Search results for "Angiogenesis"

showing 10 items of 552 documents

Versican and Tumor-Associated Macrophages Promotes Tumor Progression and Metastasis in Canine and Murine Models of Breast Carcinoma

2019

Versican and tumor-associated macrophages (TAMs) are involved in growth and metastases in several cancers. Here, we investigated the potential role of versican, a matrix proteoglycan, and its correlation with TAMs infiltrates in different stages of two different breast cancer models: spontaneous canine mammary gland carcinomas and the murine 4T1 breast cancer model. The stromal versican expression was correlated with TAMs accumulation in tumors with an advanced stage from spontaneous canine mammary carcinoma samples. Versican expression in mice, identified in late stages of tumor progression, was associated to a high number of peri-tumoral infiltrating TAMs. Indeed, TAMs were related to a p…

EXPRESSION0301 basic medicineCancer ResearchStromal cellMICROENVIRONMENTlcsh:RC254-282Metastasis03 medical and health sciencesangiogenesis0302 clinical medicineBreast cancerbreast cancerINFLAMMATIONstomatognathic systemEXTRACELLULAR-MATRIXmedicineTGF-BETA-1skin and connective tissue diseasesOriginal ResearchversicanCanine Mammary CarcinomaScience & Technologybiologybusiness.industrytumor-associated macrophageslung metastasisTGF-BETAmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensCANCERPrimary tumorcarbohydrates (lipids)030104 developmental biologyOncologyTumor progression030220 oncology & carcinogenesisCELLSbiology.proteinCancer researchGROWTHVersicanBreast carcinomabusinessLife Sciences & BiomedicineCCL2hormones hormone substitutes and hormone antagonistsFrontiers in Oncology
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Lymphatic vessels are present in human saccular intracranial aneurysms

2022

AbstractSaccular intracranial aneurysm (sIA) rupture leads to subarachnoid haemorrhage and is preceded by chronic inflammation and atherosclerotic changes of the sIA wall. Increased lymphangiogenesis has been detected in atherosclerotic extracranial arteries and in abdominal aortic aneurysms, but the presence of lymphatic vessels in sIAs has remained unexplored. Here we studied the presence of lymphatic vessels in 36 intraoperatively resected sIAs (16 unruptured and 20 ruptured), using immunohistochemical and immunofluorescence stainings for lymphatic endothelial cell (LEC) markers. Of these LEC-markers, both extracellular and intracellular LYVE-1-, podoplanin-, VEGFR-3-, and Prox1-positive…

EXPRESSIONPROX1biomarkkeritAneurysm RupturedPathology and Forensic Medicinelymphatic vesselsCellular and Molecular NeuroscienceimusuonistoHumansLymphangiogenesisCerebral aneurysmkallonsisäinen aneurysmaARTERYInflammationtulehdusaneurysma3112 NeurosciencesIntracranial AneurysmThrombosisWALLVascular Endothelial Growth Factor Receptor-3aivoverenvuotolymphangiogenesissaccular intracranial aneurysmcerebral aneurysmLymphatic vesselsinflammationADVENTITIAL LYMPHATICSMAST-CELLSNeurology (clinical)BiomarkersSaccular intracranial aneurysmActa Neuropathologica Communications
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The effects of ergot and non-ergot-derived dopamine agonists in an experimental mouse model of endometriosis

2011

Implantation of a retrogradely shed endometrium during menstruation requires an adequate blood supply, which allows the growth of endometriotic lesions. This suggests that the development of endometriosis can be impaired by inhibiting angiogenesis. The growth of endometriotic foci is impaired by commercial oncological antiangiogenic drugs used to block vascular endothelial growth factor (VEGF) signaling. The dopamine agonist cabergoline (Cb2) inhibits the growth of established endometriosis lesions by exerting antiangiogenic effects through VEGFR2 inactivation. However, the use of ergot-derived Cb2 is associated with an increased incidence of cardiac valve regurgitation. To evaluate the pot…

