Search results for "Agoni"

showing 10 items of 2493 documents

ANP (Atrial Natriuretic Peptide) presence in the heart of a tunicate, Ciona intestinalis.

2010

Atrial natriuretic peptide was found in the heart of vertebrates, we studied the ANP presence in the heart of Ciona intestinalis. This is animal is very important because of the its evolutionary position between invertebrates and vertebrates. ANP presence was only revealed in myoepithelial cells of the myocardium. Results suggest the hypothesis that ANP is present not only in the vertebrates but also in the invertebrates and in Ciona heart ANP might play a similar role like in the heart of vertebrates.

Cell physiologymedicine.medical_specialtyHistologyPathology and Forensic MedicineAtrial natriuretic peptideInternal medicinebiology.animalANP heart tunicates Ciona intestinalismedicineAnimalsCiona intestinalislcsh:QH573-671ANP - Atrial natriuretic peptidebiologylcsh:CytologyMyocardiumMyoepithelial cellVertebrateGeneral Medicinebiology.organism_classificationCiona intestinalisTunicateCionaEndocrinologyembryonic structurescardiovascular systemAtrial Natriuretic Factorhormones hormone substitutes and hormone antagonistscirculatory and respiratory physiologyFolia Histochemica et Cytobiologica
researchProduct

Comparative analysis of virtual screening approaches in the search for novel EphA2 receptor antagonists

2015

The EphA2 receptor and its ephrin-A1 ligand form a key cell communication system, which has been found overexpressed in many cancer types and involved in tumor growth. Recent medicinal chemistry efforts have identified bile acid derivatives as low micromolar binders of the EphA2 receptor. However, these compounds suffer from poor physicochemical properties, hampering their use in vivo. The identification of compounds able to disrupt the EphA2-ephrin-A1 complex lacking the bile acid scaffold may lead to new pharmacological tools suitable for in vivo studies. To identify the most promising virtual screening (VS) protocol aimed at finding novel EphA2 antagonists, we investigated the ability of…

Cell signalingDatabases Pharmaceuticaldrug designPharmaceutical ScienceComputational biologyBiologyCrystallography X-RayMolecular Docking SimulationArticleAnalytical Chemistrylcsh:QD241-441Structure-Activity RelationshipUser-Computer Interfacelcsh:Organic chemistryPPI inhibitorsDrug Discoveryshape screeningStructure–activity relationshipPhysical and Theoretical ChemistryReceptorProtein Kinase InhibitorsVirtual screeningMolecular StructureDrug discoveryReceptor EphA2EphA2 antagonistOrganic ChemistryEphrin-A1virtual screeningEPH receptor A2C700Combinatorial chemistryMolecular Docking SimulationUniPR129Chemistry (miscellaneous)Docking (molecular)dockingMolecular Medicinepharmacophore search
researchProduct

Loss of input from the mossy cells blocks maturation of newly generated granule cells.

2007

The objective of this work is to check whether the input from the mossy cells to the inner molecular layer is necessary for the integration and maturation of the newly generated granule cells of the dentate gyrus (DG) in mice, and if after status epilepticus the sprouting of the mossy fibers can substitute for this projection. Newly generated cells were labeled by administration of 5-bromo-deoxyuridine either before or after pilocarpine administration. The neuronal loss in the hippocampus after administration of pilocarpine combined with scopolamine and diazepam seemed restricted to the hilar mossy cells. The maturation of the granule cells was studied using immunohistochemistry for calreti…

Cell typeCell SurvivalCognitive NeuroscienceScopolamineConvulsantsNerve Tissue ProteinsMuscarinic Antagonistschemistry.chemical_compoundMiceS100 Calcium Binding Protein GStatus EpilepticusmedicineAnimalsCell ProliferationDiazepamEpilepsyNeuronal PlasticitybiologyChemistryDentate gyrusStem CellsGranule (cell biology)PilocarpineNuclear ProteinsCell DifferentiationImmunohistochemistryDNA-Binding Proteinsnervous systemBromodeoxyuridinePilocarpineCalbindin 2Dentate GyrusMossy Fibers HippocampalNerve Degenerationbiology.proteinAnticonvulsantsFemaleNeuNCalretininNeuroscienceBromodeoxyuridineBiomarkersSproutingmedicine.drugHippocampus
researchProduct

A systematic review of inverse agonism at adrenoceptor subtypes

2020

As many, if not most, ligands at G protein-coupled receptor antagonists are inverse agonists, we systematically reviewed inverse agonism at the nine adrenoceptor subtypes. Except for β3-adrenoceptors, inverse agonism has been reported for each of the adrenoceptor subtypes, most often for β2-adrenoceptors, including endogenously expressed receptors in human tissues. As with other receptors, the detection and degree of inverse agonism depend on the cells and tissues under investigation, i.e., they are greatest when the model has a high intrinsic tone/constitutive activity for the response being studied. Accordingly, they may differ between parts of a tissue, for instance, atria vs. ventricles…

