Search results for "Bol"

showing 10 items of 18211 documents

Mitochondrial bioenergetic pathways in blood leukocyte transcriptome decrease after intensive weight loss but are rescued following weight regain in …

2021

Prolonged periods of energy deficit leading to weight loss induce metabolic adaptations resulting in reduced energy expenditure, but the mechanisms for energy conservation are incompletely understood. We examined 42 healthy athletic females (age 27.5 +/- 4.0 years, body mass index 23.4 +/- 1.7 kg/m(2)) who volunteered into either a group dieting for physique competition (n = 25) or a control group (n = 17). The diet group substantially reduced their energy intake and moderately increased exercise levels to induce loss of fat mass that was regained during a voluntary weight regain period. The control group maintained their typical lifestyle habits and body mass as instructed. From the diet g…

0301 basic medicineBioenergeticsWeight GainruokavaliotBiochemistryTranscriptomeravitsemuskäyttäytyminen0302 clinical medicineWeight lossaineenvaihdunta2. Zero hungerexerciseAdaptation PhysiologicalMitochondriaFemalemedicine.symptomenergiankulutus (aineenvaihdunta)fyysinen aktiivisuusBiotechnologyDietingAdultmedicine.medical_specialtyleukocytesmitokondriotoxidative phosphorylationBiologypainonnousuribosomesYoung Adult03 medical and health sciencesInternal medicineWeight LossGeneticsmedicineMetabolomeHumansMolecular Biologyitsensä johtaminenlaihdutusAMPKMetabolism030104 developmental biologyEndocrinologyenergiansaanti1182 Biochemistry cell and molecular biologylihavuus3111 BiomedicineEnergy IntakeTranscriptomedietBody mass index030217 neurology & neurosurgery
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Cardiolipin synthesis in brown and beige fat mitochondria is essential for systemic energy homeostasis

2018

Summary Activation of energy expenditure in thermogenic fat is a promising strategy to improve metabolic health, yet the dynamic processes that evoke this response are poorly understood. Here we show that synthesis of the mitochondrial phospholipid cardiolipin is indispensable for stimulating and sustaining thermogenic fat function. Cardiolipin biosynthesis is robustly induced in brown and beige adipose upon cold exposure. Mimicking this response through overexpression of cardiolipin synthase (Crls1) enhances energy consumption in mouse and human adipocytes. Crls1 deficiency in thermogenic adipocytes diminishes inducible mitochondrial uncoupling and elicits a nuclear transcriptional respons…

0301 basic medicineBiologiaBioenergeticsChop-10 ; Crls1 ; Beige Adipose ; Brown Adipose ; Cardiolipin ; Insulin Resistance ; Lipid Metabolism ; Mitochondria ; Phospholipids ; ThermogenesisPhysiologyGlucose uptakeAdipose tissueTransferases (Other Substituted Phosphate Groups)MitochondrionEnergy homeostasischemistry.chemical_compoundMice0302 clinical medicineAdipose Tissue Browninsulin resistancelipid metabolismCardiolipinAdipocytesCells CulturedThermogenesisthermogenesisCell biologyMitochondriamitochondriaCHOP-10lipids (amino acids peptides and proteins)BioquímicaCardiolipinsbeige adiposeArticle03 medical and health sciencesInsulin resistanceCRLS1medicineAnimalsHumansMolecular Biologyphospholipidsbrown adiposeMembrane ProteinsCell BiologyAdipose Tissue Beigemedicine.diseaseMice Inbred C57BL030104 developmental biologychemistrycardiolipinEnergy MetabolismThermogenesis030217 neurology & neurosurgery
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NLRP3 Inflammasome Biomarker-Could Be the New Tool for Improved Cardiometabolic Syndrome Outcome.

2020

Metabolomics, the research area studying chemical processes involving metabolites, finds its utility in inflammasome biomarker discovery, thus representing a novel approach for cardiometabolic syndrome pathogeny acknowledgements. Metabolite biomarkers discovery is expected to improve the disease evolution and outcome. The activation of abundantly expressed NLRP3 inflammasome represents the background process of the diabetes mellitus disturbances like hyperglycemia and insulin resistance, as well as for myocardial cell death and fibrosis, all of them being features characteristic for cardiometabolic syndrome. Many molecules like troponins, brain natriuretic protein (BNP), ST2/IL-33, C-reacti…

