Search results for "Angiotensin-Converting Enzyme"

showing 10 items of 159 documents

Effects of clinical and laboratory variables and of pretreatment with cardiovascular drugs in acute ischaemic stroke: A retrospective chart review fr…

2009

Background: Few studies have examined the role of cardiovascular drugs on acute ischaemic stroke prognosis. Aims: To evaluate the relationship between a favourable outcome in patients with acute ischaemic stroke and specific demographic, clinical and laboratory variables and cardiovascular drug pretreatment. Methods: The 1096 patients enrolled in the GIFA study (who had a main discharge diagnosis of ischaemic stroke) represent the final patient sample used in this analysis. Drugs considered in the analysis included angiotensin converting enzyme (ACE)-inhibitors, angiotensin II receptor blockers, statins, calcium channel blockers, anti-platelet drugs, vitamin K antagonists and heparins. The …

MaleStroke prognosis pre-treatmentmedicine.medical_specialtySettore MED/09 - Medicina InternaActivities of daily livingDatabases FactualBrain IschemiaInternal medicineWhite blood cellmedicineHumansHospital MortalityStrokeAgedRetrospective StudiesAged 80 and overbiologybusiness.industryPharmacoepidemiologyCalcium channelCardiovascular AgentsAngiotensin-converting enzymeRetrospective cohort studyHeparinmedicine.diseaseStrokeTreatment Outcomemedicine.anatomical_structureBlood pressureItalybiology.proteinPhysical therapyFemaleCardiology and Cardiovascular MedicinebusinessFollow-Up Studiesmedicine.drugInternational Journal of Cardiology
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A putative placebo analysis of the effects of LCZ696 on clinical outcomes in heart failure

2015

Aims: Although active-controlled trials with renin–angiotensin inhibitors are ethically mandated in heart failure with reduced ejection fraction, clinicians and regulators often want to know how the experimental therapy would perform compared with placebo. The angiotensin receptor-neprilysin inhibitor LCZ696 was compared with enalapril in PARADIGM-HF. We made indirect comparisons of the effects of LCZ696 with putative placebos.\ud \ud Methods and results: We used the treatment-arm of the Studies Of Left Ventricular Dysfunction (SOLVD-T) as the reference trial for comparison of an ACE inhibitor to placebo and the Candesartan in Heart failure: Assessment of Reduction in Mortality and morbidit…

MaleTetrazolesAngiotensin-Converting Enzyme InhibitorsEnalaprilEnalapril/therapeutic useMedicineNatriuretic peptidesAngiotensin IIAminobutyratesHeart Failure/CardiomyopathyMiddle AgedAngiotensin Receptor Antagonists/therapeutic useHospitalizationAngiotensin-Converting Enzyme Inhibitors/therapeutic useDrug CombinationsTreatment OutcomeTetrazoles/therapeutic useCardiologyValsartanFemaleCardiology and Cardiovascular Medicinemedicine.drugBenzimidazoles/therapeutic usemedicine.medical_specialtyAngiotensin II Type 1 Receptor Blockers/therapeutic usemedicine.drug_classPlaceboAngiotensin Receptor AntagonistsInternal medicineHumansEnalaprilFASTTrack Clinical ResearchBeta blockerAgedHospitalization/statistics & numerical dataHeart Failurebusiness.industryBiphenyl Compoundsmedicine.diseaseHeart Failure/drug therapyPlacebo EffectAngiotensin IICandesartanEndocrinologyAminobutyrates/therapeutic useHeart failureACE inhibitorBenzimidazolesbusinessAngiotensin II Type 1 Receptor BlockersSacubitril ValsartanNatriuretic peptide
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The effects of telmisartan alone or with hydrochlorothiazide on morning and 24-h ambulatory BP control: results from a practice-based study (SURGE 2)

