Search results for "Benzamides"

showing 9 items of 139 documents

In vitro affinities of various halogenated benzamide derivatives as potential radioligands for non-invasive quantification of D(2)-like dopamine rece…

2007

Abstract Benzamide derivatives as radiotracers have played an important role in diagnosing malfunction in dopaminergic neurotransmission. A variety of halogenated and two unsubstituted benzamide derivatives were synthesised and their in vitro affinities to dopaminergic, serotonergic and adrenergic receptors and their lipophilicities were determined. As references IBZM (3), raclopride (4) and FLB457 (5) were tested as well. The two iodinated compounds NAE (27) and NADE (28) displayed Ki values of 0.68 and 14 nM for the D2 receptor. The well-established radiotracers FP (1) and DMFP (2) showed affinities in the same range as did the brominated compounds NABrE (29) and NABrDE (30). The log D7.4…

StereochemistryClinical BiochemistryPharmaceutical ScienceBiochemistryCell Linechemistry.chemical_compoundRadioligand AssayStructure-Activity RelationshipDopamine receptor D2Iodine IsotopesDrug DiscoverymedicineAnimalsBenzamideMolecular BiologyRacloprideReceptors Dopamine D2Organic ChemistryDopaminergicLigand (biochemistry)AffinitieschemistryDopamine receptorLipophilicityBenzamidesMolecular Medicinemedicine.drugBioorganicmedicinal chemistry
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How Does the Benzamide Antipsychotic Amisulpride get into the Brain?—An In Vitro Approach Comparing Amisulpride with Clozapine

2003

This study evaluated the disposition of the two atypical antipsychotics, amisulpride (AMS) and clozapine (CLZ), and its main metabolite N-desmethylclozapine (DCLZ), to their target structures in the central nervous system by applying an in vitro blood-brain barrier and blood-cerebrospinal fluid (CSF) barrier based on monolayers of porcine brain microvessel endothelial cells (PMEC) or porcine choroid plexus epithelial cells (PCEC). Permeation studies through PMEC- and PCEC-monolayers were conducted for 60 min at drug concentrations of 1, 5, 10, and 30 muM applied to the donor compartment. PMEC were almost impermeable for AMS (permeation coefficient, P1 x 10(-7) cm/s) in the resorptive direct…

SwineMetabolitePharmacologyBlood–brain barrierchemistry.chemical_compoundmedicineAnimalsHumansAmisulprideBenzamideClozapineClozapinePharmacologyDose-Response Relationship DrugAntagonistBrainPermeationPsychiatry and Mental healthmedicine.anatomical_structurechemistryBenzamidesChoroid plexusAmisulprideCaco-2 CellsSulpirideAntipsychotic Agentsmedicine.drugNeuropsychopharmacology
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Synthesis and biological evaluation of some new 2-phenylpropiolamidobenzamides as potential antagonists of the HDM2-p53 protein-protein interactions

2009

antitumoral activity anticancer protein-protein interactions 2-phenylpropiolamidobenzamides synthesis
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Daidzein has neuroprotective effects through ligand-binding-independent PPARγ activation.

2011

Phytoestrogens are a group of plant-derived compounds that include mainly isoflavones like daidzein. Phytoestrogens prevent neuronal damage and improve outcome in experimental stroke; however, the mechanisms of this neuroprotective action have not been fully elucidated. In this context, it has been postulated that phytoestrogens might activate the peroxisome proliferator-activated receptor-γ (PPARγ), which exerts neuroprotective effects in several settings. The aim of this study was to determine whether the phytoestrogen daidzein elicits beneficial actions in neuronal cells by mechanisms involving activation of PPARγ. Our results show that daidzein (0.05-5 μM) decreases cell death induced b…

endocrine systemmedicine.drug_classPyridinesPeroxisome proliferator-activated receptorPharmacologyLigandsNeuroprotectionCellular and Molecular Neurosciencechemistry.chemical_compoundmedicineSynaptic vesicle recyclingAnimalsReceptorCells Culturedchemistry.chemical_classificationNeuronsDaidzeinfood and beveragesCell BiologyIsoflavonesReceptor antagonistIsoflavonesRatsOxygenPPAR gammaGlucoseNeuroprotective AgentschemistryBenzamidesPhytoestrogensNeurochemistry international
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V gamma 9V delta 2 T lymphocytes efficiently recognize and kill zoledronate-sensitized, imatinib-sensitive, and imatinib-resistant chronic myelogenou…

2010

Abstract Imatinib mesylate (imatinib), a competitive inhibitor of the BCR-ABL tyrosine kinase, is highly effective against chronic myelogenous leukemia (CML) cells. However, because 20–30% of patients affected by CML display either primary or secondary resistance to imatinib, intentional activation of Vγ9Vδ2 T cells by phosphoantigens or by agents that cause their accumulation within cells, such as zoledronate, may represent a promising strategy for the design of a novel and highly innovative immunotherapy capable to overcome imatinib resistance. In this study, we show that Vγ9Vδ2 T lymphocytes recognize, trogocytose, and efficiently kill imatinib-sensitive and -resistant CML cell lines pre…

gamma delta T cells Imatinib Leukemia cellsAdultmedicine.medical_treatmentImmunologyMice SCIDLymphocyte ActivationZoledronic AcidPiperazinesMicehemic and lymphatic diseasesLeukemia Myelogenous Chronic BCR-ABL PositivemedicineImmunology and AllergyAnimalsHumansneoplasmsCells CulturedDiphosphonatesbusiness.industryImidazolesImatinibReceptors Antigen T-Cell gamma-deltaImmunotherapymedicine.diseaseIn vitroCoculture TechniquesDrug Resistance MultipleLeukemiaImatinib mesylatePyrimidinesCell cultureDrug Resistance NeoplasmImmunologyBenzamidesCancer researchImatinib MesylatebusinessK562 CellsTyrosine kinasemedicine.drugChronic myelogenous leukemiaT-Lymphocytes CytotoxicJournal of immunology (Baltimore, Md. : 1950)
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Mutant HRAS as novel target for MEK and mTOR inhibitors.

