Search results for "Adenosine"

showing 10 items of 542 documents

The rat liver foci bioassay: II. Investigations on the dose-dependent induction of ATPase-deficient foci by vinyl chloride at very low doses

1985

In order to study the dose-dependence of the genotoxic effect of vinyl chloride (VC) hepatocellular ATPase-deficient foci were evaluated after subchronic exposure of newborn rats. Wistar rats were exposed from day 1 after birth over 10 weeks to 10, 40, 70, 150, 500 and 2000 p.p.m. VC (8 h/day; 5 days/week). One week after cessation of exposure hepatic ATPase-deficient foci were quantitated. For a subsequent investigation lower dose range groups of female and male Wistar and Sprague-Dawley rats were exposed (8 h/day; 5 days/week) to 2.5, 5, 10, 20, 40 and 80 p.p.m. VC. Exposure started at day 3 of life and lasted for 3 weeks. After cessation of exposure the animals were maintained for 10 wee…

MaleCancer ResearchPathologymedicine.medical_specialtyVinyl CompoundsATPaseVinyl ChlorideDose dependenceVinyl chlorideAndrologychemistry.chemical_compoundLiver Neoplasms ExperimentalSex FactorsSpecies SpecificitymedicineAnimalsBioassayCarcinogenAdenosine TriphosphatasesDose-Response Relationship DrugbiologyLow doseRats Inbred StrainsGeneral MedicineRatsLinear relationshipLiverchemistryRat liverbiology.proteinBiological AssayFemalePrecancerous ConditionsCarcinogenesis
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The rat liver foci bioassay: I. Age-dependence of induction by vinyl chloride of ATPase-deficient foci

1985

The age-dependence of the induction of pre-neoplastic enzyme-altered hepatic foci was investigated. Rats were exposed (8 h/day, 7 days/week) to 2000 p.p.m. vinyl chloride (VC) either 'transplacentally' (exposure of pregnant females), or immediately after birth for different time intervals (5, 11, 17, 47, 83 days) or from an age of 7 or 21 days onwards. The animals were then kept without further treatment; livers were evaluated for ATPase-deficient foci at the age of 4 months. 'Transplacental' exposure and exposure from day 1 through 5 caused no increase over controls in ATPase-deficient foci, probably due to the lack of hepatocellular proliferation and the low rate of VC metabolism at this …

MaleCancer ResearchPathologymedicine.medical_specialtyVinyl CompoundsATPaseVinyl ChloridePartial hepatectomyBiologyVinyl chlorideAndrologychemistry.chemical_compoundLiver Neoplasms ExperimentalmedicineAnimalsBioassayCarcinogenAdenosine TriphosphatasesAge FactorsTransplacentalRats Inbred StrainsGeneral MedicineMetabolismRatsLiverchemistryRat liverbiology.proteinBiological AssayFemalePrecancerous ConditionsCarcinogenesis
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The ISWI chromatin remodeler organizes the hsrω ncRNA-containing omega speckle nuclear compartments.

2011

The complexity in composition and function of the eukaryotic nucleus is achieved through its organization in specialized nuclear compartments. The Drosophila chromatin remodeling ATPase ISWI plays evolutionarily conserved roles in chromatin organization. Interestingly, ISWI genetically interacts with the hsrω gene, encoding multiple non-coding RNAs (ncRNA) essential, among other functions, for the assembly and organization of the omega speckles. The nucleoplasmic omega speckles play important functions in RNA metabolism, in normal and stressed cells, by regulating availability of hnRNPs and some other RNA processing proteins. Chromatin remodelers, as well as nuclear speckles and their assoc…

MaleCancer ResearchRNA Untranslatedlcsh:QH426-470Gene ExpressionFluorescent Antibody TechniqueRNA-binding proteinBiologyEyeHeterogeneous ribonucleoprotein particleChromosomesHeterogeneous-Nuclear RibonucleoproteinsChromatin remodelingMolecular GeneticsGeneticsmedicineAnimalsDrosophila ProteinsOmega speckleBiologyMolecular BiologyTranscription factorAllelesGenetics (clinical)Ecology Evolution Behavior and SystematicsAdenosine TriphosphatasesCell NucleusGeneticsRNA-Binding ProteinsEpistasis GeneticChromatin Assembly and DisassemblyNon-coding RNAChromatinCell biologyCell nucleuslcsh:GeneticsPhenotypemedicine.anatomical_structureTandem Repeat SequencesChromatin remodeling non coding RNALarvaEpigeneticsDrosophilaRNA InterferenceResearch ArticleTranscription FactorsPLoS Genetics
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Blood flow, oxygenation, metabolic and energetic status in different clonal subpopulations of a rat rhabdomyosarcoma.

