Search results for "Anandamide"

showing 10 items of 35 documents

Oral Palmitoylethanolamide Treatment Is Associated with Reduced Cutaneous Adverse Effects of Interferon-β1a and Circulating Proinflammatory Cytokines…

2016

Palmitoylethanolamide (PEA) is an endogenous lipid mediator known to reduce pain and inflammation. However, only limited clinical studies have evaluated the effects of PEA in neuroinflammatory and neurodegenerative diseases. Multiple sclerosis (MS) is a chronic autoimmune and inflammatory disease of the central nervous system. Although subcutaneous administration of interferon (IFN)-β1a is approved as first-line therapy for the treatment of relapsing–remitting MS (RR-MS), its commonly reported adverse events (AEs) such as pain, myalgia, and erythema at the injection site, deeply affect the quality of life (QoL) of patients with MS. In this randomized, double-blind, placebo-controlled study,…

Male0301 basic medicinemyalgiaErythemaAnti-Inflammatory AgentsPalmitic AcidAdministration OralPharmacologyGastroenterologychemistry.chemical_compound0302 clinical medicineNeuroinflammationFAAHEthanolaminePharmacology (medical)SkinInterleukin-17food and beveragesAnti-Inflammatory AgentTolerabilityEthanolaminesDisease ProgressionCytokinesOriginal ArticleFemalemedicine.symptomInterferon beta-1aHumanAdultmedicine.medical_specialtyPainPalmitic AcidsProinflammatory cytokineInterferon-gamma03 medical and health sciencesMultiple Sclerosis Relapsing-RemittingDouble-Blind MethodInternal medicinemedicineHumansAdverse effectCytokinePharmacologyPalmitoylethanolamideExpanded Disability Status ScaleTumor Necrosis Factor-alphabusiness.industryMultiple sclerosisN-acylethanolamineOleoylethanolamideAnandamideNAAAmedicine.diseaseAmides030104 developmental biologychemistryNeurology (clinical)business030217 neurology & neurosurgeryNeurotherapeutics
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Reduced anxiety-like behaviour induced by genetic and pharmacological inhibition of the endocannabinoid-degrading enzyme fatty acid amide hydrolase (…

2007

Anandamide and 2-arachidonoyl glycerol, referred to as endocannabinoids (eCBs), are the endogenous agonists for the cannabinoid receptor type 1 (CB1). Several pieces of evidence support a role for eCBs in the attenuation of anxiety-related behaviours, although the precise mechanism has remained uncertain. The fatty acid amid hydrolase (FAAH), an enzyme responsible for the degradation of eCBs, has emerged as a promising target for anxiety-related disorders, since FAAH inhibitors are able to increase the levels of anandamide and thereby induce anxiolytic-like effects in rodents. The present study adopted both genetic and pharmacological approaches and tested the hypothesis that FAAH-deficient…

MaleCannabinoid receptorPolyunsaturated Alkamidesmedicine.medical_treatmentArachidonic AcidsAnxietyPharmacologyAmidohydrolasesGlyceridesMiceCellular and Molecular Neurosciencechemistry.chemical_compoundPiperidinesReceptor Cannabinoid CB1RimonabantFatty acid amide hydrolaseCannabinoid receptor type 1medicineAnimalsMaze LearningMice KnockoutPharmacologyAnalysis of VarianceBehavior AnimalAnandamideURB597Endocannabinoid systemMice Inbred C57BLDisease Models Animalnervous systemchemistryBenzamidesPyrazoleslipids (amino acids peptides and proteins)CarbamatesCannabinoidRimonabantpsychological phenomena and processesEndocannabinoidsmedicine.drugNeuropharmacology
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Inhibition by Anandamide and Synthetic Cannabimimetics of the Release of [3H]d-Aspartate and [3H]GABA from Synaptosomes Isolated from the Rat Hippoca…

2004

Cannabinoids (CB) can act as retrograde synaptic mediators of depolarization-induced suppression of inhibition or excitation in hippocampus. This mechanism may underlie the impairment of some cognitive processes produced by these compounds, including short-term memory formation in the hippocampus. In this study, we investigated several compounds known to interact with CB receptors, evaluating their effects on K +-evoked release of [ 3H]d-aspartate ([ 3H]d-ASP) and [ 3H]GABA from superfused synaptosomes isolated from the rat hippocampus. [ 3H]d-ASP and [ 3H]GABA release were inhibited to different degrees by the synthetic cannabinoids WIN 55,212-2; CP 55,940, and arachidonyl-2′- chloroethyla…

