Search results for "Array"

showing 10 items of 1264 documents

Gene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Hea…

2015

CUG-BP, Elav-like family (CELF) proteins regulate cell type- and developmental stage-specific alternative splicing in the heart. Repression of CELF-mediated splicing activity via expression of a nuclear dominant negative CELF protein in heart muscle was previously shown to induce dysregulation of alternative splicing, cardiac dysfunction, cardiac hypertrophy, and dilated cardiomyopathy in MHC-CELFΔ transgenic mice. A “mild” line of MHC-CELFΔ mice that expresses a lower level of the dominant negative protein exhibits cardiac dysfunction and myopathy at a young age, but spontaneously recovers normal cardiac function and heart size with age despite the persistence of splicing defects. To the b…

CCAAT-Enhancer-Binding Protein-deltaMaleSerum Response FactorTranscription GeneticCardiomyopathylcsh:MedicineMice Transgenic030204 cardiovascular system & hematologyBiology03 medical and health sciencesMice0302 clinical medicineGene expressionSerum response factormedicineAnimalsHumansMyocytes Cardiaclcsh:Science030304 developmental biologyOligonucleotide Array Sequence AnalysisRegulation of gene expressionHemizygote0303 health sciencesMultidisciplinaryGene Expression ProfilingMyocardiumAlternative splicinglcsh:RGene targetingHeartmedicine.diseaseMolecular biologyCell biologyGene expression profilingAlternative SplicingGene Expression RegulationRNA splicinglcsh:QCalciumFemaleCardiomyopathiesResearch ArticlePLoS ONE
researchProduct

7C: Computational Chromosome Conformation Capture by Correlation of ChIP-seq at CTCF motifs.

2019

Abstract Background Knowledge of the three-dimensional structure of the genome is necessary to understand how gene expression is regulated. Recent experimental techniques such as Hi-C or ChIA-PET measure long-range chromatin interactions genome-wide but are experimentally elaborate, have limited resolution and such data is only available for a limited number of cell types and tissues. Results While ChIP-seq was not designed to detect chromatin interactions, the formaldehyde treatment in the ChIP-seq protocol cross-links proteins with each other and with DNA. Consequently, also regions that are not directly bound by the targeted TF but interact with the binding site via chromatin looping are…

CCCTC-Binding Factorlcsh:QH426-470Protein Conformationlcsh:Biotechnologygenetic processesComputational biologyBiologyGenomeChromosomesBioconductorChromosome conformation capture03 medical and health sciences0302 clinical medicine6CHi-Clcsh:TP248.13-248.65GeneticsTranscription factorsHumansnatural sciencesNucleotide Motifs4CChIA-PET030304 developmental biologyChromatin loops0303 health sciencesThree-dimensional genome architectureChromatinChromatinChIP-seq7Clcsh:Genetics5CCTCFChromatin Immunoprecipitation SequencingHuman genomeDNA microarrayChIA-PET3CPrediction030217 neurology & neurosurgeryChromatin interactionsBiotechnologyHeLa CellsResearch ArticleBMC genomics
researchProduct

Meox2/Tcf15 Heterodimers Program the Heart Capillary Endothelium for Cardiac Fatty Acid Uptake

2015

Background— Microvascular endothelium in different organs is specialized to fulfill the particular needs of parenchymal cells. However, specific information about heart capillary endothelial cells (ECs) is lacking. Methods and Results— Using microarray profiling on freshly isolated ECs from heart, brain, and liver, we revealed a genetic signature for microvascular heart ECs and identified Meox2/Tcf15 heterodimers as novel transcriptional determinants. This signature was largely shared with skeletal muscle and adipose tissue endothelium and was enriched in genes encoding fatty acid (FA) transport–related proteins. Using gain- and loss-of-function approaches, we showed that Meox2/Tcf15 media…

CD36 AntigensHeterozygoteEndotheliumCD36Cardiac Output LowAdipose tissueLipoproteins VLDLBiologyFatty Acid-Binding ProteinsMicePhysiology (medical)Protein Interaction MappingBasic Helix-Loop-Helix Transcription FactorsmedicineAnimalsHumansRNA Small InterferingTranscription factorCells CulturedHomeodomain Proteinschemistry.chemical_classificationLipoprotein lipaseMyocardiumFatty AcidsEndothelial CellsFatty acidSkeletal muscleMetabolismCoronary VesselsCell biologyMice Inbred C57BLLipoprotein LipaseGlucosemedicine.anatomical_structureAdipose TissuechemistryBiochemistryTissue Array Analysisbiology.proteinTranscriptomeCardiology and Cardiovascular MedicineCirculation
researchProduct

Enhanced production of CCL18 by tolerogenic dendritic cells is associated with inhibition of allergic airway reactivity

