Search results for "Adhesion molecule"

showing 10 items of 525 documents

Promotion of osteogenic cell response using quasicovalent immobilized fibronectin on titanium surfaces: introduction of a novel biomimetic layer syst…

2012

Purpose Despite the undeniable potential of cell adhesion molecules such as fibronectin to support osteogenic cell responses and consecutive dental implant healing, the most beneficial mode of application onto titanium implant surfaces still requires investigation. Unspecific fibronectin adsorption on titanium dioxide (TiO2) surfaces can result in low-loading, high-desorption rates and protein–metal interactions with impaired biologic activity. The aim of the present study was to monitor the osteogenic cell responses (cell adhesion, proliferation, and differentiation) specifically to fibronectin biofunctionalized TiO2. Materials and Methods An innovative biomimetic streptavidin-biotin layer…

Time FactorsCellular differentiationOsteocalcinCell Culture TechniquesBiotinBiocompatible MaterialsCore Binding Factor Alpha 1 SubunitCell LineCyclin D1Biomimetic MaterialsOsteogenesisCell AdhesionMedicineHumansCyclin D1Cell adhesionCell ProliferationTitaniumOsteoblastsbiologyCell adhesion moleculebusiness.industryIntegrin beta1Cell DifferentiationAdhesionSilanesAlkaline PhosphataseFibronectinsFibronectinImmobilized ProteinsPhenotypeOtorhinolaryngologyBiotinylationVitamin B Complexbiology.proteinBiophysicsAlkaline phosphataseSurgeryAdsorptionStreptavidinOral SurgerybusinessJournal of oral and maxillofacial surgery : official journal of the American Association of Oral and Maxillofacial Surgeons
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LFA-1 activity state on dendritic cells regulates contact duration with T cells and promotes T-cell priming.

2010

AbstractA key event in the successful induction of adaptive immune responses is the antigen-specific activation of T cells by dendritic cells (DCs). Although LFA-1 (lymphocyte function–associated antigen 1) on T cells is considered to be important for antigen-specific T-cell activation, the role for LFA-1 on DCs remains elusive. Using 2 different approaches to activate LFA-1 on DCs, either by deletion of the αL-integrin cytoplasmic GFFKR sequence or by silencing cytohesin-1–interacting protein, we now provide evidence that DCs are able to make use of active LFA-1 and can thereby control the contact duration with naive T cells. Enhanced duration of DC/T-cell interaction correlates inversely …

Time FactorsT cellT-LymphocytesImmunologyReceptors Antigen T-CellPriming (immunology)chemical and pharmacologic phenomenaMice TransgenicCell CommunicationBiologyLymphocyte ActivationBiochemistryMiceImmune systemAntigenmedicineCell AdhesionAnimalsHypersensitivity DelayedLymphocyte function-associated antigen 1Antigen-presenting cellCells CulturedCell ProliferationMice KnockoutReverse Transcriptase Polymerase Chain ReactionMembrane Proteinshemic and immune systemsCell BiologyHematologyT lymphocyteDendritic cellDendritic CellsTh1 CellsFlow CytometryIntercellular Adhesion Molecule-1Lymphocyte Function-Associated Antigen-1Cell biologyMice Inbred C57BLmedicine.anatomical_structureImmunologyInterleukin-2RNA InterferenceCarrier ProteinsBlood
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Indicaxanthin from

2018

Oxidized low-density lipoproteins (oxLDL) play a pivotal role in the etiopathogenesis of atherosclerosis through the activation of inflammatory signaling events eventually leading to endothelial dysfunction and senescence. In the present work, we investigated the effects of indicaxanthin, a bioavailable, redox-modulating phytochemical from Opuntia ficus indica fruits, with anti-inflammatory activity, against oxLDL-induced endothelial dysfunction. Human umbilical vein cord cells (HUVEC) were stimulated with human oxLDL, and the effects of indicaxanthin were evaluated in a range between 5 and 20 μM, consistent with its plasma level after a fruit meal (7 μM). Pretreatment with indicaxanthin si…

