Search results for "Alpha"

showing 10 items of 3228 documents

Influence of extracellular matrix proteins on the development of cultured human dendritic cells.

1998

The development of dendritic cells (DC) is still only partly understood. Recently established culture systems using CD34+ cells or monocytes as precursor cells for the generation of DC indicate the necessity of pro-inflammatory cytokines for their development. In vivo the contact to other cells or to the proteins of the extracellular matrix might also be essential for their development. In our experiments we used granulocyte-macrophage colony-stimulating factor- and IL-4-treated human monocytes as precursor cells to investigate the interaction of DC at different maturation stages with the matrix proteins fibronectin, collagen type I and collagen type IV. We demonstrate a strong beta1-integr…

medicine.medical_treatmentCellular differentiationImmunologyCD34Cell CommunicationMatrix (biology)BiologyMonocytesExtracellular matrixPrecursor cellmedicineCell AdhesionImmunology and AllergyHumansCells CulturedExtracellular Matrix ProteinsTumor Necrosis Factor-alphaIntegrin beta1Cell DifferentiationDendritic cellDendritic CellsCell biologyFibronectinsUp-RegulationFibronectinCytokineAntigens Surfacebiology.proteinCollagenLymphocyte Culture Test MixedEuropean journal of immunology
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Interleukin 12 enhances deficient HCV-antigen-induced Th1-type immune response of peripheral blood mononuclear cells

1998

The aim of this study was to examine the possible immunomodulating effects of rhIL-12 on the immune response induced by different hepatitis C virus (HCV) antigens. Freshly isolated peripheral blood mononuclear cells (PBMC) of 33 patients with chronic HCV infection were stimulated with optimal concentrations of antigens from the NS3, NS4, NS5, and core region of HCV in the absence or presence of interleukin12 (IL-12). Stimulation by α-CD3 + α-CD28, lipopolysaccharide (LPS), and pokeweed mitogen (PWM) were used as controls. Proliferation and cytokine production were determined by 3H-thymidine uptake and enzyme-linked immunosorbent assay (ELISA) after 72 hr. After stimulation with antigen or a…

medicine.medical_treatmentHepatitis C virusPokeweed mitogenBiologymedicine.disease_causeVirologyPeripheral blood mononuclear cellInfectious DiseasesImmune systemCytokineAntigenVirologyImmunologyInterleukin 12medicineTumor necrosis factor alphaJournal of Medical Virology
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Pro-inflammatory cytokines and prostaglandins induce maturation of potent immunostimulatory dendritic cells under fetal calf serum-free conditions.

1998

Culture conditions for human dendritic cells (DC) have been developed by several laboratories. Most of these culture methods, however, have used conditions involving fetal calf serum (FCS) to generate DC in the presence of granulocyte-macrophage colony-stimulating factor and interleukin (IL)-4. Recently, alternative culture conditions have been described using an additional stimulation with monocyte-conditioned medium (MCM) and FCS-free media to generate DC. As MCM is a rather undefined cocktail, the yield and quality of DC generated by these cultures varies substantially. We report that a defined cocktail of tumor necrosis factor (TNF)-alpha, IL-1beta and IL-6 equals MCM in its potency to …

medicine.medical_treatmentImmunologyBiologyCulture Media Serum-FreeProinflammatory cytokineFlow cytometryImmunophenotypingInterferonCell MovementmedicineImmunology and AllergyHumansCells Culturedmedicine.diagnostic_testInterleukinCell DifferentiationImmunotherapyDendritic CellsFlow CytometryMolecular biologyCytokineImmunologyProstaglandinsCytokinesTumor necrosis factor alphaCD8medicine.drugEuropean journal of immunology
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Effects of Th1 and Th2 cytokines on cytokine production and ICAM-1 expression on synovial fibroblasts

