Search results for "BLAST"

showing 10 items of 2136 documents

Cytomegalovirus Interleukin-10 Expression in Infected Cells Does Not Impair MHC Class I Restricted Peptide Presentation on Bystanding Antigen-Present…

2006

Human cytomegalovirus (HCMV) has evolved strategies to counteract its surveillance by the immune system. Mitigation of antiviral immune responses is considered critical for establishment of viral latency and for spread. Recently, a gene encoding an interleukin-10 homologue (cmvIL-10) has been discovered in the HCMV genome. Using recombinant cmvIL-10, several mostly immunosuppressive functions of the molecule have been described. However, the role of cmvIL-10 in the context of viral infection was not addressed. To be able to analyze this issue, we generated cmvIL- 10-negative viral mutants. Using these mutants, we tested whether the expression of cmvIL-10 by infected cells would render bysta…

Gene Expression Regulation ViralHuman cytomegalovirusvirusesImmunologyCongenital cytomegalovirus infectionAntigen-Presenting CellsCytomegalovirusContext (language use)Viral ProteinsImmune systemVirologyMHC class ImedicineHumansAntigen-presenting cellCells CulturedAntigen PresentationbiologyHistocompatibility Antigens Class IBystander EffectFibroblastsmedicine.diseaseVirologyInterleukin-10CTL*Interleukin 10MutationImmunologybiology.proteinMolecular MedicineGene DeletionViral Immunology
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Epigenetic modifiers are necessary but not sufficient for reprogramming non-myelinating cells into myelin gene-expressing cells.

2010

Background Modifications on specific histone residues and DNA methylation play an essential role in lineage choice and cellular reprogramming. We have previously shown that histone modifications or combinatorial codes of transcription factors (TFs) are critical for the differentiation of multipotential progenitors into myelinating oligodendrocytes. In this study we asked whether combining global manipulation of DNA methylation and histone acetylation together with the expression of oligodendrocyte- specific TFs, was sufficient to switch the identity of fibroblasts into myelin gene-expressing cells. Methodology/Principal Findings Transfection of six oligodendrocyte-specific TFs (Olig1, Olig2…

Gene Expressionlcsh:MedicineBiologyCell LineEpigenesis GeneticHistones03 medical and health sciencesMice0302 clinical medicineHistone H1Histone methylationHistone H2ANeuroscience/Neuronal Signaling MechanismsHistone codeAnimalsCell Lineagelcsh:ScienceCells Cultured030304 developmental biologyEpigenomics0303 health sciencesMultidisciplinaryNeuroscience/Neuronal and Glial Cell BiologyMultipotent Stem Cellslcsh:RAcetylationCell DifferentiationDNA MethylationFibroblastsMolecular biologyChromatinChromatinRatsOligodendrogliaHomeobox Protein Nkx-2.2Histone methyltransferaseNIH 3T3 Cellslcsh:QNeuroscience/Neurobiology of Disease and RegenerationChromatin immunoprecipitation030217 neurology & neurosurgeryMyelin ProteinsResearch ArticleNeuroscienceTranscription FactorsPLoS ONE
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HIF-1α induces MXI1 by alternate promoter usage in human neuroblastoma cells

2009

Adaptation to low oxygen conditions is essential for maintaining homeostasis and viability in oxygen-consuming multi-cellular tissues, including solid tumors. Central in these processes are the hypoxia-inducible transcription factors, HIF-1 and HIF-2, controlling genes involved in e.g. glucose metabolism and neovascularization. Tumor hypoxia and HIF expression have also been associated with a dedifferentiated phenotype and increased aggressiveness. In this report we show that the MAX interactor-1 (MXI1) gene is directly regulated by HIF proteins in neuroblastoma and breast cancer cells. HIF-binding and transactivation were detected within MXI1 gene regulatory sequences in the vicinity of th…

Gene isoformGenes mycBreast NeoplasmsBiologyTransfectionNeuroblastomaTransactivationCell Line TumorNeuroblastomaBasic Helix-Loop-Helix Transcription FactorsmedicineHumansGenes Tumor SuppressorRNA Small InterferingPromoter Regions GeneticGeneTranscription factorOligonucleotide Array Sequence AnalysisBase SequenceTumor hypoxiaTumor Suppressor ProteinsCell BiologyHypoxia-Inducible Factor 1 alpha Subunitmedicine.diseaseCell HypoxiaUp-RegulationGene Expression Regulation NeoplasticHIF1ARegulatory sequenceCancer researchFemaleExperimental Cell Research
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Anti-Inflammatory Action of Heterogeneous Nuclear Ribonucleoprotein A2/B1 in Patients with Autoimmune Endocrine Disorders