2012

Observational studies have shown that 24-h and morning ambulatory blood pressure (BP) control is low. This large-scale, practice-based study evaluated the effects of telmisartan 40 or 80 mg alone or in combination with hydrochlorothiazide (HCTZ) 12.5 mg on these BP parameters over 8 weeks; treatment was adjusted if clinic BP remained ≥140/90 mm Hg. A total of 863 patients were evaluated (baseline mean clinic BP, morning and 24-h ambulatory BP: 155±15/93±10 mm Hg, 137±15/83±11 mm Hg, 133±14/79±10 mm Hg, respectively; 68% were previously treated at baseline). Telmisartan with/without HCTZ significantly reduced the mean morning ambulatory BP (-8.2/-4.9 mm Hg), daytime ambulatory BP (-8.0/-4.7 …

Malemedicine.medical_specialtyAmbulatory blood pressureAdolescentPhysiologyAngiotensin-Converting Enzyme InhibitorsBlood PressurePharmacologyBenzoatesYoung AdultHydrochlorothiazideReference ValuesAmbulatory CareInternal MedicinemedicineHumansTelmisartanambulatory blood pressure monitoring hidro hydrochlorothiazide morning blood pressure control telmisartanAntihypertensive AgentsAgedMorningAged 80 and overbusiness.industryMED/11 - MALATTIE DELL'APPARATO CARDIOVASCOLAREBlood Pressure Monitoring AmbulatoryMiddle AgedCircadian RhythmDrug CombinationsHydrochlorothiazideTreatment OutcomeHypertensionAmbulatoryEmergency medicineBenzimidazolesDrug Therapy CombinationFemaleTelmisartanCardiology and Cardiovascular Medicinebusinessmedicine.drugHypertension Research
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Cardiovascular risk factors, angiotensin-converting enzyme gene I/D polymorphism, and left ventricular mass in systemic hypertension.

1999

We investigated the influence of major cardiovascular risk factors (smoking, hypercholesterolemia, diabetes mellitus) on the association between angiotensin-converting enzyme (ACE) gene insertion (I)/deletion (D) polymorphism and echocardiographic left ventricular mass in 225 patients with sustained hypertension, assessed by ambulatory blood pressure monitoring. When the study population was analyzed as a whole, the 3 ACE genotypes did not differ in left ventricular mass (II, 47 g/m2.7; ID, 49 g/m2.7; DD, 51 g/m2.7; p = NS). No difference was found in subjects (n = 135) in whom at least 1 major cardiovascular risk factor was present (II, 51 g/m2.7; ID, 51 g/m2.7; DD: 52 g/m2.7; p = NS). In …

Malemedicine.medical_specialtyAmbulatory blood pressureGenotypeHeart VentriclesPeptidyl-Dipeptidase ALeft ventricular hypertrophyPolymerase Chain ReactionMuscle hypertrophyRisk FactorsInternal medicineDiabetes mellitusmedicineHumansRisk factorAllelesRetrospective StudiesPolymorphism Geneticbiologybusiness.industryAngiotensin-converting enzymeOdds ratioDNABlood Pressure Monitoring AmbulatoryMiddle Agedmedicine.diseasePrognosisEchocardiography DopplerCardiovascular DiseasesHypertensionCardiologybiology.proteinPopulation studyFemaleHypertrophy Left VentricularCardiology and Cardiovascular MedicinebusinessGene DeletionFollow-Up Studies
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Antihypertensive Mechanism of Lactoferrin-Derived Peptides: Angiotensin Receptor Blocking Effect

2013

Texto del artículo, no incluye figuras ni tablas.

