Search results for "Axons"

showing 10 items of 101 documents

Ultrastructural pathology of dermal axons and Schwann cells in lysosomal diseases

1988

Skin tissue specimens, obtained from 60 patients afflicted with a diverse range of lysosomal disorders revealed two groups of lesions within dermal axons, largely unmyelinated ones, particularly within axonal terminals: (1) non-specific mitochondria and dense bodies often enlarging the axonal terminal; and (2) disease-specific lysosomal residual bodies, the latter less frequent depending on the incidence and type of lysosomal disorders, i.e., largely only seen in GM2-gangliosidosis due to hexosaminidase A deficiency and mucolipidosis IV, while the spectrum of lysosomal residual bodies in Schwann cells appeared more variegated, especially due to the occurrence of vacuolar lysosomal residual …

Pathologymedicine.medical_specialtymedicine.diagnostic_testSchwann cellBiologyAxonsUltrastructural PathologyMitochondriaPathology and Forensic MedicineMicroscopy ElectronCellular and Molecular Neurosciencemedicine.anatomical_structureMetabolic DiseasesLysosomeBiopsymedicineAxoplasmic transportHumansHexosaminidaseSchwann CellsNeurology (clinical)EpidermisAxonLysosomesSkinActa Neuropathologica
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Syntaxin13 expression is regulated by mammalian target of rapamycin (mTOR) in injured neurons to promote axon regeneration.

2014

Injured peripheral neurons successfully activate intrinsic signaling pathways to enable axon regeneration. We have previously shown that dorsal root ganglia (DRG) neurons activate the mammalian target of rapamycin (mTOR) pathway following injury and that this activity enhances their axon growth capacity. mTOR plays a critical role in protein synthesis, but the mTOR-dependent proteins enhancing the regenerative capacity of DRG neurons remain unknown. To identify proteins whose expression is regulated by injury in an mTOR-dependent manner, we analyzed the protein composition of DRGs from mice in which we genetically activated mTOR and from mice with or without a prior nerve injury. Quantitati…

ProteomicsAxon; Proteomics; Regeneration; SNARE Proteins; mTORSNARE Proteinmedicine.medical_treatmentInbred C57BLRegenerative MedicineBiochemistryMedical and Health SciencesMiceNeurobiologyGanglia SpinalAxonCells CulturedMice KnockoutGene knockdownCulturedQa-SNARE ProteinsTOR Serine-Threonine KinasesAxotomyBiological SciencesSciatic NerveCell biologymedicine.anatomical_structureNeurologicalmTORFemaleAxotomySignal transductionmedicine.symptomSNARE ProteinsBiochemistry & Molecular BiologyPhysical Injury - Accidents and Adverse EffectsSpinalSensory Receptor CellsCellsKnockout1.1 Normal biological development and functioningBiologyAxonUnderpinning researchmedicineAnimalsRegenerationMolecular BiologyPI3K/AKT/mTOR pathwayRegeneration (biology)NeurosciencesProteomicCell BiologyNerve injuryAxonsNerve RegenerationMice Inbred C57BLnervous systemChemical SciencesAxoplasmic transportGanglia
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Metabolomic changes in the rat retina after optic nerve crush.

2013

Purpose To identify metabolic pathways and metabolites affected by optic nerve crush that can act as predictors of the disease or therapeutic targets. Methods The left optic nerve of adult rats was intraorbitally crushed and retinas were dissected 24 hours or 14 days after the lesion (n = 10 per group). Metabolic profiling analysis was carried out by Metabolon, Inc. A total of 195 metabolites were unambiguously detected. Data were normalized and the regulated metabolites were identified after comparing the different conditions. Metabolite concentration changes were analyzed using single and multivariate statistical analysis to detect discriminatory metabolites. Functional clustering and met…

Retinal Ganglion CellsNerve CrushMetaboliteProtein Array AnalysisApoptosisPharmacologymedicine.disease_causeGas Chromatography-Mass SpectrometryRetinaLesionRats Sprague-Dawleychemistry.chemical_compoundMetabolomicsTandem Mass SpectrometrymedicineAnimalsMetabolomicsAmino AcidsChemistryLipid metabolismAxotomyOptic NerveLipid MetabolismAxonsRatsMetabolic pathwayOxidative StressOptic nerveCarbohydrate MetabolismFemaleMetabolonmedicine.symptomOxidative stressChromatography LiquidSignal TransductionInvestigative ophthalmologyvisual science
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Neuroinflammation by cytotoxic T-lymphocytes impairs retrograde axonal transport in an oligodendrocyte mutant mouse.

