Search results for "Allele"

showing 10 items of 1006 documents

Parent of origin effects in attention/deficit hyperactivity disorder (ADHD): analysis of data from the international multicenter ADHD genetics (IMAGE…

2008

Contains fulltext : 71540.pdf (Publisher’s version ) (Closed access) There are conflicting reports suggesting that the parental origin of transmitted risk alleles may play a role in the etiology of attention deficit/hyperactivity disorder (ADHD). A recent report by Hawi and colleagues observed a generalized paternal over-transmission of alleles associated with ADHD. This was not replicated in more recent studies. Using data from a large multicenter study we examined the overall and gene-specific parent of origin effect in 554 independent SNPs across 47 genes. Transmission disequilibrium and explicit parent of origin test were performed using PLINK. Overall parent of origin effect was tested…

ParentsCandidate geneGenetics and epigenetic pathways of disease [NCMLS 6]2804 Cellular and Molecular NeuroscienceMedizinNeuroinformatics [DCN 3]Linkage Disequilibrium2738 Psychiatry and Mental Health0302 clinical medicineRisk FactorsPerception and Action [DCN 1]Genetics(clinical)Genetics (clinical)0303 health sciencesTPH210058 Department of Child and Adolescent PsychiatryPsychiatry and Mental healthData Interpretation StatisticalFunctional Neurogenomics [DCN 2]Clinical psychologyGenetic Markers2716 Genetics (clinical)Single-nucleotide polymorphism610 Medicine & healthMental health [NCEBP 9]Polymorphism Single NucleotideGenetic determinismGenomic disorders and inherited multi-system disorders [IGMD 3]Molecular epidemiology [NCEBP 1]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]Translational research [ONCOL 3]medicineAttention deficit hyperactivity disorderHumansFamilyGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersAlleleAlleles030304 developmental biologyChi-Square DistributionEndocrinology and reproduction [UMCN 5.2]business.industrymedicine.diseaseGenetic defects of metabolism [UMCN 5.1]Multicenter studyAttention Deficit Disorder with HyperactivityEtiologybusiness030217 neurology & neurosurgeryAmerican journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics
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Analysis of the RET, GDNF, EDN3, and EDNRB genes in patients with intestinal neuronal dysplasia and Hirschsprung disease

2001

BACKGROUNDHirschsprung disease (HSCR) is a frequent congenital disorder with an incidence of 1 in 5000 live births, characterised by the absence of parasympathetic intramural ganglion cells in the hindgut resulting in intestinal obstruction in neonates and severe constipation in infants and adults. Intestinal neuronal dysplasia (IND) shares clinical features with HSCR but the submucosal parasympathetic plexus is affected. IND has been proposed as one of the most frequent causes of chronic constipation and is often associated with HSCR.METHODSWe examined 29 patients diagnosed with sporadic HSCR, 20 patients with IND, and 12 patients with mixed HSCR/IND for mutations in the coding regions of …

Pathologymedicine.medical_specialtyGlial Cell Line-Derived Neurotrophic Factor ReceptorsHirschsprung diseaseMUTATION ANALYSISNerve Tissue ProteinsTYROSINE KINASEEDNRBArticleExonGermline mutationProto-Oncogene ProteinsNEUROTROPHIC FACTOR GDNFmedicineGlial cell line-derived neurotrophic factorDrosophila ProteinsHumansGlial Cell Line-Derived Neurotrophic FactorNerve Growth FactorsAlleleintestinal neuronal dysplasiaAllelesPolymorphism Single-Stranded ConformationalIntestinal neuronal dysplasiabiologyReceptors EndothelinSHAH-WAARDENBURG SYNDROMEProto-Oncogene Proteins c-retENDOTHELIN-B-RECEPTORMULTIGENIC INHERITANCEGastroenterologyReceptor Protein-Tyrosine KinasesSequence Analysis DNAGERMLINE MUTATIONSbiochemical phenomena metabolism and nutritionPROTOONCOGENEmedicine.diseasePHENOTYPIC-EXPRESSIONGDNFPedigreeProto-Oncogene Proteins c-retDysplasiaCase-Control StudiesMutationbiology.proteinLIGANDRETCongenital disorderEDN3
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A MULTI-LEVEL FUNCTIONAL STUDY OF A SNAP25 AT-RISK VARIANT FOR BIPOLAR DISORDER AND SCHIZOPHRENIA

