Search results for "Bioinformatics"

showing 10 items of 1632 documents

Analysis of the Thymidylate Synthase Gene Structure in Colorectal Cancer Patients and Its Possible Relation with the 5-Fluorouracil Drug Response

2009

Thymidylate synthase (TS) catalyzes methylation of dUMP to dTMP and it is the target for the 5-Fluorouracil (5-FU) activity. Barbour et al. showed that variant structural forms of TS in tumour cell lines confer resistance to fluoropyrimidines. We planned to perform the whole TS gene structure by means of sequencing techniques in human colorectal cancer (CRC) samples to try to identify the presence of any possible TS variant form that could be responsible of fluoropyrimidines drug resistance and of the worse prognosis. We performed the TS-DNA gene sequence in 68 CRC from patients of A, B, and C Dukes' stages and different histological grade, but we did not find any mutation in the TS-DNA str…

Genome instabilityArticle Subjectlcsh:QH426-470Colorectal cancerDrug resistancemedicine.disease_causeBioinformaticsBiochemistryThymidylate synthaselcsh:Biochemistrymedicinelcsh:QD415-436Molecular BiologyGeneMutationbiologybusiness.industryMethylationmedicine.diseaselcsh:GeneticsFluorouracilbiology.proteinCancer researchbusinessResearch Articlemedicine.drugJournal of Nucleic Acids
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Molecular and Cellular Insights into the Development of Uterine Fibroids

2021

Uterine leiomyomas represent the most common benign gynecologic tumor. These hormone-dependent smooth-muscle formations occur with an estimated prevalence of ~70% among women of reproductive age and cause symptoms including pain, abnormal uterine bleeding, infertility, and recurrent abortion. Despite the prevalence and public health impact of uterine leiomyomas, available treatments remain limited. Among the potential causes of leiomyomas, early hormonal exposure during periods of development may result in developmental reprogramming via epigenetic changes that persist in adulthood, leading to disease onset or progression. Recent developments in unbiased high-throughput sequencing technolog…

Genome instabilityInfertilitysteroid hormonesUterine fibroidsQH301-705.5ReviewBioinformaticsCatalysistumor-initiating cellEpigenesis GeneticInorganic Chemistrytumor bulk/single-cellsmedicineAnimalsHumansGenetic Predisposition to DiseaseEpigeneticsPhysical and Theoretical ChemistryBiology (General)Molecular BiologyQD1-999genetics/epigeneticsSpectroscopyUterine leiomyomauterine leiomyomaLeiomyomabusiness.industryOrganic ChemistryUterusMyometriumbiomarkersGeneral Medicinemedicine.diseasefemale genital diseases and pregnancy complicationsComputer Science ApplicationsGene Expression Regulation NeoplasticChemistryLeiomyomaMutationUterine Neoplasmstargetable pathwaysFemalebusinessReprogrammingInternational Journal of Molecular Sciences
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Oxidative stress and its downstream signaling in aging eyes

2014

María Dolores Pinazo-Durán,1,* Roberto Gallego-Pinazo,2,* Jose Javier García-Medina,1,3,* Vicente Zanón-Moreno,1,4 Carlo Nucci,5 Rosa Dolz-Marco,2 Sebastián Martínez-Castillo,2 Carmen Galbis-Estrada,1 Carla Marco-Ramírez,1 Maria Isabel López-Gálvez,6,* David J Galarreta,6,* Manuel Díaz-Llópis4,*1Ophthalmic Research Unit "Santiago Grisolía", Valencia, Spain; 2Department of Ophthalmology, Macula Section, The University and Polytechnic Hospital La Fe, Valencia, Spain; 3Department of Ophthalmology, University Hospital Reina Sofia, Murcia, Spain; 4Faculty of Medicine, University of Valencia, Spain; 5Uni…

GerontologyAginggenetic structuresGlaucomaDry Eye SyndromesReviewBioinformaticsmedicine.disease_causeEyeRetinal Diseasesophthalmic diseases; antioxidants; epidemiology; oxidative stress; eyes; agingMedicineHumansOcular Physiological PhenomenaPathologicalOcular Physiological PhenomenaAgedMechanism (biology)business.industrySettore MED/30 - Malattie Apparato VisivoRC952-954.6Glaucomaophthalmic diseasesGeneral MedicineDiabetic retinopathyeyesMacular degenerationmedicine.diseaseeye diseasesOxidative stress. Eyes. Aging. Ophthalmic Diseases. Antioxidants. Epidemiology.Oxidative StressantioxidantsGeriatricsDietary SupplementsDry Eye Syndromesepidemiologysense organsGeriatrics and GerontologybusinessOxidative stress
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Role of prothrombotic polymorphisms in successful or unsuccessful aging.