1998

Differentiation of a tumor plays an important role in terms of biological aggressiveness. The question arises as to whether this is reflected in differences in the metabolic and energetic status of solid tumors. The aim of this study was to analyze the influence of clonal tumor cell differentiation on the microenvironment of rat rhabdomyosarcomas. Two distinct lines of a rhabdomyosarcoma (BA-HAN-1) with different histomorphological properties were used (line F1, co-existence of mononuclear stellate cells and multinuclear myotube-like giant tumor cells; G8, polygonal, mononuclear tumor cells). Solid tumors were grown s.c. on the hind food dorsum of Lewis rats. Tumor oxygenation was measured …

MaleCancer Researchmedicine.medical_specialtyPartial PressureCellular differentiationBiologyAdenosine TriphosphateInternal medicineRhabdomyosarcomaTumor Cells CulturedmedicineAnimalsGlycolysisLactic AcidRhabdomyosarcomaOncogeneTumor OxygenationCell cyclemedicine.diseaseRatsOxygenGlucoseEndocrinologyOncologyRats Inbred LewImmunologyHepatic stellate cellFemaleSarcomaEnergy MetabolismCell DivisionNeoplasm TransplantationInternational Journal of Oncology
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A population of prenatally generated cells in the rat paleocortex maintains an immature neuronal phenotype into adulthood.

2008

New neurons in the adult brain transiently express molecules related to neuronal development, such as the polysialylated form of neural cell adhesion molecule, or doublecortin (DCX). These molecules are also expressed by a cell population in the rat paleocortex layer II, whose origin, phenotype, and function are not clearly understood. We have classified most of these cells as a new cell type termed tangled cell. Some cells with the morphology of semilunar-pyramidal transitional neurons were also found among this population, as well as some scarce cells resembling semilunar, pyramidal. and fusiform neurons. We have found that none of these cells in layer II express markers of glial cells, m…

MaleCell typeDoublecortin ProteinAntimetabolitesCognitive NeuroscienceNeurogenesisPopulationMice Inbred StrainsNeural Cell Adhesion Molecule L1Receptors N-Methyl-D-AspartateImmunophenotypingRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundMiceReceptors GlucocorticoidPregnancyAnimalsEntorhinal CortexCyclic adenosine monophosphateeducationeducation.field_of_studyArc (protein)biologyPyramidal CellsStem CellsNeurogenesisAge FactorsPhenotypeDoublecortinCell biologyRatsMicroscopy ElectronchemistryBromodeoxyuridinebiology.proteinSialic AcidsNeural cell adhesion moleculeFemaleNeuroscienceNeurogliaBiomarkersCerebral cortex (New York, N.Y. : 1991)
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Negative regulation of diacylglycerol kinase θ mediates adenosine-dependent hepatocyte preconditioning

2010

In liver ischemic preconditioning (IP), stimulation of adenosine A2a receptors (A2aR) prevents ischemia/reperfusion injury by promoting diacylglycerol-mediated activation of protein kinase C (PKC). By concerting diacylglycerol to phosphatidic acid, diacylglycerol kinases (DGKs) act as terminator of diacylglycerol signalling. This study investigates the role of DGK in the development of hepatocyte IP. DGK activity and cell viability were evaluated in isolated rat hepatocytes preconditioned by 10 min hypoxia followed by 10 min re-oxygenation or by the treatment with the A2aR agonist, CGS21680, and subsequently exposed to prolonged hypoxia. We observed that after IP or A2aR activation, a decre…

MaleDiacylglycerol Kinasemedicine.medical_specialtyAdenosineReceptor Adenosine A2Ap38 mitogen-activated protein kinasesBiologyQuinazolinonechemistry.chemical_compoundPiperidinecytoprotectionPiperidinesDownregulation and upregulationDiacylglycerol kinase thetaInternal medicinemedicineEnzyme Inhibitorhepatocytes adenosine RhoA hypoxia cytoprotectionAnimalsHepatocyteEnzyme InhibitorsRats WistarMolecular BiologyCells CulturedProtein kinase CQuinazolinonesDiacylglycerol kinaseCell DeathAnimalhypoxiaKinaseReceptors Purinergic P1RhoACell BiologyPhosphatidic acidAdenosineCell HypoxiaRatsCell biologyEndocrinologychemistryHepatocytesRatrhoA GTP-Binding Proteinmedicine.drugCell Death & Differentiation
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Regulatory properties of 6-phosphofructokinase and control of glycolysis in boar spermatozoa.

