Search results for "B1"

showing 10 items of 1109 documents

Human parvovirus B19 infection and antiphospholipid antibodies

2007

Erythema infectiosum is the main manifestation of human parvovirus B19 infections. Further B19-related diseases commonly associated with the acute infection are flue-like symptoms, transient aplastic crisis, transient arthralgias, leukopenia and thrombocytopenia, spontaneous abortion and hydrops fetalis in pregnant women. Hepatitis, myocarditis, meningitis, encephalitis as well as pure red cell anemia may occur occasionally. In addition parvovirus B19 infections have been frequently described as cause or trigger of various forms of autoimmune diseases affecting all blood cell lines, joints, connective tissue, uvea, large and small vessels. Molecular mimicry may be one major contribution to …

MyocarditisvirusesImmunologymedicine.disease_causeAutoimmune DiseasesParvoviridae InfectionsPregnancyhemic and lymphatic diseasesHydrops fetalisParvovirus B19 HumanHumansImmunology and AllergyMedicinePregnancy Complications InfectiousAnti-neutrophil cytoplasmic antibodyHepatitisbiologybusiness.industryParvovirusvirus diseasesmedicine.diseasebiology.organism_classificationVirologyMolecular mimicryErythema InfectiosumImmunologyAntibodies AntiphospholipidFemalebusinessEncephalitisAutoimmunity Reviews
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Development of aDrosophila melanogasterspliceosensor system forin vivohigh-throughput screening in myotonic dystrophy type 1

2014

AbstractAlternative splicing of pre-mRNAs is an important mechanism that regulates cellular function in higher eukaryotes. A growing number of human genetic diseases involve splicing defects that are directly connected to their pathology. In myotonic dystrophy type 1 (DM1), several clinical manifestations have been proposed to be the consequence of tissue-specific missplicing of numerous genes. These events are triggered by an RNA gain-of-function and resultant deregulation of specific RNA-binding factors, such as the nuclear sequestration of muscleblind-like family factors (MBNL1-MBNL3). Thus, the identification of chemical modulators of splicing events could lead to the development of the…

Myotonic dystrophyNeuroscience (miscellaneous)lcsh:MedicineMedicine (miscellaneous)BiologySplicingMyotonic dystrophyGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundMinigeneImmunology and Microbiology (miscellaneous)lcsh:PathologymedicineAnimalsMBNL1Resource ArticleGeneGeneticsDrug discoverylcsh:RAlternative splicingmedicine.diseasebiology.organism_classificationHigh-Throughput Screening AssaysAlternative SplicingDrosophila melanogasterchemistryIn vivo screeningRNA splicingDrosophila melanogasterLuciferaselcsh:RB1-214MinigeneDisease Models & Mechanisms
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Una nueva mirada al informe de Ley Agraria de Jovellanos doscientos años después

1995

Editada en la Fundación Empresa Pública Con motivo del bicentenario (1795-1995) de la publicación de lnforme de Ley Agraria de Jovellanos se presenta una visión retrospectiva de esa obra ya clásica del pensamiento español incluyendo un repaso de sus múltiples y desacordes intérpretes modernos y un análisis del interior del texto e incluso del título. Se concluye insistiendo en la plena inserción del Informe en la tradición ilustrada española, en que los usuales calificativos de mercantilista, liberal smithniano o fisiócrata son inadecuados y en que es preciso restituir la obra y su autor a su tiempo. Publicado

N01B12HistoriaEconomía
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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Neurobiology of cannabinoid receptor signaling
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2020

The endocannabinoid system (ECS) is a highly versatile signaling system within the nervous system. Despite its widespread localization, its functions within the context of distinct neural processes are very well discernable and specific. This is remarkable, and the question remains as to how such specificity is achieved. One key player in the ECS is the cannabinoid type 1 receptor (CBEl sistema endocannabinoide (SEC) apareció como un sistema de señalización muy versátil en el sistema nervioso. A pesar de su existencia amplia y ubicua, sus funciones están integradas en el contexto de distintos procesos neuronales y, en última instancia, son bastante bien discernibles y específicas. Esto es n…

Nervous systemCannabinoid receptormedicine.medical_treatment2-ArachidonoylglycerolContext (language use)BiologyReceptor Cannabinoid CB203 medical and health scienceschemistry.chemical_compound0302 clinical medicineNeurobiologyReceptor Cannabinoid CB1Cannabinoid receptor type 2medicineAnimalsHumansanandamideReceptorReceptors Cannabinoidbehaviorcannabinoid receptorEndocannabinoid systemState of the Art030227 psychiatry2-arachidonoylglycerolmedicine.anatomical_structurechemistryneural communicationCannabinoidNeuroscienceEndocannabinoidsSignal TransductionDialogues in clinical neuroscience
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Plexin-B1 and Semaphorin 4D Cooperate to Promote Perineural Invasion in a RhoA/ROK-Dependent Manner

2012

Perineural invasion (PNI) is a tropism of tumor cells for nerve bundles located in the surrounding stroma. It is a pathological feature observed in certain tumors, referred to as neurotropic malignancies, that severely limits the ability to establish local control of disease and results in pain, recurrent growth, and distant metastases. Despite the importance of PNI as a prognostic indicator, its biological mechanisms are poorly understood. The semaphorins and their receptors, the plexins, compose a family of proteins originally shown to be important in nerve cell adhesion, axon migration, and proper central nervous system development. Emerging evidence has demonstrated that these factors a…

