Search results for "Biochemistry"

showing 10 items of 20172 documents

Assessment of Cardiovascular Risk and Prevention of Cardiovascular Disease in Women with the Polycystic Ovary Syndrome: A Consensus Statement by the …

2010

Women with polycystic ovary syndrome (PCOS) often have cardiovascular disease (CVD) risk factors. The Androgen Excess and Polycystic Ovary Syndrome (AE-PCOS) Society created a panel to provide evidence-based reviews of studies assessing PCOS-CVD risk relationships and to develop guidelines for preventing CVD.An expert panel in PCOS and CVD reviewed literature and presented recommendations.Only studies comparing PCOS with control patients were included. All electronic databases were searched; reviews included individual studies/databases, systematic reviews, abstracts, and expert data. Articles were excluded if other hyperandrogenic disorders were not excluded, PCOS diagnosis was unclear, co…

medicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and MetabolismClinical BiochemistryMEDLINEDiseaseAndrogen ExcessRisk AssessmentBiochemistryBody Mass IndexEndocrinologyRisk FactorsDiabetes mellitusInternal medicineGlucose IntoleranceHumansMedicineWaist-Hip Ratiobusiness.industryBiochemistry (medical)nutritional and metabolic diseasesAtherosclerosismedicine.diseasePolycystic ovaryfemale genital diseases and pregnancy complicationsEndocrinologySystematic reviewDiabetes Mellitus Type 2Cardiovascular DiseasesFemaleRisk assessmentbusinessBody mass indexPolycystic Ovary SyndromeThe Journal of Clinical Endocrinology & Metabolism
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Mitochondrial complex I impairment in leukocytes from polycystic ovary syndrome patients with insulin resistance.

2009

Insulin resistance is a feature of polycystic ovary syndrome (PCOS) and is related to mitochondrial function.Our objective was to assess mitochondrial function by evaluating mitochondrial oxygen (O(2)) consumption, reactive oxygen species (ROS) production, levels of glutathione (GSH), the oxidized glutathione/GSH ratio, TNFalpha levels, and membrane potential. Additionally, we have evaluated mitochondrial complex I as a target of the oxidative stress responsible for PCOS in polymorphonuclear cells.This was a prospective controlled study conducted in an academic medical center.The study population consisted of 20 lean reproductive-age women with PCOS and 20 body composition-matched controls.…

medicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryContext (language use)BiologyMitochondrionmedicine.disease_causeBiochemistrychemistry.chemical_compoundEndocrinologyInsulin resistanceOxygen ConsumptionInternal medicinemedicineLeukocytesHumanschemistry.chemical_classificationMembrane Potential MitochondrialReactive oxygen speciesElectron Transport Complex ITumor Necrosis Factor-alphaInsulinBiochemistry (medical)nutritional and metabolic diseasesGlutathionemedicine.diseasePolycystic ovaryGlutathionefemale genital diseases and pregnancy complicationsEndocrinologychemistryFemaleInsulin ResistanceReactive Oxygen SpeciesOxidative stressPolycystic Ovary Syndrome
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Phenotypic variation in hyperandrogenic women influences the findings of abnormal metabolic and cardiovascular risk parameters.

2005

In hyperandrogenic women, several phenotypes may be observed. This includes women with classic polycystic ovary syndrome (C-PCOS), those with ovulatory (OV) PCOS, and women with idiopathic hyperandrogenism (IHA), which occurs in women with normal ovaries. Where other causes have been excluded, we categorized 290 hyperandrogenic women who were seen consecutively for this complaint between 1993 and 2004 into these three subgroups. The aim was to compare the prevalence of obesity, insulin resistance, and dyslipidemia as well as increases in C-reactive protein and homocysteine in these different phenotypes with age-matched ovulatory controls of normal weight (n = 85) and others matched for body…

