Search results for "BH"

showing 10 items of 1357 documents

Allegacion en derecho, y hecho. Por el Oficio de Sogueros de Cañamo, y Esparto, y Alpargatas todas de Cañamo,... Contra el Oficio de Esparteros y A…

1697

Sig.: []2, A-M2 Port. amb orla tip. i grav. xil.

Processos València (Regne) S.XVII Obres anteriors a 1800Gremis València (Comunitat Valenciana) S.XVIIDIG-BH
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La piazza universale di tutte le professioni del mondo / nuouamente ristampata, & posta in luce, da Thomaso Garzoni da Bagnacauallo ; Con l'Aggionta …

1592

Sig. [creu llatina]8, 2[creu llatina]4, 3[creu llatina]8, A-Ooo8 Capitals grav., frisos i altres ornaments tip. Text amb postil·les marginals. - Reclams. - Registre. - Alguna errata de pag.

Professions Diccionaris Obres anteriors a 1800DIG-BH
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Differential Roles of JNK in ConA/GalN and ConA-Induced Liver Injury in Mice

2008

Tumor necrosis factor-alpha-mediated liver injury can be induced by several different means; however, the signaling events and mechanisms of cell death are likely different. We investigated the mechanism of both apoptotic and necrotic hepatocyte cell death as well as the role of c-Jun NH2-terminal kinase (JNK) in the ConA and ConA/D-galactosamine (GalN) models of murine liver injury. ConA alone induced primarily necrotic cell death with no caspase activation, whereas ConA/GalN induced apoptosis in addition to necrotic cell death. The bi-modal death pattern in the ConA/GalN model was confirmed by the use of transgenic mice expressing a dominant-negative form of Fas-associated death domain in…

Programmed cell deathNecrosisFas-Associated Death Domain ProteinApoptosisGalactosamineMitochondria Liverchemical and pharmacologic phenomenaCaspase 8Pathology and Forensic MedicineMiceNecrosisConcanavalin AmedicineAnimalsPhosphorylationDeath domainLiver injuryCaspase 8biologyLiver DiseasesJNK Mitogen-Activated Protein Kinasesmedicine.diseaseMolecular biologyEnzyme ActivationMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureConcanavalin AApoptosisHepatocytebiology.proteinMutant ProteinsChemical and Drug Induced Liver Injurymedicine.symptomGene DeletionRegular ArticlesBH3 Interacting Domain Death Agonist ProteinThe American Journal of Pathology
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Prognostico de lo preterito, anticipacion de lo presente, y regresso de lo futuro.

1724

Sig. A4 L'autor és Diego de Torres Villarroel (Aguilar Piñal, T. VIII, 773), tal com es dedueix de la introducció Reclams. - Capital grav. i Cristus a la 1ª p

Pronòstic Poesia Obres anteriors a 1800Previsió Poesia Obres anteriors al 1800DIG-BH
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Los colloquios satiricos, con vn colloquio pastoril / Compuestos por Antonio de Torquemada ...

1584

Sig. A-Ii8 Escut heràldic xil. de Bilbao al final. - Capitals grav., frisos i orles Reclams

Prosa castellana S.XVI Obres anteriors a 1800DIG-BH
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Ara ojats queus fan saber de part del Magnifich Pere de Caspe ... Iusticia en lo Civil de la present Ciutat de Valencia ... al qual ... pertany tenir…

1645

Sign.: []2

Protocols notarials València (Comunitat Valenciana) S.XVII Obres anteriors a 1800DIG-BH
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Refranes, o Proverbios en romance / que nueuamente colligiò y glossò el Comendador Hernan Nuñez ... ; Van puestos por la orden del Abc ... ; Van t…

1555

Sig. [calderó]6, A-Q8, R6, S8 Capitals grav. i port. xil. Port. arquitectònica. - Text a dos col. - Reclams

Proverbis castellans Obres anteriors a 1800DIG-BH
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The disease-specific clinical trial network for primary ciliary dyskinesia: PCD-CTN

2022

Primary ciliary dyskinesia; Rare genetic disorder; Lung diseases Discinesia ciliar primaria; Trastorno genético raro; Enfermedades pulmonares Discinesia ciliar primària; Trastorn genètic rar; Malalties pulmonars Primary ciliary dyskinesia (PCD) is a rare genetic disorder characterised by impaired mucociliary clearance leading to irreversible lung damage. In contrast to other rare lung diseases like cystic fibrosis (CF), there are only few clinical trials and limited evidence-based treatments. Management is mainly based on expert opinions and treatment is challenging due to a wide range of clinical manifestations and disease severity. To improve clinical and translational research and facili…

