Search results for "Alleles"

showing 10 items of 478 documents

Association between neonatal temperament,SLC6A4,DRD4and a functional polymorphism located inTFAP2B

2011

Genetic studies on human personality have provided little satisfactory results to date mainly because of the complexity of this trait. Neonatal temperament using observational measures is an alternative phenotype to approach genetics to human behavior. An association study was conducted on 117 Caucasian newborns. Their temperament was evaluated using the Neonatal Behavior Assessment Scale 48 h after birth. Thirteen polymorphisms in the SLC6A4, DRD4 and TFAP2B genes were genotyped. Linear regression was performed to analyze data, and Bonferroni correction was applied. To check the functional effect of the TFAP2B Indel Intron 2 polymorphism, reporter gene luciferase assays using a mouse corti…

MaleGenotypemedia_common.quotation_subjectMinisatellite RepeatsBiologyBehavioral NeuroscienceExonGeneticsHumansAlleleTemperamentIndelGeneAllelesGenetic Association Studiesmedia_commonSerotonin Plasma Membrane Transport ProteinsGeneticsPolymorphism GeneticReceptors Dopamine D4Infant NewbornIntronVariable number tandem repeatReal-time polymerase chain reactionTranscription Factor AP-2NeurologyInfant BehaviorFemaleTemperamentGenes, Brain and Behavior
researchProduct

Enzyme polymorphisms and haemoglobin variants in Greeks

1975

Several enzyme polymorphisms and hemoglobin variants were typed in a sample of n = 219 non-related Greek blood-donors. The following gene frequencies were observed: pa = 0.201, pb = 0.701, pc = 0.098;PGDA = 0.985, PGDc = 0.015; AK1 = 0.942, AK2 = 0.058; HbA = 0.988, HbS = 0.012. No polymorphic variation was seen in LDH, s-MDH, PHI, or SOD. The population genetical aspects of these results are discussed.

MaleHemoglobins AbnormalAcid PhosphatasePopulationBlood DonorsBiologyHaemoglobin variantsGene FrequencyMalate DehydrogenaseGeneticsHumansMetabolic diseaseeducationGeneAllele frequencyAllelesGenetics (clinical)Geneticschemistry.chemical_classificationeducation.field_of_studyPolymorphism GeneticGreeceL-Lactate DehydrogenaseSuperoxide DismutasePhosphogluconate DehydrogenasePhosphotransferasesGlucose-6-Phosphate IsomeraseGenetic VariationHemoglobin variantsMolecular biologyAK2IsoenzymesPhenotypeEnzymechemistryFemaleHuman Genetics
researchProduct

Cathepsin C gene: First compound heterozygous patient with Papillon-Lefèvre syndrome and a novel symptomless mutation.

2001

Papillon-Lefevre syndrome (PLS) has recently been shown to be caused by mutations in the cathepsin C gene resulting in periodontal disease and palmoplantar keratosis. Thirteen different homozygous mutations have been characterised in PLS patients of different ethnic origin. In the present paper, a PLS patient is described who carries two novel mutations (706G>T and 872G>A) in the paternal and maternal chromosomes, respectively. This is the first compound patient described so far. In addition, a novel symptomless mutation (458C>T) in the cathepsin C gene is described in three homozygous individuals. Thus, not all mutations should be considered as a cause of disease, whether case studies or g…

MaleHeterozygoteHaim–Munk syndromeDNA Mutational AnalysisMolecular Sequence DataMutation MissensePapillon–Lefèvre syndromeBiologyCompound heterozygositymedicine.disease_causePapillon-Lefevre DiseaseCathepsin CCathepsin CPapillon-Lefevre DiseaseGene FrequencyGeneticsmedicineHumansAlleleAllele frequencyAllelesGenetics (clinical)Family HealthGeneticsMutationDNAmedicine.diseaseMutationFemale
researchProduct

Compound heterozygosity in the SPG4 gene causes hereditary spastic paraplegia

2008

The SPG4 gene is frequently mutated in autosomal dominant form of hereditary spastic paraplegia (HSP). We report that the compound heterozygous sequence variants S44L, a known polymorphism, and c.1687G>A, a novel mutation in SPG4 cause a severe form of HSP in a patient. The family members carrying solely c.1687G>A mutation are asymptomatic for HSP. The reverse transcriptase-polymerase chain reaction (RT-PCR) analysis revealed that the c.1687G>A mutation is a splice site mutation and causes skipping of the exon 15 of spastin. Furthermore, quantification of RT-PCR products by sequencing and quantification of allele-specific expression by pyrosequencing assay revealed that c.1687G>A is a leaky…