2019

Our previous studies documented that human fibroblast-limbal stem cells (f-LSCs) possess immunosuppressive capabilities, playing a role in regulating T-cell activity. This study highlights the molecular activities by which human f-LSCs can attenuate the inflammatory responses of self-reactive peripheral blood mononuclear cells (PBMCs) collected from patients with autoimmune endocrine diseases (AEDs). Anti-CD3 activated PBMCs from twenty healthy donors and fifty-two patients with AEDs were cocultured on f-LSC monolayer. 2D-DIGE proteomic experiments, mass spectrometry sequencing and functional in vitro assays were assessed in cocultured PBMCs. We identified the downmodulation of several huma…

Gene isoformInflammationfibroblast-limbal stem cells Autoimmune Endocrine DiseaseNF-ĸB interaction.Peripheral blood mononuclear cellArticleSettore MED/13 - EndocrinologiaAutoimmune Endocrine Diseases03 medical and health sciencesNF-ĸB interaction0302 clinical medicinemedicineGene silencingIL-2 receptorSettore BIO/06 - Anatomia Comparata E CitologiaHeterogeneous Nuclear Ribonucleoprotein A2/B1hnRNP A2/B1030304 developmental biology0303 health sciencesSettore MED/30 - Malattie Apparato Visivobusiness.industryautoimmunityGeneral Medicinefibroblast-limbal stem cellsSettore BIO/18 - Genetica030220 oncology & carcinogenesisCancer researchfibroblast-limbal stem cells Autoimmune Endocrine Diseasesmedicine.symptomStem cellbusinessCD8immunotoleranceJournal of Clinical Medicine
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Dendritic localization of mammalian neuralized mRNA encoding a protein with transcription repression activities.

2002

Drosophila neurogenic gene neuralized (neu) is required for the maintenance of neuroblast cell fate and differentiation. In the present study we have characterized a mouse and a rat homologue of Drosophila neu. Mammalian neu1 encodes several C-terminal RING zinc finger proteins with one or two neuralized homology repeat (NHR) domains. Mammalian neu1 mRNAs are predominantly expressed in the nervous system and in the skeletal muscle with the highest levels in the adult. In the nervous system neu1 mRNAs are expressed in neurons and dendritically localized in several brain regions, suggesting a role of neu1 in the regulation of synaptic function. Mammalian neu1 isoforms exhibit transcription re…

Gene isoformNervous systemMaleCytoplasmanimal structuresTranscription GeneticUbiquitin-Protein LigasesMolecular Sequence DataNerve Tissue ProteinsBiologyCell fate determinationRats Sprague-DawleyCellular and Molecular NeuroscienceMiceNeuroblastmedicineTumor Cells CulturedAnimalsHumansProtein IsoformsTissue DistributionAmino Acid SequenceRNA MessengerMuscle SkeletalMolecular BiologyGeneZinc fingerCell NucleusMessenger RNAMice Inbred BALB CNeurogenesisBrainGene Expression Regulation DevelopmentalCell BiologyDendritesMolecular biologyRatsRepressor Proteinsmedicine.anatomical_structureFemaleMolecular and cellular neurosciences
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Expression of the gene of the alpha-smooth muscle-actin isoform in rat liver and in rat fat-storing (ITO) cells.

1990

Fat storing cells (FSCs) in the liver represent the main site of vitamin A deposition in the body. These cells are considered to play an important role during scar formation and fibrogenesis in the liver. The putative descent of FSCs from the fibroblastic or from the myofibroblastic system have not been determined yet by morphological or immunohistochemical studies. To further define the origin of these liver cells, we analysed the pattern of expression of three structural proteins: vimentin, desmin and the α-smooth muscle (SM)-actin isoform in FSCs of the rat liver, in smooth muscle cells (SMCs) from the aorta and in rat skin fibroblasts. FSCs were studied by immunohistochemical methods im…

Gene isoformPathologymedicine.medical_specialtyFluorescent Antibody TechniqueGene ExpressionVimentinmacromolecular substancesBiologyDesminImmunoenzyme TechniquesNecrosisGene expressionmedicineAnimalsVimentinNorthern blotActinAortaCells CulturedImmunoperoxidaseNucleic Acid HybridizationMuscle SmoothRats Inbred StrainsGeneral MedicineFibroblastsLipid MetabolismMolecular biologyActinsRatsLiverHepatic stellate cellbiology.proteinRNADesminFemaleVirchows Archiv. B, Cell pathology including molecular pathology
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Kinetics of expression of prion protein in uninfected and scrapie-infected N2a mouse neuroblastoma cells.

