Search results for "Anderson"

showing 6 items of 76 documents

Fabry Disease, a Complex Pathology Not Easy to Diagnose

2015

Fabry disease is a multisystemic lysosomal storage disorder, inherited in an X-linked manner. It is a defect of metabolism of the glycosphingolipids, due to the reduction or absence of the activity of lysosomal enzyme α-galactosidase A. This reduction of activity causes the storage of globotriaosylceramide and derivatives in the lysosomes, triggering a cascade of cellular events, mainly in vascular endothelium. These events are the responsible for the systemic clinical manifestations and the renal, cardiac and cerebrovascular complications, or a combination of them. The symptomatology can lead to the premature death of patient between the fourth or fifth decade of life. The first symptoms c…

lcsh:Diseases of the circulatory (Cardiovascular) systemPathologymedicine.medical_specialtyα-galactosidase Abusiness.industryGlobotriaosylceramideDiagnostic testDiseaseEnzyme replacement therapyAnderson-Fabry diseasemedicine.diseaseFabry diseaseVascular endotheliumchemistry.chemical_compoundPremature deathchemistrylcsh:RC666-701Clinical diagnosismedicineGeneral Earth and Planetary SciencesbusinessGLA gene.General Environmental ScienceCardiogenetics
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A classical phenotype of Anderson-Fabry disease in a female patient with intronic mutations of the GLA gene: a case report

2012

Abstract Background Fabry disease (FD) is a hereditary metabolic disorder caused by the partial or total inactivation of a lysosomal hydrolase, the enzyme α-galactosidase A (GLA). This inactivation is responsible for the storage of undegraded glycosphingolipids in the lysosomes with subsequent cellular and microvascular dysfunction. The incidence of disease is estimated at 1:40,000 in the general population, although neonatal screening initiatives have found an unexpectedly high prevalence of genetic alterations, up to 1:3,100, in newborns in Italy, and have identified a surprisingly high frequency of newborn males with genetic alterations (about 1:1,500) in Taiwan. Case presentation We des…

lcsh:Diseases of the circulatory (Cardiovascular) systemPathologyα-galactosidase AAnderson-Fabry mutationBiopsyDNA Mutational AnalysisCase Reportmedicine.disease_causeGlobotriaosylceramide0302 clinical medicineSettore BIO/13 - Biologia ApplicataPromoter Regions Genetic0303 health sciencesMutationeducation.field_of_studymedicine.diagnostic_testbiologyMetabolic disorderMagnetic Resonance Imaging3. Good healthPhenotypeCardiovascular DiseasesDisease ProgressionFemaleKidney DiseasesRenal biopsyCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtyPopulation03 medical and health sciencesPredictive Value of TestsBiopsymedicineHumansHigh resolution meltingGenetic Predisposition to Diseaseeducation030304 developmental biologyFabry diseaseAlpha-galactosidasebusiness.industrymedicine.diseaseFabry diseaseIntronslcsh:RC666-701alpha-GalactosidaseMutationGLAbiology.proteinbusiness030217 neurology & neurosurgeryKidney disease
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Data from: Successive invasion-mediated interspecific hybridizations and population structure in the endangered cichlid Oreochromis mossambicus

2013

Hybridization between invasive and native species accounts among the major and pernicious threats to biodiversity. The Mozambique tilapia Oreochromis mossambicus, a widely used freshwater aquaculture species, is especially imperiled by this phenomenon since it is recognized by the IUCN as an endangered taxon due to genetic admixture with O. niloticus an invasive congeneric species. The Lower Limpopo and the intermittent Changane River (Mozambique) drain large wetlands of potentially great importance for conservation of O. mossambicus, but their populations have remained unstudied until today. Therefore we aimed (1) to estimate the autochthonous diversity and population structure among genet…

medicine and health careOreochromis andersoniiHolocenetilapiamtDNAAnthropoceneOreochromis niloticusMedicineGenetic resourceCichlidaeamplified fragment length polymorphism (AFLP)Life sciencesOreochromis mossambicus
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Downregulation of alpha-galactosidase A upregulates CD77: functional impact for Fabry nephropathy.

2009

Anderson-Fabry disease, an inherited deficiency in the lysosomal enzyme alpha-galactosidase A, is characterized by the progressive accumulation of globotriaosylceramide (Gb3), also known as CD77. We sought to clarify the pathogenesis of Fabry disease by establishing a cell model of this disorder. The expression of alpha-galactosidase A was transiently silenced by RNA interference in HK2 and primary human renal epithelial cells and stably silenced in HK2 cells by retroviral transfection with small hairpin RNA. All of the silenced cells had histological similarities to cells of patients with Fabry disease. The cells had reduced viability, significant accumulation of intracellular Gb3, and a m…

medicine.medical_specialtyGlobotriaosylceramideGb3Cell LineSmall hairpin RNAchemistry.chemical_compoundRNA interferenceDownregulation and upregulationInternal medicineMedicineGene silencingHumansGene SilencingRNA Small InterferingAnderson–Fabry diseaseGlobosidesbusiness.industryTrihexosylceramidesEpithelial CellsTransfectionEnzyme replacement therapymedicine.diseaseFabry diseaseα-galactosidaseEndocrinologychemistryGene Expression RegulationNephrologyCell culturealpha-GalactosidaseCancer researchFabry DiseaseCD77businessenzyme replacement therapyKidney international
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Anderson‐Fabry Disease: A Rare Cause of Levodopa‐Responsive Early‐Onset Parkinsonism

2021

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medicine.medical_specialtyLevodopabusiness.industryEarly onset parkinsonismGastroenterologyAnderson-Fabry DiseaseNeurologyInternal medicinemedicineAnderson-Fabry disease early onset parkinsonism levodopa response lysosomal storage diseasesSettore MED/26 - NeurologiaNeurology (clinical)businessCase Reports and Commentariesmedicine.drugMovement Disorders Clinical Practice
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UN CASO DI SINDROME DA RIVASCOLARIZZAZIONE SUCCESSIVA A INNESTO DI PROTESI DELL’AORTA PER LESIONE JATROGENA

2013

La sindrome da rivascolarizzazione (SR) comprende un insieme di eventi metabolici ed emodinamici che insorgono in un arto rivascolarizzato dopo un periodo più o meno lungo di ischemia, caratterizzata da specifiche manifestazioni cliniche sia a livello locale che sistemico. Riportiamo qui di seguito un caso di SR occorsa in una giovane donna di anni 20 con Sindrome di Turner e tiroidite autoimmune la quale veniva sottoposta ad intervento di giuntopieloplastica sinistra Robot assistita, poiché affetta da stenosi del medesimo giunto ed idronefrosi sinistra.

sindrome da rivascolarizzazione pieloplastica secondo Anderson-Hynes errore chirurgicoSettore MED/43 - Medicina Legale
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