Search results for "Allele"

showing 10 items of 1006 documents

Genetics and Beyond – The Transcriptome of Human Monocytes and Disease Susceptibility

2010

BACKGROUND: Variability of gene expression in human may link gene sequence variability and phenotypes; however, non-genetic variations, alone or in combination with genetics, may also influence expression traits and have a critical role in physiological and disease processes. METHODOLOGY/PRINCIPAL FINDINGS: To get better insight into the overall variability of gene expression, we assessed the transcriptome of circulating monocytes, a key cell involved in immunity-related diseases and atherosclerosis, in 1,490 unrelated individuals and investigated its association with >675,000 SNPs and 10 common cardiovascular risk factors. Out of 12,808 expressed genes, 2,745 expression quantitative trait …

AdultMaleChromosomes Human Pair 21Cardiovascular DisordersQuantitative Trait Locilcsh:MedicineGenome-wide association studyGenetics and Genomics/Complex TraitsBiologyPolymorphism Single NucleotideMonocytesTranscriptomeQuantitative Trait HeritableCell MovementRisk FactorsHumansGenetic Predisposition to DiseaseGenetics and Genomics/GenomicsAllelelcsh:ScienceGeneAgedGeneticsRegulation of gene expressionMultidisciplinaryBase SequenceGenome HumanGene Expression ProfilingSmokinglcsh:RImmunityGenetic VariationGenetics and GenomicsGenetics and Genomics/Gene ExpressionMiddle AgedAtherosclerosisPhenotypeHuman geneticsGene expression profilingPhenotypeGene Expression RegulationCardiovascular and Metabolic DiseasesFemalelcsh:QDNA ProbesGenome-Wide Association StudyResearch ArticlePLoS ONE
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Clock genes beyond the clock: CLOCK genotype biases neural correlates of moral valence decision in depressed patients

2007

Gene polymorphisms in the mammalian biological clock system influence individual rhythms. A single nucleotide polymorphism (SNP) in the 3' flanking region of CLOCK (3111 T/C; rs1801260) influenced diurnal preference in healthy humans and caused sleep phase delay and insomnia in patients affected by bipolar disorder. Genes of the biological clock are expressed in many brain structures other than in the 'master clock' suprachiasmatic nuclei. These areas, such as cingulate cortex, are involved in the control of many human behaviors. Clock genes could then bias 'nonclock' functions such as information processing and decision making. Thirty inpatients affected by a major depressive episode under…

AdultMaleCingulate cortexGenotypeDecision MakingCLOCK ProteinsMotor ActivityNeuropsychological TestsMoralsGyrus CinguliDevelopmental psychologyArousalBehavioral NeuroscienceImage Processing Computer-AssistedGeneticsmedicineHumansCircadian rhythmAllelesAgedDepressive Disorder MajorNeural correlates of consciousnessmedicine.diagnostic_testGenetic Carrier ScreeningHomozygoteNeuropsychologyMiddle AgedImage EnhancementMagnetic Resonance ImagingCircadian RhythmSemanticsOxygenCLOCKNeurologyTrans-ActivatorsFemaleMaster clockArousalFunctional magnetic resonance imagingPsychologyNeuroscienceGenes, Brain and Behavior
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Intestinal permeability and genetic determinants in patients, first-degree relatives, and controls in a high-incidence area of Crohn's disease in Sou…

2005

1. Am J Gastroenterol. 2005 Dec;100(12):2730-6. Intestinal permeability and genetic determinants in patients, first-degree relatives, and controls in a high-incidence area of Crohn's disease in Southern Italy. Fries W, Renda MC, Lo Presti MA, Raso A, Orlando A, Oliva L, Giofré MR, Maggio A, Mattaliano A, Macaluso A, Cottone M. Dipartimento di Medicina Interna e Terapia Medica, Università di Messina, Messina, Italy. OBJECTIVE: A defect of gastrointestinal barrier function is considered to represent an important step in the pathogenesis of Crohn's disease (CD) but the mechanisms leading to an increased intestinal permeability (IP) are poorly understood. Since IP is influenced by pro-inflammat…

AdultMaleEndemic DiseasesRisk AssessmentStatistics NonparametricPathogenesisCapillary PermeabilityCohort StudiesIntestinal mucosaCrohn DiseaseReference ValuesMedicineHumansGenetic Predisposition to DiseaseFirst-degree relativesIntestinal Mucosapermeability.crohn's disease.NOD2Allele frequencyProbabilityCrohn's diseaseIntestinal permeabilityHepatologybusiness.industryIncidenceGastroenterologyCase-control studyMiddle Agedmedicine.diseasedigestive system diseasesPedigreeToll-Like Receptor 4Genetics PopulationItalyGenetic markerCase-Control StudiesImmunologyMutationFemalebusiness
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Pathogenesis of autoimmune diseases associated with 8.1 ancestral haplotype: a genetically determined defect of C4 influences immunological parameter…

