Search results for "Arid"

showing 10 items of 1382 documents

Long-term effects of the trehalase inhibitor trehazolin on trehalase activity in locust flight muscle.

2010

SUMMARY Trehalase (EC 3.2.1.28) hydrolyzes the main haemolymph sugar of insects, trehalose, into the essential cellular substrate glucose. Trehalase in locust flight muscle is bound to membranes that appear in the microsomal fraction upon tissue fractionation, but the exact location in vivo has remained elusive. Trehalase has been proposed to be regulated by a novel type of activity control that is based on the reversible transformation of a latent (inactive) form into an overt (active) form. Most trehalase activity from saline-injected controls was membrane-bound (95%) and comprised an overt form (∼25%) and a latent form (75%). Latent trehalase could be assayed only after the integrity of …

MalePhysiologyTrehalase activityLocusta migratoriaAquatic ScienceDisaccharideschemistry.chemical_compoundIn vivoHemolymphExtracellularAnimalsTrehalaseTrehalaseEnzyme InhibitorsMuscle SkeletalMolecular BiologyEcology Evolution Behavior and SystematicsbiologyBehavior Animalbiology.organism_classificationTrehalosechemistryBiochemistryInsect ScienceFlight AnimalMicrosomeAnimal Science and ZoologyLocustThe Journal of experimental biology
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The C(-260)T gene polymorphism in the promoter of the CD14 monocyte receptor gene is not associated with acute myocardial infarction.

2003

CD surface molecules mediates cell activation and signaling. In particular, CD14 on blood monocytes mediate monocyte/macrophage activation by lipopolysaccharide. Lipopolysaccharide and its receptor, CD14, have been implicated in atherogenesis. It has been recently shown that a C(-260)T polymorphism in the promoter of the CD14 receptor may be a risk factor for coronary artery disease. Recently this association has been questioned because no increased risk was found with the T allele, even in the homozygous state. In the present study we investigated a possible association between the C(-260)T polymorphism in the CD14 promoter and acute myocardial infarction. Two hundred and thrteen patients …

MaleSettore MED/09 - Medicina InternaGenotypeCD14Clinical BiochemistryLipopolysaccharide ReceptorsMyocardial InfarctionAntigens CD14Polymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyCytosineGene FrequencyReference ValuesRisk FactorsGenotypemedicineHumansReference ValuePolymorphismAlleleReceptorPromoter Regions GeneticBiochemistry Genetics and Molecular Biology (all)business.industryRisk FactorMedicine (all)MonocyteSmokingCase-control studyGeneral MedicineMiddle AgedMolecular biologySurvival AnalysisGenotype frequencymedicine.anatomical_structureImmunologySettore MED/26 - NeurologiaFemaleSurvival AnalysiGene polymorphismCD14Cell activationbusinessThymineHumanClinical and experimental medicine
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Monocytes derived from humanized neonatal NOD/SCID/IL2Rγ(null) mice are phenotypically immature and exhibit functional impairments.

2012

Trials of immune-modulating drugs in septic patients have mostly failed to demonstrate clinical efficacy. Thus, we sought to generate a surrogate model of myelomonocytic lineage differentiation that would potentially allow sepsis induction and preclinical testing of anti-inflammatory drugs. Comparing transplantation of cord blood-derived stem cells in neonatal NOD/SCID/IL2Rγ(null) (neonatal huNSG) mice with transplantation of adult peripheral mobilized stem cells into adult NSG (adult huNSG) recipients, we demonstrate that myelomonocytic lineage differentiation in neonatal huNSG mice is retarded and monocytes are phenotypically immature with respect to HLA-DR expression and the emergence of…

MaleT-LymphocytesImmunologyPopulationLipopolysaccharide ReceptorsNodMice SCIDBiologyLymphocyte ActivationMonocytesImmunophenotypingMicePhagocytosisMice Inbred NODmedicineImmunology and AllergyAnimalsHumansCell LineageeducationCD86Mice Knockouteducation.field_of_studyMonocyteCell DifferentiationGeneral MedicineTransplantationmedicine.anatomical_structurePhenotypeImmunologyCytokinesCytokine secretionFemaleStem cellInflammation MediatorsCD80Interleukin Receptor Common gamma SubunitHuman immunology
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Impact of psychosocial factors on cardiovascular morbimortality: a prospective cohort study

2014

[Background] Whilst it is well known that psychosocial determinants may contribute to cardiovascular diseases (CVD), data from specific groups are scarce. The present study aims to determine the contribution of psychosocial determinants in increasing the risk of cardiovascular events (myocardial infarction and stroke), and death from CVD, in a high risk adult population.