Transcription GeneticCell SurvivalPyridinesNF-kappa BOpuntiaHydrogen PeroxideReactive Nitrogen SpeciesThiobarbituric Acid Reactive SubstancesBetaxanthinsUp-RegulationLipoproteins LDLHuman Umbilical Vein Endothelial CellsHumansRNA MessengerReactive Oxygen SpeciesCell Adhesion MoleculesOxidation-ReductionATP Binding Cassette Transporter 1Research ArticleOxidative medicine and cellular longevity
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Ionizing radiation-induced E-selectin gene expression and tumor cell adhesion is inhibited by lovastatin and all-trans retinoic acid

2004

E-selectin mediated tumor cell adhesion plays an important role in metastasis. Here we show that ionizing radiation (IR) induces E-selectin gene and protein expression in human endothelial cells at therapeutically relevant dose level. E-selectin expression is accompanied by an increase in the adhesion of human colon carcinoma cells to primary human umbilical vein endothelial cells (HUVEC). The HMG-CoA reductase inhibitor lovastatin impairs IR-stimulated E-selectin expression as analyzed at the level of the protein, mRNA and promoter. Inactivation of Rho GTPases either by use of Clostridium difficile toxin A or by co-expression of dominant-negative Rho blocked IR-induced E-selectin gene indu…

Transcriptional Activationrho GTP-Binding ProteinsCancer ResearchBlotting WesternIntercellular Adhesion Molecule-1Retinoic acidEnzyme-Linked Immunosorbent AssayTretinoinchemistry.chemical_compoundGenes ReporterTretinoinCell Line TumorNeoplasmsRadiation IonizingE-selectinGene expressionCell AdhesionmedicineHumansLovastatinRNA MessengerPromoter Regions GeneticCell adhesionCells CulturedDose-Response Relationship DrugbiologyReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaCell adhesion moleculeNF-kappa BDose-Response Relationship RadiationGeneral MedicineIntercellular Adhesion Molecule-1Gene Expression Regulation Neoplasticchemistrybiology.proteinCancer researchEndothelium VascularLovastatinHydroxymethylglutaryl-CoA Reductase InhibitorsE-Selectinmedicine.drugCarcinogenesis
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Longitudinal analysis of Mycobacterium tuberculosis 19-kDa antigen-specific T cells in patients with pulmonary tuberculosis: association with disease…

2003

CD8(+) T cells play a central role in immune protection against infection with Mycobacterium tuberculosis. One of the target epitopes for anti-M. tuberculosis directed CD8(+) T cells is the HLA-A2-restricted 19-kDa lipoprotein peptide VLTDGNPPEV. T cell clones directed against this epitope recognized not only the nominal peptide ligand, but also a closely related peptide (VPTDPNPPEV) from the HIV envelope gp120 (HIV(env) gp120) protein characterized by IFN-gamma release. This cross-reactivity was confirmed in ex vivo in M. tuberculosis 19-kDa tetramer-sorted T cells from patients with tuberculosis and in HIVgp120 tetramer-reactive T cells sorted from HIV(+) patients. M. tuberculosis 19-kDa …

TuberculosisHIV AntigensT cellImmunologyEpitopes T-LymphocyteHIV InfectionsCD146 AntigenBiologyCD8-Positive T-LymphocytesCross ReactionsHIV Envelope Protein gp120medicine.disease_causeEpitopeMycobacterium tuberculosisInterferon-gammaViral ProteinsAntigenBacterial ProteinsAntigens CDT-Lymphocyte SubsetsHLA-A2 AntigenmedicineImmunology and AllergyHumansTuberculosisLongitudinal StudiesNeural Cell Adhesion MoleculesAntigens BacterialMembrane GlycoproteinsMolecular MimicryGranulocyte-Macrophage Colony-Stimulating FactorT lymphocyteMycobacterium tuberculosisOncogene Proteins Viralmedicine.diseasebiology.organism_classificationVirologyPeptide FragmentsDNA-Binding ProteinsMolecular mimicrymedicine.anatomical_structureImmunologyInterleukin-4CD8BiomarkersEuropean journal of immunology
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MMP-10/stromelysin-2 promotes invasion of head and neck cancer.

