Search results for "BES"

showing 10 items of 3692 documents

Hypothalamic Apelin/Reactive Oxygen Species Signaling Controls Hepatic Glucose Metabolism in the Onset of Diabetes

2014

Aims: We have previously demonstrated that central apelin is implicated in the control of peripheral glycemia, and its action depends on nutritional (fast versus fed) and physiological (normal versus diabetic) states. An intracerebroventricular (icv) injection of a high dose of apelin, similar to that observed in obese/diabetic mice, increase fasted glycemia, suggesting (i) that apelin contributes to the establishment of a diabetic state, and (ii) the existence of a hypothalamic to liver axis. Using pharmacological, genetic, and nutritional approaches, we aim at unraveling this system of regulation by identifying the hypothalamic molecular actors that trigger the apelin effect on liver gluc…

Blood GlucoseMaleSympathetic nervous systemLIVER[SDV.BIO]Life Sciences [q-bio]/BiotechnologyGlycogenolysisPhysiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionClinical BiochemistryMice ObeseBiochemistrySYMPATHETIC-NERVE ACTIVITYAPELINBRAINGeneral Environmental ScienceINSULIN-RESISTANCE3. Good healthApelinOriginal Research CommunicationsADIPOSE-TISSUEmedicine.anatomical_structureIntercellular Signaling Peptides and ProteinsSignal TransductionEXPRESSIONmedicine.medical_specialtyGlycogenolysisHypothalamusBiologyCarbohydrate metabolismAutonomic Nervous SystemInsulin resistanceAdipokinesInternal medicineDiabetes mellitusmedicineAnimalsMolecular BiologyGluconeogenesis[ SDV.BIO ] Life Sciences [q-bio]/BiotechnologyCell Biologymedicine.diseaseMice Inbred C57BLMICEGlucoseEndocrinologyDiabetes Mellitus Type 2GluconeogenesisRATGeneral Earth and Planetary SciencesLiver functionReactive Oxygen Species[SDV.AEN]Life Sciences [q-bio]/Food and NutritionSYSTEMAntioxidants & Redox Signaling
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GLP-2 as Beneficial Factor in the Glucose Homeostasis in Mice Fed a High Fat Diet

2015

Glucagon like peptide-2 (GLP-2) is a gastrointestinal hormone released in response to dietary nutrients, which acts through a specific receptor, the GLP-2 receptor (GLP-2R). The physiological effects of GLP-2 are multiple, involving also the intestinal adaptation to high fat diet (HFD). In consideration of the well-known relationship between chronic HFD and impaired glucose metabolism, in the present study we examined if the blocking of the GLP-2 signaling by chronic treatment with the GLP-2R antagonist, GLP-2 (3-33), leads to functional consequences in the regulation of glucose metabolism in HFD-fed mice. Compared with animals fed standard diet (STD), mice at the 10th week of HFD showed hy…

Blood GlucoseMaleTime FactorsDiet High-FatPeptide FragmentsMice Inbred C57BLDisease Models AnimalHormone Antagonistsobesity insulin resistance pancreatic isletsInsulin-Secreting CellsGlucagon-Like Peptide 2AnimalsHomeostasisInsulinGLP-2; obesity insulin resistance pancreatic isletsGLP-2BiomarkersGlucose Metabolism DisordersSignal Transduction
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The Impact of Amino Acids on Postprandial Glucose and Insulin Kinetics in Humans

2020

Different amino acids (AAs) may exert distinct effects on postprandial glucose and insulin concentrations. A quantitative comparison of the effects of AAs on glucose and insulin kinetics in humans is currently lacking. PubMed was queried to identify intervention studies reporting glucose and insulin concentrations after acute ingestion and/or intravenous infusion of AAs in healthy adults and those living with obesity and/or type 2 diabetes (T2DM). The systematic literature search identified 55 studies that examined the effects of l-leucine, l-isoleucine, l-alanine, l-glutamine, l-arginine, l-lysine, glycine, l-proline, l-phenylalanine, l-glutamate, branched-chain AAs (i.e., l-leucine, l-iso…