MaleTime Factorsmodelos de riesgos proporcionalesEpidemiologyhumanosdepresiónMyocardial Infarction1471-2261Social supportdisparidades en el estado de saludClinical trialsRisk Factorsestudios prospectivosCause of Deathevaluación de riesgosMedicineLongitudinal StudiesProspective StudiesProspective cohort studyStrokemediana edadHealth inequalitiesCause of deathAged 80 and overeducation.field_of_studyancianoDepressionFactors de risc en les malaltiesHazard ratioMiddle AgedPrognosisStrokepronósticoSocioeconomic positionCardiovascular diseasesEstudi de casosEducational StatusFemaleRisk assessmentCardiology and Cardiovascular MedicinePsychosocialResearch Articlemedicine.medical_specialtyRisk factors in diseasesPopulationAcute myocardial infarctionRisk AssessmentEducational levelfactores de tiempocausas de muerteInternal medicinefactores de riesgoHumansanálisis multifactorialaccidente cerebrovascularcardiovascular diseaseseducationEpidemiologiainfarto de miocardioAgedProportional Hazards Modelsbusiness.industryProportional hazards modelMalalties cardiovascularsHealth Status Disparitiesmedicine.diseaseapoyo socialMorbiditatSpainMultivariate AnalysisPhysical therapyHousingCardiovascular deathestudios longitudinalesCase studiesMorbiditybusinessAssaigs clínics
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Cancer mortality inequalities in urban areas: a Bayesian small area analysis in Spanish cities

2011

incluye "Erratum to: Cancer mortality inequalities in urban areas: a Bayesian small area analysis in Spanish cities" BACKGROUND: Intra-urban inequalities in mortality have been infrequently analysed in European contexts. The aim of the present study was to analyse patterns of cancer mortality and their relationship with socioeconomic deprivation in small areas in 11 Spanish cities. METHODS: It is a cross-sectional ecological design using mortality data (years 1996-2003). Units of analysis were the census tracts. A deprivation index was calculated for each census tract. In order to control the variability in estimating the risk of dying we used Bayesian models. We present the RR of the censu…

MaleUrban PopulationEstudios transversalesCross-sectional studyEspaña:Health Care::Environment and Public Health::Public Health::Epidemiologic Methods::Epidemiologic Study Characteristics as Topic::Epidemiologic Studies::Cross-Sectional Studies [Medical Subject Headings]Business Management and Accounting(all)Disparidades en el estado de saludPoblación urbanaHealth informatics:Health Care::Population Characteristics::Population::Urban Population [Medical Subject Headings]NeoplasmsHuman geographyEpidemiologyCàncerUrban areasSocioeconomicsSmall-Area Analysismedia_common:Geographicals::Geographic Locations::Europe::Spain [Medical Subject Headings]Geography:Diseases::Neoplasms [Medical Subject Headings]CensusNeoplasiasGeography:Health Care::Environment and Public Health::Public Health::Epidemiologic Methods::Statistics as Topic::Probability::Bayes Theorem [Medical Subject Headings]lcsh:R858-859.7EnfermeríaFemaleRisk assessmentComputer Science(all)Riskmedicine.medical_specialtyGeneral Computer ScienceInequalitymedia_common.quotation_subjectHealth geographyeducationBayesian probabilityMedi ambientCancer mortalitylcsh:Computer applications to medicine. Medical informaticsRisk AssessmentCàncer -- MortalitatCiutatsMortalitatmedicineConfidence IntervalsTeorema de BayesHumansCancer -- MortalitySocioeconomic statusPovertyPovertybusiness.industryPublic healthResearchPublic Health Environmental and Occupational HealthCorrection:Health Care::Environment and Public Health::Public Health::Epidemiologic Measurements::Demography::Health Status::Health Status Disparities [Medical Subject Headings]Bayes TheoremHealth Status DisparitiesGeneral Business Management and AccountingSocioeconomic deprivationBayesian statistical decisionCross-Sectional StudiesEstadística bayesianaSocioeconomic FactorsSpainInequalitiesbusinessDemographyInternational Journal of Health Geographics
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Deletion of the Hunter gene and both DXS466 and DXS304 in a patient with mucopolysaccharidosis type II.

1992

Hunter syndrome is an X-linked mucopoly-saccharidosis due to deficiency of the lysosomal enzyme iduronate-2-sulfatase (IDS). A cDNA clone containing the entire coding region of the human IDS gene, mapped in Xq28, has been used as molecular probe to study a patient with Hunter syndrome. A submicroscopic deletion has been detected that spans the IDS gene as well as DXS466 and DXS304, 2 loci mapped probably not more than 900 kb from the IDS locus. A detailed clinical description of the patient is provided and his phenotype is compared to that of other patients with IDS deletion described recently. By following the segregation of a restriction fragment length polymorphism at the IDS locus in th…

MaleX ChromosomeLocus (genetics)Iduronate SulfataseBiologyGene mappingmedicineHumansMucopolysaccharidosis type IIChildGenetics (clinical)X chromosomeMucopolysaccharidosis IIGeneticsIduronate-2-sulfataseChromosome MappingHunter syndromeDNAmedicine.diseaseXq28PedigreeBlotting SouthernFemaleRestriction fragment length polymorphismChromosome DeletionPolymorphism Restriction Fragment LengthAmerican journal of medical genetics
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Gene diagnosis and carrier detection in Hunter syndrome by the iduronate-2-sulphatase cDNA probe.

