Search results for "Angiogenesi"

showing 10 items of 568 documents

Central role of IL-6 and MMP-1 for cross talk between human intestinal mast cells and human intestinal fibroblasts.

2012

Mast cells (MC) are key effector cells in allergic reactions but also involved in host defence, tissue remodeling, angiogenesis, and fibrogenesis. Here, we show that human intestinal fibroblasts (FB) suppress apoptosis in human intestinal MC dependent on IL-6. Intestinal FB produced IL-6 upon direct stimulation by intestinal MC in co-culture or by MC mediators such as TNF-α, IL-1β, tryptase or histamine. MC incubated with IL-6 survived for up to 3 weeks similar to MC co-cultured with FB and MC survival could be blocked by neutralizing anti-IL-6 Abs. Moreover, FB stimulated by MC mediators upregulated their expression of matrix metalloproteinase-1 (MMP-1), a key fibrolytic enzyme. Noteworthy…

AngiogenesisCell SurvivalImmunologyInflammationTryptaseApoptosisCell CommunicationMatrix metalloproteinaseBiologychemistry.chemical_compoundDownregulation and upregulationmedicineImmunology and AllergyHumansMast CellsIntestinal MucosaInterleukin 6Protein Kinase InhibitorsFlavonoidsInterleukin-6HematologyReceptor Cross-TalkFibroblastsMolecular biologyCoculture TechniqueschemistryApoptosisImmunologybiology.proteinmedicine.symptomMatrix Metalloproteinase 1HistamineImmunobiology
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Stimulation of Wound Revascularization by Adenosine Receptor Activation

2010

Adenosine is an endogenous mediator implicated in wound healing. The exact mechanisms and receptors involved are still under evaluation. We have observed that topical application of a selective adenosine A2A receptor agonist promotes wound healing in experimental animals, both healthy and with impaired healing. Histological analysis revealed that adenosine promoted granulation tissue formation, with increased cellularity, matrix deposition, and vessel density. Adenosine may exert effects on many different cell types that finally result in promotion of angiogenesis and, furthermore, homing of endothelial precursor cells into sites of injury. We will review the effect of adenosine receptor ac…

AngiogenesisChemistryGranulation tissueAdenosine A2A receptorPharmacologyAdenosineAdenosine receptorEndothelial stem cellmedicine.anatomical_structureAnesthesiamedicineReceptorWound healingmedicine.drug
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Cyclooxygenase inhibitors – current status and future prospects

2001

Prostaglandins are formed from arachidonic acid by the action of cyclooxygenase and subsequent downstream synthetases. Two closely related forms of the cyclooxygenase have been identified which are now known as COX-1 and COX-2. Both isoenzymes transform arachidonic acid to prostaglandins, but differ in their distribution and their physiological roles. Meanwhile, the responsible genes and their regulation have been clarified. COX-1, the pre-dominantly constitutive form of the enzyme, is expressed throughout the body and performs a number of homeostatic functions such as maintaining normal gastric mucosa and influencing renal blood flow and platelet aggregation. In contrast, the inducible for…

AngiogenesisInflammationPharmacologyStructure-Activity Relationshipchemistry.chemical_compoundIndometacinDrug DiscoverymedicineAnimalsHumansCyclooxygenase InhibitorsPharmacologyMolecular StructurebiologyAnti-Inflammatory Agents Non-SteroidalOrganic ChemistryGeneral Medicinemedicine.diseasechemistryBiochemistryEnzyme inhibitorRheumatoid arthritisbiology.proteinArachidonic acidCyclooxygenasemedicine.symptomHomeostasisForecastingmedicine.drugEuropean Journal of Medicinal Chemistry
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A recommended practical approach to the management of target therapy and angiogenesis inhibitors cardiotoxicity: an opinion paper of the working grou…

2016

The US National Cancer Institute estimates that cardiotoxicity (CTX) from target therapy refers mostly to four groups of drugs: epidermal growth factor receptor 2 inhibitors, angiogenic inhibitors, directed Abelson murine leukemia viral oncogene homolog inhibitors, and proteasome inhibitors. The main cardiotoxic side-effects related to antiepidermal growth factor receptor 2 therapy are left ventricular systolic dysfunction and heart failure. Angiogenesis inhibitors are associated with hypertension, left ventricular dysfunction/heart failure, myocardial ischemia, QT prolongation, and thrombosis. Moreover, other agents may be related to CTX induced by treatment. In this study, we review the g…