Malemedicine.medical_specialtyAngiotensin receptorPeptideLactoferrin-derived peptidesReceptor Angiotensin Type 1Angiotensin Receptor AntagonistsInternal medicineRenin–angiotensin systemmedicineAnimalsHumansAngiotensin receptor blockerRenin−angiotensin systemReceptorAntihypertensive Agentschemistry.chemical_classificationAngiotensin II receptor type 1Antihypertensive mechanismbiologyLactoferrinAngiotensin IIBovine lactoferrin hydrolysateGeneral ChemistryAngiotensin IIMolecular WeightLactoferrinEndocrinologychemistryVasoconstrictionAngiotensin-converting enzyme inhibitorHypertensionbiology.proteinRabbitsPeptidesGeneral Agricultural and Biological SciencesEx vivo
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Effects of canrenoate plus angiotensin-converting enzyme inhibitors versus angiotensin-converting enzyme inhibitors alone on systolic and diastolic f…

2004

Background Aldosterone (ALDO) exerts profibrotic effects, acting via the mineralocorticoid receptors in cardiovascular tissues. Aldosterone antagonism in combination with angiotensin-converting enzyme inhibition may better protect against the untoward effects of ALDO than angiotensin-converting enzyme inhibition alone. Methods In a double-blind randomized study, the tolerability and efficacy of canrenoate (25 mg/d) plus captopril versus captopril alone were evaluated in 510 patients with an acute anterior myocardial infarction (MI), a serum creatinine concentration 5.5 mEq/L and creatinine levels to >2.0 mg/L after 10 days of treatment were observed. At 180 days, the mitral E-wave–A-wave ra…

Malemedicine.medical_specialtyAngiotensin-Converting Enzyme Inhibitors Mineralocorticoid Receptor Antagonists/administration & dosage Myocardial Contraction/drug effects Myocardial Infarction/drug therapy Myocardial Infarction/physiopathologymedicine.drug_classMyocardial InfarctionDiastoleAngiotensin-Converting Enzyme InhibitorsPlacebochemistry.chemical_compoundDouble-Blind MethodInternal medicinemedicineHumansMineralocorticoid Receptor AntagonistsCreatinineAldosteronebiologybusiness.industryCaptoprilAngiotensin-converting enzymeMiddle AgedMyocardial ContractionchemistryTolerabilityMineralocorticoidCardiologybiology.proteinDrug Therapy CombinationFemaleCanrenoic AcidCardiology and Cardiovascular MedicinebusinessFollow-Up Studiesmedicine.drugAmerican Heart Journal
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Captopril does not affect plasma endothelin-1 during thrombolysis and reperfusion.

1995

Studies showed that endothelin-1 (ET-1) was increased in the acute myocardial infarction (AMI). Experimental studies reported that captopril was able to reduce ET-1 secretion, and that ET-1 was increased during reperfusion. This study was aimed to verify if captopril was able to reduce plasma ET-1 during thrombolysis in AMI. Seventy-three patients, hospitalized for suspected AMI within 4 h from the onset of symptoms suitable for thrombolysis (1st episode), Killip class 1-2, were randomized (double blind) into two groups: group 1 (37 pts), 8 F/29 M, received captopril, 6.25 mg, orally 15 min before thrombolysis. Group 2: (36 pts) 8 F/28 M, received placebo before thrombolysis. All patients m…

Malemedicine.medical_specialtyCaptoprilTime Factorsmedicine.medical_treatmentMyocardial InfarctionAdministration OralAngiotensin-Converting Enzyme InhibitorsBlood PressureMyocardial ReperfusionPlaceboAnginaPlacebosElectrocardiographyDouble-Blind MethodHeart RateInternal medicineFibrinolysismedicineHumansThrombolytic TherapyMyocardial infarctionAngina UnstableCreatine KinaseKillip classbusiness.industryUnstable anginaEndothelinsCaptoprilThrombolysismedicine.diseaseRecombinant ProteinsSurgeryIsoenzymesTissue Plasminogen ActivatorCardiologyFemaleCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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The combination ace-inhibitors plus canreonate in patients with anterior myocardial infarction: safety and tolerability study.