1992

Hunter disease (McKusick 309900) is an X-chromosomal mucopolysaccharidosis due to deficiency of the lysosomal enzyme iduronate-2-sulphatase (IDS; EC 3.1.6.13). Diagnosis is based on both the typical clinical features of patients and the lack/reduction of IDS activity. Female carriers show no symptoms of the disease. In the past, several different assays were elaborated for measuring enzyme activity in carriers but none of them proved to be suitable for detecting heterozygotes reliably (Zlotogora and Bach 1984)

MaleX ChromosomeMucopolysaccharidosisIduronate SulfataseBiologyGene mappingComplementary DNAGenotypeGeneticsmedicineHumansAlleleChildDeoxyribonucleases Type II Site-SpecificGenetics (clinical)Mucopolysaccharidosis IIGeneticsGenetic Carrier ScreeningHunter syndromeHeterozygote advantagemedicine.diseaseMolecular biologyEnzyme assayPedigreeBlotting Southernbiology.proteinDNA ProbesPolymorphism Restriction Fragment LengthJournal of inherited metabolic disease
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Molecular analysis in patients with mucopolysaccharidosis type II suggests that DXS466 maps within the Hunter gene

1993

Hunter disease is an X-linked mucopolysaccharidosis caused by deficiency of the lysosomal enzyme iduronate-2-sulfatase (IDS). Using the IDS cDNA and DNA probes corresponding to loci flanking the IDS locus, we performed molecular genetic studies in two patients with Hunter syndrome. An interstitial deletion spanning the middle part of the IDS gene was found in the first patient. The second patient carries a gross gene rearrangement that can be detected after HindIII or EcoRI digestion of genomic DNA, and is similar to that found recently in seven unrelated Hunter patients. Our data suggest that the structural aberration observed is a partial intragenic inversion. As the same altered hybridiz…

MaleX ChromosomeRestriction MappingLocus (genetics)Iduronate SulfataseHindIIIDeoxyribonuclease EcoRIGeneticsmedicineHumansMucopolysaccharidosis type IIChildDeoxyribonucleases Type II Site-SpecificGenetics (clinical)Mucopolysaccharidosis IIGeneticsbiologyHybridization probeHunter syndromeGene rearrangementmedicine.diseaseMolecular biologyBlotting Southerngenomic DNAChild Preschoolbiology.proteinRestriction fragment length polymorphismDNA ProbesPolymorphism Restriction Fragment LengthHuman Genetics
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Growth and emigration of third-stage larvae of Hysterothylacium aduncum (Nematoda: Anisakidae) in larval herring Clupea harengus

2000

The growth and emigration of Hystherothylacium aduncum in laboratory-reared herring larvae Clupea harengus was studied. Experimental infections of 36-day-old herring larvae resulted in 126 hosts infected with 306 H. aduncum larvae. Regression analyses showed a significant worm emigration from the rectum to the head of the fish, accompanied by an increase in worm body length. The emigration was independent of worm intensity, which suggests an ontogenetic process. Some worms departed from this pattern by moving posteriorly or by penetrating into the muscle, and in 5 cases, the larvae were observed to leave living fish. This individual variation has not been observed in previous studies and mi…

MaleZoologyGrowth:CIENCIAS DE LA VIDA [UNESCO]EmigrationFish DiseasesHerringHysterothylacium aduncum larvaeAscaridoideaparasitic diseasesUNESCO::CIENCIAS DE LA VIDAAnimalsHelminthsEcology Evolution Behavior and SystematicsLarvabiologyEcologyHost (biology)fungiFishes:CIENCIAS DE LA VIDA::Biología animal (Zoología) ::Parasitología animal [UNESCO]Aquatic animalClupeabiology.organism_classificationAscaridida InfectionsUNESCO::CIENCIAS DE LA VIDA::Biología animal (Zoología) ::Parasitología animalAnisakidaeClupeidaeLarvaFemaleParasitologyLarval herring clupea harengusGrowth ; Emigration ; Hysterothylacium aduncum larvae ; Larval herring clupea harengus
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Behavioural and physiological effects of the trophically transmitted cestode parasite, Cyathocephalus truncatus, on its intermediate host, Gammarus p…

2007

SUMMARYSome parasites with complex life-cycles are able to manipulate the behaviour of their intermediate hosts in a way that increases their transmission to the next host. Gammarids infected by the tapeworm Cyathocephalus truncatus (Cestoda: Spathebothriidea) are known to be more predated by fish than uninfected ones, but potential behavioural manipulation by the parasite has never been investigated. In this study, we tested the hypothesis that C. truncatus is able to manipulate the behaviour of one of its intermediate hosts, Gammarus pulex (Crustacea: Amphipoda). To assess if any behavioural change was linked to other phenotypic alterations, we also measured the immunity of infected and u…

Male[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyAmphipodaCestodaZoologyBiologyHost-Parasite InteractionsPredationOxygen Consumption[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisPhototaxisAnimalsParasite hostingAmphipodacestodeSwimmingEnzyme Precursors[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyBehavior AnimalMonophenol MonooxygenaseHost (biology)EcologyIntermediate hostbiology.organism_classificationSurvival AnalysisimmunityGammarus pulexInfectious DiseasesCestodaAnimal Science and ZoologyParasitologypathogenic effectsCatechol OxidaseGammaridaebehavioural manipulationParasitology
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