2007

Glycolysis is crucial for sperm functions (motility and fertilization), but how this pathway is regulated in spermatozoa is not clear. This prompted to study the location and the regulatory properties of 6-phosphofructokinase (PFK, EC 2.7.1.11), the most important element for control of glycolytic flux. Unlike some other glycolytic enzymes, PFK showed no tight binding to sperm structures. It could readily be extracted from ejaculated boar spermatozoa by sonication and was then chromatographically purified. At physiological pH, the enzyme was allosterically inhibited by near-physiological concentrations of its co-substrate ATP, which induced co-operativity, i.e. reduced the affinity for the …

MaleEmbryologySwinePhosphofructokinase-1Allosteric regulationImmunoblottingMotilityBiologychemistry.chemical_compoundEndocrinologyAdenosine TriphosphateAllosteric RegulationFructosediphosphatesAnimalsGlycolysisCitrateschemistry.chemical_classificationObstetrics and GynecologyFructoseCell BiologyHydrogen-Ion ConcentrationSpermImmunohistochemistrySpermatozoaAdenosine MonophosphateEnzymeReproductive MedicinechemistryBiochemistryFlagellaElectrophoresis Polyacrylamide GelFlux (metabolism)AcrosomeGlycolysisPhosphofructokinaseReproduction (Cambridge, England)
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Modulation of induced reversion frequency by nucleotide pool imbalance as a tool for mutant characterization.

1987

Addition of thymidine (TdR) or deoxycytidine (CdR) to the culture medium during posttreatment incubation affected the frequency of mutagen-induced reversion in three hypoxanthine-guanine phosphoribosyl transferase-deficient mutants of V79 Chinese hamster cells. With two of the mutants (E20 and I3), reversions induced by N-ethylnitrosourea, ethyl methanesulfonate, and methyl methanesulfonate were enhanced by TdR and were either decreased (E20) or not affected (I3) by CdR. With the third mutant (E21), alkylating agent-induced reversions were enhanced by CdR and decreased by TdR. Finally, 6-amino-2-hydroxypurine induced reversions were enhanced by TdR in mutant I3 and were decreased by TdR or …

MaleEthyl methanesulfonateEpidemiologyHealth Toxicology and MutagenesisMutantReversionMutagenesis (molecular biology technique)BiologyDeoxycytidineCell Linechemistry.chemical_compoundCricetulusDeoxyadenosineCricetinaeAnimalsGenetics (clinical)DeoxyadenosinesNucleotidesPoint mutationFibroblastsMethyl methanesulfonatechemistryBiochemistryMutationThymidineMutagensThymidineEnvironmental and molecular mutagenesis
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Cordycepin protects against cerebral ischemia/reperfusion injury in vivo and in vitro.

2011

Cordycepin, (3'-deoxyadenosine), a bioactive compound of Cordyceps militaris, has been shown to exhibit many pharmacological actions, such as anti-inflammatory, antioxidative and anticancer activities. Little is known about the neuroprotective action of cordycepin as well as its molecular mechanisms. In this study, cordycepin was investigated for its neuroprotective potential in mice with ischemia following 15 min of the bilateral common carotid artery occlusion and 4h of reperfusion. The effect of cordycepin was also studied in mice brain slices treated with oxygen-glucose deprivation (OGD) injury. Our results showed that cordycepin was able to prevent postischemic neuronal degeneration an…

MaleExcitatory Amino AcidsIschemiaCell CountPharmacologyNeuroprotectionHippocampusBrain IschemiaSuperoxide dismutaseBrain ischemiachemistry.chemical_compoundMiceIn vivoMalondialdehydemedicineAnimalsPharmacologyNeuronsbiologyCordycepinDeoxyadenosinesbusiness.industrySuperoxide DismutaseGlutamate receptormedicine.diseaseOxygenGlucoseBiochemistrychemistryReperfusion Injurybiology.proteinMatrix Metalloproteinase 3businessReperfusion injuryPropidiumEuropean journal of pharmacology
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Anti-B-50 (GAP-43) antibodies decrease exocytosis of glutamate in permeated synaptosomes.

1999

Abstract The involvement of the protein kinase C substrate, B-50 (GAP-43), in the release of glutamate from small clear-cored vesicles in streptolysin-O-permeated synaptosomes was studied by using anti-B-50 antibodies. Glutamate release was induced from endogenous as well as 3 H -labelled pools in a [Ca2+]-dependent manner. This Ca2+-induced release was partially ATP dependent and blocked by the light-chain fragment of tetanus toxin, demonstrating its vesicular nature. Comparison of the effects of anti-B-50 antibodies on glutamate and noradrenaline release from permeated synaptosomes revealed two major differences. Firstly, Ca2+-induced glutamate release was decreased only partially by anti…

MaleGlutamic AcidBiologyIn Vitro TechniquesSynaptic vesicleExocytosisExocytosischemistry.chemical_compoundNorepinephrineAdenosine TriphosphateGAP-43 ProteinAnimalsEnzyme InhibitorsRats WistarNeurotransmitterProtein kinase CProtein Kinase CPharmacologySynaptosomeVesicleGlutamate receptorAntibodies MonoclonalIntracellular MembranesRatschemistryBiochemistryStreptolysinsBiophysicsLiberationCalciumSynaptosomesEuropean journal of pharmacology
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