Search results for "Anacetrapib"

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Beyond Statins: New Lipid Lowering Strategies to Reduce Cardiovascular Risk

2014

Statins are the first-line therapy in LDL-Cholesterol (LDL-C) reduction and its clinical use has contributed to significant prevention and treatment of atherosclerotic vascular disease. Yet, a significant proportion of patients remain at high risk. Recently, a number of new therapies have been developed to further lower LDL-C. These agents may provide clinical benefit on top of statin therapy in patients with high residual risk, severe hypercholesterolemia or as an alternative for patients who are intolerant to statins. We review four novel approaches based on the inhibition of proprotein convertase subtilisin/kexin type 9 (PCSK9), apolipoprotein-B100 (apoB), Cholesteryl ester transport pro…

medicine.medical_specialtySettore MED/09 - Medicina InternaApolipoprotein BHypercholesterolemiaMipomersenFamilial hypercholesterolemiaBioinformaticschemistry.chemical_compoundRisk FactorsAnacetrapibInternal medicinemedicineHumansStatins Novel LDL-C lowering drugs Mipomersen Lomitapide Anacetrapib Evacetrapib PCSK9 inhibitorsbiologybusiness.industryAnticholesteremic AgentsPCSK9nutritional and metabolic diseasesLipid Metabolismmedicine.diseaseLomitapideResidual riskEndocrinologychemistryCardiovascular Diseasesbiology.proteinlipids (amino acids peptides and proteins)Hydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicinebusinessEvacetrapibCurrent Atherosclerosis Reports
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Vascular effects and safety of dalcetrapib in patients with or at risk of coronary heart disease: the dal-VESSEL randomized clinical trial

2012

Aims High-density lipoprotein cholesterol (HDL-C) is inversely associated with cardiovascular (CV) events and thus an attractive therapeutic target. However, in spite of marked elevations in HDL-C, the first cholesterol transport protein (CETP) inhibitor torcetrapib raised blood pressure (BP), impaired endothelial function, and increased CV mortality and morbidity. Dalcetrapib is a novel molecule acting on CETP with a different chemical structure to torcetrapib. As HDL stimulates nitric oxide (NO), suppresses inflammation, and exerts protective CV effects, we investigated the effects of dalcetrapib on endothelial function, blood pressure, inflammatory markers, and lipids in patients with, o…

MaleBrachial ArteryBlood PressureCoronary Diseasechemistry.chemical_compoundAnacetrapibTorcetrapibMedicineLipoproteinbiologyAnticholesteremic AgentsEstersMiddle AgedVasodilationTreatment OutcomeCardiologyFemalelipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicineBlood Flow Velocitymedicine.medical_specialtyAmbulatory blood pressureDalcetrapibHypercholesterolemia610 Medicine & healthPlacebo142-005 142-0052705 Cardiology and Cardiovascular MedicineCholesterol (HDL-C)Double-Blind MethodInternal medicineCholesterylester transfer proteinDalcetrapibHumansSulfhydryl CompoundsTriglyceridesAgedbusiness.industryCholesterol HDLTorcetrapibCholesterol LDLAmidesFasttrack ClinicalCholesterol Ester Transfer ProteinsEndocrinologyBlood pressurechemistrybiology.proteinHigh-density570 Life sciences; biologyEndothelium VascularbusinessBiomarkersEvacetrapibEuropean heart journal
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