medicine.medical_specialtyendocrine system diseasesHomocysteineEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryBiochemistryBody Mass IndexNORMAL MENSESchemistry.chemical_compoundEndocrinologyInsulin resistanceRisk FactorsInternal medicineparasitic diseasesmedicineHumansInsulinANDROGEN EXCESSDEHYDROEPIANDROSTERONE SULFATEPLASMAbusiness.industryInsulinBiochemistry (medical)Hyperandrogenismnutritional and metabolic diseasesCholesterol LDLPOLYCYSTIC-OVARY-SYNDROMELuteinizing Hormonemedicine.diseaseObesityPolycystic ovaryfemale genital diseases and pregnancy complicationsEndocrinologyC-Reactive ProteinPhenotypechemistryCardiovascular DiseasesFemaleSENSITIVITYInsulin ResistancebusinessBody mass indexDyslipidemiaPolycystic Ovary SyndromeThe Journal of clinical endocrinology and metabolism
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Determinants of elevated carbohydrate antigen 125 in patients with severe symptomatic aortic valve stenosis referred for transcatheter aortic valve i…

2018

Elevated carbohydrate antigen 125 (CA125) predicts adverse outcome after transcatheter aortic valve implantation (TAVI). While known underlying pathophysiological mechanisms of elevated CA125 include serosal effusions and inflammatory stimuli, clinical determinants associated with elevated CA125 in patients referred for TAVI remain unknown. Therefore, we investigated clinical, laboratory and echocardiographic determinants of elevated CA125 in patients with severe aortic valve stenosis referred for TAVI.This study includes 650 patients with severe aortic stenosis referred for TAVI. Baseline CA125 was determined by an immunoassay and dichotomized (elevated versus normal) based on the manufact…

medicine.medical_specialtyendocrine system diseasesTranscatheter aorticAdverse outcomesHealth Toxicology and MutagenesisClinical Biochemistry030204 cardiovascular system & hematologyRisk AssessmentBiochemistryVentricular Function LeftNyha classTranscatheter Aortic Valve ReplacementHemoglobins03 medical and health sciences0302 clinical medicineRisk FactorsInternal medicineHumansMedicineIn patient030212 general & internal medicineAgedAged 80 and overbusiness.industryAortic Valve Stenosismedicine.diseaseTricuspid Valve Insufficiencyfemale genital diseases and pregnancy complicationsPathophysiologyStenosisC-Reactive ProteinCA-125 AntigenAortic valve stenosisCardiologybusinessCarbohydrate antigenBiomarkers
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DHEA, DHEAS and PCOS.

2014

Approximately 20-30% of PCOS women demonstrate excess adrenal precursor androgen (APA) production, primarily using DHEAS as a marker of APA in general and more specifically DHEA, synthesis. The role of APA excess in determining or causing PCOS is unclear, although observations in patients with inherited APA excess (e.g., patients with 21-hydroxylase deficient congenital classic or non-classic adrenal hyperplasia) demonstrate that APA excess can result in a PCOS-like phenotype. Inherited defects of the enzymes responsible for steroid biosynthesis, or defects in cortisol metabolism, account for only a very small fraction of women suffering from hyperandrogenism or APA excess. Rather, women wi…

medicine.medical_specialtyendocrine system diseasesmedicine.drug_classEndocrinology Diabetes and Metabolismmedicine.medical_treatmenteducationClinical BiochemistryPopulationSingle-nucleotide polymorphismSteroid biosynthesisBiochemistryBody Mass IndexEndocrinologyRisk FactorsInternal medicinemental disordersmedicinePrevalenceAnimalsHumanseducationMolecular Biologyeducation.field_of_studybusiness.industryDehydroepiandrosterone SulfateInsulinHyperandrogenismCell BiologyDehydroepiandrosteroneHyperplasiaAndrogenmedicine.diseaseObesityEndocrinologyPhenotypeCardiovascular DiseasesAndrogensMolecular MedicineFemaleSteroidsbusinessHyperandrogenismpsychological phenomena and processesPolycystic Ovary SyndromeThe Journal of steroid biochemistry and molecular biology
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Controlled Ovarian Stimulation Induces a Functional Genomic Delay of the Endometrium with Potential Clinical Implications

