Search results for "Apamin"

showing 9 items of 39 documents

Dual effect mediated by protease-activated receptors on the mechanical activity of rat colon

2002

1. The present study examined the mechanical effects of agonist enzymes and receptor-activating peptides for protease-activated receptor (PAR)-1 and PAR-2 on longitudinal and circular muscle of rat isolated colonic segments in the attempt to clarify the PAR functional role in intestinal motility. 2. The responses to PAR-1 and PAR-2 activation were examined in vitro by recording simultaneously the changes of endoluminal pressure (index of circular muscle activity) and of isometric tension (index of longitudinal muscle activity). 3. Both PAR-1 agonists, thrombin (0.1 nM - 3 microM) and SFLLRN-NH2 (1 nM - 3 microM), and PAR-2 agonists, trypsin (0.1 nM - 10 microM) and SLIGRL-NH2 (1 nM - 10 mic…

PharmacologyAgonistmedicine.medical_specialtymedicine.drug_classMotilityBiologyApaminPotassium channelchemistry.chemical_compoundEndocrinologychemistryInternal medicineTetrodotoxinmedicineProtease-activated receptormedicine.symptomReceptorMuscle contractionBritish Journal of Pharmacology
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Arachidonic acid relaxes human pulmonary arteries through K+ channels and nitric oxide pathways.

2004

We aimed to investigate the role of K(+) channels and nitric oxide (NO) on the relaxant effects of arachidonic acid in the human intralobar pulmonary arteries. Arachidonic acid produced a concentration-dependent relaxation (E(max)=93+/-3% of maximal relaxation induced by papaverine 0.1 mM;-log EC(30)=7.03+/-0.09) that was antagonized by the cyclooxygenase inhibitor indomethacin (1 microM), by the combination of cyclooxygenase blockade and cytochrome P450 (CYP) blockade with 17-octadecynoic acid (17-ODYA, 10 microM), by the combination of cyclooxygenase inhibition and NO synthase (NOS) inhibition with N(omega)-nitro-l-arginine (l-NOARG, 100 microM), by the simultaneous inhibition of CYP and …

PharmacologyArachidonic AcidPotassium ChannelsCharybdotoxinDose-Response Relationship DrugStereochemistryPharmacologyIn Vitro TechniquesPulmonary ArteryApaminNitric OxidePotassium channelNitric oxideGlibenclamideVasodilationchemistry.chemical_compoundchemistrymedicineHumansArachidonic acidChannel blockerUnsaturated fatty acidmedicine.drugSignal TransductionEuropean journal of pharmacology
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Tonic inhibitory action by nitric oxide on spontaneous mechanical activity in rat proximal colon: involvement of cyclic GMP and apamin-sensitive K+ c…

1999

The cellular mechanisms by which endogenous nitric oxide (NO) modulates spontaneous motility were investigated in rat isolated proximal colon. The mechanical activity was detected as changes in intraluminal pressure. Apamin (1–100 nM) produced a concentration-dependent increase in the amplitude of the spontaneous pressure waves. The maximal contractile effect was of the same degree as that produced by Nω-nitro-L-arginine methyl ester (L-NAME) (100 μM) and the joint application of apamin plus L-NAME had no additive effects. Apamin (0.1 μM) reduced the inhibitory effects (i.e. reduction in the amplitude of the pressure waves) induced by sodium nitroprusside (SNP) (1 nM–10 μM) or 8-Br-cyclic G…

PharmacologyMembrane potentialmedicine.medical_specialtybiologyChemistryHyperpolarization (biology)Inhibitory postsynaptic potentialApaminPotassium channelNitric oxide synthasechemistry.chemical_compoundEndocrinologyInternal medicinebiology.proteinmedicinemedicine.symptomZaprinastMuscle contractionBritish Journal of Pharmacology
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NANC inhibitory neurotransmission in mouse isolated stomach: involvement of nitric oxide, ATP and vasoactive intestinal polypeptide

