Search results for "Alleles"

showing 10 items of 478 documents

The ATC1 gene encodes a cell wall-linked acid trehalase required for growth on trehalose in Candida albicans.

2004

After screening a Candida albicans genome data base, the product of an open reading frame (IPF 19760/CA2574) with 41% identity to Saccharomyces cerevisiae vacuolar acid trehalase (Ath1p) was identified and named Atc1p. The deduced amino acid sequence shows that Atc1p contains an N-terminal hydrophobic signal peptide and 20 potential sites for N-glycosylation. C. albicans homozygous mutants that lack acid trehalase activity were constructed by gene disruption at the two ATC chromosomal alleles. Analysis of these null mutants shows that Atc1p is localized in the cell wall and is required for growth on trehalose as a carbon source. An Atc1p endowed with acid trehalase activity was obtained by …

Saccharomyces cerevisiae ProteinsTime FactorsTranscription GeneticMutantBlotting WesternMolecular Sequence DataTrehalase activityBiologyBiochemistrychemistry.chemical_compoundOpen Reading FramesCell WallCandida albicansAmino Acid SequenceRNA MessengerTrehalaseTrehalaseCandida albicansMolecular BiologyPeptide sequenceAlleleschemistry.chemical_classificationCell-Free SystemModels GeneticSequence Homology Amino AcidReverse Transcriptase Polymerase Chain ReactionStructural geneHomozygoteNuclear ProteinsTrehaloseCell BiologyDNAbiology.organism_classificationPhosphoproteinsTrehaloseCarbonAmino acidProtein Structure TertiaryGlucosechemistryBiochemistryProtein BiosynthesisMutationElectrophoresis Polyacrylamide GelCell DivisionPlasmidsThe Journal of biological chemistry
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Specific Defects in Different Transcription Complexes Compensate for the Requirement of the Negative Cofactor 2 Repressor in Saccharomyces cerevisiae

2007

Abstract Negative cofactor 2 (NC2) has been described as an essential and evolutionarily conserved transcriptional repressor, although in vitro and in vivo experiments suggest that it can function as both a positive and a negative effector of transcription. NC2 operates by interacting with the core promoter and components of the basal transcription machinery, like the TATA-binding protein (TBP). In this work, we have isolated mutants that suppress the growth defect caused by the depletion of NC2. We have identified mutations affecting components of three different complexes involved in the control of basal transcription: the mediator, TFIIH, and RNA pol II itself. Mutations in RNA pol II in…

Saccharomyces cerevisiae ProteinsTranscription GeneticRepressorRNA polymerase IISaccharomyces cerevisiaeInvestigationsGeneticsPromoter Regions GeneticTranscription factorAllelesGeneticsAdenosine TriphosphatasesTATA-Binding Protein Associated FactorsbiologyGeneral transcription factorDNA HelicasesPromoterPhosphoproteinsRepressor ProteinsProtein SubunitsTranscription Factor TFIIHMutationTranscription factor II Hbiology.proteinTrans-ActivatorsTranscription Factor TFIIBMutant ProteinsTranscription Factor TFIIDRNA Polymerase IITranscription factor II BTranscription Factor TFIIHTranscription Factors
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ABO genotyping by PCR-RFLP and cloning and sequencing

2005

A refined PCR-RFLP based method was established to genotype ABO blood groups. The main objective of this study was to make the techniques also suitable for working with degraded DNA. Specific primer design was carried out to choose fragments shorter than 200 bp as necessary in forensic and archaeological applications. Four fragments of exon 6 and 7 of the ABO gene were amplified and digested by in total 7 restriction endonucleases. Particular attention was paid to the base changes at nucleotide positions 261(delG), 297, 526, 703, 721, 771, 796 and 1060(delC) in order to distinguish the six common alleles A101, A201, B, O01, O02 and O03. Furthermore, this method also enables determination of…

Sequence analysisBiologyPolymerase Chain ReactionABO Blood-Group Systemlaw.inventionlawABO blood group systemGenotypeHumansCloning MolecularGenotypingAllelesHistory AncientEcology Evolution Behavior and SystematicsPolymerase chain reactionGeneticsReproducibility of ResultsSequence Analysis DNAGeneral MedicineForensic MedicineRestriction enzymePhenotypeAncient DNAArchaeologyBlood StainsPostmortem ChangesAnthropologyDNA Transposable ElementsAnimal Science and ZoologyChromosome DeletionRestriction fragment length polymorphismToothPolymorphism Restriction Fragment LengthAnthropologischer Anzeiger
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Identification of self(in)compatibility genotypes and microsatellite marker based fingerprinting of traditional italian sweet cherry accessions