Cell typeDrug Inverse AgonismAdrenergic receptorDrug discoveryChemistryinverse agonismReviewpharmacology_toxicology570 Life sciencesBasal (phylogenetics)lcsh:Biology (General)Drug DevelopmentDrug developmentHumansInverse agonistAgonismReceptors Adrenergic beta-2Receptorlcsh:QH301-705.5adrenoceptorconstitutive activityNeuroscience570 Biowissenschaften
researchProduct

Nicotinic Receptors in Human Brain

1994

A vast knowledge is currently available on the molecular biology and the pharmacology of nicotinic acetylcholine receptors (nAChR) in the mammalian central nervous system (CNS) (Sargent, 1993). Only few attempts have been made to approach the expression of nAChRs at the level of functional systems, considering the different cell types involved and their connectivity. This aspect is of particular importance in order to evaluate nAChR expression under pathological conditions. Histochemical techniques have proven to be useful since immunohistochemistry and in situ hybridization can be performed on human autopsy tissue and allow for a cell type-specific localization of nAChR proteins and nAChR …

Cell typeReceptor expressionCentral nervous systemHuman brainIn situ hybridizationBiologyNicotinic acetylcholine receptorNicotinic agonistmedicine.anatomical_structurenervous systemmedicinesense organsNeuroscienceAcetylcholine receptor
researchProduct

Axonal control of the adult neural stem cell niche.

2014

SummaryThe ventricular-subventricular zone (V-SVZ) is an extensive germinal niche containing neural stem cells (NSCs) in the walls of the lateral ventricles of the adult brain. How the adult brain’s neural activity influences the behavior of adult NSCs remains largely unknown. We show that serotonergic (5HT) axons originating from a small group of neurons in the raphe form an extensive plexus on most of the ventricular walls. Electron microscopy revealed intimate contacts between 5HT axons and NSCs (B1) or ependymal cells (E1) and these cells were labeled by a transsynaptic viral tracer injected into the raphe. B1 cells express the 5HT receptors 2C and 5A. Electrophysiology showed that acti…

Cellular differentiationMessengerRegenerative MedicineMedical and Health SciencesImmunoenzyme TechniquesLateral ventriclesMice0302 clinical medicineNeural Stem CellsReceptor Serotonin 5-HT2C5-HT2CStem Cell NicheNeurons0303 health sciencesMicroscopyBlottingReverse Transcriptase Polymerase Chain ReactionNeurogenesisBrainCell DifferentiationAnatomyBiological SciencesNeural stem cellCell biologySerotonin Receptor AgonistsElectrophysiologyNeurologicalMolecular MedicineStem Cell Research - Nonembryonic - Non-HumanWesternReceptorSerotoninEpendymal CellNeurogenesis1.1 Normal biological development and functioningBlotting WesternBiologySerotonergicReal-Time Polymerase Chain ReactionElectronArticle03 medical and health sciencesUnderpinning researchGeneticsAnimalsRNA Messenger030304 developmental biologyCell ProliferationRapheNeurosciencesCell BiologyStem Cell ResearchAxonsMicroscopy Electronnervous systemRaphe NucleiRNARaphe nuclei030217 neurology & neurosurgeryDevelopmental Biology
researchProduct

Leukocyte Redistribution: Effects of Beta Blockers in Patients with Chronic Heart Failure

2009

BACKGROUND:Overproduction of pro-inflammatory cytokines is a well established factor in the progression of chronic heart failure (CHF). Changes in cellular immunity have not been widely studied, and the impact of standard medication is uncertain. Here we investigate whether a leukocyte redistribution occurs in CHF and whether this effect is influenced by beta-blocker therapy. METHODOLOGY:We prospectively studied 75 patients with systolic CHF (age: 68+/-11 years, left ventricular ejection fraction 32+/-11%, New York Heart Association class 2.5+/-0.7) and 20 age-matched healthy control subjects (age: 63+/-10 years). We measured the response of cells to endotoxin exposure in vitro, analysed su…

Cellular immunityAdrenergic beta-AntagonistsCardiovascular Disorders/Heart Failurelcsh:MedicineVentricular Function LeftAdrenergic beta-AntagonistsLeukocytesHumansMedicineIn patientProspective Studiescardiovascular diseaseslcsh:ScienceProspective cohort studyAgedHeart FailureMultidisciplinaryVentricular functionbusiness.industrylcsh:RCase-control studyMiddle AgedFlow Cytometrymedicine.diseaseCase-Control StudiesImmunology/Leukocyte ActivationHeart failureImmunologylcsh:QbusinessImmunology/Leukocyte DevelopmentResearch ArticlePLoS ONE
researchProduct

