Search results for "Antioxidants."

showing 10 items of 824 documents

Oxidative stress in marathon runners: interest of antioxidant supplementation

2006

We have recently reported that xanthine oxidase is involved in the generation of free radicals in exhaustive exercise. Allopurinol, an inhibitor of xanthine oxidase, prevents it. The aim of the present work was to elucidate the role of exercise-derived reactive oxygen species in the cell signalling pathways involved in the adaptation to exercise in man. We have found that exercise causes an increase in the activity of plasma xanthine oxidase and an activation of NF-κB in peripheral blood lymphocytes after marathon running. This activation is dependent on free radical formation in exercise: treatment with allopurinol completely prevents it. In animal models, we previously showed that NF-κB a…

AdultMaleXanthine Oxidasemedicine.medical_specialtyAntioxidantAllopurinolmedicine.medical_treatmentPhysical ExertionMedicine (miscellaneous)AllopurinolPhysical exerciseLymphocyte Activationmedicine.disease_causeAntioxidantsRunningLipid peroxidationSuperoxide dismutasechemistry.chemical_compoundMalondialdehydeInternal medicinemedicineHumansLymphocytesMuscle SkeletalXanthine oxidasechemistry.chemical_classificationReactive oxygen speciesNutrition and DieteticsbiologyChemistryNF-kappa BMiddle AgedAdaptation PhysiologicalOxidative StressEndocrinologyMuscle Fatiguebiology.proteinLipid PeroxidationReactive Oxygen SpeciesOxidative stressSignal Transductionmedicine.drugBritish Journal of Nutrition
researchProduct

Increased oxidative stress levels and normal antioxidant enzyme activity in circulating mononuclear cells from patients of familial hypercholesterole…

2010

Familial hypercholesterolemia (FH) is a clinical condition with high risk for developing atherosclerosis. Increased oxidative stress (OS) and FH have been related to atherosclerosis, but no data are available on levels of OS and antioxidant enzyme activity in circulating mononuclear cells (CMCs) from FH patients. Circulating mononuclear cells are important mediators in atherosclerosis development, and chronically increased blood OS present in FH can induce modification in CMC activity. The objective of the study was to analyze the OS levels in CMCs from FH patients and controls. We have selected 30 nonrelated FH index patients and 30 normoglycemic and normocholesterolemic controls matched b…

AdultMaleXanthine Oxidasemedicine.medical_specialtyAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentFamilial hypercholesterolemiamedicine.disease_causeAntioxidantsHyperlipoproteinemia Type IISuperoxide dismutasechemistry.chemical_compoundEndocrinologyMalondialdehydeInternal medicinemedicineHumansXanthine oxidasechemistry.chemical_classificationGlutathione PeroxidaseGlutathione DisulfidebiologySuperoxide DismutaseGlutathione peroxidaseGlutathioneMiddle AgedAtherosclerosisCatalasemedicine.diseaseGlutathioneOxidative StressEndocrinologychemistryCatalaseLeukocytes Mononuclearbiology.proteinFemaleOxidation-ReductionOxidative stressMetabolism
researchProduct

Effects of the association of aging and obesity on lipids, lipoproteins and oxidative stress biomarkers: a comparison of older with young men.

2011

In this study, plasma lipids, lipoproteins and markers of oxidant/antioxidant status were investigated in young (n = 45) and older (n = 40) obese men and compared to those in young (n = 65) and older (n = 55) normal weight controls. The purpose was to determine whether obesity exacerbates or not lipid, lipoprotein abnormalities and oxidative stress in older men. Our findings showed that all obese patients had increased plasma triglyceride, cholesterol, LDL-cholesterol, -triglyceride and HDL-triglyceride levels concentrations compared to controls (P < 0.01). However, the younger obese men had relatively larger and accentuated changes in plasma lipids and lipoproteins than the older patients.…

AdultMalemedicine.medical_specialtyAgingLipid PeroxidesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentLipoproteinsMedicine (miscellaneous)medicine.disease_causeAntioxidantsBody Mass IndexProtein Carbonylationchemistry.chemical_compoundInternal medicineMedicineHumansObesityAgedchemistry.chemical_classificationNutrition and DieteticsTriglyceridebusiness.industryCholesterolSuperoxide DismutaseGlutathione peroxidaseMiddle Agedmedicine.diseaseObesityLipidsOxidative StressEndocrinologychemistrylipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicinebusinessBody mass indexOxidative stressBiomarkersLipoproteinNutrition, metabolism, and cardiovascular diseases : NMCD
researchProduct

Antenatal Steroids and Antioxidant Enzyme Activity in Preterm Infants: Influence of Gender and Timing

2009

Antenatal steroids have improved the survival of preterm infants; however, the mechanism of action is not fully understood. We aimed to establish an association between antenatal steroids and antioxidant activity and postnatal oxidative stress. In a prospective cohort study, extremely preterm neonates receiving antenatal steroids (CORT) or not (NOCORT) were enrolled. An association between antenatal steroids and activities of antioxidant enzymes and glutathione cycle enzymes in cord blood was found. In addition, reduced oxidative stress (GSH/GSSG ratio, CORT vs. NOCORT, 35.68 + or - 12.20 vs. 28.38 + or - 9.92; p < 0.01) and, decreased oxidation of proteins (ortho-tyrosine/phenylalanine rat…