2001

There is recent evidence that aldosterone (ALDO) exerts pro-fibrotic effects, acting via the mineral-corticoid receptors in cardiovascular tissues and partial aldosterone escape during ACE-inhibition treatment occurs.A double blind randomised study was performed to evaluate the feasibility, and tolerability of the administration of the 25 mg/day of canreonate plus captopril versus captopril alone in patients with anterior AMI unsuitable for thrombolysis and/or not receiving thrombolytic treatment, and unreperfused after thrombolysis. Fifty five patients hospitalised for anterior AMI,with a serum creatinine concentration2.0 mg/dl and a serum K concentration5.0 mmol per liter were randomised …

Malemedicine.medical_specialtyCaptoprilmedicine.medical_treatmentAldosterone escapeUrologyMyocardial InfarctionAngiotensin-Converting Enzyme Inhibitorschemistry.chemical_compoundDouble-Blind MethodmedicineHumanscardiovascular diseasesMyocardial infarctionAgedCreatinineE/A ratiobusiness.industryCaptoprilThrombolysisMiddle Agedmedicine.diseaseSurgerychemistryTolerabilityACE inhibitorFeasibility StudiesDrug Therapy CombinationFemaleCanrenoic AcidCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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Novel antihypertensive hexa- and heptapeptides with ACE-inhibiting properties: From the in vitro ACE assay to the spontaneously hypertensive rat

2011

Bioactive ACE inhibiting peptides are gaining interest in hypertension treatment. We have designed and screened six synthetic heptapeptides (PACEI48 to PACEI53) based on two hexapeptide leads (PACEI32 and PACEI34) to improve ACE inhibitory properties and assess their antihypertensive effects. ACE activity was assayed in vitro and ex vivo. Selected peptides were administered to spontaneously hypertensive rats (SHRs) and normotensive Wistar-Kyoto (WKY) rats. In vitro cytotoxicity was assessed with the MTT reduction test. The six heptapeptides at low micromolar concentration produced different degrees of in vitro inhibition of ACE activity using the synthetic substrate HHL or the natural subst…

Malemedicine.medical_specialtyCell SurvivalPhysiologyAdministration OralAngiotensin-Converting Enzyme InhibitorsBlood PressurePeptidyl-Dipeptidase APharmacologyRats Inbred WKYBiochemistryTissue Culture TechniquesMiceCellular and Molecular NeuroscienceEndocrinologySpontaneously hypertensive ratOral administrationRats Inbred SHRInternal medicineRenin–angiotensin systemmedicineAnimalsHumansInfusions IntravenousAntihypertensive AgentsbiologyChemistryAngiotensin-converting enzyme3T3 CellsHep G2 CellsIn vitroRatsCarotid ArteriesEndocrinologyVasoconstrictionHypertensionACE inhibitorbiology.proteinRabbitsAngiotensin Imedicine.symptomOligopeptidesVasoconstrictionEx vivomedicine.drug
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Importance of HDL cholesterol levels and the total/ HDL cholesterol ratio as a risk factor for coronary heart disease in molecularly defined heterozy…

2001

Aims To assess the relationship of the lipid profile to coronary heart disease in a group of heterozygous familial hypercholesterolaemic subjects with similar age, sex, body mass index, prevalence of angiotensin converting enzyme DD genotype and type of low density lipoprotein receptor mutation. Methods and Results A total of 66 molecularly defined heterozygous familial hypercholesterolaemic subjects, 33 of whom had coronary heart disease, were studied. Clinical features, cardiovascular risk factors and lipid parameters were compared in both groups. Familial hypercholesterolaemic patients with coronary heart disease showed significantly lower values of mean plasma HDL cholesterol and a high…

Malemedicine.medical_specialtyGenotypeCoronary DiseasePeptidyl-Dipeptidase AHyperlipoproteinemia Type IIchemistry.chemical_compoundRisk FactorsStatistical significanceInternal medicinemedicineHumansRisk factorReceptors Lipoproteinbiologymedicine.diagnostic_testCholesterolbusiness.industryCholesterol HDLCase-control studyAngiotensin-converting enzymeMiddle AgedEndocrinologyBlood pressurechemistrySpainCase-Control StudiesMutationCardiologybiology.proteinRegression AnalysisFemalelipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicinebusinessLipid profileBody mass indexEuropean Heart Journal
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