Hunting for the high-affinity state of G-protein-coupled receptors with agonist tracers: Theoretical and practical considerations for positron emissi…

2019

Abstract The concept of the high‐affinity state postulates that a certain subset of G‐protein‐coupled receptors is primarily responsible for receptor signaling in the living brain. Assessing the abundance of this subset is thus potentially highly relevant for studies concerning the responses of neurotransmission to pharmacological or physiological stimuli and the dysregulation of neurotransmission in neurological or psychiatric disorders. The high‐affinity state is preferentially recognized by agonists in vitro. For this reason, agonist tracers have been developed as tools for the noninvasive imaging of the high‐affinity state with positron emission tomography (PET). This review provides an…

Central Nervous SystemBETA-ADRENERGIC-RECEPTORpositron emission tomographyagonist high-affinity stateD-2/3 AGONISTG-protein-coupled receptorsReview ArticleReceptors G-Protein-Coupledchemistry.chemical_compound0302 clinical medicineDrug DiscoveryReceptorNeurotransmitterReview Articles0303 health sciencesmedicine.diagnostic_testNONHUMAN PRIMATE BRAINEndocytosisTEST-RETEST REPRODUCIBILITYPositron emission tomographyG‐protein‐coupled receptors030220 oncology & carcinogenesisENDOGENOUS OPIOID RELEASEMolecular MedicineIN-VIVO BINDINGSignal TransductionAgonistNoninvasive imagingexperimental designmedicine.drug_classNeurotransmissionRAT-BRAINneurotransmittersagonist high‐affinity state03 medical and health sciencesIn vivomedicineAnimalsHumanshuman brain030304 developmental biologyG protein-coupled receptorPharmacologyDOPAMINE D2(HIGH) RECEPTORS5-HT1A RECEPTORSchemistryPositron-Emission TomographyPET RADIOLIGANDRadiopharmaceuticalsNeuroscienceMedicinal research reviews
researchProduct

Control of spasticity in a multiple sclerosis model using central nervous system-excluded CB1 cannabinoid receptor agonists

2014

The purpose of this study was the generation of central nervous system (CNS)-excluded cannabinoid receptor agonists to test the hypothesis that inhibition of spasticity, due to CNS autoimmunity, could be controlled by affecting neurotransmission within the periphery. Procedures included identification of chemicals and modeling to predict the mode of exclusion; induction and control of spasticity in the ABH mouse model of multiple sclerosis; conditional deletion of CB1 receptor in peripheral nerves; side-effect profiling to demonstrate the mechanism of CNS-exclusion via drug pumps; genome-wide association study in N2(129×ABH) backcross to map polymorphic cannabinoid drug pump; and sequencing…

Central Nervous SystemCannabinoid receptorEncephalomyelitis Autoimmune ExperimentalMultiple Sclerosismedicine.medical_treatmentCentral nervous systemPharmacologyBiologyBiochemistryMiceReceptor Cannabinoid CB1GeneticsmedicineAnimalsSpasticityMolecular BiologyCannabinoid Receptor AgonistsCannabinoidsMultiple sclerosisExperimental autoimmune encephalomyelitisCannabinoid Receptor Agonistsmedicine.disease3. Good healthmedicine.anatomical_structureAjulemic acidMuscle SpasticityFemaleCannabinoidmedicine.symptomMultidrug Resistance-Associated ProteinsBiotechnologymedicine.drug
researchProduct

The Process-inducing Activity of Transmembrane Agrin Requires Follistatin-like Domains

2009

Clustering or overexpression of the transmembrane form of the extracellular matrix proteoglycan agrin in neurons results in the formation of numerous highly motile filopodia-like processes extending from axons and dendrites. Here we show that similar processes can be induced by overexpression of transmembrane-agrin in several non-neuronal cell lines. Mapping of the process-inducing activity in neurons and non-neuronal cells demonstrates that the cytoplasmic part of transmembrane agrin is dispensable and that the extracellular region is necessary for process formation. Site-directed mutagenesis reveals an essential role for the loop between beta-sheets 3 and 4 within the Kazal subdomain of t…

Central Nervous SystemFollistatinanimal structuresBiologyCytoplasmic partPC12 CellsBiochemistryProtein Structure SecondaryNeuromuscular junctionCell membraneExtracellular matrixMolecular Basis of Cell and Developmental BiologyProtein structureChlorocebus aethiopsmedicineAnimalsHumansAgrinMolecular BiologyNeuronsAgrinCell MembraneCell BiologyTransmembrane proteinProtein Structure TertiaryRatsCell biologymedicine.anatomical_structurenervous systemProteoglycanBiochemistryCOS CellsMutagenesis Site-Directedbiology.proteinFemaleChickenshormones hormone substitutes and hormone antagonistsJournal of Biological Chemistry
researchProduct