Search results for "Atrophy"

showing 10 items of 385 documents

Brain atrophy and lesion load in a large population of patients with multiple sclerosis

2005

Objective: To measure white matter (WM) and gray matter (GM) atrophy and lesion load in a large population of patients with multiple sclerosis (MS) using a fully automated, operator-independent, multiparametric segmentation method. Methods: The study population consisted of 597 patients with MS and 104 control subjects. The MRI parameters were abnormal WM fraction (AWM-f), global WM-f (gWM-f), and GM fraction (GM-f). Results: Significant differences between patients with MS and control subjects included higher AWM-f and reduced gWM-f and GM-f. MRI data showed significant differences between patients with relapsing-remitting and secondary progressive forms of MS. Significant correlations bet…

AdultMalePathologymedicine.medical_specialtyAdolescentBrain mappingNerve Fibers MyelinatedCentral nervous system diseaseWhite matterMultiple sclerosisAtrophySex FactorsPredictive Value of TestsNeural PathwaysmedicineHumansAge of OnsetMultiple Sclerosis/physiopathologyAgedCross-Sectional StudieBrain MappingExpanded Disability Status Scalemedicine.diagnostic_testBrain/physiopathologybusiness.industryMultiple sclerosisBrainMagnetic resonance imagingInterferon-betaMiddle Agedmedicine.diseasePrognosislesion loadMagnetic Resonance ImagingMultiple Sclerosis/diagnosimedicine.anatomical_structureCross-Sectional Studiesmultiple sclerosiLinear ModelsDisease ProgressionEducational StatusFemaleNeurology (clinical)Age of onsetAtrophybusinessMultiple Sclerosis/complicationbrain atrophyMRI
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Brain atrophy evolution and lesion load accrual in multiple sclerosis: a 2-year follow-up study

2009

Background To investigate in a large cohort of patients with multiple sclerosis (MS), lesion load and atrophy evolution, and the relationship between clinical and magnetic resonance imaging (MRI) correlates of disease progression. Methods Two hundred and sixty-seven patients with MS were studied at baseline and two years later using the same MRI protocol. Abnormal white matter fraction, normal appearing white matter fraction, global white matter fraction, gray matter fraction and whole brain fraction, T2-hyperintense, and T1-hypointense lesions were measured at both time points. Results The majority of patients were clinically stable, whereas MRI-derived brain tissue fractions were signifi…

AdultMalePathologymedicine.medical_specialtyAdolescentCentral nervous systemmultiple sclerosisSeverity of Illness IndexLesion loadWhite matterCentral nervous system diseaseYoung AdultDegenerative diseaseAtrophyMultiple Sclerosis Relapsing-RemittingatrophyRisk FactorsT2 lesionsmedicinefollow upHumansAgedmedicine.diagnostic_testbusiness.industryMultiple sclerosisBrain AtrophyBrainMagnetic resonance imagingMiddle AgedMultiple Sclerosis Chronic Progressivelesion loadmedicine.diseaseMagnetic Resonance Imagingmedicine.anatomical_structureCross-Sectional StudiesLogistic ModelsNeurologymultiple sclerosiMultivariate AnalysisDisease ProgressionFemaleSettore MED/26 - NeurologiaNeurology (clinical)businessFollow-Up StudiesMRI
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Detection of gastric atrophy by circulating pepsinogens: A comparison of three assays.

2017

Background Circulating levels of pepsinogens have been used in high gastric cancer-risk Asian and European populations to triage endoscopic evaluation for more severe pathology. There are different analytic methods with uncertain correlations. We therefore compared diagnostic performance of three commonly used pepsinogen assays to detect histologically confirmed gastric atrophy. Methods We tested plasma samples from adult patients with (n=50) and without (n=755) moderate or severe gastric corpus atrophy, as determined histologically by consensus of three expert pathologists. A single laboratory measured pepsinogens I (PgI) and II (PgII) using commercially available assays: two ELISA assays …

AdultMalePathologymedicine.medical_specialtyAdolescentStomach DiseasesEnzyme-Linked Immunosorbent AssayGastroenterologyArticle03 medical and health sciencesYoung Adult0302 clinical medicineAtrophyPepsinInternal medicineMedicineHumansAgedAged 80 and overAdult patientsPlasma samplesReceiver operating characteristicbiologyPepsinogensbusiness.industryDiagnostic Tests RoutineHistocytochemistryGastric AtrophyStomachGastroenterologyGeneral MedicineMiddle Agedmedicine.diseasedigestive system diseasesInfectious Diseasesmedicine.anatomical_structureROC CurveGastric Mucosa030220 oncology & carcinogenesisbiology.protein030211 gastroenterology & hepatologyTest performanceFemaleAtrophybusinessLatex Fixation TestsHelicobacter
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DNA-fragmentation and apoptosis-related proteins of muscle cells in motor neuron disorders