2019

Background The synaptosomal associated protein SNAP25 is crucial for synaptic vesicle docking and fusion and has been associated with multiple psychiatric conditions. We recently identified a promoter variant in SNAP25, rs6039769, associated with bipolar disorder and gene expression in prefrontal cortex. Methods Here, we performed a genetic association study using this variation on two independent cohorts of 288 and 173 subjects with schizophrenia and 315 unaffected control individuals. We replicated our results using data from the schizophrenia group of the Psychiatric Genomics Consortium (PGC). Functional consequences combined both in vitro and post-mortem gene expression analysis on 30 p…

Pharmacologymedicine.diagnostic_testbusiness.industryImaging geneticsSNAP25medicine.diseaseBioinformaticsPsychiatry and Mental healthNeurologySynaptic vesicle dockingMedicinePharmacology (medical)Neurology (clinical)Bipolar disorderAllelebusinessFunctional magnetic resonance imagingPrefrontal cortexBiological PsychiatryGenetic associationEuropean Neuropsychopharmacology
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Association study of affective disorders with genetic polymorphisms of monoamine oxidases

2000

Introduction: Monoamine oxidases (MAO) catalyze the oxidative deamination of monoamines like norepinephrine, serotonin and dopamine. The existing MAOs (A and B) have distinct although partially overlapping biological functions and distributions in the brain. MAO A is mainly expressed in catecholaminergic neurons. Thirty-fold differences in enzyme activity of MAO A can be found in cultured cells from different individuals suggesting a genetic determination of enzyme activity. Indeed, a point mutation in the coding region of the gene which creates a restriction site for Fnu4HI alters the activity. Moreover, the pharmacological inhibition of monoamine oxidase A activity is one of the most effe…

Pharmacologymedicine.medical_specialtybiologybusiness.industrymedicine.diseaseGenotype frequencySubstance abusePsychiatry and Mental healthMonoamine neurotransmitterEndocrinologyNeurologyMood disordersInternal medicineGenetic variationmedicinebiology.proteinPharmacology (medical)Neurology (clinical)Monoamine oxidase BAlleleMonoamine oxidase AbusinessBiological PsychiatryEuropean Neuropsychopharmacology
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Genetic and morphological differentiation in Tephritis bardanae (Diptera: Tephritidae): evidence for host-race formation

2003

The fruit fly Tephritis bardanae infests flower heads of two burdock hosts, Arctium tomentosum and A. minus. Observations suggest host-associated mating and behavioural differences at oviposition indicating host-race status. Previously, flies from each host plant were found to differ slightly in allozyme allele frequencies, but these differences could as well be explained by geographical separation of host plants. In the present study, we explicitly test whether genetic and morphological variance among T. bardanae are explained best by host-plant association or by geographical location, and if this pattern is stable over a 10-year period. Populations of A. tomentosum flies differed signific…

Phenotypic plasticitybiologyHost (biology)fungiTephritis bardanaebiology.organism_classificationArctium tomentosumGene flowEvolutionary biologyTephritidaeBotanyGenetic variationAllele frequencyEcology Evolution Behavior and SystematicsJournal of Evolutionary Biology
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Molecular Basis Of Mild Hyperphenylalaninaemia In Turkey

2000

Öz bulunamadı.

Phenylalanine hydroxylaseGenotypeTurkeyPhenylalanineDNA Mutational AnalysisMEDLINEHyperphenylalaninemiaGene FrequencyPhenylketonuriasGenotypeDNA Mutational AnalysisGeneticsmedicineHumansChildAllele frequencyGenetics (clinical)Geneticsbiologybusiness.industryInfantPhenylalanine Hydroxylasemedicine.diseasePhenotypeHuman geneticsPhenotypeChild Preschoolbiology.proteinbusiness
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A new self-compatibility haplotype in the sweet cherry 'Kronio', S5' attributable to a pollen-part mutation in the SFB gene

2008

‘Kronio’ is a Sicilian cultivar of sweet cherry (Prunus avium), nominally with the incompatibility genotype S 5 S 6 , that is reported to be naturally self-compatible. In this work the cause of its self-compatibility was investigated. Test selfing confirmed self-compatibility and provided embryos for analysis; PCR with consensus primers designed to amplify S-RNase and SFB alleles showed that the embryos were of two types, S 5 S 5 and S 5 S 6 , indicating that S 6 pollen failed, but S 5 succeeded, perhaps because of a mutation in the pollen or stylar component. Stylar RNase analysis indicated active S-RNases for both S 5 and S 6 . The S-RNase alleles were cloned and sequenced; and sequences …