AdultMalemedicine.medical_specialtyAntioxidantAdolescentPhysiologymedicine.medical_treatmentClinical BiochemistryBiologymedicine.disease_causeBiochemistryAntioxidantsYoung Adultchemistry.chemical_compoundSex FactorsAdrenal Cortex HormonesPregnancyInternal medicinemedicineHumansProspective StudiesProspective cohort studyMolecular BiologyGeneral Environmental SciencePregnancySuperoxide DismutaseInfant NewbornRetinopathy of prematurityCell BiologyGlutathioneCatalasemedicine.diseaseOxidative StressEndocrinologyBronchopulmonary dysplasiachemistryCord bloodGeneral Earth and Planetary SciencesFemaleInfant PrematureOxidative stressAntioxidants &amp; Redox Signaling
researchProduct

Enhanced oxidative susceptibility and reduced antioxidant content of metabolic precursors of small, dense low-density lipoproteins.

2001

Abstract PURPOSE: Elevated plasma concentrations of low-density lipoproteins (LDL) increase risk for coronary heart disease. However, lipoprotein profiles rich in small, dense LDL particles confer greater risk than those that mainly consist of large, buoyant LDL. This may be due, in part, to the greater oxidative susceptibility of small, dense LDL. In the current studies, we tested whether differences in the oxidative behavior of buoyant and dense LDL arise from differences in their immediate metabolic precursors, intermediate-density lipoproteins. SUBJECTS AND METHODS: We compared the properties of intermediate-density lipoproteins and buoyant and dense LDL subfractions in 9 subjects with …

AdultMalemedicine.medical_specialtyAntioxidantParinaric Acidmedicine.medical_treatmentOxidative phosphorylationAntioxidantsReference ValuesInternal medicineMedicineHumansPolyacrylamide gel electrophoresisFluorescent DyesIntermediate-density lipoproteinbusiness.industryGeneral MedicineLipoproteins LDLEndocrinologyPhenotypeFatty Acids Unsaturatedlipids (amino acids peptides and proteins)Electrophoresis Polyacrylamide GelFemaleUltracentrifugebusinessQuantitative analysis (chemistry)Oxidation-ReductionLipoproteinThe American journal of medicine
researchProduct

Nitric oxide metabolites and oxidative stress in mild essential hypertension

2010

Many papers have showed non univocal data about oxidative stress status and nitric oxide metabolites in essential hypertension. Considering this preamble we examined the total antioxidant status (TAS), the lipid peroxidation (LP), expressed as thiobarbituric acid-reactive substances (TBARS), the stable end products of nitric oxide (NOx) and LP/NOx ratio in 25 subjects with untreated mild essential hypertension. The obtained data show a significant increase in TBARS (p < 0.001) and NOx (p < 0.001) in hypertensives and no variation in TAS and in TBARS/NOx ratio. None of these parameters was statistically related to the metabolic parameters or to the blood pressure values. The high level of li…

AdultMalemedicine.medical_specialtyAntioxidantPhysiologymedicine.medical_treatmentBlood PressureEssential hypertensionmedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsNitric oxideEssential hypertension.Lipid peroxidationchemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineTBARSHumansNOxOxidate streNitric oxideHematologyMiddle Agedmedicine.diseaseOxidative StressBlood pressureEndocrinologychemistryBiochemistryHypertensionFemaleLipid PeroxidationCardiology and Cardiovascular MedicineOxidative stressClinical Hemorheology and Microcirculation
researchProduct

Factors related to the impact of antihypertensive treatment in antioxidant activities and oxidative stress by-products in human hypertension

2004

The objective was to study factors related to the changes induced by antihypertensive treatment on oxidative status, antioxidant activities, and reactive oxygen species by-products in whole blood and mononuclear peripheral cells. Eighty-nine hypertensive patients (mean age 46 years, 46 men, average 24-h blood pressure 139/88 mm Hg, body mass index 29) were included. After 3 months of nonrandomized allocation to antihypertensive treatment (20 nonpharmacologic, 36 beta-blockers, 33 angiotensin receptor blocker), oxidized/reduced glutathione ratio and malondialdehyde were significantly reduced, and the activity of superoxide dismutase, catalase, and glutathione peroxidase was significantly inc…

AdultMalemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentAngiotensin-Converting Enzyme Inhibitorsmedicine.disease_causeBenzoatesAntioxidantsSuperoxide dismutasechemistry.chemical_compoundInternal medicineInternal MedicinemedicineHumansTelmisartanAntihypertensive AgentsWhole bloodchemistry.chemical_classificationReactive oxygen speciesbiologybusiness.industryGlutathione peroxidaseGlutathioneMiddle AgedMalondialdehydeOxidative StressHydrochlorothiazideTreatment OutcomeEndocrinologyAtenololchemistryHypertensionbiology.proteinBenzimidazolesDrug Therapy CombinationFemalebusinessOxidative stressDNA DamageAmerican Journal of Hypertension
researchProduct

AZT treatment induces molecular and ultrastructural oxidative damage to muscle mitochondria. Prevention by antioxidant vitamins.

