Search results for "BLAST"

showing 10 items of 2136 documents

Age-related and tissue-specific accumulation of oxidative DNA base damage in 7,8-dihydro-8-oxoguanine-DNA glycosylase (Ogg1) deficient mice.

2001

Mutations that influence the repair of oxidative DNA modifications are expected to increase the steady-state (background) levels of these modifications and thus create a mutator phenotype that predisposes to malignant transformation. We have analysed the steady-state levels and repair kinetics of oxidative DNA modifications in cells of homozygous ogg1(-/-) null mice, which are deficient in Ogg1 protein, a DNA repair glycosylase that removes the miscoding base 8-hydroxyguanine (8-oxoG) from the genome. Oxidative purine modifications including 8-oxoG were quantified by means of an alkaline elution assay in combination with Fpg protein, the bacterial functional analogue of Ogg1 protein. In pri…

PurineMaleCancer ResearchGuanineDNA RepairOxidative phosphorylationBiologymedicine.disease_causeMalignant transformationchemistry.chemical_compoundMiceTranscription (biology)medicineAnimalsN-Glycosyl HydrolasesMice KnockoutCell growthAge FactorsGeneral MedicineDNAFibroblastsMolecular biologyOxygenOxidative StresschemistryDNA-Formamidopyrimidine GlycosylaseDNA glycosylaseOrgan SpecificityImmunologyHepatocytesOxidative stressDNACell DivisionDNA DamageCarcinogenesis
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Is the repair of oxidative DNA base modifications inducible by a preceding DNA damage induction?

2007

In mammalian cells, 7,8-dihydro-8-oxoguanine (8-oxoG) and some other oxidative guanine modifications are removed from the DNA by base excision repair, which is initiated by OGG1 protein. We have tested whether this repair is inducible in mouse embryonic fibroblasts (MEFs), MCF-7 breast cancer cells and primary human fibroblasts by a pretreatment with the photosensitizer Ro19-8022 plus light, which generates predominantly 8-oxoG, or with methyl methanesulfonate (MMS), which generates alkylated bases and abasic sites (AP sites). The results indicate that the repair rate of the oxidative guanine modifications induced by the photosensitizer was not increased if a priming dose of the oxidative o…

PyrrolidinesTime FactorsDNA RepairDNA repairGuanineDNA damageBiologymedicine.disease_causeBiochemistryMicechemistry.chemical_compoundTumor Cells CulturedmedicineAnimalsHumansheterocyclic compoundsAntineoplastic Agents AlkylatingBase PairingMolecular BiologyPhotosensitizing AgentsGuanosineDNACell BiologyBase excision repairGlutathioneFibroblastsMethyl MethanesulfonateGlutathioneMolecular biologyMethyl methanesulfonateOxidative StresschemistryFemaleOxidation-ReductionQuinolizinesDNAOxidative stressDNA DamageDNA Repair
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Classification of Congeneric and QSAR of Homologous Antileukemic S–Alkylcysteine Ketones

2021

Based on a set of six vector properties, the partial correlation diagram is calculated for a set of 28 S-alkylcysteine diazomethyl- and chloromethyl-ketone derivatives. Those with the greatest antileukemic activity in the same class correspond to high partial correlations. A periodic classification is performed based on information entropy. The first four characteristics denote the group, and the last two indicate the period. Compounds in the same period and, especially, group present similar properties. The most active substances are situated at the bottom right. Nine classes are distinguished. The principal component analysis of the homologous compounds shows five subclasses included in t…