2012

Mice overexpressing proteolipid protein (PLP) develop a leukodystrophy-like disease involving cytotoxic, CD8+ T-lymphocytes. Here we show that these cytotoxic T-lymphocytes perturb retrograde axonal transport. Using fluorogold stereotactically injected into the colliculus superior, we found that PLP overexpression in oligodendrocytes led to significantly reduced retrograde axonal transport in retina ganglion cell axons. We also observed an accumulation of mitochondria in the juxtaparanodal axonal swellings, indicative for a disturbed axonal transport. PLP overexpression in the absence of T-lymphocytes rescued retrograde axonal transport defects and abolished axonal swellings. Bone marrow tr…

Retinal Ganglion CellsProteolipid protein 1MouseCD8-Positive T-LymphocytesGranzymesMyelinMiceBone Marrow TransplantationNeuronsddc:616MultidisciplinarybiologyQRNeurodegenerative DiseasesAnimal ModelsCell biologyOligodendrogliamedicine.anatomical_structureNeurologyMedicineResearch ArticleHeterozygoteMultiple SclerosisProteolipidsScienceImmunologyMice Transgenicchemical and pharmacologic phenomenaAutoimmune DiseasesModel OrganismsmedicineAnimalsBiologyNeuroinflammationInflammationImmunityDemyelinating DisordersOligodendrocyteAxonsGranzyme BPerforinGranzymenervous systemImmune SystemImmunologyMutationAxoplasmic transportbiology.proteinClinical ImmunologyMolecular NeuroscienceT-Lymphocytes CytotoxicNeurosciencePLoS ONE
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The Innervation of Taste Buds in the Soft Palate and Circumvallate Papilla of the Rat as Revealed by the Zinc Iodide-Osmium Tetroxide Technique.

1998

The taste buds in the soft palate and the circumvallate papillae of the rat were investigated by the zinc iodide-osmium tetroxide technique. In addition, electron micrographs of taste buds stained with this method were presented for the first time. Differences in taste bud structures were found between the examined regions. The taste buds of the soft palate showed a complicated plexus of intragemmal nerve fibers. Some fibers exhibited terminal polymorphic swellings. Single branches could be traced close to the space of the taste pore. In the soft palate, the taste bud cells remained unstained, whereas in the circumvallate papillae of the tongue, a subpopulation of taste bud cells could be s…

TasteHistologyOsmium TetroxideRats Sprague-DawleyFixativeschemistry.chemical_compoundNerve FibersTongueTongueTaste budmedicineAnimalsColoring AgentsLingual papillaSoft palateAnatomyIodidesTaste BudsAxonsRatsCell biologyStainingmedicine.anatomical_structureOsmium tetroxidechemistryZinc CompoundsCytoplasmPalate SoftArchives of Histology and Cytology
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Medullary respiratory-related neurons with axonal connections to rostral pons and their function in termination of inspiration.

1985

In urethane-anaesthetized, paralyzed and artificially ventilated rabbits, medullary respiration-related neurons (RRU) were classified according to the phase relation of their burst discharge to phrenic nerve activity. Phase-bound inspiratory (I) or expiratory (E) neurons were discriminated from phase-spanning expiratory-inspiratory (EI) or inspiratory-expiratory (IE) units. Mechanisms of termination of inspiration by electrical stimulation of rostral pontine nuclei (Nc. parabrachialis medialis; Lc. coeruleus) were examined firstly to demonstrate whether RRU receive descending excitatory and inhibitory afferents as well as ascending efferents and secondly to analyse the time course of the ne…

Time FactorsPhysiologyClinical BiochemistryStimulationInhibitory postsynaptic potentialPhysiology (medical)PonsNeural PathwaysMedicineAnimalsNeurons AfferentPhrenic nerveNeuronsMedulla Oblongatabusiness.industryRespirationPontine nucleiPons VaroliiAnatomyPonsAxonsElectric Stimulationnervous systemMedulla oblongataExcitatory postsynaptic potentialRabbitsbusinessNeurosciencePflugers Archiv : European journal of physiology
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α-Synuclein expression levels do not significantly affect proteasome function and expression in mice and stably transfected PC12 cell lines

2004

α-Synuclein (α-syn) is a small protein of unknown function that is found aggregated in Lewy bodies, the histopathological hallmark of sporadic Parkinson disease and other synucleinopathies. Mutations in the α-syn gene and a triplication of its gene locus have been identified in early onset familial Parkinson disease. α-Syn turnover can be mediated by the proteasome pathway. A survey of published data may lead to the suggestion that overexpression of α-syn wild type, and/or their variants (A53T and A30P), may produce a decrease in proteasome activity and function, contributing to α-syn aggregation. To investigate the relationship between synuclein expression and proteasome function we have s…