2003

Abstract Subjects with certain HLA alleles have a higher risk of specific autoimmune diseases than those without these alleles. The 8.1 ancestral haplotype (AH) is a common Caucasoid haplotype carried by most people who type for HLA-B8,DR3. It is unique in its association with a wide range of immunopathological diseases. To gain insight into the identification of the mechanism(s) of disease susceptibility of 8.1 AH carriers, we have investigated the prevalence of circulating immune complexes and non-organ-specific autoantibodies in healthy carriers of the haplotype. The results show that carriers of 8.1 AH display both a significant increased prevalence of immune complexes and higher titers…

AdultMaleEnzyme-Linked Immunosorbent AssayHuman leukocyte antigenBiologyAutoimmune DiseasesHLA-B8 AntigenImmune systemHLA-DR3 AntigenAntigenGene FrequencyHLA AntigensGenetic predispositionmedicineHumansAlleleAllelesPharmacologyAutoimmune diseaseGeneticsHaplotypeAutoantibodyComplement C4General MedicineMiddle Agedmedicine.diseaseHaplotypesImmunologyFemaleBiomedicinepharmacotherapy = Biomedecinepharmacotherapie
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Microsatellite allele A5.1 of MHC class I chain-related gene A is associated with latent autoimmune diabetes in adults in Latvia.

2006

NIDDM is one of the most common forms of diabetes. The diagnosis is based on WHO classification, which is a clinical classification and misses the autoimmune diabetes in adults. Therefore, among the clinically diagnosed NIDDM cases, there can be a certain number of patients with latent autoimmune diabetes in adults (LADA). The MICA gene is located in the MHC class I region and is expressed by monocytes, keratinocytes, and endothelial cells. Sequence determination of the MICA gene identifies trinucleotide repeat (GCT) microsatellite polymorphism, which identifies 5 alleles with 4, 5, 6, and 9 repetitions of GCT (A4, A5, A6, and A9) or 5 repetitions of GCT with 1 additional G insertion for al…

AdultMaleGeneral Biochemistry Genetics and Molecular Biologylaw.inventionHistory and Philosophy of ScienceGene FrequencylawDiabetes mellitusMHC class ImedicineHumansGenetic Predisposition to DiseaseAlleleAge of OnsetPolymerase chain reactionAllelesbiologyGeneral NeuroscienceHistocompatibility Antigens Class Imedicine.diseaseLatviastomatognathic diseasesDiabetes Mellitus Type 2HaplotypesImmunologybiology.proteinMicrosatelliteFemaleAge of onsetAntibodyTrinucleotide repeat expansionMicrosatellite RepeatsAnnals of the New York Academy of Sciences
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Cholinesterase variants: rapid characterisation by PCR/SSCP and evidence for molecular homogeneity.

1995

We have applied the technique of PCR-SSCP (polymerase chain reaction-single stranded conformation polymorphism) to characterise the molecular basis of cholinesterase deficiency and variants in a Jordanian family. PCR-SSCP proved to be a quick and sensitive method of screening cholinesterase variants in a clinical setting. An AG insertion at position 351 was found to cause a silent allele, for which the parents were heterozygous and three children homozygous. In addition, the father and two sons were heterozygous for an A to G transition at position 209, known to cause the dibucaine resistant variant. No linkage to the K variant was found, which has been reported previously in white populati…

AdultMaleGenotypeGenetic LinkageMolecular Sequence DataDibucainePolymerase Chain ReactionFrameshift mutationlaw.inventionlawGenetic linkageGenotypeGeneticsCholinesterasesHumansPoint MutationGenetic TestingAlleleFrameshift MutationGenetics (clinical)PolymerasePolymerase chain reactionAllelesPolymorphism Single-Stranded ConformationalCholinesteraseGeneticsJordanbiologyBase SequencePoint mutationSequence Analysis DNAMolecular biologyPedigreebiology.proteinFemaleMetabolism Inborn ErrorsResearch ArticleJournal of medical genetics
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The val158met polymorphism of human catechol-O-methyltransferase (COMT) affects anterior cingulate cortex activation in response to painful laser sti…

2010

Background: Pain is a complex experience with sensory, emotional and cognitive aspects. Genetic and environmental factors contribute to pain-related phenotypes such as chronic pain states. Genetic variations in the gene coding for catechol-O-methyltransferase ( COMT) have been suggested to affect clinical and experimental pain-related phenotypes including regional μ-opioid system responses to painful stimulation as measured by ligand-PET (positron emission tomography). The functional val158met single nucleotide polymorphism has been most widely studied. However, apart from its impact on pain-induced opioid release the effect of this genetic variation on cerebral pain processing has not been…