AngiogenesisLeftAngiogenesis Inhibitors030204 cardiovascular system & hematologyVentricular Dysfunction Left0302 clinical medicinetyrosine kinase inhibitorNeoplasmstyrosine kinase inhibitorsVentricular DysfunctionMolecular Targeted TherapyEpidermal growth factor receptorSocieties Medicalangiogenesis inhibitors; HER2/epidermal growth factor receptor 2; tyrosine kinase inhibitorABLbiologyDisease ManagementGeneral MedicineItalyCardiovascular DiseasesSupplement Submission030220 oncology & carcinogenesisangiogenesis inhibitors; HER2/epidermal growth factor receptor 2; tyrosine kinase inhibitors; Angiogenesis Inhibitors; Antineoplastic Agents; Cardiology; Cardiomyopathies; Cardiotoxicity; Heart Failure; Humans; Italy; Neoplasms; Practice Guidelines as Topic; Societies Medical; Ventricular Dysfunction Left; Disease ManagementPractice Guidelines as TopicCardiologyCardiology and Cardiovascular MedicineCardiomyopathiesmedicine.medical_specialtyCardiologyAntineoplastic AgentsRisk AssessmentQT interval03 medical and health sciencesGrowth factor receptorInternal medicineMedicalmedicineHumansMonitoring PhysiologicHeart FailureCardiotoxicitybusiness.industryCancerHER2/epidermal growth factor receptor 2medicine.diseaseangiogenesis inhibitors; HER2/epidermal growth factor receptor 2; tyrosine kinase inhibitors; Cardiology and Cardiovascular MedicineCardiotoxicityangiogenesis inhibitorHeart failurebiology.proteinbusinessSocietiesJournal of cardiovascular medicine (Hagerstown, Md.)
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The transmembrane receptor Uncoordinated5 (Unc5) is essential for heart lumen formation in Drosophila melanogaster

2011

AbstractTransport of liquids or gases in biological tubes is fundamental for many physiological processes. Our knowledge on how tubular organs are formed during organogenesis and tissue remodeling has increased dramatically during the last decade. Studies on different animal systems have helped to unravel some of the molecular mechanisms underlying tubulogenesis. Tube architecture varies dramatically in different organs and different species, ranging from tubes formed by several cells constituting the cross section, tubes formed by single cells wrapping an internal luminal space or tubes that are formed within a cell. Some tubes display branching whereas others remain linear without interse…

AngiogenesisLumen (anatomy)Receptors Cell SurfaceOrganogenesisLumen formationBiologyLigandsUnc5AnimalsDrosophila ProteinsDrosophila heart morphogenesisMyocytes CardiacNerve Growth FactorsReceptorMolecular BiologyCardiogenesisTumor Suppressor ProteinsHeartCell BiologyAnatomyNetrin-1Tubulogenesisbiology.organism_classificationTransmembrane proteinCell biologyDrosophila melanogasterNetrinBSignal transductionDrosophila melanogasterNetrin ReceptorsDrosophila ProteinDevelopmental BiologyDevelopmental Biology
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Rapid vascularization of starchâ poly(caprolactone) in vivo by outgrowth endothelial cells in co-culture with primary osteoblasts

2011

The successful integration of in vitro-generated tissues is dependent on adequate vascularization in vivo. Human outgrowth endothelial cells (OECs) isolated from the mononuclear cell fraction of peripheral blood represent a potent population of circulating endothelial progenitors that could provide a cell source for rapid anastomosis and scaffold vascularization. Our previous work with these cells in co-culture with primary human osteoblasts has demonstrated their potential to form perfused vascular structures within a starch–poly(caprolactone) biomaterial in vivo. In the present study, we demonstrate the ability of OECs to form perfused vascular structures as early as 48 h following subcut…

AngiogenesisPolyestersPopulationBiomedical EngineeringNeovascularization PhysiologicMedicine (miscellaneous)02 engineering and technologyBiologyBiomaterialsNeovascularization03 medical and health sciencesTissue engineeringIn vivoIn vivomedicineHumansVimentinProgenitor celleducationCells CulturedCell Proliferation030304 developmental biologyPericyte0303 health scienceseducation.field_of_studyOsteoblastsScience & TechnologyOsteoblastEndothelial CellsOutgrowth endothelial cellStarchOsteoblast021001 nanoscience & nanotechnologyImmunohistochemistryCoculture Techniques3. Good healthCell biologyPlatelet Endothelial Cell Adhesion Molecule-1medicine.anatomical_structureBlood VesselsPericyteAngiogenesismedicine.symptomCo-culture0210 nano-technologyBiomedical engineering
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Angiogenic activity of breast cancer patients' monocytes reverted by combined use of systems modeling and experimental approaches.

2015

Angiogenesis plays a key role in tumor growth and cancer progression. TIE-2-expressing monocytes (TEM) have been reported to critically account for tumor vascularization and growth in mouse tumor experimental models, but the molecular basis of their pro-angiogenic activity are largely unknown. Moreover, differences in the pro-angiogenic activity between blood circulating and tumor infiltrated TEM in human patients has not been established to date, hindering the identification of specific targets for therapeutic intervention. In this work, we investigated these differences and the phenotypic reversal of breast tumor pro-angiogenic TEM to a weak pro-angiogenic phenotype by combining Boolean m…

AngiogenesisQH301-705.5In silicoBreast NeoplasmsMice TransgenicKaplan-Meier EstimateBiologyModels BiologicalMonocytesCell Line03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineBreast cancerGeneticsmedicineAnimalsHumansBiology (General)Molecular BiologyEcology Evolution Behavior and Systematics030304 developmental biology0303 health sciencesTumor microenvironmentEcologyNeovascularization PathologicComputational BiologyNeoplasms ExperimentalTumor-DerivedMiddle Agedmedicine.diseasePhenotype3. Good healthGene expression profilingPhenotypeComputational Theory and Mathematics030220 oncology & carcinogenesisModeling and SimulationImmunologyCancer researchCytokinesFemaleSignal transductionResearch ArticleSignal TransductionPLoS Computational Biology
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Inhibition of tumor angiogenesis by antibodies, synthetic small molecules and natural products.