Time Factorsanimal diseasesmedicine.disease_causePC12 CellsBiochemistryMicechemistry.chemical_compoundTransgenesPromoter Regions GeneticMice KnockoutGeneticsMutationInnervationBrainParkinson DiseaseProteasome complexAmyloidosisCell biologyInnervacióalpha-SynucleinAdditions and CorrectionsPèptidsPlasmidsProteasome Endopeptidase ComplexPrionsProtein subunitBlotting WesternImmunoblottingSynucleinsMice TransgenicNerve Tissue ProteinsBiologyTransfectionBacterial ProteinsMultienzyme ComplexesmedicineAnimalsImmunoprecipitationMolecular BiologyAlpha-synucleinSynucleinopathiesEpilepsyWild typeGenetic VariationCell BiologyAxonsRatsnervous system diseasesMice Inbred C57BLEpilèpsiaDisease Models AnimalLuminescent ProteinschemistryProteasomenervous systemSinapsiMutationSynapsesSynucleinAmiloïdosiPeptides
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Hardwiring the Brain: Endocannabinoids Shape Neuronal Connectivity

2007

The roles of endocannabinoid signaling during central nervous system development are unknown. We report that CB 1 cannabinoid receptors (CB 1 Rs) are enriched in the axonal growth cones of γ-aminobutyric acid–containing (GABAergic) interneurons in the rodent cortex during late gestation. Endocannabinoids trigger CB 1 R internalization and elimination from filopodia and induce chemorepulsion and collapse of axonal growth cones of these GABAergic interneurons by activating RhoA. Similarly, endocannabinoids diminish the galvanotropism of Xenopus laevis spinal neurons. These findings, together with the impaired target selection of cortical GABAergic interneurons lacking CB 1 Rs, identify endoc…

medicine.medical_specialtyCannabinoid receptorGrowth ConesSynaptogenesisXenopus ProteinsBiologyRats Sprague-DawleyMiceXenopus laevisReceptor Cannabinoid CB1ChemorepulsionCell MovementInterneuronsInternal medicineCannabinoid Receptor ModulatorsmedicineAnimalsAxonGrowth coneCells CulturedIn Situ Hybridizationgamma-Aminobutyric AcidUltrasonographyCerebral CortexMicroscopy ConfocalMultidisciplinaryStem Cellsmusculoskeletal neural and ocular physiologyEndocannabinoid systemAxonsRatsMice Inbred C57BLEndocrinologymedicine.anatomical_structurenervous systemSynapsesGABAergiclipids (amino acids peptides and proteins)Axon guidanceNeuroscienceEndocannabinoidsSignal TransductionScience
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Combined pre- and postnatal ethanol exposure in rats disturbs the myelination of optic axons.

2011

Aims: To analyse myelination and outgrowth of the optic axons in relation to the neuro-ophthalmological manifestations of ethanol (EtOH) abuse during pregnancy. Methods: An experimental model of chronic EtOH exposure was developed in rats and their offspring by subjecting the dams to a liquid diet (35% of the daily total calories as either EtOH or maltose-dextrose nutritional controls (Con). Eyeballs and optic nerves were obtained at key developmental stages and processed for morphologic, immunocytochemical and immunoblotting procedures, using alternatively antibodies against myelin basic protein (MBP) or neurofilament (NF) protein, and image analysing. Results: A significant delay in onset…

medicine.medical_specialtyNeurofilamentTime Factorsgenetic structuresOffspringEyeRetinaMyelinchemistry.chemical_compoundNeurofilament ProteinsPregnancyInternal medicinemedicineAnimalsAxonRats WistarMyelin SheathRetinabiologyEthanolChemistryBody WeightCentral Nervous System DepressantsRetinalMyelin Basic ProteinOptic NerveGeneral MedicineAnatomyeye diseasesAxonsMyelin basic proteinRatsDisease Models Animalmedicine.anatomical_structureEndocrinologynervous systemAnimals NewbornPrenatal Exposure Delayed Effectsbiology.proteinOptic nerveFemaleAlcohol and alcoholism (Oxford, Oxfordshire)
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A new window in multiple sclerosis pathology: non-conventional quantitative magnetic resonance imaging outcomes

2010

Recent findings suggest that neuronal pathology occurs early in the course of multiple sclerosis and seems to be responsible for accumulation of disability. Nonetheless, the nervous system has an intrinsic potential for repair and compensation in the neuronal component. Disease-modifying drugs such as glatiramer acetate interfere with, and down-regulate, inflammatory pathology and slow neurodegeneration. Moreover, certain regulatory cytokines and neurotrophic factors have the capacity to promote neuronal and axonal repair. Given the importance of neuronal injury in multiple sclerosis and the potential of certain treatments for neuronal repair, it is important to possess adequate and sensiti…

medicine.medical_specialtyPathologyMultiple SclerosisNeuroprotectionNeurotrophic factorsmedicineHumansGlatiramer acetateNeuronsmedicine.diagnostic_testbusiness.industryMultiple sclerosisNeurodegenerationBrainMagnetic resonance imagingAnatomical pathologymedicine.diseaseMagnetic Resonance ImagingAxonsnervous systemNeurologyNeurology (clinical)businessNeuroscienceDiffusion MRImedicine.drugJournal of the Neurological Sciences
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