MaleCannabinoid receptorSettore BIO/14 - FARMACOLOGIAPolyunsaturated Alkamidesmedicine.medical_treatmentHippocampusArachidonic AcidsPharmacologyHippocampal formationDepolarization-induced suppression of inhibitionHippocampusBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundglutamate releasemedicineAnimalsRats WistarCannabinoidgamma-Aminobutyric AcidCannabinoid Receptor AgonistsAspartic AcidCannabinoidsChemistryGeneral MedicineAnandamideCyclohexanolsgaba releaseEndocannabinoid systemRatsKineticsnervous systemBiochemistryAnimals Arachidonic Acids Aspartic Acid Calcium Cannabinoids Capsaicin Cyclohexanols gamma-Aminobutyric Acid Hippocampus Kinetics Polyunsaturated Alkamides Potassium Rats Receptors Cannabinoid SynaptosomesPotassiumCalciumlipids (amino acids peptides and proteins)CannabinoidCapsaicinCapsazepineEndocannabinoidsSynaptosomesNeurochemical Research
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Serotonin, the Prefrontal Cortex, and the Antidepressant-Like Effect of Cannabinoids

2007

Preclinical and clinical studies show that cannabis modulates mood and possesses antidepressant-like properties, mediated by the agonistic activity of cannabinoids on central CB1 receptors (CB1Rs). The action of CB1R agonists on the serotonin (5-HT) system, the major transmitter system involved in mood control and implicated in the mechanism of action of antidepressants, remains however poorly understood. In this study, we demonstrated that, at low doses, the CB1R agonist WIN55,212-2 [R(+)-[2,3-dihydro-5-methyl-3-[(morpholinyl)]pyrrolo[1,2,3-de]-1,4-benzoxazinyl]-(1-naphthalenyl) methanone mesylate] exerts potent antidepressant-like properties in the rat forced-swim test (FST). This effect …

MaleSerotoninJournal ClubMorpholinesmedicine.medical_treatmentPrefrontal CortexNaphthalenesPharmacologyEuphoriantAntidepressant likeRats Sprague-Dawleychemistry.chemical_compoundReceptor Cannabinoid CB1mental disordersAnimalsEthanolamideMedicineReceptorPrefrontal cortexNeuronsCannabinoidsDepressionbusiness.industryorganic chemicalsGeneral NeuroscienceAnandamideAntidepressive AgentsBenzoxazinesRatschemistryCannabinoidSerotoninbusinessNeuroscienceThe Journal of Neuroscience
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Presynaptic effects of anandamide and WIN55,212-2 on glutamatergic nerve endings isolated from rat hippocampus

2006

We examined the effects of the endocannabinoide-anandamide (AEA), the synthetic cannabinoid, WIN55,212-2, and the active phorbol ester, 4-beta-phorbol 12-myristate 13-acetate (4-beta-PMA), on the release of [(3)H]d-Aspartate ([(3)H]d-ASP) from rat hippocampal synaptosomes. Release was evoked with three different stimuli: (1) KCl-induced membrane depolarization, which activates voltage-dependent Ca(2+) channels and causes limited neurotransmitter exocytosis, presumably from ready-releasable vesicles docked in the active zone; (2) exposure to the Ca(2+) ionophore-A23187, which causes more extensive transmitter release, presumably from intracellular reserve vesicles; and (3) K(+) channel block…

MaleSettore BIO/14 - FARMACOLOGIAPolyunsaturated AlkamideshippocampusMorpholinesmedicine.medical_treatmentPresynaptic TerminalsArachidonic AcidsNaphthalenesExocytosisCellular and Molecular Neurosciencechemistry.chemical_compoundGlutamatesglutamate releasemedicineAnimalsanandamideActive zoneRats WistarNeurotransmitterCannabinoidCalcimycinProtein kinase CSynaptosomeArachidonic AcidChemistrysynaptosomesDepolarizationCell BiologyAnandamideHippocampal synaptosomeCalcium Channel BlockersBenzoxazinesRatsBiochemistryBiophysicsTetradecanoylphorbol AcetateCannabinoidCapsaicinEndocannabinoidsNeurochemistry International
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Differential diurnal variations of anandamide and 2-arachidonoyl-glycerol levels in rat brain.

2004

The endogenous ligands of cannabinoid receptors, also known as endocannabinoids, have been implicated in many physiological and pathological processes of the central nervous system. Here we show that the levels of the two major endocannabinoids, anandamide and 2-arachidonoyl-glycerol (2-AG), in four areas of the rat brain, change dramatically between the light and dark phases of the day. While anandamide levels in the nucleus accumbens, pre-frontal cortex, striatum and hippocampus were significantly higher in the dark phase, the opposite was observed with 2-AG, whose levels were significantly higher during the light phase in all four regions. We found that the activity of the fatty acid ami…

Malemedicine.medical_specialtyDiacylglycerol lipaseCannabinoid receptorPolyunsaturated Alkamidesmedicine.medical_treatmentPhotoperiod2-ArachidonoylglycerolArachidonic AcidsAmidohydrolasesGlyceridesRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundFatty acid amide hydrolaseInternal medicineCannabinoid Receptor ModulatorsmedicineanandamideAnimals2-arachidonoylglycerol; anandamide; cannabinoid; circadian; faahMolecular BiologyPharmacologybiologyBrainCell BiologyAnandamidefaahcannabinoidEndocannabinoid system2-arachidonoylglycerolCircadian RhythmRatsMonoacylglycerol lipaseEndocrinologycircadianchemistryBiochemistrybiology.proteinMolecular MedicineCannabinoidEndocannabinoidsCellular and molecular life sciences : CMLS
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Endocannabinoid Levels in Newborns in Relation to the Mode of Delivery.