2011

BackgroundPeriostin, IFN-induced transmembrane protein 1 (IFITM1) and Wingless-type MMTV integration site family, member 5B (Wnt-5b) were previously identified as the invasion promoted genes of head and neck squamous cell carcinoma (HNSCC) by comparing the gene expression profiles between parent and a highly invasive clone. We have previously reported that Periostin and IFITM1 promoted the invasion of HNSCC cells. Here we demonstrated that Wnt-5b overexpression promoted the invasion of HNSCC cells. Moreover, stromelysin-2 (matrix metalloproteinase-10; MMP-10) was identified as a common up-regulated gene among Periostin, IFITM1 and Wnt-5b overexpressing HNSCC cells by using microarray data s…

Tumor PhysiologyClone (cell biology)p38 Mitogen-Activated Protein KinasesMetastasisMetastasisMolecular Cell BiologyBasic Cancer ResearchNeoplasm MetastasisRegulation of gene expressionGene knockdownMultidisciplinaryHead and Neck cancerQRTransfectionHead and Neck TumorsExtracellular MatrixUp-RegulationGene Expression Regulation NeoplasticOncologyHead and Neck NeoplasmsGene Knockdown TechniquesCarcinoma Squamous CellMedicineResearch ArticleScience490Oral MedicineBiologyPeriostinHead and Neck Squamous Cell CarcinomaMatrix Metalloproteinase 10stomatognathic systemSettore MED/28 - Malattie OdontostomatologicheCell Line TumormedicineCancer Detection and Diagnosisotorhinolaryngologic diseasesHumansNeoplasm Invasiveness490BiologyExtracellular Matrix AdhesionsProtein Kinase InhibitorsneoplasmsMicroarray analysis techniquesCancers and Neoplasmsmedicine.diseaseMolecular biologyHead and neck squamous-cell carcinomaAntigens DifferentiationWnt Proteinsstomatognathic diseasesCancer researchCell Adhesion MoleculesPLoS ONE
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Antioxidant betalains from cactus pear (Opuntia ficus-indica) inhibit endothelial ICAM-1 expression.

2004

It has been suggested that some pigments would have antioxidant properties and that their presence in dietary constituents would contribute to reduce the risk of oxidative stress–correlated diseases. Among others, inflammatory response depends on redox status and may implicate oxidative stress. Vascular endothelial cells are a direct target of oxidative stress in inflammation. We have tested the impact of the free radical scavenger and antioxidant properties of betalains from the prickle pear in an in vitro model of endothelial cells. Here we show the capacity of betalains to protect endothelium from cytokine- induced redox state alteration, through ICAM-1 inhibition. KEYWORDS: endothelial …

Umbilical VeinsAntioxidantEndotheliumICAM-1Pyridinesmedicine.medical_treatmentAnti-Inflammatory AgentsBetalainsInflammationOxidative phosphorylationBiologymedicine.disease_causeModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyAntioxidantschemistry.chemical_compoundHistory and Philosophy of ScienceSettore BIO/10 - BiochimicamedicineHumansCells CulturedInflammationICAM-1Dose-Response Relationship DrugPlant ExtractsGeneral NeurosciencebetalainOpuntiaFree radical scavengerFlow CytometryIntercellular Adhesion Molecule-1BetaxanthinsQuaternary Ammonium CompoundsOxidative Stressmedicine.anatomical_structurechemistryBiochemistryendothelial cellendothelial cells; ICAM-1; betalains; antiinflammatory drugsCytokinesEndothelium Vascularantiinflammatory drugsmedicine.symptomIndicaxanthinOxidation-ReductionOxidative stressAnnals of the New York Academy of Sciences
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Prostacyclin inhibits adhesion of polymorphonuclear leukocytes to human vascular endothelial cells due to adhesion molecule independent regulatory me…

2002

Prostacyclin is an important endothelial mediator involved in the interaction of neutrophils (PMN) with the vessel wall. Many studies have shown the beneficial effects of prostacyclin in ischemia and reperfusion. However, no previous study has investigated the direct effects of the prostacyclin analogs iloprost (ILO) and alprostadil (PGE(1)) on the endothelial part of the adhesion process. Human umbilical vein endothelial cells (HUVECs) were grown to confluence, stimulated with 300 U/ml TNF-alpha and treated with increasing concentrations of ILO and PGE(1). The cells were washed to remove TNF and the inhibitors and adhesion of fluorescence-green labeled PMN was determined microscopically. I…

