Search results for "BPS"

showing 8 items of 28 documents

Melanoma cells release extracellular vesicle which contain H1° linker histone as well as RNA-binding proteins which bind to the H1° mRNA

2015

We previously demonstrated that G26/24 oligodendroglioma cells release EVs that contain proteins, such as FasL and TRAIL, which induce apoptosis in rat cortical neurons [1] and astrocytes [2]. We also reported that cancer cells use EVs for transferring, into the environment [3], proteins such as extracellular matrix remodelling proteases [4], and H1°, a differentiation-specific histone [5]. In particular, by releasing H1°, cells could escape differentiation cues [5]. To verify the role of EVs in releasing specific proteins and mRNAs, in this study we used as a model A375 melanoma cells. METHODS EVs were purified from cell culture media as previously reported [1, 2]. T1 RNase-protection assa…

Settore BIO/10 - BiochimicaOligodendroglioma cells extracellular vesicles (EVS) histone H1.0 RNA-binding proteins (RBPs) myelin expression factor-2 (MYEF2)Settore BIO/06 - Anatomia Comparata E Citologia
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Expression and intracellular localization of H1° mRNA-containing complexes in developing rat brain and astrocytes

2015

INTRODUCTION: Post-transcriptional regulation of gene expression relies on RNA-binding proteins (RBPs), which regulate intracellular transport, stability, and translation of mRNAs [1]. We previously identified a set of proteins which interact with mRNAs encoding H1° and H3.3 histones [2-5]. All these proteins are probably part of a ribonucleoprotein particle [6]. Here we report more details on the expression and intracellular localization of some of these RBPs, during rat brain development and in isolated rat astrocytes. METHODS: Affinity chromatography was performed as already described [6]. Preparation of total lysates and cellular sub-fractions was done as reported in [3]. Possible co-lo…

Settore BIO/10 - BiochimicaPost-transcriptional regulation RNA-binding proteins (RBPs) H1° and H3.3 histones variants CSD-C2.Settore BIO/06 - Anatomia Comparata E Citologia
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Rateless Codes Performance Analysis in Correlated Channel Model for GEO Free Space Optics Downlinks

2012

Settore ING-INF/03 - TelecomunicazioniFree Space Optics (FSO) technologies for satellite communications offer several advantages: wide bandwidth high rate capability immunity to electromagnetic interference and small equipment size. Thus they are suitable for inter-satellite links deep space communications and also for high data rate ground-to-satellite/satellite-to-ground communications. Nevertheless FSO links suffer impairments that cause power signal degradation at the receiver. Scattering and absorption cause power signal attenuations predictable by suitable deterministic models. Optical turbulence causes random irradiance fluctuations which can generate signal fading events and can thereby only be predicted by statistical models. Attenuation and fading events can corrupt FSO links and so it would be recommended to add mitigation error codes on the communication link. FSO channel can be described as an erasure channel: fading events can cause erasure errors. We have identified in rateless codes (RCs) a suitable solution to be employed in FSO links. RCs do not need feedback and they add a redundant coding on the source data that allows the receiver to recover the whole payload despite erasure errors. We implemented two different of rateless codes: Luby Transform (LT) and Raptor. We analyzed their performances on a simulated turbulent GEO FSO downlink (1 Gbps - OOK modulation) at a 106 μm wavelength and for different values of zenith angles. Assuming a plane-wave propagation and employing Hufnagel-Valley we modeled the downlink using: 1) a temporal correlated channel model based on Gamma-Gamma probability distribution and 2) an irradiance covariance function that we converted on a time function using Taylor frozen eddies hypothesis. Our new channel model is able to simulate irradiance fluctuations at different turbulence conditions as it will be shown in the full paper. We will also report performance results of LT and Raptor codes at overhead range varying between 0 and 50% and for different values of source packets.Settore ING-INF/01 - Elettronica
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Extracellular membrane vesicles can mediate intercellular transfer of molecules

2012

Many eukaryotic cell types, including neural cells, release into the extracellular environment vesicles of different sizes and composition. Neurons and astrocytes shed extracellular vesicles which contain FGF2 and VEGF and could be involved in interaction with endothelial cells, to form the blood-brain barrier. Also brain tumor cells, such as glioblastomas, release vesicles in the extracellular space. Microvesicles (MVs) shed from G26/24 oligodendro¬glioma cells were previously reported to contain FAS-L and to cause a reproducible, dose-dependent, inhibitory effect on neurite outgrowth, and neuronal apoptosis, when added to primary cultures of rat cortical neurons. More recently, they were …

apoptosis astrocyteRBPsSettore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E Citologiaoligodendrogliomashedding vesicle
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RNA-Binding Proteins as Epigenetic Regulators of Brain Functions and Their Involvement in Neurodegeneration.

