MMP-13 in remodeling of three-dimensional (3D) collagenous matrix by primary adult human skin fibroblasts. The high level of human MMP-13 expression by fibroblasts achieved by adenoviral gene delivery resulted in potent enhancement of r…

Pathologymedicine.medical_specialtyCell SurvivalHuman skinDermatologyMatrix Metalloproteinase InhibitorsMatrix metalloproteinaseBiologyBiochemistryFilamentous actinAdenoviridaeDermal fibroblastExtracellular matrixMatrix Metalloproteinase 13medicineHumansFibroblastMolecular BiologyCells CulturedCell ProliferationWound HealingTissue Inhibitor of Metalloproteinase-1integumentary systemCell BiologyFibroblastsActinsCell biologyEnzyme ActivationCollagen type I alpha 1medicine.anatomical_structureCollagenaseCollagenmedicine.drugJournal of Investigative Dermatology
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Novel contributions of neutrophils in the pathogenesis of abdominal aortic aneurysm, the role of neutrophil extracellular traps: A systematic review.

2020

Abdominal aortic aneurysm (AAA) is a vascular disorder caused by a dilatation of the aortic diameter that can be potentially lethal in case of rupture. Molecular mechanisms underlying the development of AAA are complex and not completely understood. However, inflammation plays a pivotal role in AAA development. Infiltration of inflammatory cells, especially macrophages, has been widely observed in lesion areas. Nevertheless, neutrophils have been gaining importance in the context of AAA. The release of neutrophil extracellular traps (NETs), extracellular structures formed by DNA, histones, granular and cytoplasmic proteins, is a recently discovered mechanism of neutrophil activation that ca…

Pathologymedicine.medical_specialtyNeutrophilsInflammationmacromolecular substances030204 cardiovascular system & hematologyExtracellular TrapsNeutrophil ActivationLesionPathogenesis03 medical and health sciences0302 clinical medicinemedicineExtracellularHumanscardiovascular diseasesbusiness.industryThrombosisHematologyNeutrophil extracellular trapsmedicine.diseaseAbdominal aortic aneurysm030220 oncology & carcinogenesisVascular Disordercardiovascular systemmedicine.symptombusinessInfiltration (medical)Aortic Aneurysm AbdominalThrombosis research
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Fibroblast activation protein inhibitor (FAPi) positive tumour fraction on PET/CT correlates with Ki-67 in liver metastases of neuroendocrine tumours

2021

Abstract Aim Gallium-68-labelled inhibitors of the fibroblast activation protein (FAPi) enable positron emission tomography/computed tomography (PET/CT) imaging of fibroblast activation. We evaluated if [68Ga]Ga-DATA5m.SA.FAPi PET/CT is related to Ki-67 as a marker of tumour aggressiveness in patients with liver metastases of NET. Methods Thirteen patients with liver metastases of a histologically confirmed NET who underwent PET/CT with [68Ga]Ga-DATA5m.SA.FAPi, [18F]FDG and [68Ga]Ga-DOTA-TOC were retrospectively analyzed. PET-positive liver tumour volumes were segmented for calculation of volume, SUVmax and PET-positive tumour fraction (TF). PET parameters were correlated with Ki-67. Result…

Pathologymedicine.medical_specialtyProliferation index030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineFibroblast activation protein alphaPositron Emission Tomography Computed TomographymedicineHumansRadiology Nuclear Medicine and imagingIn patientFibroblastGrading (tumors)Retrospective StudiesPET-CTbiologymedicine.diagnostic_testbusiness.industryLiver NeoplasmsGeneral MedicineFibroblastsNeuroendocrine TumorsKi-67 Antigenmedicine.anatomical_structurePositron emission tomography030220 oncology & carcinogenesisKi-67biology.proteinbusinessNuklearmedizin - NuclearMedicine
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Acute Vestibular Nucleus Lesion Affects Cortical Activation Pattern during Caloric Irrigation in PET

2003

Pathologymedicine.medical_specialtybusiness.industryGeneral NeuroscienceCaloric theoryAnatomyVestibular cortexActivation studyGeneral Biochemistry Genetics and Molecular BiologyActivation patternLesionHistory and Philosophy of ScienceVestibular nucleimedicinemedicine.symptomCaloric stimulationbusinessAnnals of the New York Academy of Sciences
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