Search results for "Activation"

showing 10 items of 2079 documents

Velocity-specific training in elbow flexors.

1999

The purpose of this study was to show that velocity-specific training may be implicated in modifications in the level of coactivation of agonist and antagonist muscles. Healthy males (n = 20) were randomly placed in to two groups: one group trained using concentric contractions (n = 12), the other was an untrained control group (n = 8). The training group underwent unilateral resistance training at a level of 35 (5)% of a one-repetition maximal contraction of the elbow flexors, executed at maximal angular velocity. Training sessions consisted of six sets of eight consecutive elbow flexions, three times per weak for a total of seven weeks. The velocity of the ballistic movements executed dur…

AgonistAdultMaleWeight LiftingPhysiologymedicine.drug_classElbowIsometric exerciseConcentricBicepsIsometric ContractionmedicineElbowEccentricHumansMuscle Skeletalbusiness.industryElectromyographyPublic Health Environmental and Occupational HealthAnatomyCoactivationBiomechanical Phenomenamedicine.anatomical_structureTorquePhysical Fitnessmedicine.symptomNuclear medicinebusinessMuscle contractionMuscle ContractionEuropean journal of applied physiology and occupational physiology
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Antagonist mechanical contribution to resultant maximal torque at the ankle joint in young and older men.

2007

Abstract A recorded muscular torque at one joint is a resultant torque corresponding to the participation of both agonist and antagonist muscles. This study aimed to examine the effect of aging on the mechanical contributions of both plantar- and dorsi-flexors to the resultant maximal voluntary contraction (MVC) torques exerted at the ankle joint, in dorsi-flexion (DF) and plantar-flexion (PF). The estimation of isometric agonist and antagonist torques by means of an EMG biofeedback technique was made with nine young (mean age 24 years) and nine older (mean age 80 years) men. While there was a non-significant age-related decline in the measured resultant DF MVC torque (−15%; p =0.06), there…

AgonistAdultMalemedicine.medical_specialtyAgingmedicine.drug_classPhysical ExertionBiophysicsNeuroscience (miscellaneous)Isometric exerciseVoluntary contractionPhysical medicine and rehabilitationmedicineTorqueHumansEmg biofeedbackMuscle SkeletalPostural BalanceAged 80 and overbusiness.industryAntagonistmusculoskeletal systemCoactivationbody regionsmedicine.anatomical_structureTorquePhysical therapyPhysical EnduranceNeurology (clinical)Anklebusinesshuman activitiesAnkle JointMuscle ContractionJournal of electromyography and kinesiology : official journal of the International Society of Electrophysiological Kinesiology
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5-HT3 receptor-channels coupled with Na+ influx in human T cells: role in T cell activation.

1999

The study was conducted on a human (Jurkat) T cell line, loaded with a Na+ fluorescent probe, SBFI/AM. Serotonin and an agonist of 5-HT3 receptor-channels, 2-methyl-5HT, evoked Na+ influx, whereas the agonists of other serotonergic receptor subtypes, i.e., 5-HT1A and 5-HT1B receptors, failed to induce Na+ influx in these cells. By using 3H-BRL43694, an agonist of 5-HT3 receptor-channels, we characterized 5-HT3 lymphocyte receptors which exhibited a density (Bmax) of 300 +/- 20 fmol/10(6) cells and a Kd of 30 nM in Jurkat T cells. The T-cell 5-HT3 receptor-channel is not regulated either by the protein kinase C or by the free intracellular calcium concentrations as the agents known to activa…

AgonistSerotoninmedicine.drug_classMetoclopramideT cellT-LymphocytesImmunologyBiologyLymphocyte ActivationJurkat cellsCalcium in biologyPiperazinesSodium ChannelsGranisetronJurkat CellsQuinoxalinesmedicineImmunology and AllergyHumansCalcium SignalingPhytohemagglutininsReceptorProtein kinase C5-HT receptorProtein Kinase C8-Hydroxy-2-(di-n-propylamino)tetralinIon TransportRyanodineCell CycleSodiumCell biologyNeoplasm ProteinsSerotonin Receptor AgonistsEnzyme Activationmedicine.anatomical_structureNeurologyReceptors SerotoninReceptor Serotonin 5-HT1BThapsigarginNeurology (clinical)Serotonin AntagonistsReceptors Serotonin 5-HT3Ion Channel GatingReceptors Serotonin 5-HT1IntracellularJournal of neuroimmunology
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Characterisation of serotonin transport mechanisms in rainbow trout peripheral blood lymphocytes: role in PHA-induced lymphoproliferation