AdultMaleGenotypePainSingle-nucleotide polymorphismStimulationCatechol O-MethyltransferaseGyrus CinguliCellular and Molecular NeuroscienceYoung Adultmedicinelcsh:PathologyHumansddc:610AlleleAnterior cingulate cortexCerebral CortexCatechol-O-methyl transferasePolymorphism Geneticmedicine.diagnostic_testResearchLasersChronic painMiddle Agedmedicine.diseaseMagnetic Resonance ImagingAnesthesiology and Pain Medicinemedicine.anatomical_structureCerebral cortexPositron-Emission TomographyMolecular MedicineFemaleFunctional magnetic resonance imagingPsychologyNeurosciencelcsh:RB1-214Molecular Pain
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Increased frequency of the CTLA-4 49 A/G polymorphism in patients with acquired haemophilia A compared to healthy controls

2007

Acquired haemophilia (AH) is an autoimmune disorder characterized by autoantibodies against endogenous factor VIII (FVIII). Half of the patients present with an underlying disease known to cause the FVIII autoantibodies whereas in the other half the disease is of idiopathic nature. Recently, it has been shown that variants of the polymorphic cytotoxic T lymphocyte antigen-4 (CTLA-4) gene are associated with autoimmune diseases and also represent a risk factor for inhibitor formation in inherited haemophilia A. In the present study, we investigated whether CTLA-4 variants also play a role in the pathogenesis of AH. Therefore, we analyzed three single nucleotide polymorphisms (SNPs) of the CT…

AdultMaleGenotypeSingle-nucleotide polymorphismHemophilia AHaemophiliaPolymorphism Single NucleotideGene FrequencyAntigens CDGenotypemedicineHumansCTLA-4 AntigenGenetic Predisposition to DiseaseAlleleAllele frequencyGenetics (clinical)AgedAutoantibodiesAged 80 and overAutoimmune diseaseFactor VIIIbusiness.industryAutoantibodyHematologyGeneral MedicineMiddle Agedmedicine.diseaseAntigens DifferentiationCase-Control StudiesImmunologyFemaleGene polymorphismbusinessHaemophilia
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The -308G/A polymorphism of TNF-alpha influences immunological parameters in old subjects affected by infectious diseases.

2005

Abnormal increments of pro-inflammatory cytokines (IL-6 and TNF-alpha) characterize the outbreak of infectious diseases, which are the major cause of death in the elderly. A counterbalance to the inflammation is exerted by IL-10 with an inhibitory role on TNF-alpha production. As is well known, some cytokine gene polymorphisms influence the cytokine production, playing a role as susceptibility or resistance factors against immune-mediated and infectious disease. Genetic variations in the -308A/G locus for TNF-alpha seems to affect the clinical outcome of some infectious diseases. In fact, the -308A allele is associated with severe septic shock and death. On this basis, we have screened heal…

AdultMaleGenotypemedicine.medical_treatmentImmunologyCommunicable DiseasesGene FrequencyGenotypeGeneticsmedicineBronchopneumoniaHumansAlleleInterleukin 6Molecular BiologyAllele frequencyGenetics (clinical)AgedPolymorphism GeneticbiologyInterleukin-6Tumor Necrosis Factor-alphaOutbreakGeneral MedicineMiddle AgedBronchitis ChronicKiller Cells NaturalZincCytokineInfectious disease (medical specialty)Immunologybiology.proteinTumor necrosis factor alphaFemaleMetallothioneinInternational journal of immunogenetics
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A functional polymorphism in theIL-10 promoter influences the response after vaccination with HBsAg and hepatitis A

2005

The immune response to hepatitis B surface antigen (HBsAg) is mostly genetically determined. Interleukin 10 (IL-10) is a central immunoregulatory cytokine with important effects on B-cells. We have studied the influence of IL-10 promoter polymorphisms on the immune response to HBsAg and hepatitis A vaccination. We vaccinated 202 twin pairs in an open prospective study with a combined recombinant HBsAg/inactivated hepatitis A vaccine. IL-10 promoter polymorphisms were investigated in all individuals and their influence on anti-HBs, and anti-HAV responsiveness was studied. In the multiple regression analysis accounting for smoking, gender, body mass index and age, the ACC haplotype (-1082, -8…

AdultMaleHBsAgHepatitis A vaccineTwinsBiologyAntigenmedicineHumansProspective StudiesAllelePromoter Regions GeneticAntigens ViralHepatitisHepatitis A VaccinesHepatitis B Surface AntigensPolymorphism GeneticHepatologyHaplotypevirus diseasesHepatitis Amedicine.diseaseVirologydigestive system diseasesInterleukin-10VaccinationImmunologyFemaleHepatology
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