Nervous systemPathologymedicine.medical_specialtyCell typeanimal structuresRHOANervous System NeoplasmsTransplantation HeterologousPerineural invasionRetraction NoticeMice NudeNerve Tissue ProteinsReceptors Cell SurfaceSemaphorinsPathology and Forensic Medicine03 medical and health sciencesMice0302 clinical medicineSemaphorinAntigens CDCell MovementCell Line TumorSettore BIO/10 - BiochimicamedicineAnimalsHumansNeoplasm InvasivenessAxonRNA Small InterferingCell adhesion030304 developmental biologyMice Knockout0303 health sciencesbiologyDrug SynergismAxonsTransplantationMice Inbred C57BLmedicine.anatomical_structure030220 oncology & carcinogenesisembryonic structuresbiology.proteinCancer researchperineural invasion tumor cells Rho kinase-dependent manner plexin B1rhoA GTP-Binding ProteinNeoplasm TransplantationSignal TransductionThe American Journal of Pathology
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Neuronal populations mediating the effects of endocannabinoids on stress and emotionality

2011

An adequate emotional response to stress is essential for survival and requires the fine-tuned regulation of several distinct neuronal circuits. Therefore, a precise control of these circuits is necessary to prevent behavioral imbalances. During the last decade, numerous investigations have evidenced that the endocannabinoid (eCB) system is able to crucially control stress coping. Its central component, the cannabinoid type 1 receptor (CB1 receptor), is located at the presynapse, where it is able to attenuate neurotransmitter release after its activation by postsynaptically produced and released eCBs. To date, the eCB system has been found to control the neurotransmitter release from severa…

NeuronsHypothalamo-Hypophyseal SystemGeneral NeuroscienceEmotionsGlutamate receptorPituitary-Adrenal SystemContext (language use)Endocannabinoid systemAmygdalaPresynapsemedicine.anatomical_structureReceptor Cannabinoid CB1nervous systemStress PhysiologicalCannabinoid Receptor ModulatorsSynapsesmedicineAnimalsLocus coeruleusPremovement neuronal activityPsychologyPrefrontal cortexNeuroscienceStress PsychologicalEndocannabinoidsNeuroscience
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EFFECT OF ACETALDEHYDE INTOXICATION AND WITHDRAWAL ON NEUROPEPTIDE Y EXPRESSION. FOCUS ON CB1 RECEPTOR ROLE.

2014

Neuropeptide Y (NPY) acts as an endogenous anxiolytic, and, like endocannabinoids, plays an important role in the regulation of neuronal excitability during ethanol withdrawal (Rubio et al., 2011). Since acetaldehyde is considered a mediator of ethanol central effects, this research aims at investigating, following intoxication and during withdrawal, the effects of acetaldehyde on NPY expression in brain areas particularly vulnerable to alcohol, and the influence of cannabinoid system on it. Rats underwent acetaldehyde intoxication (450mg/kg, i.g., 4 times daily for 4 days); AM281, a CB1 selective antagonist (2,5 mg/kg, i.p.) was administered during abstinence. Immunohistochemical analysis …

Neuropeptide YAcetaldehydeCB1 antagonist
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Formation of fumonisin B(1)-glucose reaction product, in vitro cytotoxicity, and lipid peroxidation on kidney cells.

2010

Fumonisin B(1) (FB(1)) content in corn products decreases during the heating process in foods containing reducing sugars, mainly because of the formation of N-(carboxymethyl)fumonisin B(1). In this study, a rapid method has been developed for the determination of both compounds in corn products using a high-speed blender, Ultra-Turrax, for solvent extraction and liquid chromatography-tandem mass spectrometry. The kinetics of FB(1) degradation and the formation of the Maillard adduct were studied in a model system constituted by corn bread spiked with FB(1) and heated at 160, 180, and 200 degrees C for 3, 6, 10, 15, and 20 min. FB(1) decreased from 0.96 to 0.3 mg/kg and N-(carboxymethyl)fumo…

Neutral redFood HandlingKidneyFumonisinsZea maysLipid peroxidationsymbols.namesakechemistry.chemical_compoundfumonisinFumonisinChlorocebus aethiopskidney cellAnimalsIC50Vero CellsFumonisin B1ChromatographyChemistryfood and beveragesGeneral ChemistryCarbohydrateMalondialdehydeMaillard reactionKineticsGlucosesymbolsLipid PeroxidationGeneral Agricultural and Biological SciencesJournal of agricultural and food chemistry
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Maternal vitamin deficiency mimicking multiple acyl-CoA dehydrogenase deficiency on newborn screening

2021

Abstract Background In infancy multiple acyl-CoA dehydrogenase deficiency (MADD) is commonly a severe inherited metabolic disease caused by genetic defects in electron transfer flavoprotein (ETF) or ETF ubiquinone oxidoreductase. Both enzymes require flavin adenine dinucleotide (FAD) as a cofactor. Riboflavin (vitamin B2) is a precursor in the synthesis of FAD. MADD can be detected by newborn screening (NBS) based on elevation of multiple acylcarnitines. Methods We present the results of two children whose NBS results and subsequent confirmatory testing resulted in a suspected diagnosis of MADD. In parallel in both children vitamin B12 deficiency was detected. Results Biochemical profiles n…

Newborn screeningMedicine (General)medicine.medical_specialtyQH301-705.5FlavoproteinRiboflavinMaternalCofactorchemistry.chemical_compoundR5-920EndocrinologyMultiple acyl-CoA dehydrogenase deficiencyInternal medicineGeneticsmedicineVitamin B12Biology (General)Multiple Acyl-CoA Dehydrogenase DeficiencyMother and child healthMolecular Biologychemistry.chemical_classificationFlavin adenine dinucleotideNewborn screeningbiologybusiness.industryfood and beveragesEnzymeEndocrinologyVitamin B12 deficiencychemistrybiology.proteinbusinessResearch PaperMolecular Genetics and Metabolism Reports
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