1999

Abstract AbstractIn this study we investigated the serotonin transport mechanisms in rainbow trout (Oncorhynchus mykiss) peripheral blood lymphocytes We have observed that the transport of serotonin is a membrane transport process that have the properties of a secondary active transport system The binding isotherm of [3H]-paroxetine a serotonin transport blocker demonstrated a high-affinity binding site with a positive type of cooperativity Hill coefficient being higher than unity Known specific inhibitors of the mammalian serotonin transporter significantly inhibited the uptake process in fish lymphocytes In order to demonstrate the physiological relevance of the serotonin transporter in T…

AgonistSerotoninmedicine.medical_specialtySerotonin uptakemedicine.drug_classImmunologySerotonin transportNerve Tissue ProteinsLymphocyte ActivationInternal medicineCyclic AMPmedicineAnimalsLymphocytesPhytohemagglutininsSerotonin Uptake InhibitorsSerotonin transporterSerotonin Plasma Membrane Transport ProteinsMembrane GlycoproteinsbiologyMembrane Transport ProteinsBiological TransportMembrane transportEndocrinologyOncorhynchus mykissActive transportbiology.proteinSerotoninCarrier ProteinsSelective Serotonin Reuptake InhibitorsDevelopmental BiologyDevelopmental & Comparative Immunology
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Activation of phospholipases C and D by the novel metabotropic glutamate receptor agonist tADA.

1997

Abstract In hippocampal slices taken from 8-day-old rats, trans-azetidine-2,4-dicarboxylic acid (tADA), a novel glutamatergic agonist acting preferentially at class I mGluR receptors, activates phosphoinositide and phosphatidylcholine hydrolysis with widely different potencies. Inositol phosphate formation was maximally increased at 10 μM tADA ( ec 50: 1.2 μM), while phospholipase D activation was observed at a tADA concentration of 1 mM. This is the first report of a tADA-induced phospholipase D activity. © 1997 Elsevier Science Ltd. All rights reserved.

Agonistmedicine.drug_classInositol PhosphatesPhospholipaseBiologyIn Vitro TechniquesReceptors Metabotropic GlutamateHippocampusRats Sprague-DawleyCellular and Molecular NeurosciencemedicineExcitatory Amino Acid AgonistsPhospholipase DPhospholipase D activityAnimalsInositol phosphatePharmacologychemistry.chemical_classificationPhospholipase CPhospholipase DRatsEnzyme ActivationMetabotropic receptorBiochemistrychemistryMetabotropic glutamate receptorType C PhospholipasesAzetidinecarboxylic AcidNeuropharmacology
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Induction of Transglutaminase 2 by a Liver X Receptor/Retinoic Acid Receptor α Pathway Increases the Clearance of Apoptotic Cells by Human Macrophages

2009

Rationale: Liver X receptors (LXRs) are oxysterol-activated nuclear receptors that are involved in the control of cholesterol homeostasis and inflammatory response. Human monocytes and macrophages express high levels of these receptors and are appropriate cells to study the response to LXR agonists. Objective: The purpose of this study was to identify new LXR targets in human primary monocytes and macrophages and the consequences of their activation. Methods and Results: We show that LXR agonists significantly increase the mRNA and protein levels of the retinoic acid receptor (RAR)α in primary monocytes and macrophages. LXR agonists promote RARα gene transcription through binding to a spec…

Agonistmedicine.medical_specialtyReceptors Retinoic AcidPhysiologymedicine.drug_classResponse elementReceptors Cytoplasmic and NuclearApoptosisBiologyCell LinePhagocytosisGTP-Binding ProteinsInternal medicinemedicineHumansMacrophageProtein Glutamine gamma Glutamyltransferase 2ReceptorLiver X receptorLiver X ReceptorsTransglutaminasesMacrophagesRetinoic Acid Receptor alphaMacrophage ActivationAtherosclerosisOrphan Nuclear ReceptorsCell biologyDNA-Binding ProteinsRetinoic acid receptorEndocrinologyNuclear receptorRetinoic acid receptor alphaEnzyme InductionCardiology and Cardiovascular MedicineCirculation Research
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Platelet Reactivity in Hemophilia a. Flow Cytometry Study to Detect the Hemorragic Phenotype

