Search results for "Agoni"

showing 10 items of 2493 documents

Effect of budesonide/formoterol maintenance and reliever therapy on asthma exacerbations

2007

This randomised, double-blind, 6-month study compared budesonide/formoterol for maintenance and relief with salmeterol/fluticasone and a fixed maintenance dose of budesonide/formoterol, both with terbutaline for relief. Following a 2-week run-in, 3335 symptomatic adults and adolescents (mean FEV1 73% predicted, mean inhaled corticosteroid dose 745 μg/day) received budesonide/formoterol 160/4.5 μg one inhalation bid plus additional inhalations as needed, salmeterol/fluticasone 25/125 μg two inhalations bid plus as-needed terbutaline or budesonide/formoterol 320/9 μg one inhalation bid plus as-needed terbutaline. Budesonide/formoterol for maintenance and relief prolonged the time to first sev…

Budesonidemedicine.drug_classbusiness.industryTerbutalineGeneral Medicinerespiratory systemrespiratory tract diseasesBudesonide/formoterolimmune system diseasesBronchodilatorAnesthesiamedicineFormoterol FumarateFormoterolSalmeterolbusinesshormones hormone substitutes and hormone antagonistscirculatory and respiratory physiologymedicine.drugFluticasoneInternational Journal of Clinical Practice
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Plasma membrane and lysosomal localization of CB1 cannabinoid receptor are dependent on lipid rafts and regulated by anandamide in human breast cance…

2005

AbstractIn this report we show, by confocal analysis of indirect immunofluorescence, that the type-1 cannabinoid receptor (CB1R), which belongs to the family of G-protein-coupled receptors, is expressed on the plasma membrane in human breast cancer MDA-MB-231 cells. However, a substantial proportion of the receptor is present in lysosomes. We found that CB1R is associated with cholesterol- and sphyngolipid-enriched membrane domains (rafts). Cholesterol depletion by methyl-β-cyclodextrin (MCD) treatment strongly reduces the flotation of the protein on the raft-fractions (DRM) of sucrose density gradients suggesting that CB1 raft-association is cholesterol dependent. Interestingly binding of …

CB1 receptorCannabinoid receptorMESH: Membrane MicrodomainsMESH: Receptor Cannabinoid CB1Biochemistrychemistry.chemical_compoundRaftsMESH: Cholesterol0302 clinical medicineReceptor Cannabinoid CB1Structural BiologyReceptorLipid raft0303 health sciencesChemistrybeta-CyclodextrinsAnandamideEndocannabinoid system3. Good healthCell biologyCholesterollipids (amino acids peptides and proteins)AgonistMESH: beta-CyclodextrinsMESH: Cell Line TumorPolyunsaturated Alkamidesmedicine.drug_classBiophysicsBreast NeoplasmsArachidonic Acids03 medical and health sciencesMembrane MicrodomainsCell Line TumorGeneticsmedicineMESH: Arachidonic AcidsHumansMolecular Biology030304 developmental biologyG protein-coupled receptorMESH: HumansMESH: Polyunsaturated AlkamidesCell Membrane[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyAnandamideCell BiologyCaveolin 1LysosomesIntracellular traffickingMESH: Breast Neoplasms030217 neurology & neurosurgeryMESH: Cell MembraneMESH: LysosomesEndocannabinoids
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Evidence for a modulatory role of cannabinoids on the excitatory NANC neurotransmission in mouse colon

2007

Abstract It is well accepted that endogenous cannabinoids and CB1 receptors are involved in the regulation of smooth muscle contractility and intestinal motility, through a mechanism mainly related to reduction of acetylcholine release from cholinergic nerve endings. Because, few data exist on a possible modulatory action of the cannabinoid agents on the non-adrenergic non-cholinergic (NANC) excitatory and inhibitory neurotransmission, the aim of the present study was to investigate the effects of cannabinoid drugs on the NANC responses elicited by electrical field stimulation (EFS) in the circular muscle of mouse proximal colon. Colonic contractions were monitored as changes in endoluminal…

CB1 receptorIndolesCannabinoid receptormedicine.medical_treatmentSynaptic TransmissionSettore BIO/09 - FisiologiaEnteric Nervous SystemReceptor Cannabinoid CB2Micechemistry.chemical_compoundPiperidinesReceptor Cannabinoid CB1Fatty acid amide hydrolaseCannabinoid receptor type 2musculoskeletal neural and ocular physiologyAnandamideSmooth muscle contractionRimonabantAgonistmedicine.medical_specialtyColonPolyunsaturated Alkamidesmedicine.drug_classMorpholinesNeuromuscular JunctionArachidonic AcidsIn Vitro TechniquesNaphthalenesTachykininsInternal medicineCannabinoid Receptor ModulatorsIntestinal motilitymedicineAnimalsCannabinoidReceptors TachykininPharmacologyDose-Response Relationship DrugCannabinoidsExcitatory Postsynaptic PotentialsNANC relaxationURB597Electric StimulationBenzoxazinesMice Inbred C57BLEndocrinologyInhibitory Postsynaptic PotentialschemistryPyrazolesNANC contractionCannabinoidGastrointestinal MotilityEndocannabinoidsPharmacological Research
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Allergen-induced IgE-dependent gut inflammation in a human PBMC-engrafted murine model of allergy.