2003

1. The neurotransmitters involved in NANC relaxation and their possible interactions were investigated in mouse isolated stomach, recording the motor responses as changes of endoluminal pressure from whole organ. 2. Field stimulation produced tetrodotoxin-sensitive, frequency-dependent, biphasic responses: rapid transient relaxation followed by a delayed inhibitory component. 3. The inhibitor of the synthesis of nitric oxide (NO), l-NAME, abolished the rapid relaxation and significantly reduced the slow relaxation. Apamin, blocker of Ca2+-dependent K+ channels, or ADPbetaS, which desensitises P2y purinoceptors, reduced the slow relaxation to 2-8 Hz, without affecting that to 16-32 Hz or the…

Pharmacologymedicine.medical_specialtyRelaxation (psychology)Vasoactive intestinal peptideStimulationNeurotransmissionApaminchemistry.chemical_compoundMuscle relaxationEndocrinologychemistryInternal medicinemedicineBiophysicsSodium nitroprussideNeurotransmittermedicine.drugBritish Journal of Pharmacology
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Tachykinergic neurotransmission is enhanced in duodenum from dystrophic (mdx ) mice

2005

1 Duodenal longitudinal muscle of mdx mice, an animal model for Duchenne muscular dystrophy, showed a decrease in the electrically evoked nonadrenergic, noncholinergic (NANC) inhibitory responses associated with a reduction of the participation of nitric oxide (NO). In this study, we investigated whether the impairment of NO could also lead to alterations in the NANC excitatory transmission. 2 Nerve-evoked responses consisted of an inhibitory phase followed, at the end of stimulation, by an excitatory response characterised by an increase in amplitude of the spontaneous contractions. In mdx mice, the amplitude of the nerve-evoked contractions was significantly higher than in normals. 3 N(om…

Pharmacologymedicine.medical_specialtySubstance PStimulationBiologyNeurotransmissionApaminchemistry.chemical_compoundEndocrinologychemistryInternal medicineExcitatory postsynaptic potentialmedicineSodium nitroprussideNeurokinin ASoluble guanylyl cyclasemedicine.drugBritish Journal of Pharmacology
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Inhibitory influence of chromogranin A N-terminal fragment (vasostatin-1) on the spontaneous contractions of rat proximal colon

2005

Very little is known about the role played by CGA and its fragments in the gastrointestinal physiology. We have studied the role of CGA N-terminal fragments in the regulation of intestinal smooth muscle contractility by measuring the influence of recombinant CGA 1-78 (VS-1) and synthetic CGA 7-57 peptides on the spontaneous mechanical activity of rat proximal colon in vitro. The mechanical activity was recorded as changes in the intraluminal pressure. VS-1 (0.1-30 nM) and CGA 7-57 (10-300 nM) produced concentration-dependent inhibitory effects, characterized by a progressive decrease in the mean amplitude of circular muscle spontaneous contractions, without affecting the resting tone. The r…

Time FactorsPhysiologyClinical BiochemistrySettore BIO/09 - FisiologiaBiochemistrylaw.inventionchemistry.chemical_compoundEndocrinologylawEnzyme InhibitorsIntestinal smooth muscleOxadiazolesCGA-derived peptideVasostatin-1Chromogranin ASmooth muscle contractionRecombinant ProteinsNG-Nitroarginine Methyl EsterRecombinant DNATetrodotoxinMuscle Contractionendocrine systemmedicine.medical_specialtyColonTetrodotoxinBiologyInhibitory postsynaptic potentialApaminNitric oxideCellular and Molecular NeuroscienceQuinoxalinesInternal medicineChromograninsPressuremedicineAnimalsRats WistarDose-Response Relationship DrugMuscle SmoothNitric oxidePeptide FragmentsIn vitroProtein Structure TertiaryRatsGastrointestinal TractEndocrinologyApaminchemistrybiology.proteinChromogranin ACalreticulinPeptidesRegulatory Peptides
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Apigenin‐induced nitric oxide production involves calcium‐activated potassium channels and is responsible for antiangiogenic effects