2011

The study of the genetic profile of centenarians aims to identify the genes and allelic variants which may influence a greater life expectancy and that can be considered as predisposing factors associated to the aging diseases, such as Alzheimer. Centenarians, that represent a cohort of selected survivors, show an hypercoagulability state characterised by striking signs of high coagulation enzyme activity, as directly assessed by the tested higher plasma level of some important factors involved in the haemostasis balance. Anyway, these individuals seem to have a reduced susceptibility to dementia, as well as to cardiovascular events. In this study we analyze the frequencies of Leiden Factor…

GerontologyMaleAging.media_common.quotation_subjectDiseaseBioinformaticsLeiden factor V; Prothrombin; Nonagenarians; Alzheimer disease; Aging.Polymorphism (computer science)Alzheimer DiseasemedicineDementiaHumansNonagenarianAlleleAllelesmedia_commonAgedSettore MED/04 - Patologia GeneralePolymorphism GeneticSuccessful agingbusiness.industryALZHEIMER’S DISEASEagingLongevityFactor Vmedicine.diseaseCase-Control StudiesCohortFemaleProthrombinGeriatrics and GerontologyAlzheimer's diseasebusinessGerontologyLeiden Factor V
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Epigenetics As The Driving Force In Long-Term Immunosuppression

2016

Epigenetics is an emerging frontier of biology, with the potential for deciphering the intricate molecular and transcriptional cellular programs, therefore contributing to explain the pathological evolution of sepsis, one of the most elusive syndromes in medicine. The evolution of sepsis depends not only on the pathogen which originated the infection but also on the genetic and epigenetic background of the host. Short-term mortality of sepsis and septic shock is high, being considered a public health concern worldwide. Immunosuppression is the predominant driving force for morbidity and mortality in late deaths and long-term deaths of survivors from a sepsis episode. In this regard, apoptos…

GerontologySeptic shockbusiness.industrymedicine.medical_treatmentImmunosuppressionmedicine.diseaseBioinformaticsSepsisPathogenesisImmune systemmedicineEpigeneticsProgenitor cellbusinessReprogrammingJournal of Clinical Epigenetics
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Big Data in Medical Science–a Biostatistical View

2015

Big data” is a universal buzzword in business and science, referring to the retrieval and handling of ever-growing amounts of information. It can be assumed, for example, that a typical hospital generates hundreds of terabytes (1 TB = 1012 bytes) of data annually in the course of patient care (1). For instance, exome sequencing, which results in 5 gigabytes (1 GB = 109 bytes) of data per patient, is on the way to becoming routine (2). The analysis of such enormous volumes of information, i.e., organization and description of the data and the drawing of (scientifically valid) conclusions, can already hardly be accomplished with the traditional tools of computer science and statistics. For ex…

Gigabytebusiness.industrymedia_common.quotation_subjectBig dataByteCloud computingGeneral MedicineTerabyteBioinformaticsData scienceData analysisMedicinebusinessFunction (engineering)media_commonDatasets as TopicDeutsches Ärzteblatt international
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Strategies to Reduce Oxidative Stress in Glaucoma Patients

2018

Background Primary open-angle glaucoma (POAG) is a multifactorial pathology involving a variety of pathogenic mechanisms, including oxidative/nitrosative stress. This latter is the consequence of the imbalance between excessive formation and insufficient protection against reactive oxygen/nitrogen species. Objective Our main goal is to gather molecular information to better managing pathologic variants that may determine the individual susceptibility to oxidative/nitrosative stress (OS/NS) and POAG. Method An extensive search of the scientific literature was conducted using PUBMED, the Web of Science, the Cochrane Library, and other references on the topic of POAG and OS/NS from human and a…

Gingko bilobaIntraocular pressurePathologymedicine.medical_specialtygenetic structuresGlaucomaessential fatty acidsOxidative phosphorylationmedicine.disease_causeBioinformaticsArticlePathogenesisMelatonin03 medical and health scienceschemistry.chemical_compound0302 clinical medicinenatural compoundsAnimalsHumansMedicinePharmacology (medical)PharmacologyCoenzyme Q10business.industryGlaucomaGeneral Medicineoxidative stress.medicine.diseasenitrosative stresseye diseasesOxidative StressPsychiatry and Mental healthantioxidantsNeurologychemistry030221 ophthalmology & optometryNeurology (clinical)business030217 neurology & neurosurgeryOxidative stressmedicine.drugCurrent Neuropharmacology
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Co-factors, Microbes, and Immunogenetics in Celiac Disease to Guide Novel Approaches for Diagnosis and Treatment.