Quantitative structure–activity relationshipLogarithmStereochemistryprincipal component analysisLymphoblastic LeukemiaPharmaceutical Science01 natural sciencesAnalytical Chemistrylcsh:QD241-44103 medical and health sciences0302 clinical medicinelcsh:Organic chemistryGroup (periodic table)Drug DiscoveryPhysical and Theoretical ChemistryPartial correlationperiodic classificationChemistrypartial correlation diagramOrganic ChemistryDiagraminformation entropy0104 chemical sciences010404 medicinal & biomolecular chemistryChemistry (miscellaneous)030220 oncology & carcinogenesisPrincipal component analysisLipinski's rule of fiveMolecular MedicineMolecules
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Harmonization of QSAR Best Practices and Molecular Docking Provides an Efficient Virtual Screening Tool for Discovering New G-Quadruplex Ligands

2015

Telomeres and telomerase are key players in tumorogenesis. Among the various strategies proposed for telomerase inhibition or telomere uncapping, the stabilization of telomeric G-quadruplex (G4) structures is a very promising one. Additionally, G4 stabilizing ligands also act over tumors mediated by the alternative elongation of telomeres. Accordingly, the discovery of novel compounds able to act on telomeres and/or inhibit the telomerase enzyme by stabilizing DNA telomeric G4 structures as well as the development of approaches efficiently prioritizing such compounds constitute active areas of research in computational medicinal chemistry and anticancer drug discovery. In this direction, we…

Quantitative structure–activity relationshipTelomeraseGeneral Chemical EngineeringDrug Evaluation PreclinicalQuantitative Structure-Activity RelationshipComputational biologyLibrary and Information SciencesBiologyG-quadruplexCrystallography X-RayLigandsMolecular Docking Simulationchemistry.chemical_compoundDrug DiscoveryHumansCell ProliferationGeneticsVirtual screeningMolecular StructureDrug discoveryQSARGeneral ChemistryFibroblastsTelomereComputer Science ApplicationsTelomereG-QuadruplexesMolecular Docking SimulationchemistryAcridinesDNAHeLa Cells
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Age-dependent regulation of antioxidant genes by p38α MAPK in the liver

2018

p38α is a redox sensitive MAPK activated by pro-inflammatory cytokines and environmental, genotoxic and endoplasmic reticulum stresses. The aim of this work was to assess whether p38α controls the antioxidant defense in the liver, and if so, to elucidate the mechanism(s) involved and the age-related changes. For this purpose, we used liver-specific p38α-deficient mice at two different ages: young-mice (4 months-old) and old-mice (24 months-old). The liver of young p38α knock-out mice exhibited a decrease in GSH levels and an increase in GSSG/GSH ratio and malondialdehyde levels. However, old mice deficient in p38α had higher hepatic GSH levels and lower GSSG/GSH ratio than young p38α knock-…

ROS Reactive oxygen species;RSK1 Ribosomal S6 kinase10301 basic medicineMAPK/ERK pathwayAgingHPLC High-performance liquid chromatographyAntioxidantmedicine.medical_treatmentTBP TATA-binding proteinClinical BiochemistryDEN Diethyl nitrosamine;MKP-1 MAPK phosphatase-1IκB kinaseGCLc Glutamate cysteine ligase catalytic subunitp38 Mitogen-Activated Protein KinasesG6PDH Glucose-6-phosphate dehydrogenaseBiochemistryAntioxidantsMicechemistry.chemical_compoundSuperoxide Dismutase-1Akt Protein kinase B0302 clinical medicineNrf2 Nuclear factor erythroid 2-related factor-2IL InterleukinSOD1 Cu/Zn-superoxide dismutaselcsh:QH301-705.5Mice KnockoutMK2 MAP-activated protein kinase 2;PGC-1α Peroxisome proliferator-activated receptor gamma coactivator 1-alphachemistry.chemical_classificationlcsh:R5-920Trx ThioredoxinGlutathione DisulfideTNF-α Tumor necrosis factor-alphabiologyLPS Lipopolysaccharide;GSSG Oxidized glutathione;MEF Mouse embryonic fibroblastsNF-kappa BGstm1 Glutathione S-transferase mu 1CatalaseEndoplasmic Reticulum StressGlutathioneLiverGSH Reduced glutathione;Catalase030220 oncology & carcinogenesisJNK c-Jun N-terminal kinaselcsh:Medicine (General)Research Papermedicine.medical_specialtyNF-E2-Related Factor 2Glutamate-Cysteine LigaseMKK MAPK kinaseAP-1 Activator protein-1IKK IƙB KinaseGene Expression Regulation EnzymologicSuperoxide dismutase03 medical and health sciencesInternal medicineGlutamate cysteine ligaseEGFR Epidermal growth factor receptormedicineAnimalsNuclear factor ƙBAnd catalaseChIP Chromatin immunoprecipitation;Protein kinase BNF-ƙB Nuclear factor kappa BSuperoxide DismutaseSuperoxide dismutase 1Superoxide dismutase 2Organic ChemistryGlutathioneASK1 Apoptosis signal-regulating kinase 1ATF2 activating transcription factor 2;030104 developmental biologyEndocrinologyEnzymeHsp Heat shock proteinlcsh:Biology (General)chemistrybiology.proteinSOD2 Mn-superoxide dismutaseMAPK mitogen activated protein kinaseNEM N-ethyl maleimide;Redox Biology
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The radiosensitization effect of titanate nanotubes as a new tool in radiation therapy for glioblastoma: A proof-of-concept