MaleHeterozygoteSpastinHereditary spastic paraplegiaDNA Mutational AnalysisMolecular Sequence DataMutantIntracellular SpaceBiologyCompound heterozygositySpastinPolymorphism Single NucleotideWhite PeopleLoss of heterozygosity03 medical and health sciencesExon0302 clinical medicineGermanyGeneticsmedicineHumansRNA MessengerAllelesGenetics (clinical)030304 developmental biologyAdenosine TriphosphatasesRegulation of gene expressionGenetics0303 health sciencesSplice site mutationBase SequenceSpastic Paraplegia HereditaryComputational BiologyExonsmedicine.diseasePedigreeProtein TransportAmino Acid SubstitutionGene Expression RegulationMutationFemaleRNA Splice Sites030217 neurology & neurosurgeryHeLa CellsClinical Genetics
researchProduct

A Single Copy of the Recently Identified Dual Oxidase Maturation Factor (DUOXA) 1 Gene Produces Only Mild Transient Hypothyroidism in a Patient with …

2011

Dual oxidases (DUOX1 and DUOX2) play a crucial role in the generation of hydrogen peroxide required in the oxidation of iodide and the synthesis of thyroid hormone. Heterodimerization with specific maturation factors (DUOXA1 and DUOXA2) is essential for the maturation and function of the DUOX enzyme complexes. Biallelic loss-of-function mutations of DUOX2 result in congenital hypothyroidism (CH), whereas a single reported case of homozygous DUOXA2 mutation (Y246X) has been associated with mild CH.We now report an infant with transient CH due to a complex genetic alteration of the DUOX/DUOXA system.Our patient was born to euthyroid nonconsanguineous parents and presented with an elevated TSH…

MaleHeterozygoteendocrine system diseasesEndocrinology Diabetes and MetabolismBlotting WesternGenetic VectorsClinical BiochemistryGene DosageMutation MissenseThyrotropinBiologyTransfectionmedicine.disease_causePolymorphism Single NucleotideBiochemistryGene dosageEndocrinologyHypothyroidismPolymorphism (computer science)medicineHumansMissense mutationAlleleGeneAllelesCells CulturedOligonucleotide Array Sequence AnalysisGeneticsMutationfungiBiochemistry (medical)Infant NewbornMembrane ProteinsNADPH OxidasesNucleic Acid Hybridizationfood and beveragesHeterozygote advantageJCEM Online: Brief ReportsDNADual OxidasesMolecular biologyMembrane proteinGene DeletionThe Journal of Clinical Endocrinology & Metabolism
researchProduct

H-ficolin (ficolin-3) concentrations and FCN3 gene polymorphism in neonates

2011

Serum H-ficolin (ficolin-3) concentrations (n=613) and FCN3 genotypes (n=529) from a large group of neonates are presented. Both pre-term deliveries and low birthweight (independently of gestational age) were significantly associated with low H-ficolin concentrations but not with heterozygosity for the FCN3 1637delC frameshift mutation. The presence of the variant allele, however, apparently influenced the protein level. No association of FCN3 gene heterozygosity or relative functional H-ficolin insufficiency (determined as serum level ≤8.6 μg/ml) with perinatal infections was found. One premature newborn, with confirmed infection caused by Streptococcus agalactiae, was H-ficolin-deficient …

MaleHeterozygotemedicine.medical_specialtyGenotypeImmunologyGestational AgeBiologymedicine.disease_causeMannose-Binding LectinStreptococcus agalactiaeFrameshift mutationLoss of heterozygosityPolymorphism (computer science)LectinsStreptococcal InfectionsInternal medicineGenotypemedicineHumansImmunology and AllergyFrameshift MutationAllelesGlycoproteinsMannan-binding lectinPolymorphism GeneticHomozygoteInfant NewbornHematologyInfant Low Birth WeightEndocrinologyStreptococcus agalactiaeMannose-Binding Protein-Associated Serine ProteasesImmunologyPremature BirthFemaleGene polymorphismFicolinImmunobiology
researchProduct

Risk of chemotherapy-associated liver injury (CALI) in PNPLA3 p.148M allele carriers: Preliminary results of a transient elastography-based study

2019

Liver steatosis is one of the side effects of chemotherapy. The PNPLA3 p.I148M, TM6SF2 p.E167K and MBOAT7 p.G17E variants represent genetic determinants for progressive liver diseases. Here, we investigate their association with chemotherapy-associated steatosis.Prospectively, we recruited 87 patients undergoing systemic chemotherapy for gastrointestinal cancers. Hepatic fat (controlled attenuation parameter, CAP) and liver stiffness (LSM) were measured non-invasively before the initiation of chemotherapy (T0) and after at least two (T1) and four cycles (T2). Genetic variants were genotyped using allelic discrimination assays.In the final dataset (n = 60) patients demonstrated the following…