2021

Celiac disease (CeD) is a frequent immune-mediated disease that affects not only the small intestine but also many extraintestinal sites. The role of gluten proteins as dietary triggers, HLA-DQ2 or -DQ8 as major necessary genetic predisposition, and tissue transglutaminase (TG2) as mechanistically involved autoantigen, are unique features of CeD. Recent research implicates many cofactors working in synergism with these key triggers, including the intestinal microbiota and their metabolites, nongluten dietary triggers, intestinal barrier defects, novel immune cell phenotypes, and mediators and cytokines. In addition, apart from HLA-DQ2 and -DQ8, multiple and complex predisposing genetic fact…

GlutensTissue transglutaminaseHuman leukocyte antigenDiseaseGut floraImmunologic Testsmedicine.disease_causeBioinformaticsAutoimmunityImmune systemPredictive Value of TestsRisk FactorsGenetic predispositionMedicineAnimalsHumansGenetic Predisposition to DiseaseImmunogenetic PhenomenaIrritable bowel syndromeHepatologybiologyBacteriabusiness.industryfungiGastroenterologynutritional and metabolic diseasesmedicine.diseasebiology.organism_classificationPrognosisGastrointestinal MicrobiomeIntestinesCeliac DiseaseDisease Models AnimalPhenotypeHost-Pathogen Interactionsbiology.proteinbusinessGastroenterology
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The mucopolysaccharidoses: Inborn errors of glycosaminoglycan catabolism

1976

The mucopolysaccharidoses are genetic disorders of glycosaminoglycan metabolism. Patients with these diseases accumulate within the lysosomes of most tissues excessive amounts of dermatan and/or heparan sulfates, or of keratan sulfate. The clinical consequences of such glycosaminoglycan storage range from skeletal abnormalities to cardiovascular problems, and to motor and mental retardation. In all mucopolysaccharidoses, except Morquio disease, an excessive accumulation of sulfate-labeled glycosaminoglycans has been demonstrated in fibroblasts cultured from the patient's skin. It was subsequently shown that this was due to the deficiency of specific proteins which were named "corrective fac…

Glycoside HydrolasesKeratan sulfateMucopolysaccharidosisPrenatal diagnosisDiseaseMucopolysaccharidosesBiologyBioinformaticsmedicine.diseaseHuman geneticsEnzyme assayGlycosaminoglycanTissue culturechemistry.chemical_compoundPhenotypechemistryGeneticsmedicinebiology.proteinHumansSulfatasesLysosomesGenetics (clinical)GlycosaminoglycansHuman Genetics
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Pharmacogenetic considerations for optimizing tacrolimus dosing in liver and kidney transplant patients

2013

The introduction of tacrolimus in clinical practice has improved patient survival after organ transplant. However, despite the long use of tacrolimus in clinical practice, the best way to use this agent is still a matter of intense debate. The start of the genomic era has generated new research areas, such as pharmacogenetics, which studies the variability of drug response in relation to the genetic factors involved in the processes responsible for the pharmacokinetics and/or the action mechanism of a drug in the body. This variability seems to be correlated with the presence of genetic polymorphisms. Genotyping is an attractive option especially for the initiation of the dosing of tacrolim…

Graft Rejectionmedicine.medical_specialtyCYP3A5ATP Binding Cassette Transporter Subfamily BCYP3A4Genotypemedicine.medical_treatmentPharmacologyLiver transplantationBioinformaticsOrgan transplantationTacrolimusCalcineurin inhibitorMedicineCytochrome P-450 CYP3AHumansDrug Dosage CalculationsDosingATP Binding Cassette Transporter Subfamily B Member 1Topic HighlightKidney transplantLiver transplantKidney transplantationBiotransformationPolymorphism Geneticbusiness.industryPharmacogeneticGraft SurvivalGastroenterologyABCB1General Medicinemedicine.diseaseKidney TransplantationTacrolimusLiver TransplantationSingle nucleotide polymorphismTransplantationsurgical procedures operativePhenotypeTreatment OutcomePharmacogeneticsTacrolimuSettore BIO/14 - FarmacologiaPersonalized medicinebusinessPharmacogeneticsImmunosuppressive Agents
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