2011

Background Humanized murine models comprise a new tool to analyze novel therapeutic strategies for allergic diseases of the intestine. Objective In this study we developed a human PBMC–engrafted murine model of allergen-driven gut inflammation and analyzed the underlying immunologic mechanisms. Methods Nonobese diabetic (NOD)– scid -γc −/− mice were injected intraperitoneally with human PBMCs from allergic donors together with the respective allergen or not. Three weeks later, mice were challenged with the allergen orally or rectally, and gut inflammation was monitored with a high-resolution video miniendoscopic system, as well as histologically. Results Using the aeroallergens birch or gra…

CD4-Positive T-LymphocytesAllergymedicine.medical_treatmentImmunologyHistamine AntagonistsAdministration OralInflammationNodMice SCIDPlatelet Membrane GlycoproteinsBiologymedicine.disease_causeImmunoglobulin ELymphocyte ActivationReceptors G-Protein-Coupledchemistry.chemical_compoundMiceAllergenimmune system diseasesAdministration RectalAntibody Specificityotorhinolaryngologic diseasesmedicineHypersensitivityImmunology and AllergyAnimalsHumansColitisMice KnockoutReceptors IgEAllergensImmunoglobulin Emedicine.diseaseDisease Models AnimalCytokinechemistryGastritisImmunologybiology.proteinLeukocytes MononuclearCytokinesPollenmedicine.symptomHistamineSpleenThe Journal of allergy and clinical immunology
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SOCS3 transactivation by PPARγ prevents IL-17-driven cancer growth.

2013

Abstract Activation of the transcription factor PPARγ by the n-3 fatty acid docosahexaenoic acid (DHA) is implicated in controlling proinflammatory cytokine secretion, but the intracellular signaling pathways engaged by PPARγ are incompletely characterized. Here, we identify the adapter-encoding gene SOCS3 as a critical transcriptional target of PPARγ. SOCS3 promoter binding and gene transactivation by PPARγ was associated with a repression in differentiation of proinflammatory T-helper (TH)17 cells. Accordingly, TH17 cells induced in vitro displayed increased SOCS3 expression and diminished capacity to produce interleukin (IL)-17 following activation of PPARγ by DHA. Furthermore, naïve CD4…

CD4-Positive T-LymphocytesCancer ResearchAngiogenesisMammary Neoplasms Experimental/genetics/pathology/prevention & controlSuppressor of Cytokine Signaling Proteinsddc:616.07BioinformaticsTransactivationMice0302 clinical medicineTumor Burden/drug effects/geneticsSOCS3Docosahexaenoic Acids/administration & dosage/pharmacologyPromoter Regions GeneticMice Knockout0303 health sciencesMice Inbred BALB CChemistryReverse Transcriptase Polymerase Chain ReactionInterleukin-17InterleukinCell DifferentiationCell biologyTumor BurdenOncology030220 oncology & carcinogenesisFemaleRNA InterferenceInterleukin 17Th17 Cells/drug effects/metabolismTranscriptional ActivationDocosahexaenoic AcidsBlotting WesternMice NudeCD4-Positive T-Lymphocytes/drug effects/metabolismProinflammatory cytokine03 medical and health sciencesSuppressor of Cytokine Signaling Proteins/genetics/metabolismCell Line TumorAnimalsTranscription factor030304 developmental biologyMammary Neoplasms ExperimentalPromoter Regions Genetic/geneticsDietMice Inbred C57BLPPAR gammaInterleukin-17/metabolismCell cultureSuppressor of Cytokine Signaling 3 ProteinCell Differentiation/drug effectsPPAR gamma/agonists/genetics/metabolismTh17 CellsCancer research
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Efficacy of tiotropium and olodaterol fixed-dose combination in patients with COPD on β-blockers

2015

Introduction: The efficacy and safety of a new once-daily (QD) fixed-dose combination (FDC) with tiotropium (T), a long-acting muscarinic antagonist, and olodaterol (O), a long-acting β 2 -agonist, was established for the treatment of COPD in the TONADO studies (NCT01431274; NCT01431287). This analysis evaluates the efficacy of the FDC in a subpopulation of patients receiving β-blockers (BBs) in these studies. Methods: Two replicate, randomised, double-blind, parallel-group, 52-week, Phase III trials assessed the efficacy and safety of T+O FDC (2.5/5 μg; 5/5 μg; Respimat ® inhaler) QD compared to the monocomponents. Key primary end point data for the combined analysis of the replicate trial…