2015

HRAS is a frequently mutated oncogene in cancer. However, mutant HRAS as drug target has not been investigated so far. Here, we show that mutant HRAS hyperactivates the RAS and the mTOR pathway in various cancer cell lines including lung, bladder and esophageal cancer. HRAS mutation sensitized toward growth inhibition by the MEK inhibitors AZD6244, MEK162 and PD0325901. Further, we found that MEK inhibitors induce apoptosis in mutant HRAS cell lines but not in cell lines lacking RAS mutations. In addition, knockdown of HRAS by siRNA blocked cell growth in mutant HRAS cell lines. Inhibition of the PI3K pathway alone or in combination with MEK inhibitors did not alter signaling nor had an imp…

mTOR inhibitorMutantBlotting Western610 Medicine & healthApoptosisMice SCIDCell LineProto-Oncogene Proteins p21(ras)chemistry.chemical_compoundCell Line TumorNeoplasmsMedicineAnimalsHumansHRASHRAS mutationsProtein Kinase InhibitorsPI3K/AKT/mTOR pathwayCell ProliferationGeneticsMitogen-Activated Protein Kinase KinasesMEK inhibitorOncogeneCell growthbusiness.industryMEK inhibitorTOR Serine-Threonine KinasesDiphenylamineXenograft Model Antitumor AssaysTumor Burdenlung cancer10219 Clinic for Gastroenterology and HepatologyCell Transformation NeoplasticOncologychemistry10032 Clinic for Oncology and HematologyBenzamidesMutationCancer researchbladder cancer2730 OncologyBenzimidazolesRNA InterferenceSignal transductionGrowth inhibitionbusinessSignal TransductionResearch PaperOncotarget
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Endocannabinoid anandamide mediates hypoxic pulmonary vasoconstriction

2013

Endocannabinoids are important regulators of organ homeostasis. Although their role in systemic vasculature has been extensively studied, their impact on pulmonary vessels remains less clear. Herein, we show that the endocannabinoid anandamide (AEA) is a key mediator of hypoxic pulmonary vasoconstriction (HPV) via fatty acid amide hydrolase (FAAH)-dependent metabolites. This is underscored by the prominent vasoconstrictive effect of AEA on pulmonary arteries and strongly reduced HPV in FAAH(-/-) mice and wild-type mice upon pharmacological treatment with FAAH inhibitor URB597. In addition, mass spectrometry measurements revealed a clear increase of AEA and the FAAH-dependent metabolite arac…

medicine.medical_specialtyPolyunsaturated Alkamidesmedicine.medical_treatmentHypertension PulmonaryBlotting WesternMyocytes Smooth MuscleArachidonic AcidsBiologyAmidohydrolaseschemistry.chemical_compoundMiceFatty acid amide hydrolaseInternal medicineHypoxic pulmonary vasoconstrictionmedicineAnimalsHypoxiaLungDNA PrimersMice KnockoutAnalysis of VarianceMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionAnandamideHypoxia (medical)URB597Biological Sciencesmedicine.diseaseEndocannabinoid systemPulmonary hypertensionImmunohistochemistryEndocrinologynervous systemchemistryVasoconstrictionBenzamideslipids (amino acids peptides and proteins)CannabinoidCarbamatesmedicine.symptompsychological phenomena and processesChromatography LiquidEndocannabinoidsSignal Transduction
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D2-receptor imaging with [123I]IBZM and Single Photon Emission Tomography in psychiatry: a survey of current status

1995

D2-dopamine receptors can be visualized in the human brain in vivo by Single Photon Emission Tomography (SPECT) and the radiolabeled benzamide [123I]IBZM. The present paper reviews the current status of this type of functional brain imaging with respect to basic methodological aspects, data analysis and quantification. The results from published clinical studies in different psychiatric patient populations and normal controls with [123I]IBZM are reviewed. [123I]IBZM-SPECT is a powerful tool for the investigation of D2-dopamine receptor status in psychiatric disorders, different types of drug treatment as well as therapeutic and side effects of pharmacologic agents. However, there still is a…

medicine.medical_specialtyReceptor StatusPyrrolidinesNeurologyContrast MediaDrug treatmentDopamine receptor D2Image Processing Computer-AssistedmedicineSingle Photon Emission TomographyHumansPsychiatryBiological PsychiatryBrain ChemistryTomography Emission-Computed Single-Photonmedicine.diagnostic_testReceptors Dopamine D2business.industryBrainHuman brainPsychiatry and Mental healthmedicine.anatomical_structureNeurology123i ibzmPositron emission tomographyBenzamidesDopamine AntagonistsNeurology (clinical)Nuclear medicinebusinessPsychologyJournal of Neural Transmission
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Synthesis and biological evaluation of some new 2-phenylpropiolamidobenzamides as potential antagonists of the HMDM2-p53 protein-protein interactions

2009

phenylpropiolamidobenzamides/antiproliferative activitySettore CHIM/08 - Chimica Farmaceutica
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