Blood GlucoseMaleinsulin secretionobesitymedicine.medical_treatmentAdministration OralReviewType 2 diabetes0302 clinical medicinesystematic reviewInsulinIngestionGlucose homeostasis030212 general & internal medicineInfusions IntravenousNutrition and DieteticsL-ARGININEINTRAVENOUS ARGININEFREE FATTY-ACIDSBLOOD-GLUCOSEdynamicsPostprandial PeriodPostprandialSECRETIONFemaletype 2 diabetesLeucineGROWTH-HORMONElcsh:Nutrition. Foods and food supplyAdultORAL ALANINEmedicine.medical_specialtyMETABOLIC-RESPONSElcsh:TX341-641030209 endocrinology & metabolism03 medical and health sciencesInternal medicinemedicineHumansglucose homeostasisinsulin sensitivityamino acidsbusiness.industryInsulinmedicine.diseaseObesityGlucoseEndocrinologyDiabetes Mellitus Type 2kineticsPLASMA-GLUCAGON RESPONSEtime series dataIsoleucinebusinessFood ScienceINGESTIONNutrients
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Associations of the FTO rs9939609 and the MC4R rs17782313 polymorphisms with type 2 diabetes are modulated by diet, being higher when adherence to th…

2012

Background: Although the Fat Mass and Obesity (FTO) and Melanocortin-4 Receptor (MC4R) genes have been consistently associated with obesity risk, the association between the obesity-risk alleles with type 2 diabetes is still controversial. In some recent meta-analyses in which significant results have been reported, the associations disappeared after adjustment for body mass index (BMI). However gene-diet interactions with dietary patterns have not been investigated. Our main aim was to analyze whether these associations are modulated by the level of adherence to the Mediterranean Diet (MedDiet). Methods: Case-control study in 7,052 high cardiovascular risk subjects (3,430 type 2 diabetes c…

Blood GlucoseMalelcsh:Diseases of the circulatory (Cardiovascular) systemmodelos logísticoscumplimiento del pacienteMediterranean dietEndocrinology Diabetes and MetabolismhumanosType 2 diabetesfrecuencia génicaMC4RDiet MediterraneanBody Mass IndexNutrigenomicsGene FrequencySurveys and QuestionnairesOdds Ratiomediana edadNutrigeneticsOriginal InvestigationAged 80 and overeducation.field_of_studyancianonutrigenómicaDiabetisdietamodelos linealesDiabetesayunodistribución de la ji al cuadradoFastingMiddle Agedcociente de probabilidades relativasestado nutricionalPhenotypeinteracción gen-ambientediabetes mellitusfenotipoReceptor Melanocortin Type 4FemaleDietaCardiology and Cardiovascular MedicineFTONutrigenòmicamedicine.medical_specialtyglucosa sanguíneaGene-diet interactionsPopulationestudios de casos y controlesAlpha-Ketoglutarate-Dependent Dioxygenase FTONutritional StatusDiabetes mellitusInternal medicineMediterranean dietmedicineGenetic predispositionHumansGenetic Predisposition to Diseaseanálisis multifactorialeducationAgedChi-Square DistributionPolymorphism Geneticbusiness.industryproteínasíndice de masa corporalCase-control studyProteinsnutritional and metabolic diseasespredisposición genética a la enfermedadmedicine.diseaseObesityDietEndocrinologyLogistic ModelsDiabetes Mellitus Type 2lcsh:RC666-701SpainCase-Control StudiesMultivariate AnalysisLinear ModelsPatient ComplianceGene-Environment InteractionbusinessBody mass index
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Association of obesity with proteasomal gene polymorphisms in children.