2012

Italy is one of the main European sweet cherry producers and it has a rich germplasm, including many minor local varieties that have not been well studied or used in breeding programmes. Sweet cherry is self-incompatible, with few exceptions; its incompatibility is controlled by a multi-allelic S locus, which is gametophytically expressed. The knowledge of S-alleles and cross-incompatibility groups of sweet cherry genotypes and cultivars is important for growers and breeders. In this work we analysed 48 traditional sweet cherry varieties and cultivars, mainly from Calabria and Emilia Romagna regions, together with eight Sicilian cultivars and the standard set of reference genotypes proposed…

Settore AGR/03 - Arboricoltura Generale E Coltivazioni ArboreePrunus avium S-alleles breeding programmes genetic resources
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CR1 allelic distribution in healthy italians and SLE patients

2010

Complement receptor type 1 (CR1; CD35), the receptor for C3b/C4b, has a molecular weight polymorphism involving four co-dominantly expressed alleles CR1*1, CR1*2, CR1*3 and CR1*4. The most common allotype in the Caucasian population is CR1*1. Previous studies on the frequency of the CR1 allele in Systemic Lupus Erythematosus (SLE) patients reported conflicting results. Recently an increased frequency of the CR1*2 allele was reported in Caucasian SLE patients. The aim of our study is to assess the frequency of CR1 structural alleles in a sample of the Italian population and in a cohort of Italian SLE patients equally distributed nationwide. We found that the most common phenotype was CR1*1.1…

Settore BIO/18 - GeneticaComplement CR1 structural alleles SLE
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Effect of family structure and TPH2 G-703T on the stability of dysregulation profile throughout adolescence

2015

Abstract Background Two different polymorphisms (TPH2 G-703T and 5-HTTLPR) involved in the serotonergic pathway have been reported to play a role, both alone and in interaction with the environment, in early and adult emotion regulation. As most of these studies are cross-sectional, we know little about the impact of these polymorphisms over time, particularly during adolescence. Methods Because we were interested in the effects of these polymorphisms and environment (i.e., family structure) at different time-points on the emotional dysregulation profile, we performed a path analysis model in a general adolescent population sample of a five-year follow-up study. Results We found a high stab…

Settore M-PSI/01 - Psicologia GeneraleMaleAdolescentGenotypeEmotional dysregulationTryptophan Hydroxylase5-HTTLPRSerotonergicDevelopmental psychologyDysregulation profileYoung Adult03 medical and health sciences0302 clinical medicinePolymorphism (computer science)TPH2HumansAffective SymptomsAlleleGene–environment interactionYoung adultAllelesTPH2; 5-HTTLPR; Emotional dysregulation; Adolescence; Family structure; Dysregulation profileSerotonin Plasma Membrane Transport ProteinsPolymorphism GeneticTPH2Emotional dysregulationAdolescence030227 psychiatryPsychiatry and Mental healthClinical Psychology5-HTTLPRFemaleGene-Environment InteractionFamily RelationsPsychologyFamily structure030217 neurology & neurosurgeryFollow-Up StudiesClinical psychologyJournal of Affective Disorders
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Missense and nonsense mutations in melanocortin 1 receptor (MC1R) gene of different goat breeds: association with red and black coat colour phenotype…

2009

Abstract Background Agouti and Extension loci control the relative amount of eumelanin and pheomelanin production in melanocytes that, in turn, affects pigmentation of skin and hair. The Extension locus encodes the melanocortin 1 receptor (MC1R) whose permanent activation, caused by functional mutations, results in black coat colour, whereas other inactivating mutations cause red coat colour in different mammals. Results The whole coding region of the MC1R gene was sequenced in goats of six different breeds showing different coat colours (Girgentana, white cream with usually small red spots in the face; Maltese, white with black cheeks and ears; Derivata di Siria, solid red; Murciano-Granad…