Embryologymedicine.medical_specialtyCabergolineProliferation indexAngiogenesisEndometriosisEndometriosisCell CountPharmacologyDopamine agonistClavicepsNeovascularizationEndometriumMicechemistry.chemical_compoundEndocrinologyInternal medicineCabergolinemedicineAnimalsHumansErgolinesCell ProliferationUterine DiseasesNeovascularization PathologicReceptors Dopamine D2business.industryQuinagolideObstetrics and GynecologyCell Biologymedicine.diseaseVascular Endothelial Growth Factor Receptor-2Vascular endothelial growth factorDisease Models AnimalEndocrinologyReproductive MedicinechemistryDopamine AgonistsBlood VesselsFemalemedicine.symptombusinessmedicine.drugREPRODUCTION
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Lactate adversely affects the in vitro formation of endothelial cell tubular structures through the action of TGF-beta1.

2006

When lactate accumulation in a tumor microenvironment reaches an average concentration of 10-20 mM, it tends to reflect a high degree of malignancy. However, the hypothesis that tumor-derived lactate has a number of partially adverse biological effects on malignant and tumor-associated host cells requires further evidence. The present study attempted to evaluate the impact of lactate on the process of angiogenesis, in particular on the formation of tubular structures. The endothelial cell (EC) network in desmoplastic breast tumors is primarily located in areas of reactive fibroblastic stroma. We employed a fibroblast-endothelial cell co-culture model as in vitro angiogenesis system normally…

EndotheliumAngiogenesisCell SurvivalCellPopulationNeovascularization PhysiologicBiologyTransforming Growth Factor beta1Cell MovementNeutralization TestsmedicineHumansLactic AcidRNA MessengerFibroblasteducationCell ProliferationTumor microenvironmenteducation.field_of_studyCell growthEndothelial CellsCell BiologyFibroblastsActinsCoculture TechniquesCell biologyEndothelial stem cellmedicine.anatomical_structureGene Expression RegulationImmunologyExperimental cell research
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Tumour vascularization via endothelial differentiation of glioblastoma stem-like cells

2010

Glioblastoma is a highly angiogenetic malignancy, the neoformed vessels of which are thought to arise by sprouting of pre-existing brain capillaries. The recent demonstration that a population of glioblastoma stem-like cells (GSCs) maintains glioblastomas indicates that the progeny of these cells may not be confined to the neural lineage. Normal neural stem cells are able to differentiate into functional endothelial cells. The connection between neural stem cells and the endothelial compartment seems to be critical in glioblastoma, where cancer stem cells closely interact with the vascular niche and promote angiogenesis through the release of vascular endothelial growth factor(VEGF) and str…

EndotheliumAngiogenesisTransplantation HeterologousSettore MED/27 - NEUROCHIRURGIAMice TransgenicMice SCIDBiologyModels BiologicalMiceVasculogenesisNeural Stem CellsMice Inbred NODCell Line TumormedicineAnimalsHumansCell LineageVasculogenic mimicryglioblastoma tumor vascularizationIn Situ Hybridization FluorescenceChromosome AberrationsMultidisciplinaryNeovascularization PathologicEndothelial CellsCell DifferentiationVascular endothelial growth factor BEndothelial stem cellVascular endothelial growth factor Amedicine.anatomical_structureVascular endothelial growth factor CTumor Markers BiologicalImmunologyCancer researchEndothelium VascularGlioblastomaNeoplasm TransplantationNature
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Expression Profile of Endoglin in Different Grades of Endometrial Cancer

2019

Background Endoglin is a marker of active, proliferating endothelial cells of blood vessels. In many cancers, it is present in both peripheral vessels and vessels located inside the tumor. Endoglin is more specific and sensitive compared to other tumor angiogenesis markers. It is suggested that endoglin can be considered a reliable marker of disease outcome. Objective The aim of the study was to assess the expression of endoglin and to determine its potential usefulness as a complementary molecular marker of endometrial cancer. Method The study included 60 women who underwent hysterectomy: 45 with endometrioid endometrial cancer (study group) and 15 without neoplastic changes (control group…