2013

The aim of this study was to ascertain possible associations between childhood obesity, its anthropometric and clinical parameters, and three loci of proteasomal genes rs2277460 (PSMA6c.-110C>A), rs1048990 (PSMA6c.-8C>G), and rs2348071 (PSMA3c. 543+138G>A) implicated in obesity-related diseases. Obese subjects included 94 otherwise healthy children in Latvia. Loci were genotyped and then analyzed using polymerase chain reactions, with results compared to those of 191 nonobese controls.PSMA3SNP frequency differences between obese children and controls, while not reaching significance, suggested a trend. These differences, however, proved highly significant (P<0.002) in the subset…

Blood GlucoseMalelcsh:Internal medicinePediatric ObesityProteasome Endopeptidase ComplexArticle SubjectAdolescentGenotypeEndocrinology Diabetes and MetabolismPSMA6Blood PressurePSMA3Polymorphism Single NucleotideChildhood obesityBody Mass IndexmedicineHumansGenetic Predisposition to Diseaselcsh:RC31-1245ChildGeneGeneticsAnalysis of Variancebusiness.industryCholesterol LDLAnthropometrymedicine.diseaseObesityCase-Control StudiesChild PreschoolFemalebusinessResearch ArticleJournal of obesity
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Novel genetic variant in FTO influences insulin levels and insulin resistance in severely obese children and adolescents.

2008

Background: The global prevalence of obesity and overweight is increasing rapidly among adults as well as among children and adolescents. Recent genome-wide association studies have provided strong support for association between variants in the FTO gene and obesity. We sequenced regions of the FTO gene to identify novel variants that are associated with obesity and related metabolic traits. Results: We screened exons 3 and 4 including exon-intron boundaries in FTO in 48 obese children and adolescents and identified three novel single nucleotide polymorphism in the fourth intronic region, (c.896 + 37A > G, c.896 + 117C > G and c.896 + 223A > G). We further genotyped c.896 + 223A > G in 962 …

Blood GlucoseMalemedicine.medical_specialtyAdolescentEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMedicine (miscellaneous)Single-nucleotide polymorphismOverweightFTO genePolymorphism Single NucleotideBody Mass IndexYoung AdultInsulin resistanceInternal medicinemedicineHumansGenetic Predisposition to DiseaseObesityChildNutrition and Dieteticsbusiness.industryInsulinGenetic Variationmedicine.diseaseObesityGenotype frequencyEndocrinologyPhenotypeFemalemedicine.symptomInsulin ResistancebusinessBody mass indexGenome-Wide Association StudyInternational journal of obesity (2005)
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Association between WHO cut-offs for childhood overweight and obesity and cardiometabolic risk

2012

AbstractObjectiveTo examine the association between cardiovascular risk and childhood overweight and obesity using the BMI cut-offs recommended by the WHO.DesignChildren were classified as normal weight, overweight and obese according to the WHO BMI-for-age reference. Blood pressure, lipids, glucose, insulin, homeostasis model assessment–insulin resistance (HOMA-IR) and uric acid levels were compared across BMI groups. ANOVA and tests of linearity were used to assess overall mean differences across groups. Crude and adjusted odds ratios were calculated for adverse plasma levels of biochemical variables.SettingPaediatric care centres.SubjectsChildren (n149) aged 8–18 years.ResultsAbout 37 %,…

Blood GlucoseMalemedicine.medical_specialtyAdolescentmedicine.medical_treatmentMedicine (miscellaneous)Blood PressureOverweightWorld Health OrganizationBody Mass Indexchemistry.chemical_compoundInsulin resistanceRisk FactorsInternal medicinemedicineCluster AnalysisHumansInsulinObesityProspective StudiesChildNutrition and Dieteticsbusiness.industryInsulinMonitoring and SurveillanceCholesterol HDLPublic Health Environmental and Occupational Healthnutritional and metabolic diseasesCholesterol LDLOdds ratioOverweightmedicine.diseaseObesityUric AcidLogistic ModelsEndocrinologyBlood pressurechemistryCardiovascular DiseasesSpainHypertensionMultivariate AnalysisUric acidFemaleInsulin Resistancemedicine.symptombusinessBody mass indexFollow-Up Studies
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Plasma glycosphingolipids in diabetics and normals