2008

Context: Controlled ovarian stimulation induces morphological, biochemical, and functional genomic modifications of the human endometrium during the window of implantation. Objective: Our objective was to compare the gene expression profile of the human endometrium in natural vs. controlled ovarian stimulation cycles throughout the early-mid secretory transition using microarray technology. Method: Microarray data from 49 endometrial biopsies obtained from LH+1 to LH+9 (n = 25) in natural cycles and from human chorionic gonadotropin (hCG) +1 to hCG+9 in controlled ovarian stimulation cycles (n = 24) were analyzed using different methods, such as clustering, profiling of biological processes…

medicine.medical_specialtyendocrine systemEndocrinology Diabetes and Metabolismmedia_common.quotation_subjectClinical BiochemistryStimulationLuteal PhaseBiologyEndometriumChorionic GonadotropinBiochemistryHuman chorionic gonadotropinEndometriumEndocrinologyOvulation InductionReference ValuesInternal medicinemedicineHumansMenstrual CycleMenstrual cycleOligonucleotide Array Sequence Analysismedia_commonRegulation of gene expressionGlutathione PeroxidaseGenome HumanReverse Transcriptase Polymerase Chain ReactionMicroarray analysis techniquesurogenital systemBiochemistry (medical)Luteinizing HormoneInsulin-Like Growth Factor Binding ProteinsGene expression profilingInsulin-Like Growth Factor Binding Protein 3Endocrinologymedicine.anatomical_structureGene Expression RegulationGene chip analysisRNAFemaleAlgorithms
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Oncostatin M, leukaemia-inhibitory factor and interleukin 6 trigger different effects on alpha1-proteinase inhibitor synthesis in human lung-derived …

1998

Interleukin 6 (IL-6), oncostatin M (OSM) and leukaemia-inhibitory factor (LIF) share a common signal-transducing subunit in each of their receptors and thus mediate an overlapping spectrum of biological activities. Although all of these cytokines stimulate the production of α1-proteinase inhibitor (α1-PI) in hepatocyte-derived cells, only OSM is able to up-regulate levels of this inhibitor in epithelial cells originating from the lung. In this study we characterized human lung-derived epithelial-like HTB58 cells for their ability to synthesize α1-PI after treatment with IL-6, OSM and LIF. The results demonstrate that the resistance of HTB58 cells to the effects of IL-6 and LIF was not becau…

medicine.medical_specialtyendocrine systemProtein subunitBlotting WesternOncostatin MInhibitory postsynaptic potentialBiochemistryLeukemia Inhibitory FactorInternal medicinemedicineTumor Cells CulturedHumansInterleukin 6ReceptorMolecular BiologyLungLymphokinesbiologyChemistryInterleukin-6fungiOncostatin MOncostatin M receptorEpithelial CellsCell BiologyGlycoprotein 130Blotting NorthernGrowth InhibitorsCell biologyInterleukin 31Endocrinologyalpha 1-Antitrypsinbiology.proteinPeptidesResearch ArticleThe Biochemical journal
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Thyrotropin Receptor Blocking Antibodies.