PhysiologyMolecular Sequence DataPlant ScienceFlowersBiologyPolymerase Chain ReactionPrunusRibonucleasesChromosome SegregationGenotypeAmino Acid SequencePollinationGeneAllelesCrosses GeneticGeneticsF-Box ProteinsHaplotypeIntronfood and beveragesSelfingSequence Analysis DNAHaplotypesSeedlingsMutationMicrosatellitePrunusPloidy
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Genome-wide parent-of-origin DNA methylation analysis reveals the intricacies of human imprinting and suggests a germline methylation-independent mec…

2014

Genomic imprinting is a form of epigenetic regulation that results in the expression of either the maternally or paternally inherited allele of a subset of genes (Ramowitz and Bartolomei 2011). This imprinted expression of transcripts is crucial for normal mammalian development. In humans, loss-of-imprinting of specific loci results in a number of diseases exemplified by the reciprocal growth phenotypes of the Beckwith-Wiedemann and Silver-Russell syndromes, and the behavioral disorders Angelman and Prader-Willi syndromes (Kagami et al. 2008; Buiting 2010; Choufani et al. 2010; Eggermann 2010; Kelsey 2010; Mackay and Temple 2010). In addition, aberrant imprinting also contributes to multige…

PlacentaADNGene ExpressionBiologyMethylationGenomic ImprintingPregnancyGerm cellsGeneticsmedicineHumansEpigeneticsRNA-Directed DNA MethylationAllelesEmbryonic Stem CellsGenetics (clinical)GeneticsGenome HumanResearchDNAGenomicsDNA Methylationmedicine.diseaseUniparental disomyCèl·lules germinalsGenòmicaGerm CellsDifferentially methylated regionsDNA methylationIllumina Methylation AssayCpG IslandsFemaleMetilacióGenomic imprintingReprogrammingGenome Research
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Identification of two new alleles at k-casein (CSN3) gene in Girgetana goat breed

2013

In the milk of ruminants, more than 95% of proteins are synthesized by six structural genes, four caseins and two whey proteins. The κ-casein fraction plays an important role in the formation, stabilization and aggregation on casein micelles and thus affects technological and nutritional properties of milk. Goat κ-casein (CSN3) gene comprises five exons with the mRNA coding region for mature protein spanning from exon 3 to exon 4. The κ-casein locus has been widely investigated and several genetic variants have been found in different goat breeds. At protein level, AIEF and BIEF groups corresponding to isoelectric point 5.53 and 5.78, respectively, have been identified. In this study, exon …

Polymorphisms new alleles CSN3 gene Girgentana goat breedSettore AGR/17 - Zootecnica Generale E Miglioramento Genetico
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Genetic diversity within the R408W phenylketonuria mutation lineages in Europe

2003

The R408W phenylketonuria mutation in Europe has arisen by recurrent mutation in the human phenylalanine hydroxylase (PAH) locus and is associated with two major PAH haplotypes. R408W-2.3 exhibits a west-to-east cline of relative frequency reaching its maximum in the Balto–Slavic region, while R408W-1.8 exhibits an east-to-west cline peaking in Connacht, the most westerly province of Ireland. Spatial autocorrelation analysis has demonstrated that the R408W-2.3 cline, like that of R408W-1.8, is consistent with a pattern likely to have been established by human dispersal. Genetic diversity within wild-type and R408W chromosomes in Europe was assessed through variable number tandem repeat (VNT…

Population geneticsEurope; PAH; Phenylalanine hydroxylase; Phenylketonuria; PKU; Population genetics; STR; VNTR;VNTRPopulation geneticsLocus (genetics)Minisatellite RepeatsBiologyArginineSTRPhenylketonuriasGeneticsHumansPhenylketonuriaGenetic TestingAlleleGenetics (clinical)GeneticsGenetic diversityPhenylalanine hydroxylaseHaplotypeTryptophanGenetic VariationCline (biology)PAHFounder EffectEuropeVariable number tandem repeatAmino Acid SubstitutionPKUMutationMicrosatelliteMicrosatellite Repeats
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