1975

In diabetic patients with hyperlipoproteinemia type IV the monohexosyl ceramide concentration in blood plasma is significantly elevated. This augmentation can be attributed to an increased monohexosyl ceramide content of the BLDL plasma fraction. In contrast, the di-, tri-, the tetrahexosyl ceramide levels remain within normal limits. In normolipidemic diabetics of comparable age, sex, and weight classes and of comparable metabolic control no elevations of glycolipid fractions could be found. However, patients with primary hyperlipoproteinemia type IV show an increase of monohexosyl ceramide concentrations in blood plasma. Therefore, the augmentation of monohexosyl ceramide levels in plasma…

Blood GlucoseMalemedicine.medical_specialtyCeramideVery low-density lipoproteinHyperlipidemiasLipoproteins VLDLCeramidesGlycosphingolipidsDiabetes Complicationschemistry.chemical_compoundGlycolipidInternal medicineDiabetes mellitusDrug DiscoveryBlood plasmaDiabetes MellitusmedicineHumansObesityTriglyceridesGenetics (clinical)HexosesChemistrynutritional and metabolic diseasesGeneral MedicineMiddle Agedmedicine.diseaseMolecular medicineNormal limitEndocrinologyMetabolic control analysisMolecular MedicineFemaleGlycolipidsKlinische Wochenschrift
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Effects of heparin treatment on hemostatic abnormalities in obese non-insulin-dependent diabetic patients.

1997

This study was conducted to identify the mechanisms responsible for coagulative and fibrinolytic alterations and to study the effects of a short-term treatment with low-dose heparin on hemostatic abnormalities in obese non-insulin-dependent diabetes mellitus (NIDDM) patients. Four groups of age- and sex-matched patients were studied: (1) lean nondiabetic subjects (n = 30) with a body mass index (BMI) less than 25 kg/m2 (lean control subjects), (2) obese nondiabetic subjects (n = 30) with a BMI greater than 30 kg/m2 (obese control subjects), (3) lean NIDDM patients (n = 30), and (4) obese NIDDM patients (n = 30). All subjects were tested on the following parameters: fibrinogen, factor VII, p…

Blood GlucoseMalemedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentFibrinogenTissue plasminogen activatorBody Mass Indexchemistry.chemical_compoundEndocrinologyInternal medicineFibrinolysismedicineHumansInsulinObesityBlood CoagulationHemostasisFactor VIIbusiness.industryHeparinFibrinolysisnutritional and metabolic diseasesAnticoagulantsHeparinMiddle AgedLipidsBlood Coagulation FactorsEndocrinologychemistryDiabetes Mellitus Type 2HemostasisCase-Control StudiesFemalebusinessPlasminogen activatorBody mass indexmedicine.drugMetabolism: clinical and experimental
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Time course of changes in serum oxidant/antioxidant status in overfed obese rats and their offspring.

2009

The aim of the present study was to determine the time course of changes in oxidant/antioxidant status, as well as serum glucose, insulin, leptin and lipid levels, liver adipose tissue and muscle lipid and protein contents, in cafeteria-diet-fed dams during gestation and lactation, and in their offspring throughout adulthood. Food intake was also evaluated. The cafeteria diet induced a significant increase in maternal body and relative adipose tissue weights, daily energy intake, and plasma glucose, insulin, leptin and lipid levels at parturition (day 0) and at the end of lactation (day 21). Plasma total antioxidant status [ORAC (oxygen radical absorbance capacity)], erythrocyte catalase an…

Blood GlucoseMalemedicine.medical_specialtyErythrocytesOffspringmedicine.medical_treatmentAdipokineAdipose tissueBiologyAntioxidantschemistry.chemical_compoundBlood serumPregnancyInternal medicinemedicineAnimalsInsulinObesityRats WistarPrenatal Nutritional Physiological PhenomenaTriglycerideInsulinLeptinBody WeightProteinsGeneral Medicinemedicine.diseaseOxidantsLipidsRatsPregnancy ComplicationsOxidative StressEndocrinologychemistryPrenatal Exposure Delayed EffectsFemaleMetabolic syndromeEnergy IntakeClinical science (London, England : 1979)
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