MaleHeterozygotemedicine.medical_specialtymedicine.medical_treatmentAntineoplastic AgentsGastroenterology03 medical and health sciences0302 clinical medicineFat accumulationInternal medicinemedicineHumansGenetic Predisposition to DiseaseAdiponutrinProspective StudiesAlleleeducationAllelesTriglyceridesAgedLiver injuryChemotherapyeducation.field_of_studyPolymorphism GeneticHepatologybusiness.industryGastroenterologyMembrane ProteinsLipaseMiddle Agedmedicine.diseaseFatty LiverLiver030220 oncology & carcinogenesisElasticity Imaging TechniquesFemale030211 gastroenterology & hepatologySteatosisTransient elastographybusinessAcyltransferasesTM6SF2Digestive and Liver Disease
researchProduct

Human Y-chromosome variation in the Western Mediterranean area: Implications for the peopling of the region

2001

Y-chromosome variation was analyzed in a sample of 1127 males from the Western Mediterranean area by surveying 16 biallelic and 4 multiallelic sites. Some populations from Northeastern Europe and the Middle East were also studied for comparison. All Y-chromosome haplotypes were included in a parsimonious genealogic tree consisting of 17 haplogroups, several of which displayed distinct geographic specificities. One of the haplogroups, HG9.2, has some features that are compatible with a spread into Europe from the Near East during the Neolithic period. However, the current distribution of this haplogroup would suggest that the Neolithic gene pool had a major impact in the eastern and central …

MaleImmunologyMediterranean BasinHaplogroupGene flowMiddle Eastwest mediterranean basinAfrica NorthernY ChromosomeGenetic variationHumansImmunology and Allergyy-chromosome polymorphismsAllelesRecombination GeneticGeneticsPolymorphism GeneticMiddle EastMediterranean Regioneuropean populationsy-chromosome haplogroupsHaplotypeGenetic VariationGeneral MedicinehumanitiesEuropeGeographyHaplotypesEvolutionary biologyMultivariate AnalysisPeriod (geology)Gene poolgeographic locationseuropean populations; west mediterranean basin; y-chromosome haplogroups; y-chromosome polymorphismsMicrosatellite Repeats
researchProduct

HLA-A, -B and -DRB1 allele frequencies in Cyrenaica population (Libya) and genetic relationships with other populations.

2011

Abstract The frequencies of HLA-A, HLA-B and HLA-DRB1 alleles in 118 unrelated Libyans from Benghazi (Cyrenaica) were analysed using high resolution typing and compared with other populations. Their relatedness has been tested by correspondence analyses and principal component analysis. The most frequent HLA-A alleles were A ∗ 02:01:01:01 (15.7%), A ∗ 01:01:01:01 (11.4%) and A ∗ 03:01:01:01 (9.3%). For the HLA-B locus, the commonest allele was HLA-B ∗ 50:01:01 (14.4%) followed by B ∗ 51:01:01 (9.8%) and B ∗ 08:01:01 (6.4%). For the HLA-DRB1 locus, the commonest was HLA-DRB1 ∗ 07:01:01:01 (16.9%) followed by DRB1 ∗ 03:01:01:01 (13.6%) and DRB1 ∗ 13:02:01 (9.3%). The most frequent two-locus h…

MaleImmunologyPopulationDisease AssociationLocus (genetics)Human leukocyte antigenLibyaBiologySettore BIO/08Gene FrequencyEthnicityImmunology and AllergyHumansAlleleeducationChildAllele frequencyAllelesGeneticseducation.field_of_studyPrincipal Component AnalysisPolymorphism GeneticHLA-A AntigensHaplotypeInfantGeneral MedicineHLA-ASettore BIO/18 - GeneticaGenetics PopulationSettore MED/03 - Genetica MedicaHaplotypesHLA-B AntigensChild PreschoolFemaleHLA-DRB1 ChainsHuman immunology
researchProduct

The Demographic Costs of Sexually Antagonistic Selection in Partially Selfing Populations

2022

When selection differs between the sexes, genes expressed by both males and females can experience sexually antagonistic (SA) selection, where beneficial alleles for one sex are deleterious for the other. Classic population genetics theory has been fundamental to understanding how and when SA genetic variation can be maintained by balancing selection, but these models have rarely considered the demographic consequences of coexisting alleles with deleterious fitness effects in each sex. In this article, we develop a stage-structured Mendelian matrix model and jointly analyze the evolutionary and demographic consequences of SA selection in obligately outcrossing (i.e., dioecious/gonochorous) …

MaleInbreeding DepressionPolymorphism Geneticluonnonvalintasuvullinen lisääntyminenpolymorphismGenetics Populationevolutionary demographypartial selfingpopulaatiogenetiikkaintralocus sexual conflictkasvitsukusiitosHumanskaksineuvoisuusFemaleSelection Geneticsuvuton lisääntyminenAllelesDemographyinbreeding depressionMimulus guttatus
researchProduct