2015

Objective  This study aims to examine the endocannabinoid levels in newborns in relation to the mode of delivery. Study Design  In this study, the concentrations of the endocannabinoids anandamide (AEA), 2-arachidonylglycerol (2-AG), palmitoylethanolamine (PEA), and the metabolite arachidonic acid (AA) in umbilical cord vein blood of 49 newborns was determined by quantitative mass spectrometry using liquid chromatography multiple reaction monitoring. The newborns were divided by their mode of delivery. Only healthy newborns born after 34 0/7 gestational weeks without birth complications were included. Results  The concentration of AEA, PEA, and AA was significantly higher in vaginal deliver…

Malemedicine.medical_specialtyPolyunsaturated AlkamidesMetaboliteArachidonic AcidsMass SpectrometryGlycerideschemistry.chemical_compoundInternal medicineMedicineEndocrine systemHumansFetusbusiness.industryVaginal deliveryInfant NewbornObstetrics and GynecologyAnandamideDelivery ObstetricEndocannabinoid systemMode of deliveryEndocrinologychemistryAnesthesiaPediatrics Perinatology and Child Healthlipids (amino acids peptides and proteins)Arachidonic acidFemalebusinessChromatography LiquidEndocannabinoidsAmerican journal of perinatology
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Neurobiology of cannabinoid receptor signaling
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2020

The endocannabinoid system (ECS) is a highly versatile signaling system within the nervous system. Despite its widespread localization, its functions within the context of distinct neural processes are very well discernable and specific. This is remarkable, and the question remains as to how such specificity is achieved. One key player in the ECS is the cannabinoid type 1 receptor (CBEl sistema endocannabinoide (SEC) apareció como un sistema de señalización muy versátil en el sistema nervioso. A pesar de su existencia amplia y ubicua, sus funciones están integradas en el contexto de distintos procesos neuronales y, en última instancia, son bastante bien discernibles y específicas. Esto es n…

Nervous systemCannabinoid receptormedicine.medical_treatment2-ArachidonoylglycerolContext (language use)BiologyReceptor Cannabinoid CB203 medical and health scienceschemistry.chemical_compound0302 clinical medicineNeurobiologyReceptor Cannabinoid CB1Cannabinoid receptor type 2medicineAnimalsHumansanandamideReceptorReceptors Cannabinoidbehaviorcannabinoid receptorEndocannabinoid systemState of the Art030227 psychiatry2-arachidonoylglycerolmedicine.anatomical_structurechemistryneural communicationCannabinoidNeuroscienceEndocannabinoidsSignal TransductionDialogues in clinical neuroscience
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The endocannabinoid system: emotion, learning and addiction

2008

The identification of the cannabinoid receptor type 1 (CB1 receptor) was the milestone discovery in the elucidation of the behavioural and emotional responses induced by the Cannabis sativa constituent Delta(9)-tetrahydrocannabinol. The subsequent years have established the existence of the endocannabinoid system. The early view relating this system to emotional responses is reflected by the fact that N-arachidonoyl ethanolamine, the pioneer endocannabinoid, was named anandamide after the Sanskrit word 'ananda', meaning 'bliss'. However, the emotional responses to cannabinoids are not always pleasant and delightful. Rather, anxiety and panic may also occur after activation of CB1 receptors.…

PharmacologyCannabinoid receptorAddictionmedia_common.quotation_subjectmedicine.medical_treatmentMedicine (miscellaneous)AnandamideEndocannabinoid systemPsychiatry and Mental healthchemistry.chemical_compoundnervous systemchemistryCannabinoid receptor type 1medicinelipids (amino acids peptides and proteins)CannabinoidAversive StimulusPsychologyTetrahydrocannabinolNeurosciencemedia_commonmedicine.drugAddiction Biology
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Differential effects of anandamide on acetylcholine release in the guinea-pig ileum mediated via vanilloid and non-CB1 cannabinoid receptors

2001

The effects of anandamide on [3H]-acetylcholine release and muscle contraction were studied on the myenteric plexus-longitudinal muscle preparation of the guinea-pig ileum preincubated with [3H]-choline. Anandamide increased both basal [3H]-acetylcholine release (pEC50 6.3) and muscle tone (pEC50 6.3). The concentration-response curves for anandamide were shifted to the right by 1 μM capsazepine (pKB 7.5 and 7.6), and by the combined blockade of NK1 and NK3 tachykinin receptors with the antagonists CP99994 plus SR142801 (each 0.1 μM). The CB1 and CB2 receptor antagonists, SR141716A (1 μM) and SR144528 (30 nM), did not modify the facilitatory effects of anandamide. Anandamide inhibited the e…

Pharmacologymedicine.medical_specialtyCannabinoid receptormedicine.medical_treatmentTRPV1AnandamideMuscarinic agonistchemistry.chemical_compoundEndocrinologychemistryInternal medicinemedicineCannabinoidCapsazepineTachykinin receptorAcetylcholinemedicine.drugBritish Journal of Pharmacology
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