Silent mutationCoatlcsh:QH426-470GenotypeMolecular Sequence DataNonsense mutationPopulationMutation MissenseMELANISMBiologyPolymorphism Single NucleotideAGOUTI PROTEINSettore AGR/17 - Zootecnica Generale E Miglioramento GeneticoMSH RECEPTORBREEDSMC1RGeneticsAnimalsMissense mutationGenetics(clinical)Amino Acid Sequencecoat colour; MC1R; goatAlleleHair ColoreducationAllele frequencyPOPULATIONPOLYMORPHISMSAllelesGenetics (clinical)Geneticseducation.field_of_studySTIMULATING-HORMONE-RECEPTORGoatsgoatCATTLE BREEDSSequence Analysis DNAMolecular biologyCOAT COLORlcsh:GeneticsPhenotypeCodon NonsensePIGMENTATIONWHITEReceptor Melanocortin Type 1EXTENSIONcoat colourResearch ArticleMelanocortin 1 receptorBMC Genetics
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Systematic screening for mutations in the human serotonin-2A (5-HT2A) receptor gene: Identification of two naturally occurring receptor variants and …

1996

A statistically significant association between a silent mutation (102T/C) in the serotonin-2A (5-HT2A) receptor gene and schizophrenia has recently been reported in a sample of Japanese patients and healthy controls. This finding suggests that genetic predisposition to schizophrenia may be affected by a functional 5-HT2A receptor variant that is in linkage disequilibrium with 102T/C. In the present study, we have sought to identify genetic variation in the 5-HT2A receptor gene by screening genomic DNA samples from 91 unrelated subjects comprising 45 patients with schizophrenia and 46 healthy controls by using single-strand conformation analysis. We have identified four nucleotide sequence …

Silent mutationLinkage disequilibriumMolecular Sequence DataRestriction MappingBiologymedicine.disease_causePolymerase Chain ReactionReference ValuesGenetic variationConfidence IntervalsGeneticsGenetic predispositionmedicineHumansPoint MutationReceptor Serotonin 5-HT2AAmino Acid SequenceAlleleAllele frequencyAllelesGenetics (clinical)DNA PrimersGenetic associationGeneticsMutationPolymorphism GeneticBase SequenceChromosomes Human Pair 13Chromosome MappingGenetic VariationExonsReceptors SerotoninSchizophreniaHuman Genetics
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Identification of HLA-A*0201-Restricted T Cell Epitopes Derived from the Novel Overexpressed Tumor Antigen Calcium-Activated Chloride Channel 2

2002

Abstract Vaccination against tumor Ags may become a promising treatment modality especially in cancer types where other therapeutic approaches fail. However, diversity of tumors requires that a multitude of Ags become available. Differential expression in normal vs cancerous tissues, both at the mRNA and the protein level, may identify Ag candidates. We have previously compared transcripts from squamous cell lung cancer and normal lung tissue using differential display analysis, and found a transcript that was overexpressed in malignant cells and was identical with the calcium-activated chloride channel 2 (CLCA2) gene. We have now selected HLA-A2-restricted peptides from CLCA2, and have gen…

T cellImmunologyAntigen presentationEpitopes T-LymphocyteStreptamerCD8-Positive T-LymphocytesBiologyEpitopeCell LineInterleukin 21AntigenAntigens NeoplasmChloride ChannelsHLA-A2 AntigenmedicineHumansImmunology and AllergyCytotoxic T cellAntigen-presenting cellAllelesAntigen PresentationHLA-A AntigensMolecular biologyCoculture TechniquesPeptide FragmentsPancreatic Neoplasmsmedicine.anatomical_structureCalciumOligopeptidesProtein BindingThe Journal of Immunology
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A G468-T AMPD1 mutant allele contributes to the high incidence of myoadenylate deaminase deficiency in the Caucasian population.

2002

Myoadenylate deaminase deficiency is the most common metabolic disorder of skeletal muscle in the Caucasian population, affecting approximately 2% of all individuals. Although most deficient subjects are asymptomatic, some suffer from exercise-induced myalgia suggesting a causal relationship between a lack of enzyme activity and muscle function. In addition, carriers of this derangement in purine nucleotide catabolism may have an adaptive advantage related to clinical outcome in heart disease. The molecular basis of myoadenylate deaminase deficiency in Caucasians has been attributed to a single mutant allele characterized by double C to T transitions at nucleotides +34 and +143 in mRNA enco…

ThreonineDNA ComplementaryGenotypeBlotting WesternGlycineMetabolic myopathyBiologyCompound heterozygosityPolymerase Chain ReactionWhite PeopleAMP DeaminaseMetabolic DiseasesMuscular DiseasesGenotypemedicineHumansAlleleTransversionMuscle SkeletalGenetics (clinical)AllelesElectromyographyPoint mutationMetabolic disorderAMP deaminasemedicine.diseaseMolecular biologyPhenotypeNeurologyPediatrics Perinatology and Child HealthMutationNeurology (clinical)DNA ProbesNeuromuscular disorders : NMD
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