2016

Abstract INTRODUCTION Hemophilia A (HA) is a rare inherited genetic disorder linked to the X chromosome, characterized by joint bleeding in patients with severe disease with <1% of factor VIII (FVIII)(White et al. Thromb Haemost 2001). However, there are cases of severe HA with less bleeding than usual and it has been postulated that there must be other contributors to the hemorrhagic phenotype, including a platelet dysfunction (Van Bladel et al. Haematologica 2011). In this study we analyzed platelet reactivity in a cohort of 16 male patients with severe HA in prophylactic treatment by flow cytometry. The control group was 15 healthy male subjects. METHODS A longitudinal prospective obs…

Agonistmedicine.medical_specialtyVenipuncturemedicine.diagnostic_testmedicine.drug_classbusiness.industryImmunologyCell BiologyHematologyBiochemistryGastroenterologyFlow cytometryBasal (phylogenetics)Internal medicineStatistical significanceImmunologymedicinePlateletPlatelet activationbusinessWhole bloodBlood
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Mechanical impact of the antagonist muscles during maximal dorsiflexion is action mode dependent

2019

Maximal voluntary contraction (MVC) has long been considered as the best measure of neuromuscular performance. However, when MVC is paired with electromyographic (EMG) activity recording it has bee...

Agonistmedicine.medical_specialtybusiness.industrymedicine.drug_classMechanical impactBiomedical EngineeringAntagonistMeasure (physics)BioengineeringGeneral Medicinemusculoskeletal systemCoactivationComputer Science Applicationsbody regionsHuman-Computer InteractionVoluntary contractionPhysical medicine and rehabilitationAction (philosophy)Medicinebusinesshuman activitiesComputer Methods in Biomechanics and Biomedical Engineering
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Coactivation at the ankle joint is not sufficient to estimate agonist and antagonist mechanical contribution

2009

The aim of this study was to assess, via an elec- tromyographic (EMG) biofeedback method, the mechanical con- tribution of both agonist and antagonist muscles during maximal voluntary contraction (MVC). We compared this original method with the MVC-EMGmax ratio and the torque/EMG relationship method, both of which are commonly used to estimate antago- nist torque. The plantarflexion (PF) and dorsiflexion (DF) MVCs were measured simultaneously with EMG activity of triceps surae (TS) and tibialis anterior in 15 young adults (mean age 23 years). Antagonist torques obtained from the torque/EMG relationship and EMG biofeedback methods appeared to be similar. TS antagonist torque had a major mech…

Agonistmedicine.medical_specialtymedicine.diagnostic_testPhysiologyChemistrymedicine.drug_classAntagonistBiomechanicsAnatomyElectromyographymusculoskeletal systemCoactivationbody regionsCellular and Molecular NeuroscienceElectrophysiologyPhysical medicine and rehabilitationmedicine.anatomical_structurePhysiology (medical)medicineNeurology (clinical)Anklemedicine.symptomMuscle contractionMuscle & Nerve
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How the ankle joint angle alters the antagonist and agonist torques during maximal efforts in dorsi- and plantar flexion

2011

The aim of this study was to assess, via an EMG bio-feedback method, the ankle joint angle effect on the agonist and antagonist torques in plantar- (PF) and dorsi-flexion (DF). The isometric PF and DF maximal voluntary contractions (MVCs) torques were measured simultaneously with surface EMG activity of triceps surae (TS) and tibialis anterior (TA) muscles in 12 young adults (mean age 27) at five different ankle joint angles. Our results showed that: (i) The coactivation level does not properly reflect the mechanical effect of the antagonist muscle, (ii) TS antagonist torque significantly altered the DF MVC-angle relationship, whereas TA antagonist torque did not influence this MVC-angle re…

Agonistmedicine.medical_specialtymedicine.drug_classbusiness.industryBiomechanicsAntagonistPhysical Therapy Sports Therapy and Rehabilitation030229 sport sciencesAnatomyIsometric exercisemusculoskeletal systemCoactivationbody regions03 medical and health sciences0302 clinical medicinePhysical medicine and rehabilitationmedicine.anatomical_structureJoint anglemedicineTorqueOrthopedics and Sports MedicineAnklebusiness030217 neurology & neurosurgeryScandinavian Journal of Medicine & Science in Sports
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