Search results for "Agonist-antagonist"

showing 4 items of 4 documents

Coupling between agonist and chloride ionophore sites of the GABA(A) receptor: agonist/antagonist efficacy of 4-PIOL.

2000

Eight gamma-aminobutyric acid (GABA) mimetics were tested on their ability to differentiate native GABA(A) receptor subtypes present in various rat brain regions. In rat brain cryostat sections, little regional variations by the agonistic actions of muscimol, thiomuscimol, 4,5,6,7-tetrahydroisoazolo(5,4-c)pyridin-3-ol, piperidine-4-sulphonic acid, taurine and beta-alanine on [35S]t-butylbicyclophosphorothionate ([35S]TBPS) binding to GABA(A) receptor channels were found. They were very similar to those found for GABA itself and indicated no direct correlation with single subunit distributions for any of these compounds. Only the low-efficacy GABA mimetic 5-(4-piperidyl)isoxazol-3-ol (4-PIOL…

AgonistMalemedicine.medical_specialtyAgonist-antagonistmedicine.drug_classBiologyLigandsPartial agonistGABAA-rho receptorCell Linechemistry.chemical_compoundPiperidinesInternal medicinemedicineAnimalsHumansRats WistarReceptorGABA AgonistsPharmacologyIonophoresGABAA receptorBrainIsoxazolesBridged Bicyclo Compounds HeterocyclicReceptors GABA-ARatsEndocrinologyMuscimolchemistryThiomuscimolEuropean journal of pharmacology
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Biogenic Amines Modulate Olfactory Receptor Neurons Firing Activity in Mamestra brassicae

2001

The modulatory effects of the biogenic amines octopamine and serotonin on pheromonal receptor neurons of Mamestra brassicae were investigated. The responses to sex pheromone components of two cells types (A and B) in single male long sensilla trichodea were monitored. Cell types A and B do not respond to the same compound. The response of type A to a pulse of the major sex pheromone component increased 5 min after octopamine injection. Responses of type B to other odorants increased after 30 min. In the absence of any pheromone stimulation the background firing activity of type A increased following octopamine injection. This background activity was used to evaluate the kinetics of octopami…

Agonistmedicine.medical_specialtyTime FactorsPhysiologymedicine.drug_classAgonist-antagonistMothsBiologySensory receptorClonidineOlfactory Receptor NeuronsBehavioral Neurosciencechemistry.chemical_compoundPhysiology (medical)Internal medicineBiogenic aminemedicineAnimalsAminesSex AttractantsNeurotransmitterOctopamineComputingMilieux_MISCELLANEOUSchemistry.chemical_classificationOlfactory receptorDose-Response Relationship Drug[SCCO.NEUR]Cognitive science/Neuroscience[SCCO.NEUR] Cognitive science/NeuroscienceOctopamine (drug)Sensory Systemsmedicine.anatomical_structureEndocrinologychemistry[ SCCO.NEUR ] Cognitive science/NeuroscienceSerotoninChemical Senses
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Central nicotinic receptors, neurotrophic factors and neuroprotection

2000

The multiple combinations of nAChR subunits identified in central nervous structures possess distinct pharmacological and physiological properties. A growing number of data have shown that compounds interacting with neuronal nAChRs have, both in vivo and in vitro, the potential to be neuroprotective and that treatment with nAChR agonists elicit long-lasting improving of cognitive performance in a variety of behavioural tests in rats, monkeys and humans. Epidemiological and clinical studies suggested also a potential neuroprotective/trophic role of (-)-nicotine in neurodegenerative disease, such as Alzheimer's and Parkinson's disease. Taken together experimental and clinical data largely ind…

Cell SurvivalAgonist-antagonistCentral nervous systemReceptors Nicotiniccomplex mixturesNeuroprotectionBehavioral NeuroscienceNeurotrophic factorsmental disordersmedicineAnimalsHumansNerve Growth FactorsAcetylcholine receptorNeuronsRegulation of gene expressionbiologymusculoskeletal neural and ocular physiologyBrainHaplorhinimedicine.diseaseRatsNeuroprotective Agentsmedicine.anatomical_structurenervous systembiology.proteinFibroblast Growth Factor 2sense organsAlzheimer's diseasePsychologyNeuroscienceNeurotrophinBehavioural Brain Research
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Raloxifene promotes prostacyclin release in human endothelial cells through a mechanism that involves cyclooxygenase-1 and -2

2005

Objective To examine the effects of raloxifene on prostacyclin production by human umbilical vein endothelial cells (HUVEC) and to shed light on the molecular details of that action. Design Cell culture for 4, 8, 16, 24, and 48 hours. Setting University research laboratory. Patient(s) Source of HUVEC. Intervention(s) Measurement of prostacyclin production and of protein levels and mRNA expression of cyclooxygenase (COX)-1 and -2. Main Outcome Measure(s) Prostacyclin production was measured by enzyme immunoassay, the mRNA expression of COX-1 was measured by quantitative real time-polymerase chain reaction, and the protein levels of COX-1 and -2 were measured by immunoblotting. Result(s) Ralo…

medicine.medical_specialtyEndotheliumAgonist-antagonistEstrogen receptorProstacyclinPharmacologyGene Expression Regulation EnzymologicUmbilical veinInternal medicinemedicineHumansCyclooxygenase InhibitorsRaloxifeneCells CulturedDose-Response Relationship DrugbiologyChemistryEndothelial CellsObstetrics and GynecologyEpoprostenolEndothelial stem cellmedicine.anatomical_structureEndocrinologyReproductive MedicineCyclooxygenase 2Raloxifene HydrochlorideCyclooxygenase 1biology.proteinCyclooxygenasemedicine.drugFertility and Sterility
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