Endotheliummedicine.drug_classAngiogenesisPharmaceutical ScienceReceptors Cell SurfaceMonoclonal antibodyMiceangiogenesisEndometrial cancerAntigens CDhemic and lymphatic diseasesBiomarkers Tumorotorhinolaryngologic diseasesmedicineAnimalsHumansmolecular markerendoglinNeovascularization Pathologicbusiness.industryEndometrial cancerCancerEndoglinmedicine.diseaseEndometrial Neoplasmsmedicine.anatomical_structureCase-Control StudiesCancer cellimmunohistochemistryCancer researchImmunohistochemistryFemaleEndothelium VascularNeoplasm Gradingbusinessvascular endotheliumSignal TransductionBiotechnologyCurrent Pharmaceutical Biotechnology
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Angiogenesis, immune system and growth factors: new targets in colorectal cancer therapy.

2005

Colorectal cancer is the second most common malignant human neoplasia. Over recent years, many efforts have been performed in order to develop and improve therapeutic protocols, and many advances have been accomplished in both the field of adjuvant and palliative therapy. Most of the chemotherapic agents currently used in the clinical setting are the products of decades of research aimed at inhibiting the uncontrolled growth of dysplastic cells. However, new frontiers in this field have recently been opened, with the identification of key molecules involved in physiologic mechanisms that are of fundamental importance for cancer development and progression. Tumor-induced angiogenesis, the ca…

Epidermal Growth FactorNeovascularization PathologicAngiogenesisbusiness.industryColorectal cancerGrowth factormedicine.medical_treatmentAngiogenesis Inhibitorsmedicine.diseasePalliative TherapyNeovascularizationImmune systemCytokineOncologyImmune SystemImmunologymedicineCancer researchHumansPharmacology (medical)medicine.symptombusinessColorectal NeoplasmsAdjuvantSignal TransductionExpert review of anticancer therapy
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Structural Basis of Tumoral Angiogenesis

2003

Mammalian cells require oxygen and nutrients for metabolism and growth. In all cases tissues possess a vascular and lymphatic network assuring the supply of these needs within 200 to 250µm. Multicellular organisms that grow beyond this size require the recruitment of new blood vessels, although some normal tissues are devoid of specific vascularization (cartilage, cornea, epidermis), obtaining their oxygen and metabolic supply through perfusion

Epidermis (botany)AngiogenesisCartilagegovernment.form_of_governmentBiologyCell biologyVascular endothelial growth factorchemistry.chemical_compoundLymphatic EndotheliumMulticellular organismmedicine.anatomical_structureLymphatic systemchemistrymedicinegovernmentVasculogenic mimicry
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Potential Anti-Metastatic Role of the Novel miR-CT3 in Tumor Angiogenesis and Osteosarcoma Invasion

2022

Osteosarcoma (OS) is the most common primary bone tumor mainly occurring in young adults and derived from primitive bone-forming mesenchyme. OS develops in an intricate tumor microenvironment (TME) where cellular function regulated by microRNAs (miRNAs) may affect communication between OS cells and the surrounding TME. Therefore, miRNAs are considered potential therapeutic targets in cancer and one of the goals of research is to accurately define a specific signature of a miRNAs, which could reflect the phenotype of a particular tumor, such as OS. Through NGS approach, we previously found a specific molecular profile of miRNAs in OS and discovered 8 novel miRNAs. Among these, we deepen our …

Epithelial-Mesenchymal TransitionQH301-705.5MAP Kinase Signaling SystemEMT proteinBone NeoplasmsArticleCatalysisCell LineInorganic ChemistryCell Line TumorHuman Umbilical Vein Endothelial CellsmetastasisHumansNeoplasm InvasivenessBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopyOsteosarcomaOsteoblastsmicroRNANeovascularization PathologicOrganic ChemistryEMT proteinstumor angiogenesisGeneral MedicinemicroRNAsComputer Science ApplicationsGene Expression Regulation NeoplasticChemistryosteosarcoma; microRNAs; tumor angiogenesis; metastasis; EMT proteinsmetastasitumor angiogenesiInternational Journal of Molecular Sciences
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Role of exosomes released by chronic myelogenous leukemia cells in angiogenesis

2011

ExosomeSettore BIO/13 - Biologia ApplicataAngiogenesisCML
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