1995

OBJECTIVES--To investigate the influence of the Th1 and Th2 lymphokines interleukins (IL)-4 and IL-13, interferon gamma (IFN gamma), and several monokines on the adhesion of mononuclear cells to synovial fibroblasts and intercellular adhesion molecule-1 (ICAM-1) expression and cytokine production of synovial fibroblasts in patients with osteoarthritis. METHODS--Synovial fibroblasts were isolated from patients with osteoarthritis and stimulated with IL-1 beta, IL-4, IL-6, IL-10, IL-12, IL-13, tumour necrosis factor alpha (TNF alpha), and IFN gamma. Subsequently, we determined the production of IL-1 alpha, IL-1 beta, IL-6, IL-10, IL-12, IFN alpha and TNF alpha, and the expression of ICAM-1 ly…

medicine.medical_treatmentImmunologyIntercellular Adhesion Molecule-1BiologyGeneral Biochemistry Genetics and Molecular BiologyTh2 CellsRheumatologyOsteoarthritisCell AdhesionmedicineHumansImmunology and AllergyInterferon gammaCell adhesionCells CulturedInterleukin-13Interleukin-6Cell adhesion moleculeSynovial MembraneLymphokineReceptors InterleukinFibroblastsTh1 CellsIntercellular Adhesion Molecule-1Molecular biologyRecombinant ProteinsReceptors Interleukin-4Cytokinemedicine.anatomical_structureImmunologyCytokinesTumor necrosis factor alphaInterleukin-4Synovial membraneResearch Articlemedicine.drug
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Major histocompatibility complex regulation of cytokine production.

1996

This review describes the phenomenon of the major histocompatibility complex (MHC) control of cytokine production both in experimental animals and in humans. H-2 (mouse MHC) regulates which type of cytokine is selectively produced in response to the hapten trinitrophenyl (TNP). T cells from TNP-immune H-2k mice produce interferon-gamma (IFN-gamma), interleukin-2 (IL-2), IL-3, IL-5, tumor necrosis factor-alpha (TNF-alpha), IL-10, and very low levels of IL-4 on reexposure to the specific antigen in vitro. By contrast, T cells from H-2d mice produce IL-3, TNF-alpha, IL-10, and IL-4 but very low levels of IL-2, IL-5 and IFN-gamma. As MHC-congenic matched strains (BALB/k and BALB/c) are used, th…

medicine.medical_treatmentImmunologychemical and pharmacologic phenomenaMajor histocompatibility complexPeripheral blood mononuclear cellMajor Histocompatibility ComplexInterferon-gammaMiceImmune systemAntigenVirologyImmunopathologymedicineAnimalsHumansbiologyTumor Necrosis Factor-alphaInterleukinsH-2 AntigensCell BiologyCytokineImmunologyAntibody Formationbiology.proteinCytokinesTumor necrosis factor alphaHapten
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INFLAMMATORY CYTOKINES IN ACUTE ISCHEMIC STROKE.

2008

Three major cytokines, namely, tumor necrosis factor (TNF-alpha), interleukin (IL)-1, and IL-6 are produced by cultured brain cells after various stimuli such as ischemia. Neurones, astrocytes, microglia and oligodendrocytes can produce inflammatory mediators, and cytokine receptors are expressed constitutionally throughout the Central Nervous System (CNS), albeit at low levels. Cytokines are involved in virtually every facet of stroke and they have numerous pro-inflammatory and pro-coagulant effects on endothelium. TNF-alpha expression after stroke stimulates expression of tissue factor and adhesion molecules for leukocytes, release of interleukin-1 (IL-1), nitric oxide, factor VIII/von Wi…

medicine.medical_treatmentInflammationProinflammatory cytokineBrain IschemiaBrain ischemiaTissue factorDrug DiscoverymedicineHumansStrokePharmacologyInflammationMicrogliabusiness.industrymedicine.diseasePrognosisStrokeCytokinemedicine.anatomical_structureImmunologyAcute DiseaseCytokinesTumor necrosis factor alphamedicine.symptomStroke cytokinesbusinessBiomarkers
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Interaction Between Cytokines and Oxidative Stress in Acute Pancreatitis

2006

Acute pancreatitis is an inflammation initially localized in the pancreatic gland which may lead to local and systemic complications. The development of severe acute pancreatitis is mediated by pathophysiological mechanisms involved in the systemic inflammatory response, cytokines and oxidative stress being their components of major importance. Nevertheless, it is still unknown why an episode of acute pancreatitis remains mild or progresses to a severe form. Activated leukocytes are the main source of cytokines. Interleukin 1beta and tumor necrosis factor alpha (TNF-alpha) initiate and propagate almost all the consequences of the systemic inflammatory response syndrome, leading to amplifica…

medicine.medical_treatmentInflammationmedicine.disease_causeBiochemistryProinflammatory cytokineDrug DiscoverymedicineAnimalsHumansPharmacologyChemistryOrganic ChemistryModels Immunologicalmedicine.diseaseSystemic inflammatory response syndromeOxidative StressCytokinePancreatitisAcute DiseaseImmunologyCytokinesMolecular MedicineAcute pancreatitisPancreatitisTumor necrosis factor alphamedicine.symptomOxidative stressCurrent Medicinal Chemistry
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Regulation of cytochrome P450 IID by acute phase mediators in C3H/HeJ mice.