2022

A central aspect of nervous system development and function is the post-transcriptional regulation of mRNA fate, which implies time- and site-dependent translation, in response to cues originating from cell-to-cell crosstalk. Such events are fundamental for the establishment of brain cell asymmetry, as well as of long-lasting modifications of synapses (long-term potentiation: LTP), responsible for learning, memory, and higher cognitive functions. Post-transcriptional regulation is in turn dependent on RNA-binding proteins that, by recognizing and binding brief RNA sequences, base modifications, or secondary/tertiary structures, are able to control maturation, localization, stability, and tr…

learningsynaptic plasticityOrganic ChemistryneurodegenerationRNA-Binding ProteinsBrainGeneral MedicineCatalysisComputer Science ApplicationsmemoryInorganic ChemistryIntrinsically Disordered ProteinsGene Expression RegulationSettore BIO/10 - BiochimicaRNA-binding proteins (RBPs)Settore MED/26 - NeurologiaNervous System Physiological PhenomenaRNA Messengerpost-transcriptional regulation of gene expressionSettore BIO/06 - Anatomia Comparata E CitologiaPhysical and Theoretical ChemistryEVsMolecular Biologyintrinsically disordered regions (IDRs)SpectroscopyInternational journal of molecular sciences
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Thyroid hormones induce sumoylation of the cold shock domain-containing protein PIPPin in developing rat brain and in cultured neurons.

2006

We previously identified a cold shock domain (CSD)-containing protein (PIPPin), expressed at high level in brain cells. PIPPin has the potential to undergo different post-translational modifications and might be a good candidate to regulate the synthesis of specific proteins in response to extracellular stimuli. Here we report the effects of thyroid hormone (T3) on PIPPin expression in developing rat brain. We found that a significant difference among euthyroid- and hypothyroid- newborn rats concerns sumoylation of nuclear PIPPin, that is abolished by hypothyroidism. Moreover, T3-dependence of PIPPin sumoylation has been confirmed in cortical neurons purified from brain cortices and culture…

medicine.medical_specialtySUMO-1 ProteinSUMO proteinDeveloping rat brainNerve Tissue ProteinsEndocrinologyAntithyroid AgentsHypothyroidismPregnancyInternal medicinemedicineExtracellularAnimalsRats WistarCells CulturedCell NucleusCerebral CortexNeuronsbiologyRNA-Binding ProteinsCold-shock domainChromatinProtein Structure TertiaryRatsThyroid hormoneChemically defined mediumCell nucleusmedicine.anatomical_structureHistoneEndocrinologyAnimals NewbornPropylthiouracilPrenatal Exposure Delayed Effectsbiology.proteinTriiodothyronineRNA-binding proteins (RBPs)FemaleRabbitsNucleusEndocrinology
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Onset of MRONJ in Breast Cancer Patients after Switching from Low to High Dose of Bone Modifying Agents Due to Bone Metastases Development: A Single …

2022

Background: Medication-Related Osteonecrosis of the Jaw (MRONJ) is an adverse drug reaction mainly associated to bone modifying agents (BMAs). Breast cancer (BC) is the most frequent cancer worldwide. Its therapy can cause cancer treatment-induced bone loss (CTIBL), commonly treated with BMAs. The aims of this retrospective study are: to describe characteristics of BC patients under BMAs for CTIBL; to record any switch to high-dose BMAs; to assess MRONJ onset and to identify any factors associated with it. Patients: Authors included patients referred for MRONJ prevention to the Unit of Oral Medicine (University Hospital of Palermo). Results: Fourteen female BC patients under low-dose BMAs f…

osteonecrosis of the jawbisphosphonatebreast cancerBPosteonecrosis of the jaw; MRONJ; cancer treatment-induced bone loss; CTIBL; breast cancer; osteoporosis; bone metastases; bisphosphonates; BPs; denosumabcancer treatment-induced bone loosteoporosidenosumabPharmacology (medical)MRONJCTIBLbone metastaseOral; Volume 2; Issue 4; Pages: 274-285
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Mathematical models and simulation of Costas loops

2013

vaihelukitut silmukatCostas-silmukatCostas loopQPSK Costas loopPLLphase-locked loopGPSelektroniset piiritsimulointicarrier trackingmatemaattiset mallitBPSK Costas loop
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