2011

Cancer remains one of the major causes of death worldwide. The switch to pathological angiogenesis is a key process in the promotion of cancer and consequently provides several new and promising targets to anticancer therapy. Thus, antagonizing angiogenesis cuts off the tumor's oxygen and nutrition supply. This review focuses on angiogenesis inhibitors as option for cancer treatment. Modes of action, adverse effects, mechanisms of resistance as well as new developments are highlighted. One approach in angiogenesis inhibition is intermitting the further VEGF (vascular endothelial growth factor) signal pathway with monoclonal antibodies. Bevacizumab is a highly specific recombinant humanized …

Angiogenesismedicine.drug_classGenisteinAngiogenesis InhibitorsAntineoplastic AgentsBiologyPharmacologyMonoclonal antibodyBiochemistryReceptor tyrosine kinaseNeovascularizationSmall Molecule Librarieschemistry.chemical_compoundGrowth factor receptorNeoplasmsDrug DiscoverymedicineAnimalsHumansPharmacologyBiological ProductsNeovascularization PathologicVascular Endothelial Growth FactorsOrganic ChemistryCancerAntibodies Monoclonalmedicine.diseaseAntineoplastic Agents PhytogenicVascular endothelial growth factorchemistrybiology.proteinMolecular Medicinemedicine.symptomCurrent medicinal chemistry
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Priming with proangiogenic growth factors and endothelial progenitor cells improves revascularization in linear diabetic wounds

2014

In the present study, we investigated whether proangiogenic growth factors and endothelial progenitor cells (EPCs) induce favourable effects on cutaneous incisional wound healing in diabetic mice. The proangiogenic effects of human EPCs were initially analyzed using a HUVEC in vitro angiogenesis assay and an in vivo Matrigel assay in nude mice (n=12). For the diabetic wound model, 48 Balb/c mice with streptozotocin (STZ)-induced diabetes were divided randomly into 4 groups (12 mice in each group). Subsequently, 3, 5 and 7 days before a 15-mm full-thickness incisional skin wound was set, group 1 was pre-treated subcutaneously with a mixture of vascular endothelial growth factor (VEGF)/basic …

Angiogenesismedicine.medical_treatmentBasic fibroblast growth factorMice NudeNeovascularization Physiologicwound healingdiabetic miceDiabetes Mellitus ExperimentalAndrologychemistry.chemical_compoundMiceTensile StrengthGeneticsHuman Umbilical Vein Endothelial CellsMedicineAnimalsHumansProgenitor cellprimingendothelial progenitor cellsMatrigelMice Inbred BALB Cbiologybusiness.industryGrowth factorStem CellsEndothelial CellsGeneral MedicineArticlesVascular endothelial growth factorproangiogenicDrug CombinationschemistryImmunologyMicrovesselsbiology.proteincardiovascular systemIntercellular Signaling Peptides and ProteinsBiological AssayProteoglycansCollagenLamininbusinessWound healingPlatelet-derived growth factor receptorStem Cell TransplantationInternational Journal of Molecular Medicine
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The erythropoietin and regenerative medicine: a lesson from fish

2009

Background Erythropoietin (EPO), the main haematopoietic growth factor for the proliferation and differentiation of erythroid progenitor cells, is also known for its angiogenic and regenerative properties. Materials and methods In this study, we aimed to test the regenerative effects of EPO administration in an experimental model of Sea bass (Dicentrarchus labrax) subjected to amputation of the caudal fin. Results Erythropoietin-treated fishes (3000 UI of human recombinant EPO-alpha immediately after cutting and after 15 days) showed an increased growth rate of their fins compared with those untreated (ANOVA variance: P :0 AE01 vs. P :0 AE04). By analysing fin length at established times (1…

Angiogenesismedicine.medical_treatmentClinical BiochemistryNeovascularization PhysiologicRegenerative MedicineModels BiologicalBiochemistryAndrologyangiogenesisfin growthFibrosismedicineAnimalsSea bassbiologySettore MED/27 - NeurochirurgiaMedicine (all)Growth factorFishesGeneral MedicineSea bamedicine.diseasebiology.organism_classificationImmunohistochemistryEPO regenerative medicineangiogenesis; Erythropoietin; tissue regeneration; fishAngiogenesiHaematopoiesisErythropoietinregenerationImmunologyBassDicentrarchuserythropoietinStem cellsea bassmedicine.drugEuropean Journal of Clinical Investigation
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