2018

AbstractAutoantibodies (Ab) against the thyroid-stimulating hormone receptor (TSHR) are frequently found in autoimmune thyroid disease (AITD). Autoantibodies to the TSHR (anti-TSHR-Ab) may mimic or block the action of TSH or be functionally neutral. Measurement of anti-TSHR-Ab can be done either via competitive-binding immunoassays or with functional cell-based bioassays. Antibody-binding assays do not assess anti-TSHR-Ab functionality, but rather measure the concentration of total anti-TSHR binding activity. In contrast, functional cell-based bioassays indicate whether anti-TSHR-Ab have stimulatory or blocking activity. Historically bioassays for anti-TSHR-Ab were research tools and were u…

medicine.medical_specialtyendocrine systemendocrine system diseasesEndocrinology Diabetes and MetabolismGraves' diseaseClinical Biochemistry030209 endocrinology & metabolismHashimoto DiseaseReviewBiochemistryThyroiditisThyrotropin receptor03 medical and health sciences0302 clinical medicineEndocrinologyInternal medicineBlocking antibodymedicineAnimalsHumansReceptorAntibodies BlockingAutoantibodiesbinding assaycell-based bioassaybiologybusiness.industryBiochemistry (medical)AutoantibodyReceptors ThyrotropinGeneral MedicineHashimoto’s thyroiditismedicine.diseaseTSH receptor blocking autoantibodieseye diseasesEndocrinologyHormone receptor030220 oncology & carcinogenesisImmunologybiology.proteinBiological AssayAntibodybusinessGraves’ diseasehormones hormone substitutes and hormone antagonistsHormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme
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Mutations in the NKX2.5 Gene and the PAX8 Promoter in a Girl with Thyroid Dysgenesis

2011

Screening of the known candidate genes involved in thyroid organogenesis has revealed mutations in a small subset of patients with congenital hypothyroidism due to thyroid dysgenesis (TD).We studied a girl with TD who had mutations in two transcription factors involved in thyroid development.Sequencing analysis of candidate genes involved in thyroid gland development revealed a new paternally inherited heterozygous mutation in the NKX2.5 gene (S265R) and a new maternally inherited heterozygous mutation in the PAX8 promoter region (-456CT). Both parents and a brother, who was also heterozygous for both mutations, were phenotypically normal. Immunofluorescence microscopy showed a correct nucl…

medicine.medical_specialtyendocrine systemendocrine system diseasesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryBiologyGene mutationDominant-Negative Mutationmedicine.disease_causeBiochemistryThyroid dysgenesisPAX8 Transcription FactorEndocrinologyInternal medicinemedicineCongenital HypothyroidismHumansPaired Box Transcription FactorsPromoter Regions GeneticGeneticsHomeodomain ProteinsMutationBiochemistry (medical)ThyroidJCEM Online: Brief Reportsmedicine.diseaseCongenital hypothyroidismmedicine.anatomical_structureEndocrinologyMutationThyroid DysgenesisCancer researchHomeobox Protein Nkx-2.5ThyroglobulinFemalePAX8Transcription Factors
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The Impact of Epigallocatechin Gallate and Coconut Oil Treatment on Cortisol Activity and Depression in Multiple Sclerosis Patients

2021

(1) Background: Multiple sclerosis (MS) is pathogenically characterized by high oxidative stress and symptomatically by progressive muscle loss and increased body fat associated with the presence of depression. Epigallocatechin gallate (EGCG) (particularly present in green tea) and ketone bodies (in particular beta-hydroxybutyrate (BHB)), whose main source is coconut oil, have shown emotional benefits and body fat loss. The aim of this study was to assess the impact of EGCG and coconut oil on cortisol activity related to fat loss and depression in MS patients. (2) Methods: The study involved 51 MS patients who were randomly divided into an intervention group or a control group. The interven…

medicine.medical_specialtyepigallocatechin gallatefood.ingredientScience030209 endocrinology & metabolismEpigallocatechin gallatePlacebomedicine.disease_causemultiple sclerosisGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicinefoodInternal medicinefatMedicineEcology Evolution Behavior and SystematicsDepression (differential diagnoses)coconut oilbusiness.industryQCoconut oilAlbuminBeck Depression InventoryPaleontologyfood and beveragesEndocrinologychemistrySpace and Planetary SciencedepressionKetone bodiesbusiness030217 neurology & neurosurgeryOxidative stressLife
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