2012

Background IL-10–treated dendritic cells (DCs) have been shown to inhibit T-cell responses through induction of anergy and regulatory T cells in various model systems, including allergic inflammation, but the factors being involved in this inhibition are still unclear. Objective This study set out to analyze such factors produced or induced by IL-10–treated DCs by using gene expression profiling and to explore their function. Methods CD4 + T cells from allergic donors were stimulated with autologous monocyte-derived allergen-pulsed mature DCs or IL-10–treated DCs. After 24 hours, the transcriptional profile was analyzed by using Affymetrix technology. Results were validated by using quantit…

CD4-Positive T-LymphocytesChemokinemedicine.medical_treatmentImmunologyT-Lymphocytes RegulatoryAllergic inflammationMiceMice Inbred NODImmune ToleranceRespiratory HypersensitivitymedicineAnimalsHumansImmunology and AllergyCCL17dendritic cellsCells CulturedT(H)1/T(H)2 cellsMice KnockoutbiologyCCL18FOXP3regulationDendritic cellMicroarray AnalysisallergyCoculture TechniquesInterleukin-10Disease Models Animalhumanized miceCytokineChemokines CCImmunologyHumanized mousebiology.proteinChemokinesTranscriptomeJournal of Allergy and Clinical Immunology
researchProduct

The receptor NLRP3 is a transcriptional regulator of TH2 differentiation.

2015

The receptor NLRP3 is involved in the formation of the NLRP3 inflammasome that activates caspase-1 and mediates the release of interleukin 1β (IL-1β) and IL-18. Whether NLRP3 can shape immunological function independently of inflammasomes is unclear. We found that NLRP3 expression in CD4(+) T cells specifically supported a T helper type 2 (TH2) transcriptional program in a cell-intrinsic manner. NLRP3, but not the inflammasome adaptor ASC or caspase-1, positively regulated a TH2 program. In TH2 cells, NLRP3 bound the Il4 promoter and transactivated it in conjunction with the transcription factor IRF4. Nlrp3-deficient TH2 cells supported melanoma tumor growth in an IL-4-dependent manner and …

CD4-Positive T-LymphocytesInflammasomesImmunologyBlotting WesternBiologyInterleukin 21MiceTh2 CellsCell Line TumorNLR Family Pyrin Domain-Containing 3 ProteinImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorPromoter Regions GeneticInterleukin 3Oligonucleotide Array Sequence AnalysisMice KnockoutCD40integumentary systemReverse Transcriptase Polymerase Chain ReactionZAP70Gene Expression ProfilingCell DifferentiationNeoplasms ExperimentalAsthmaCell biologyGene Expression Regulation NeoplasticMice Inbred C57BLInterleukin 10Interferon Regulatory FactorsInterleukin 12biology.proteinNIH 3T3 CellsTrans-ActivatorsFemaleInterleukin-4Carrier ProteinsProtein BindingSignal TransductionNature immunology
researchProduct

miR-155 inhibition sensitizes CD4+ Th cells for TREG mediated suppression.

2009

BackgroundIn humans and mice naturally occurring CD4(+)CD25(+) regulatory T cells (nTregs) are a thymus-derived subset of T cells, crucial for the maintenance of peripheral tolerance by controlling not only potentially autoreactive T cells but virtually all cells of the adaptive and innate immune system. Recent work using Dicer-deficient mice irrevocably demonstrated the importance of miRNAs for nTreg cell-mediated tolerance.Principal findingsDNA-Microarray analyses of human as well as murine conventional CD4(+) Th cells and nTregs revealed a strong up-regulation of mature miR-155 (microRNA-155) upon activation in both populations. Studying miR-155 expression in FoxP3-deficient scurfy mice …

CD4-Positive T-LymphocytesScienceImmunology/ImmunomodulationBiologyModels BiologicalT-Lymphocytes RegulatoryImmune tolerancemiR-155MiceDownregulation and upregulationImmune ToleranceAnimalsHumansIL-2 receptorOligonucleotide Array Sequence AnalysisMultidisciplinaryInnate immune systemGenetics and Genomics/Functional GenomicsQInterleukin-2 Receptor alpha SubunitRPeripheral toleranceFOXP3Forkhead Transcription FactorsTransfectionImmunity InnateCell biologyUp-RegulationKineticsMicroRNAsImmunologyImmunology/Immune ResponseMedicineGenetics and Genomics/Genetics of the Immune SystemResearch ArticlePLoS ONE
researchProduct

Búsqueda e identificación de nuevas causas genéticas o epigenéticas de trastornos del neurodesarrollo