COPDmedicine.medical_specialtyRespimatbusiness.industryInhalerOlodaterolFixed-dose combinationMuscarinic antagonistmedicine.diseasechemistry.chemical_compoundchemistryInternal medicinemedicinePhysical therapyClinical endpointIn patientbusinessmedicine.drug5.1 Airway Pharmacology and Treatment
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Cachexia: a therapeutic approach beyond cytokine antagonism

2002

Cachexia is seen in a number of chronic diseases, and it is always associated with a poor prognosis. Irrespective of etiology, the development of cachexia appears to share a common pathophysiological pathway. This includes induction of proteasome-dependent myofibril-degradation, which is thought to be secondary to stimulation by enhanced levels of pro-inflammatory cytokines. Elevation of tumor necrosis factor-alpha (TNFalpha) and other plasma cytokines has been demonstrated in many conditions associated with cachexia. Despite improved pathophysiological understanding, a specific treatment for cachexia has not yet been established. Whilst direct TNFalpha antagonism has therapeutic appeal, th…

CachexiaTumor Necrosis Factor-alphabusiness.industrymedicine.medical_treatmentNF-kappa BImmunosuppressionNF-κBmedicine.diseaseCachexiaTranscription Factor AP-1PathogenesisTherapeutic approachchemistry.chemical_compoundTreatment OutcomeCytokinechemistryImmunologyCytokinesHumansMedicineTumor necrosis factor alphaCardiology and Cardiovascular MedicineAntagonismbusinessImmunosuppressive AgentsInternational Journal of Cardiology
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Activin-A, myostatin and interleukin-6 in cancer associated cachexia

2017

Cachexia is a muscle wasting condition associated with multiple different chronic illnesses, such as cancer, diabetes and AIDS. In cancer, approximately 80% of patients with advanced disease have symptoms of muscle wasting, and around 25% of cancer mortality concerns cachexia. Elevated serum levels of different cytokines and TGF-β protein family members, such as Interleukin-6, Myostatin and Activin-A, have been observed in cachetic patients and test animals. However, the mechanistic role and the relative contribution of these molecules to muscle loss in the syndrome have not yet been fully elucidated. In this thesis, the gene-expression levels of Activin-A, Myostatin and Interleukin-6 was a…

CachexiamyostatiiniInterleukin-6interleukiinitaktiviini-aActivin-AsyöpätauditkakeksiaC2C12proteiinitMyostatinmusculoskeletal systemhormones hormone substitutes and hormone antagonists
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Microbial-mediated pre-salt carbonate deposition during the Messinian salinity crisis (Calcare di Base fm., Southern Italy)

2017

Abstract A multi-scale analysis of sedimentary carbonate facies and post-sedimentary diagenetic features of the Calcare di Base Formation, the precursor to evaporites in Upper Messinian successions of Northern Calabria and Central Sicily, has revealed their microbial bio-mediated origin. Massive to laminated microbial boundstones represent the most common sedimentary facies forming flat to low relief cm to m scale stromatolitic and thrombolitic bodies. The fabric of the micrite varies from peloidal to aphanitic, and almost always preserves filamentous bacteria which characterized the original microbial mat. The mat was dominated by sulphur-oxidizing bacteria belonging to the Thiotrichaceae,…

Calcite010504 meteorology & atmospheric sciencesMicriteEvaporiteStratigraphyAragoniteDolomiteGeochemistryGeologyengineering.material010502 geochemistry & geophysicsOceanography01 natural scienceschemistry.chemical_compoundPaleontologyGeophysicschemistryengineeringCarbonateEconomic GeologySedimentary rockMicrobial matMicrobialite Pre-salt carbonate Messinian salinity crisis Sulphur bacteria EvaporiteGeology0105 earth and related environmental sciencesMarine and Petroleum Geology
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‘Not All That Is White Is Lime’-White Substances from Archaeological Burial Contexts: Analyses and Interpretations

2019

Archaeological burial contexts may include a variety of white substances, but few analyses have been published. This study reports on the physico‐chemical characterization of such residues from seven archaeological sites. It is often assumed that white materials from burial contexts are lime. Our findings demonstrate that they can be gypsum, calcite (chalk), aragonite, brushite, degraded metal, natural (gum) resins or synthetic polymer–based products. These may be present as the result of diagenetic processes, funerary practices or modern contamination. This paper provides an analytical approach for the holistic investigation of white materials encountered in burial contexts.

Calcite010506 paleontologyArcheologyHistoryTaphonomyWhite (horse)Gypsum060102 archaeologyAragonite06 humanities and the artsengineering.material01 natural sciencesArchaeologyNatural (archaeology)Diagenesischemistry.chemical_compoundchemistryengineering0601 history and archaeologyGeology0105 earth and related environmental sciencesLimeArchaeometry
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