1998

AIDS patients who receive zidovudine (AZT) frequently suffer from myopathy. This has been attributed to mitochondrial (mt) damage, and specifically to the loss of mtDNA. This study examines whether AZT causes oxidative damage to DNA in patients and to skeletal muscle mitochondria in mice, and whether this damage may be prevented by supranutritional doses of antioxidant vitamins. Asymptomatic HIV-infected patients treated with AZT have a higher urinary excretion (355+/-100 pmol/kg/d) of 8-oxo-7, 8-dihydro-2'-deoxyguanosine (8-oxo-dG) (a marker of oxidative damage to DNA) than untreated controls (asymptomatic HIV-infected patients) (182+/-29 pmol/kg/d). This was prevented (110+/-79 pmol/kg/d)…

AdultMalemedicine.medical_specialtyDNA damageAnti-HIV Agentsmedicine.medical_treatmentAscorbic AcidBiologyDNA MitochondrialAntioxidantsZidovudinechemistry.chemical_compoundMiceInternal medicinemedicineDeoxyguanosineAnimalsHumansVitamin Eheterocyclic compoundsMyopathyVitamin ESkeletal musclevirus diseasesDeoxyguanosineGeneral MedicineGlutathioneHydrogen PeroxideAscorbic acidMitochondria Musclemedicine.anatomical_structureEndocrinologychemistryBiochemistry8-Hydroxy-2'-Deoxyguanosinemedicine.symptomZidovudinemedicine.drugDNA DamageResearch ArticleThe Journal of clinical investigation
researchProduct

Different patterns of in vivo pro-oxidant states in a set of cancer- or aging-related genetic diseases

2008

A comparative evaluation is reported of pro-oxidant states in 82 patients with ataxia telangectasia (AT), Bloom syndrome (BS), Down syndrome (DS), Fanconi anemia (FA), Werner syndrome (WS), and xeroderma pigmentosum (XP) vs 98 control donors. These disorders display cancer proneness, and/or early aging, and/or other clinical features. The measured analytes were: (a) leukocyte and urinary 8-hydroxy-2'-deoxyguanosine (8-OHdG), (b) blood glutathione (GSSG and GSH), (c) plasma glyoxal (Glx) and methylglyoxal (MGlx), and (d) some plasma antioxidants [uric acid (UA) and ascorbic acid (AA)]. Leukocyte 8-OHdG levels ranked as follows: WS>BS approximately FA approximately XP>DS approximately AT appr…

AdultMalemedicine.medical_specialtyDown syndromeXeroderma pigmentosumAdolescentmedicine.disease_causeBiochemistrychemistry.chemical_compoundAtaxia TelangiectasiaPhysiology (medical)Internal medicinemedicineHumansBloom syndromeChildAgedXeroderma PigmentosumMethylglyoxalDeoxyguanosineGlutathioneGlyoxalMiddle AgedAscorbic acidmedicine.diseasePyruvaldehydeGlutathioneEndocrinologyFanconi AnemiaantioxidantschemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineUric acidOxidative streFemaleWerner SyndromeDown SyndromeReactive Oxygen SpeciesOxidative stressBloom SyndromeDNA Damage
researchProduct

Altered glutathione system is associated with the presence of distal symmetric peripheral polyneuropathy in type 2 diabetic subjects.

2015

Abstract Distal symmetric peripheral polyneuropathy (DSPN) is a highly prevalent complication of diabetes. However, underlying pathophysiological mechanisms are multiple and not well understood. The aim of our study was to analyze the oxidative stress levels in circulating mononuclear cells by measuring the glutathione system, malondialdehyde and oxidized-LDL, in 60 type 2 diabetic patients from a well-characterized cohort of 196 type 2 diabetic patients. Using a nested case–control design, we studied 30 type 2 diabetic patients with distal symmetric polyneuropathy and 30 diabetic controls without this complication, according to the Neuropathy Disability Score. We have found that diabetic p…

AdultMalemedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.disease_causePeripheral blood mononuclear cellRisk AssessmentSeverity of Illness IndexAntioxidantschemistry.chemical_compoundEndocrinologyDiabetic NeuropathiesInternal medicineDiabetes mellitusMalondialdehydeInternal MedicineMedicineHumansAgedChi-Square Distributionbusiness.industryIncidenceType 2 Diabetes MellitusGlutathioneMiddle AgedMalondialdehydemedicine.diseasePrognosisGlutathionePathophysiologyOxidative StressEndocrinologychemistryDiabetes Mellitus Type 2Case-Control StudiesFemalebusinessComplicationOxidative stressJournal of diabetes and its complications
researchProduct