1992

Abstract Cytochrome P450 IID6 is a drug metabolizing enzyme and the major target antigen in LKM-1 antibody positive chronic active hepatitis. The histological hallmark of chronic active hepatitis is a lymphocytic infiltrate in the liver. It is unknown whether and how cytokines produced and secreted by these tissue infiltrating mononuclear cells regulate the cellular expression of cytochrome P450 IID6. To study the effect of interleukin 1, tumor necrosis factor and interleukin 6 on the hepatocellular RNA expression of cytochrome P450 IID, we injected each of the cytokines in C3H HeJ mice. We found a time-dependent suppression of the cytochrome in the liver. Six hours after the intraperitonea…

medicine.medical_treatmentIntraperitoneal injectionBiophysicsBiochemistryMixed Function OxygenasesMiceCytochrome P-450 Enzyme SystemmedicineEscherichia coliAnimalsHumansInterleukin 6Molecular BiologyHepatitisMice Inbred C3HbiologyInterleukin-6Tumor Necrosis Factor-alphaCytochrome P450InterleukinCell Biologymedicine.diseaseBlotting NorthernRecombinant ProteinsEndotoxinsCytokineCytochrome P-450 CYP2D6LiverImmunologybiology.proteinRNATumor necrosis factor alphaFemaleAntibodyInterleukin-1Biochemical and biophysical research communications
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Ex vivoevidence for PGE2and LTB4involvement in cutaneous leishmaniasis: relation with infection status and cytokine production

1996

SUMMARYEx vivoculture of spleen cells from BALB/c mice infected with 2 × 106Leishmania major(L.major) promastigotes were cultured with ConcanavalinA (ConA) or leishmanial antigen (L. Ag) and tested for prostaglandin E2(PGE2) and for leukotriene B4(LTB4), in order to study their involvement in the evolution of cutaneous leishmaniasis and the connexion with lymphokine-mediated responses. The data were compared with those obtained in BALB/c mice protected againstL. majorby sublethal irradiation (550 rad; cured mice). In the unprotected BALB/c mice the levels of PGE2that were responsible for the depression of interferon-γ (IFN-γ) and tumour necrosis factor-α (TNFα) Th1-associated cytokines and …

medicine.medical_treatmentLeishmaniasis CutaneousInflammationLeukotriene B4DinoprostoneInterferon-gammaMiceTh2 CellsImmune systemCutaneous leishmaniasismedicineAnimalsLeishmania majorInterferon gammaCells CulturedLeishmania majorMice Inbred BALB CbiologyTumor Necrosis Factor-alphaTh1 Cellsbiology.organism_classificationmedicine.diseaseInfectious DiseasesCytokineImmunologyFemaleAnimal Science and ZoologyParasitologyTumor necrosis factor alphaInterleukin-4medicine.symptomSpleenWhole-Body IrradiationEx vivomedicine.drugParasitology
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Cytokine expression profile in idiopathic inflammatory myopathies.

1996

Cytokines have been shown to be potent inducers of major histocompatibility complexes (MHC) class I and II as well as of cell adhesion molecules in muscle tissue cultures, indicating that cytokines may play a role in mediating muscle fiber damage in inflammatory myopathies. We found in 21 cases of autoimmune myositis various amounts of inflammatory cells expressing interleukin (IL)-1 alpha and -beta, IL-2, IL-4, tumor necrosis factor (TNF) -alpha and -beta, and interferon (IFN)-gamma and its receptor. Muscle fibers displayed enhanced expression of IL-1 alpha and -beta, IL-2, and TNF-alpha. Upregulation of cytokines was strongest at sites of cellular infiltration typical for the respective m…

medicine.medical_treatmentMuscle Fibers SkeletalInflammationCytokine Expression ProfileBiologyMuscular DystrophiesPathology and Forensic MedicineCellular and Molecular NeuroscienceInterferonmedicineCytotoxic T cellHumansInterleukin 4InflammationInterleukinGeneral MedicineImmunohistochemistryPolymyositisCytokineNeurologyImmunologyCytokinesTumor necrosis factor alphaNeurology (clinical)medicine.symptommedicine.drugJournal of neuropathology and experimental neurology
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