2013

Abstract Background and purpose One of the new challenges to improve radiotherapy is to increase the ionizing effect by using nanoparticles. The interest of titanate nanotubes (TiONts) associated with radiotherapy was evaluated in two human glioblastoma cell lines (SNB-19 and U87MG). Materials and methods Titanate nanotubes were synthetized by the hydrothermal treatment of titanium dioxide powder in a strongly basic NaOH solution. The cytotoxicity of TiONts was evaluated on SNB-19 and U87MG cell lines by cell proliferation assay. The internalization of TiONts was studied using Transmission Electron Microscopy (TEM). Finally, the effect of TiONts on cell radiosensitivity was evaluated using …

Radiation-Sensitizing AgentsCell SurvivalDNA repairCellApoptosisFlow cytometryCell Line TumormedicineHumansRadiology Nuclear Medicine and imagingRadiosensitivityClonogenic assayCytotoxicityTitaniumNanotubesmedicine.diagnostic_testBrain NeoplasmsChemistryCell growthCell CycleHematologyCell cyclemedicine.anatomical_structureOncologyBiophysicsGlioblastomaReactive Oxygen SpeciesDNA DamageRadiotherapy and Oncology
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A radiosensitizing effect of artesunate in glioblastoma cells is associated with a diminished expression of the inhibitor of apoptosis protein surviv…

2011

Abstract Background and purpose Novel strategies to overcome an irradiation resistant phenotype may help to increase therapeutic efficacy in glioblastoma multiforme. The present study aimed to elucidate radiation sensitizing properties of artesunate, a semi synthetic derivate of artemisinin and to assess factors involved in this effect. Materials and methods LN229 and U87MG cells were treated with various concentrations of artesunate and radiation response was determined by a colony forming assay. Cell numbers, apoptosis induction, cell cycle distribution, and DNA repair following combined modality treatment were monitored by MTT-, caspase 3/7 assay, cytofluorometry, and γ-H2AX foci formati…

Radiation-Sensitizing AgentsDNA RepairCell SurvivalSurvivinArtesunateDown-RegulationCaspase 3ApoptosisInhibitor of apoptosisInhibitor of Apoptosis Proteinschemistry.chemical_compoundCell Line TumorSurvivinHumansRadiology Nuclear Medicine and imagingClonogenic assayDose-Response Relationship DrugBrain NeoplasmsCell CycleHematologyCell cycleArtemisininsXIAPNeoplasm ProteinsOncologychemistryArtesunateApoptosisCancer researchGlioblastomaRadiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology
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Ra223 in bone metastases with osteolytic activity