1993

The scrapie prion protein, PrPSc, is formed from its isoform, the cellular PrPc. There is evidence available indicating that PrPSc is necessary component of the infectious prion particle to cause a series of transmissible spongiform encephalopathies. We have used immunocytochemistry and RNA blotting techniques to investigate if infection with prions results in an increased PrP gene expression. For the experiments we used N2a cells which had been infected with prions (ScN2a cells). We demonstrated by confocal laser scanning microscopy that PrP-protein was present in the nucleus (predominantly in the nucleoli) of ScN2a cells. Analysis of the PrP-mRNA levels both in N2a- and in ScN2a cells usi…

Gene isoformPrPSc ProteinsTranscription GeneticNucleolusPrionsanimal diseasesClinical BiochemistryCellImmunocytochemistryGene ExpressionScrapieNerve Tissue ProteinsBiologyBiochemistryMiceNeuroblastomaGene expressionmedicineTumor Cells CulturedAnimalsNorthern blotRNA MessengerCell NucleusMessenger RNACell BiologyGeneral MedicineMolecular biologynervous system diseasesKineticsmedicine.anatomical_structureCell NucleolusCell biochemistry and function
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Selective Activation of Trophoblast-specific PLAC1 in Breast Cancer by CCAAT/Enhancer-binding Protein β (C/EBPβ) Isoform 2

2009

The trophoblast-specific gene PLAC1 (placenta-specific 1) is ectopically expressed in a wide range of human malignancies, most frequently in breast cancer, and is essentially involved in cancer cell proliferation, migration, and invasion. Here we show that basal activity of the PLAC1 promoter is selectively controlled by ubiquitous transcription factor SP1 and isoform 2 of CCAAT/enhancer-binding protein beta that we found to be selectively expressed in placental tissue and cancer cells. Binding of both factors to their respective elements within the PLAC1 promoter was essential to attain full promoter activity. Estrogen receptor alpha (ERalpha) signaling further augmented transcription and …

Gene isoformSp1 Transcription FactorMolecular Sequence DataEstrogen receptorBreast NeoplasmsPregnancy ProteinsBiologyBiochemistryTransactivationMolecular Basis of Cell and Developmental BiologyTranscription (biology)Cell Line TumorGene expressionHumansProtein IsoformsPromoter Regions GeneticMolecular BiologyCell ProliferationSp1 transcription factorBase SequenceCcaat-enhancer-binding proteinsCCAAT-Enhancer-Binding Protein-betaEstrogen Receptor alphaEstrogensCell BiologyMolecular biologyTrophoblastsGene Expression Regulation NeoplasticChromatin immunoprecipitationJournal of Biological Chemistry
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Hsp70 is required for optimal cell proliferation in mouse A6 mesoangioblast stem cells.

2009

Mouse Hsp70 (70 kDa heat shock protein) is preferentially induced by heat or stress stimuli. We previously found that Hsp70 is constitutively expressed in A6 mouse mesoangioblast stem cells, but its possible role in these cells and the control of its basal transcription remained unexplored. Here we report that in the absence of stress, Ku factor is able to bind the HSE (heat shock element) consensus sequence in vitro, and in vivo it is bound to the proximal hsp70 promoter. In addition, we show that constitutive hsp70 transcription depends on the co-operative interaction of different factors such as Sp1 (specificity protein 1) and GAGA-binding protein with Ku factor, which binds the HSE cons…

Gene knockdownMesoangioblastBinding SitesGeneral transcription factorCell growthStem CellsCell BiologyBiologyFlow CytometryBiochemistryMolecular biologyHsp70MiceTranscription (biology)Heat shock proteinAnimalsBlood VesselsHSP70 Heat-Shock ProteinsRNA InterferenceStem cellmesoangioblast RNAi doubling timePromoter Regions GeneticMolecular BiologyCell ProliferationTranscription FactorsThe Biochemical journal
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Subtypes of glial cells in the Drosophila embryonic ventral nerve cord as related to lineage and gene expression

2008

In the Drosophila embryonic CNS several subtypes of glial cells develop, which arrange themselves at characteristic positions and presumably fulfil specific functions. The mechanisms leading to the specification and differentiation of glial subtypes are largely unknown. By DiI labelling in glia-specific Gal4 lines we have clarified the lineages of the lateral glia in the embryonic ventral nerve cord and linked each glial cell to a specific stem cell. For the lineage of the longitudinal glioblast we show that it consists of 9 cells, which acquire at least four different identities. A large collection of molecular markers (many of them representing transcription factors and potential Gcm targ…

Genetic MarkersEmbryologyLineage (genetic)CellBiologyNervous SystemCell LineGlioblastCell MovementPeripheral Nervous SystemmedicineAnimalsCluster AnalysisCell LineageTranscription factorIn Situ HybridizationCell MembraneGene Expression Regulation DevelopmentalCell DifferentiationAnatomyEmbryonic stem cellCell biologyNeuroepithelial cellDrosophila melanogastermedicine.anatomical_structureGenetic Techniquesnervous systemVentral nerve cordStem cellNeurogliaDevelopmental BiologyMechanisms of Development
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