2009

Apoptosis has been described as one of the mechanisms of muscle fiber loss in infantile spinal muscular atrophy. In order to investigate if muscle fiber-apoptosis plays a role in other denervating disorders as well, we studied DNA-fragmentation, a hallmark of apoptosis, by the TUNEL-method and, moreover, the expression patterns of apoptosis-related proteins in 2 patients suffering from ALS and in 6 patients with polyneuropathy. We identified DNA-cleavage in muscle fibers of all these patients. Furthermore, we found strong expression of bax and ICE promoting apoptosis in muscle fibers. However, also strong expression of the anti-apoptotic factor bcl-2 was found. Our findings indicate that de…

AdultMalePathologymedicine.medical_specialtyMuscle Fibers SkeletalApoptosisCell Cycle ProteinsDNA FragmentationBiologyProto-Oncogene ProteinsGene expressionmedicineHumansMyocytefas ReceptorMotor Neuron DiseaseAmyotrophic lateral sclerosisMuscle SkeletalActinAgedReceptors Leukocyte-AdhesionAmyotrophic Lateral SclerosisPeripheral Nervous System DiseasesGeneral MedicineMiddle AgedMotor neuronmedicine.diseaseCell biologyCysteine Endopeptidasesmedicine.anatomical_structureNeurologyApoptosisNerve DegenerationDNA fragmentationFemaleNeurology (clinical)AtrophyPolyneuropathyActa Neurologica Scandinavica
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Serum neurofilament light chain is a biomarker of acute and chronic neuronal damage in early multiple sclerosis.

2018

Background: Monitoring neuronal injury remains one key challenge in early relapsing-remitting multiple sclerosis (RRMS) patients. Upon axonal damage, neurofilament – a major component of the neuro-axonal cytoskeleton – is released into the cerebrospinal fluid (CSF) and subsequently peripheral blood. Objective: To investigate the relevance of serum neurofilament light chain (sNfL) for acute and chronic axonal damage in early RRMS. Methods: sNfL levels were determined in 74 patients (63 therapy-naive) with recently diagnosed clinically isolated syndrome (CIS) or RRMS using Single Molecule Array technology. Standardized 3 T magnetic resonance imaging (MRI) was performed at baseline and 1–3 con…

AdultMalePathologymedicine.medical_specialtyNeurofilamentMultiple SclerosisNeurofilament lightIntermediate FilamentsSeverity of Illness IndexDisease activity03 medical and health sciencesYoung Adult0302 clinical medicineNeuronal damageNeurofilament ProteinsMedicineHumans030212 general & internal medicineNeuronsbusiness.industryMultiple sclerosisNeurodegenerationBrainMiddle Agedmedicine.diseaseNeurologyBiomarker (medicine)FemaleNeurology (clinical)Atrophybusiness030217 neurology & neurosurgeryClinical progressionBiomarkersMultiple sclerosis (Houndmills, Basingstoke, England)
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Cognitive impairment in multiple sclerosis can be predicted by imaging early in the disease.

2008

Background: Cognitive impairment is common in multiple sclerosis (MS) and adds significantly to the burden of the disease. The ability to predict future cognitive impairment from imaging obtained at disease onset has not been investigated. Methods: 62 patients imaged within 3 months of a clinically isolated syndrome were assessed neuropsychologically 7 years later. Baseline and periodic MRI measures of lesions, atrophy and normal-appearing white and grey matter were regressed against neuropsychological scores to explore the best predictors of cognitive outcome. Results: 28 patients had developed clinically definite MS at follow-up and a further nine met revised McDonald criteria for MS. Def…

AdultMalePathologymedicine.medical_specialtyPediatricsMagnetic Resonance SpectroscopyAdolescentGrey matterNeuropsychological TestsCerebral VentriclesWhite matterCohort StudiesDisability EvaluationAtrophyMultiple Sclerosis Relapsing-RemittingPredictive Value of TestsmedicineHumansProspective StudiesNeurologic ExaminationAspartic AcidClinically isolated syndromeMultiple sclerosisCognitive disorderNeuropsychologyBrainMcDonald criteriaDipeptidesMiddle Agedmedicine.diseaseCognitive impairment: multiple sclerosisMagnetic Resonance ImagingPsychiatry and Mental healthmedicine.anatomical_structureSurgeryFemaleNeurology (clinical)AtrophyPsychologyCognition DisordersInositolFollow-Up StudiesJournal of neurology, neurosurgery, and psychiatry
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IgA anti-actin antibodies ELISA in coeliac disease: A multicentre study.