2018

Radium 223 dichloride (Ra223) is the only targeted alpha therapy able to extend survival in patients with bone metastases from prostate cancer. Mechanism of action and data currently available focused mainly on osteoblastic metastases from prostate cancer. In our institution, a patient with breast cancer affected by osteolytic metastases was treated with off-label use of Ra223. The evaluation of the deposit areas of Ra223 showed a perfect overlap with the regions of osteolysis previously detected by scintigraphy, indicating a possible therapeutic effect. This case report is the first document attesting Ra223 deposit in osteolytic metastases opening new opportunity of therapeutic development…

Radium-223lcsh:Medical physics. Medical radiology. Nuclear medicinemedicine.medical_specialtyOsteolysislcsh:R895-920Case ReportScintigraphy030218 nuclear medicine & medical imagingradium 22303 medical and health sciencesProstate cancer0302 clinical medicineBreast cancerbreast cancerosteoblastic bone metastasesmedicineIn patientRadium-223 Dichlorideosteolytic bone metastasesmedicine.diagnostic_testbusiness.industryBone metastasesTherapeutic effectmedicine.disease030220 oncology & carcinogenesisRadiologybusinessmedicine.drugWorld Journal of Nuclear Medicine
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Two distinct Ca2+ influx pathways activated by the bradykinin B2 receptor.

1996

The hormone-induced depletion of cellular Ca stores provides a signal for the Ca2+ influx into electrically non-excitable cells; however, the underlying molecular mechanisms remain elusive. Therefore, we analyzed bradykinin-activated Ca2+ influx into human foreskin fibroblast cells, HF-15, by fura-2 and 45Ca labeling to discriminate between Ca2+ influx into the fura-sensitive compartment and Ca uptake into fura-insensitive Ca stores. Bradykinin-activated CaZt influx into the fura-sensitive compartment was blocked by inhibitors of NO synthases. These inhibitors also suppressed bradykinin-activated increases in cGMP, indicating that the NO-dependent increase in cGMP is involved in the activat…

Receptor Bradykinin B2BradykininBradykininNitric OxideBiochemistryNitric oxideCell Linechemistry.chemical_compoundmedicineCyclic GMP-Dependent Protein KinasesHumansFibroblastCyclic GMPInterphaseFluorescent DyesIon TransportCell growthChemistryKinaseReceptors BradykininCa2 influxCompartment (chemistry)Calcium Channel BlockersCell biologymedicine.anatomical_structureBiochemistryCytoplasmCalciumFura-2Cell DivisionEuropean journal of biochemistry
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Ligand-induced phosphorylation/dephosphorylation of the endogenous bradykinin B2 receptor from human fibroblasts.

1996

We have studied the ligand-induced phosphorylation/dephosphorylation of the bradykinin B2 receptor endogenously expressed in human HF-15 fibroblasts. An antiserum (AS346) to a synthetic peptide (CRS36), derived from the extreme carboxyl terminus of the human B2 receptor, precipitated the receptor from solubilized membranes of HF-15 cells that had been labeled with [32P]orthophosphate. A low basal level of B2 receptor phosphorylation was found in the absence of a ligand. Stimulation of the cells with the B2 receptor agonists bradykinin, [Lys0,Hyp3]bradykinin, kallidin, and T-kinin resulted in a rapid and efficient phosphorylation of the receptor. The B2 receptor antagonist HOE140 and the B1 …

Receptor Bradykinin B2Receptors BradykininCell BiologyBiologyFibroblastsInterleukin-13 receptorBradykininBiochemistryTropomyosin receptor kinase CMolecular biologyPhosphoric Monoester HydrolasesCell LineEstrogen-related receptor alphaCOS CellsEnzyme-linked receptorConcanavalin AAnimalsHumansProtease-activated receptorProtein phosphorylationElectrophoresis Polyacrylamide GelBradykinin receptorPhosphorylationMolecular BiologyProtease-activated receptor 2The Journal of biological chemistry
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