2015

Los trastornos del neurodesarrollo engloban desordenes cognitivos, de aprendizaje, comportamiento, comunicación y motores, que aparecen en edad temprana, como la discapacidad intelectual (DI) o los trastornos del espectro autista (TEA). Se trata de patologías complejas, debidas a alteraciones en el desarrollo del sistema nervioso central (SNC), que pueden presentan agregación familiar o asociarse a otras anomalías congénitas o rasgos dismórficos. Pese a que la causa de este tipo de trastornos en muchos casos es desconocida, existen claras evidencias sobre sus bases genéticas, con un gran número de síndromes descritos que cursan con DI asociados a diversas variantes en número de copias (CNVs…

CGH-arrayUNESCO::CIENCIAS DE LA VIDA::GenéticaTrastornos del espectro autista:CIENCIAS DE LA VIDA::Genética [UNESCO]Genética HumanaTrastornos del neurodesarrollometilación del ADNEpigenéticaGenética MolecularDiscapacidad intelectual
researchProduct

Aplicación de nuevas tecnologías para la detección de reordenamientos genómicos en pacientes con retraso del neurodesarrollo y anomalías congénitas

2014

El retraso del neurodesarrollo o discapacidad intelectual (DI) es una de las causas de minusvalías más frecuente en niños, afecta a cuatro de cada mil nacidos vivos en su forma más grave, aunque el retraso leve podría afectar a 1-3% de la población. La mayoría de los casos de DI son de origen desconocido. Las variantes de número de copias (CNVs) son una causa importante de discapacidad intelectual sindrómica. A lo largo de estos años se han desarrollado diferentes herramientas para el estudio genético de la discapacidad intelectual sindrómica, tales como la FISH (hibridación in situ fluorescente), el MLPA (multiplex ligation-dependent probe amplification) o la CGH-array (Hibridación genómic…

CGH-arraydiscapacidadUNESCO::CIENCIAS DE LA VIDA::Genética:CIENCIAS DE LA VIDA::Genética [UNESCO]FISHUNESCO::CIENCIAS MÉDICASgenética:CIENCIAS MÉDICAS [UNESCO]
researchProduct

Defense Responses in Two Ecotypes of Lotus japonicus against Non-Pathogenic Pseudomonas syringae

2013

Lotus japonicus is a model legume broadly used to study many important processes as nitrogen fixing nodule formation and adaptation to salt stress. However, no studies on the defense responses occurring in this species against invading microorganisms have been carried out at the present. Understanding how this model plant protects itself against pathogens will certainly help to develop more tolerant cultivars in economically important Lotus species as well as in other legumes. In order to uncover the most important defense mechanisms activated upon bacterial attack, we explored in this work the main responses occurring in the phenotypically contrasting ecotypes MG-20 and Gifu B-129 of L. ja…

CIENCIAS MÉDICAS Y DE LA SALUDSTRESSLotus japonicusLotusInmunologíaDefence mechanismslcsh:MedicinePseudomonas syringaePlant disease resistanceCiencias BiológicasSYRINGAE//purl.org/becyt/ford/1 [https]Gene Expression Regulation PlantTRANSCRIPTOMICBotanyPseudomonas syringaePlant defense against herbivoryArabidopsis thalianalcsh:Science//purl.org/becyt/ford/1.6 [https]Ciencias de las Plantas BotánicaDisease ResistanceOligonucleotide Array Sequence AnalysisPlant DiseasesEcotypeMultidisciplinarybiologyEcotypeLOTUSGene Expression Profilinglcsh:Rfungifood and beverages//purl.org/becyt/ford/3.1 [https]biology.organism_classificationBIOTICMedicina BásicaJAPONICUSLotuslcsh:Q//purl.org/becyt/ford/3 [https]PSEUDOMONASCIENCIAS NATURALES Y EXACTASResearch ArticlePLoS ONE
researchProduct

Design and evaluation of a 32-channel phased-array coil for lung imaging with hyperpolarized 3-helium

2010

Imaging with hyperpolarized 3-helium is becoming an increasingly important technique for MRI diagnostics of the lung but is hampered by long breath holds (>20 sec), which are not always applicable in patients with severe lung disease like chronic obstructive pulmonary disease (COPD) or α-1-anti-trypsin deficiency. Additionally, oxygen-induced depolarization decay during the long breath holds complicates interpretation of functional data such as apparent diffusion coefficients. To address these issues, we describe and validate a 1.5-T, 32-channel array coil for accelerated 3He lung imaging and demonstrate its ability to speed up imaging 3He. A signal-to-noise ratio increase of up to a factor…

COPDLungChannel (digital image)Image qualitybusiness.industrymedicine.diseasePhased array coilmedicine.anatomical_structureNuclear magnetic resonanceLung imagingArray coilmedicineRadiology Nuclear Medicine and imagingbusinessImage resolutionMagnetic Resonance in Medicine
researchProduct