2007

Summary. Background: The dietary flavonoid apigenin (Api) has been demonstrated to exert multiple beneficial effects upon the vascular endothelium. The aim of this study was to examine whether Ca2+-activated K+ channels (KCa) are involved in endothelial nitric oxide (NO) production and antiangiogenic effects.Methods: Endothelial NO generation was monitored using a cyclic guanosine monophosphate radioimmunoassay. KCa activity and changes of the intracellular Ca2+ concentration [Ca2+]i were analyzed using the fluorescent dyes bis-barbituric acid oxonol, potassium-binding benzofuran isophthalate, and fluo-3. The endothelial angiogenic parameters measured were cell proliferation, [3H]-thymidine…

Umbilical VeinsPotassium ChannelsTime FactorsRadioimmunoassayAngiogenesis InhibitorsNitric OxideApaminModels BiologicalNitric oxidechemistry.chemical_compoundCell MovementHumansApigeninPhosphorylationProtein kinase BCyclic guanosine monophosphateCells CulturedChemistryHematologyHyperpolarization (biology)IberiotoxinCalcium-activated potassium channelBiochemistryBiophysicsCalciumEndothelium VascularIntracellularSignal TransductionJournal of Thrombosis and Haemostasis
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Relaxation induced by N-terminal fragments of chromogranin A in mouse gastric preparations.

2007

Abstract A definitive role for chromogranin A (CGA)-derived fragments in the control of the gastrointestinal smooth muscle contractility has not been yet established. The purpose of the present study was to evaluate, in vitro , the effects of the recombinant vasostatin 1–78 (VS-1), CGA 7–57 and CGA 47–66 on the mouse gastric mechanical activity, recording the changes of intraluminal pressure. VS-1, CGA 7–57 and CGA 47–66 produced concentration-dependent relaxations. Mouse anti-vasostatin-1 monoclonal antibody 5A8, recognising the region 53–57, abolished the relaxation induced by VS-1, indicating the specificity of the effect. The relaxation was significantly reduced by tetrodotoxin (TTX), b…

endocrine systemmedicine.medical_specialtyPhysiologyMuscle RelaxationClinical BiochemistryBiologyIn Vitro TechniquesApaminInhibitory postsynaptic potentialBiochemistrySettore BIO/09 - FisiologiaNitric oxideContractilityGastric relaxationCellular and Molecular Neurosciencechemistry.chemical_compoundMiceEndocrinologyInternal medicinemedicineAnimalsGastrointestinal tractCGA-derived peptideDose-Response Relationship DrugStomachChromogranin ANitric oxideMuscle SmoothMolecular biologyIn vitroPeptide FragmentsRecombinant ProteinsMice Inbred C57BLEndocrinologychemistryTetrodotoxinbiology.proteinVasostatinChromogranin ACalreticulinRegulatory peptides
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Basal release of nitric oxide in the mesenteric artery in portal hypertension and cirrhosis: Role of dimethylarginine dimethylaminohydrolase

2013

Background and Aim Increased basal release of nitric oxide (NO) in the splanchnic circulation contributes to elevated plasma levels of NO observed in decompensated cirrhosis. We evaluated in rat mesenteric arteries whether the differences in basal release of NO, revealed by asymmetric dimethylarginine (ADMA)- and NG-nitro-L-arginine methyl ester (L-NAME)-induced contractions, were associated with changes in messenger RNA (mRNA) expression of endothelial NO synthase (eNOS) and dimethylarginine dimethylaminohydrolases (DDAHs). Methods Rat small mesenteric arteries from 14 Sham-control, from 14 with partial portal vein ligation (PPVL), and from 14 with bile duct excision (BDE)-induced cirrhosi…

medicine.medical_specialtyHepatologybiologybusiness.industryGastroenterologyVasodilationmedicine.diseasebiology.organism_classificationApaminNitric oxidechemistry.chemical_compoundEndocrinologymedicine.anatomical_structurechemistryEnosInternal medicinemedicinePortal hypertensionbusinessAsymmetric dimethylarginineMesenteric arteriesArteryJournal of Gastroenterology and Hepatology
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