2007

Previous studies have demonstrated that serum anti-actin antibodies are a reliable marker of intestinal damage severity in coeliac disease.To validate in a multicentre study the clinical usefulness of serum IgA anti-actin antibody ELISA and its possible use in monitoring intestinal mucosa lesions during gluten-free diet.Four centres recruited 205 newly diagnosed coeliac disease patients with villous atrophy, 80 healthy controls and 81 "disease" controls. Twelve coeliac disease patients on gluten-free diet but with persistent symptoms underwent serum IgA anti-actin antibody assay and intestinal histology evaluation. IgA anti-actin antibody ELISA was performed with a commercial kit. All coeli…

AdultMalePathologymedicine.medical_specialtySettore MED/09 - Medicina InternaAdolescentEnzyme-Linked Immunosorbent AssaySerum igaDiseaseCommercial kitSensitivity and SpecificityCoeliac diseaseIgA anti-actin antibodies; coeliac disease; multicentre studyIntestinal mucosaHumansMedicineIntestinal MucosaVillous atrophyChildAgedAutoantibodiesHepatologybiologybusiness.industryGastroenterologyInfantnutritional and metabolic diseasesMiddle AgedIgA anti-actin antibodiemedicine.diseaseActinsmulticentre studyImmunoglobulin ACeliac DiseaseIntestinal histologyChild Preschoolbiology.proteinFemaleAntibodybusinessBiomarkerscoeliac disease
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Unilateral absence of submandibular gland secondary to stones. Aplasia versus early atrophy.

2009

Major salivary gland absence is a rare disorder. The cause of congenital absence of the salivary glands has not been determined, but it may be associated with ectodermal defects of the first and second branchial arches. Isolated absence of a unilateral submandibular gland is an unusual entity with less than ten cases reported in the literature. The etiopathogenesis of isolated absence of a major salivary gland without other developmental anomalies is still unclear. The formation of a sialolith within the remaining Wharton?s duct, associated with isolated aplasia (versus atrophy) of a unilateral submandibular gland has been recently reported. We describe in this work two cases of sialolithia…

AdultMalePathologymedicine.medical_specialtySubmandibular Gland DiseasesSubmandibular GlandAtrophystomatognathic systemMajor Salivary GlandSubmandibular Gland DiseasesmedicineHumansIn patientGeneral DentistryAgedSalivary Gland CalculiSalivary Gland Calculusbusiness.industryAplasiaAnatomy:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseSubmandibular glandmedicine.anatomical_structureOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASSurgeryFemaleAtrophybusinessDuct (anatomy)Medicina oral, patologia oral y cirugia bucal
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Tissue hypoxia in complex regional pain syndrome.

2003

Untreated complex regional pain syndrome (CRPS) may progress from acute stages with increased hair and nail growth in the affected limb to chronic stages with atrophy of the skin, muscles and bones. The aim of this study was to investigate whether tissue hypoxia could be one mechanism responsible for this late CRPS symptoms. Nineteen patients with CRPS and two control groups (healthy control subjects, surgery patients with edema) participated in this study. Skin capillary hemoglobin oxygenation (HbO(2)) was measured non-invasively employing micro-lightguide spectrophotometry (EMPHO). The EMPHO probe was mounted force-controlled onto the skin of the affected and unaffected hand. HbO(2) was m…

AdultMalePilot ProjectsAtrophyEdemaMedicineHumansReactive hyperemiaAgedSkinAnalysis of Variancebusiness.industryBlood flowOxygenationHypoxia (medical)Middle Agedmedicine.diseaseCell HypoxiaAnesthesiology and Pain MedicineComplex regional pain syndromeNeurologyAnesthesiaOxyhemoglobinsFemaleNeurology (clinical)Hemoglobinmedicine.symptombusinessComplex Regional Pain SyndromesPain
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Apoptosis-related Proteins in Skeletal Muscle Fibers of Spinal Muscular Atrophy

1997

There is evidence that apoptosis in spinal muscular atrophies (SMA) is not restricted to motor neurons but also affects muscle fibers. Studying the expression of several apoptosis-associated proteins we found constant expression of bax in muscle fibers, which promoted cell death. The expression of bax correlated with defective innervation of muscle fibers was also indicated by upregulation of N-CAM. While in early-onset SMA atrophic as well as normo- and hypertrophic muscle fibers displayed expression of bax, muscle fibers in late-onset SMA and peripheral neuropathies showed bax-expression only in atrophic fibers. Other investigated apoptosis-associated factors comprised interleukin-1 beta …

AdultMaleProgrammed cell deathPathologymedicine.medical_specialtyMuscle Fibers Skeletalbcl-X ProteinMuscle ProteinsApoptosisBiologyMicrofilamentPathology and Forensic MedicineMuscular Atrophy SpinalCellular and Molecular NeuroscienceReference ValuesProto-Oncogene ProteinsmedicineHumansMyocyteMuscle SkeletalActinAgedbcl-2-Associated X ProteinCaspase 1InfantPeripheral Nervous System DiseasesGeneral MedicineSpinal muscular atrophyMiddle AgedSMA*Spinal muscular atrophiesmedicine.diseaseCell biologyCysteine EndopeptidasesProto-Oncogene Proteins c-bcl-2NeurologyFemaleNeural cell adhesion moleculeNeurology (clinical)Journal of Neuropathology and Experimental Neurology
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