Pulmonary and Respiratory Medicine:enfermedades respiratorias::trastornos de la motilidad ciliar [ENFERMEDADES]:Health Occupations::Medicine::Pediatrics [DISCIPLINES AND OCCUPATIONS]Respiratory SystemSağlık BilimleriClinical Medicine (MED)SOLUNUM SİSTEMİRespiratory CareHealth SciencesMANAGEMENTKlinik Tıp (MED)Chest Diseases and Allergy:profesiones sanitarias::medicina::pediatría [DISCIPLINAS Y OCUPACIONES]:Otros calificadores::/terapia [Otros calificadores]:Respiratory Tract Diseases::Ciliary Motility Disorders [DISEASES]Internal Medicine SciencesScience & TechnologyKlinik TıpMUTATIONSRESPIRATORY SYSTEM:Other subheadings::/therapy [Other subheadings]Dahili Tıp BilimleriGöğüs Hastalıkları ve AllerjiCLINICAL MEDICINECèl·lules - Motilitat:Congenital Hereditary and Neonatal Diseases and Abnormalities::Genetic Diseases Inborn [DISEASES]TıpPulmons - Malalties - TractamentAkciğer ve Solunum TıbbıMedicineMalalties congènites:enfermedades y anomalías neonatales congénitas y hereditarias::enfermedades genéticas congénitas [ENFERMEDADES]Solunum BakımıLife Sciences & BiomedicinePulmons Malalties
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Rho protein inactivation induced apoptosis of cultured human endothelial cells.

2002

Small GTP-binding Rho GTPases regulate important signaling pathways in endothelial cells, but little is known about their role in endothelial cell apoptosis. Clostridial cytotoxins specifically inactivate GTPases by glucosylation [ Clostridium difficile toxin B-10463 (TcdB-10463), C. difficile toxin B-1470 (TcdB-1470)] or ADP ribosylation ( C. botulinum C3 toxin). Exposure of human umbilical cord vein endothelial cells (HUVEC) to TcdB-10463, which inhibits RhoA/Rac1/Cdc42, or to C3 toxin, which inhibits RhoA, -B, -C, resulted in apoptosis, whereas inactivation of Rac1/Cdc42 with TcdB-1470 was without effect, suggesting that Rho inhibition was responsible for endothelial apoptosis. Disruptio…

Pulmonary and Respiratory Medicinerac1 GTP-Binding Proteinrho GTP-Binding ProteinsProgrammed cell deathUmbilical VeinsEndotheliumPhysiologyBacterial ToxinsCASP8 and FADD-Like Apoptosis Regulating ProteinApoptosisBcl-2-associated X proteinBacterial ProteinsPhysiology (medical)Proto-Oncogene ProteinsmedicineCyclic AMPIn Situ Nick-End LabelingHumanscdc42 GTP-Binding ProteinCells Culturedbcl-2-Associated X ProteinAdenosine Diphosphate RibosebiologyCaspase 3Intracellular Signaling Peptides and ProteinsCell BiologyCaspase 9Cell biologyNeoplasm ProteinsEndothelial stem cellmedicine.anatomical_structureCdc42 GTP-Binding ProteinProto-Oncogene Proteins c-bcl-2Cell cultureApoptosisCaspasesbiology.proteinMyeloid Cell Leukemia Sequence 1 ProteinEndothelium VascularSignal transductionCarrier ProteinsrhoA GTP-Binding ProteinBH3 Interacting Domain Death Agonist ProteinSignal TransductionAmerican journal of physiology. Lung cellular and molecular physiology
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Improved Bounds for Hermite–Hadamard Inequalities in Higher Dimensions

2019

Let $\Omega \subset \mathbb{R}^n$ be a convex domain and let $f:\Omega \rightarrow \mathbb{R}$ be a positive, subharmonic function (i.e. $\Delta f \geq 0$). Then $$ \frac{1}{|\Omega|} \int_{\Omega}{f dx} \leq \frac{c_n}{ |\partial \Omega| } \int_{\partial \Omega}{ f d\sigma},$$ where $c_n \leq 2n^{3/2}$. This inequality was previously only known for convex functions with a much larger constant. We also show that the optimal constant satisfies $c_n \geq n-1$. As a byproduct, we establish a sharp geometric inequality for two convex domains where one contains the other $ \Omega_2 \subset \Omega_1 \subset \mathbb{R}^n$: $$ \frac{|\partial \Omega_1|}{|\Omega_1|} \frac{| \Omega_2|}{|\partial \Ome…

Pure mathematicsInequalitymedia_common.quotation_subject01 natural sciencesConvexitysymbols.namesakeMathematics - Metric GeometrySettore MAT/05 - Analisi MatematicaHadamard transformHermite–Hadamard inequality0103 physical sciencesClassical Analysis and ODEs (math.CA)FOS: Mathematics[MATH.MATH-AP]Mathematics [math]/Analysis of PDEs [math.AP]Hermite-Hadamard inequality subharmonic functions convexity.0101 mathematicsComputingMilieux_MISCELLANEOUSsubharmonic functionsmedia_commonMathematicsSubharmonic functionHermite polynomialsconvexity010102 general mathematicsMetric Geometry (math.MG)Functional Analysis (math.FA)Mathematics - Functional AnalysisMSC : 26B25 28A75 31A05 31B05 35B50Mathematics::LogicHermite-Hadamard inequalityDifferential geometryMathematics - Classical Analysis and ODEsFourier analysissymbols010307 mathematical physicsGeometry and TopologyThe Journal of Geometric Analysis
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