Umbilical VeinsEndotheliumNeutrophilsPhysiologyVascular Cell Adhesion Molecule-1ProstacyclinPharmacologyUmbilical veinPhysiology (medical)Cell AdhesionmedicineHumansIloprostAlprostadilChemoattractant activityCells CulturedCell NucleusTumor Necrosis Factor-alphaChemistryChemotaxisAdhesionrespiratory systemEpoprostenolrespiratory tract diseasesEndothelial stem cellmedicine.anatomical_structureBiochemistrycardiovascular systemlipids (amino acids peptides and proteins)Tumor necrosis factor alphaEndothelium VascularCardiology and Cardiovascular MedicineCell Adhesion MoleculesIloprostmedicine.drugBasic Research in Cardiology
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Heavy metal ion induction of adhesion molecules and cytokines in human endothelial cells: the role of NF-kappaB, I kappaB-alpha and AP-1.

1997

We analyzed the influence of heavy-metal ions on human umbilical vein endothelial cells (HUVEC) in comparison to proinflammatory cytokines (TNF-alpha, IL-1beta) and lipopolysaccharide (LPS). Adhesion molecule and cytokine expressions are upregulated by heavy-metal exposure. Expression of E-selectin on the cell surface was strongly induced by 1-mM concentrations of NiCl2 and CoCl2, whereas ZnCl2 and CrCl3 had no influence. Furthermore, it is shown that NiCl2 induces mRNA expression of E-selectin, intercellular adhesion molecule-1, IL-6 and IL-8 in a 1-mM concentration. The transcription factor NF-kappaB is known to be involved in the regulation of adhesion molecule expression in endothelial …

Umbilical VeinsLipopolysaccharideBlotting WesternUmbilical veinPathology and Forensic MedicineProinflammatory cytokineMetalchemistry.chemical_compoundNF-KappaB Inhibitor alphaMetals HeavyHumansRNA MessengerMolecular BiologyCells CulturedCell adhesion moleculeChemistrySingle-Strand Specific DNA and RNA EndonucleasesNF-kappa BNF-κBCell BiologyGeneral MedicineAdhesionBlotting NorthernMolecular biologyCell biologyUp-RegulationDNA-Binding ProteinsTranscription Factor AP-1Gene Expression Regulationvisual_artcardiovascular systemvisual_art.visual_art_mediumCytokinesTetradecanoylphorbol AcetateI-kappa B ProteinsEndothelium VascularSignal transductionDNA ProbesCell Adhesion MoleculesPathobiology : journal of immunopathology, molecular and cellular biology
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Enzymatically modified, nonoxidized LDL induces selective adhesion and transmigration of monocytes and T-lymphocytes through human endothelial cell m…

1999

Abstract —Circulating monocytes and T lymphocytes extravasate through the endothelium at sites of developing atheromatous lesions, where they tend to accumulate and mediate the progression of the disease. We have previously demonstrated the presence of an enzymatically degraded, nonoxidized form of LDL (E-LDL) in early human fatty streaks, which possesses major biological properties of an atherogenic lipoprotein. The effects of E-LDL on human endothelial cells have now been studied with respect to adhesion and transmigration of monocytes and T lymphocytes. E-LDL induced a rapid and dose-dependent selective adhesion of monocytes and T lymphocytes to endothelial cell monolayers within 30 min…

Umbilical VeinsP-selectinArteriosclerosisT-LymphocytesIntercellular Adhesion Molecule-1HL-60 CellsBiologyMonocytesMuscle Smooth VascularCell MovementE-selectinmedicineCell AdhesionHumansLymphocyte homing receptorCell adhesionDose-Response Relationship DrugMonocyteT lymphocyteCholesterol LDLIntercellular Adhesion Molecule-1Molecular biologyEndothelial stem cellLipoproteins LDLPlatelet Endothelial Cell Adhesion Molecule-1KineticsP-Selectinmedicine.anatomical_structureImmunologybiology.proteinlipids (amino acids peptides and proteins)Endothelium VascularCardiology and Cardiovascular MedicineE-SelectinArteriosclerosis, thrombosis, and vascular biology
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