0301 basic medicineBiomarkers Cardiometabolic syndrome Inflammasome Metabolomics NLRP3 inflammasome Outcome Targeted therapyMyocarditisEndocrinology Diabetes and Metabolismmedicine.medical_treatmentReviewBioinformaticsBiochemistryTargeted therapy03 medical and health sciences0302 clinical medicineInsulin resistanceinflammasomeDiabetes mellitusmedicineMyocardial infarctionBiomarker discoveryMolecular Biologybusiness.industrybiomarkersInflammasomemedicine.diseasetargeted therapymetabolomicsNLRP3 inflammasome030104 developmental biology030220 oncology & carcinogenesisoutcomeBiomarker (medicine)cardiometabolic syndromebusinessmedicine.drugMetabolites
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Polyphosphate as a metabolic fuel in Metazoa: A foundational breakthrough invention for biomedical applications

2015

In animals, energy-rich molecules like ATP are generated in the intracellular compartment from metabolites, e.g. glucose, taken up by the cells. Recent results revealed that inorganic polyphosphates (polyP) can provide an extracellular system for energy transport and delivery. These polymers of multiple phosphate units, linked by high-energy phosphoanhydride bonds, use blood platelets as transport vehicles to reach their target cells. In this review it is outlined how polyP affects cell metabolism. It is discussed that polyP influences cell activity in a dual way: (i) as a metabolic fuel transferring metabolic energy through the extracellular space; and (ii) as a signaling molecule that amp…

0301 basic medicineBiomedical TechnologyMitochondrionBiologyEndocytosisApplied Microbiology and Biotechnology03 medical and health scienceschemistry.chemical_compoundAdenosine TriphosphateTissue engineeringPolyphosphatesExtracellularHumansBlood CellsPolyphosphateGeneral MedicineCell biologyMitochondriaMetabolic pathway030104 developmental biologychemistryBiochemistryMolecular MedicineNanoparticlesAdenosine triphosphateIntracellularMetabolic Networks and PathwaysBiotechnology Journal
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Mutant p53 induces Golgi tubulo-vesiculation driving a prometastatic secretome

2020

TP53 missense mutations leading to the expression of mutant p53 oncoproteins are frequent driver events during tumorigenesis. p53 mutants promote tumor growth, metastasis and chemoresistance by affecting fundamental cellular pathways and functions. Here, we demonstrate that p53 mutants modify structure and function of the Golgi apparatus, culminating in the increased release of a pro-malignant secretome by tumor cells and primary fibroblasts from patients with Li-Fraumeni cancer predisposition syndrome. Mechanistically, interacting with the hypoxia responsive factor HIF1α, mutant p53 induces the expression of miR-30d, which in turn causes tubulo-vesiculation of the Golgi apparatus, leading …

0301 basic medicineBiopsyGeneral Physics and AstronomyGolgi ApparatusAnimals Biopsy Breast Neoplasms Cell Line Tumor Cell Transformation Neoplastic Female Fibroblasts Gene Expression Regulation Neoplastic Golgi Apparatus Humans Hypoxia-Inducible Factor 1 alpha Subunit Li-Fraumeni Syndrome Mice MicroRNAs Microtubules Mutation Primary Cell Culture Secretory Vesicles Signal TransductionSkin Tumor Microenvironment Tumor Suppressor Protein p53 Xenograft Model Antitumor Assays02 engineering and technologymedicine.disease_causeCell TransformationMicrotubulesSettore BIO/09 - FisiologiaMetastasisLi-Fraumeni SyndromeMiceTumor MicroenvironmentGolgisecretory machinerySuper-resolution microscopyAnimals; Biopsy; Breast Neoplasms; Cell Line Tumor; Cell Transformation Neoplastic; Female; Fibroblasts; Gene Expression Regulation Neoplastic; Golgi Apparatus; Humans; Hypoxia-Inducible Factor 1 alpha Subunit; Li-Fraumeni Syndrome; Mice; MicroRNAs; Microtubules; Mutation; Primary Cell Culture; Secretory Vesicles; Signal Transduction; Skin; Tumor Microenvironment; Tumor Suppressor Protein p53; Xenograft Model Antitumor Assayslcsh:ScienceSkinMultidisciplinaryTumorChemistrymutant p53QCell migrationMicroRNASecretomics021001 nanoscience & nanotechnologyCell biologyGene Expression Regulation NeoplasticCell Transformation NeoplasticsymbolsFibroblastmiR-30dFemaleHypoxia-Inducible Factor 10210 nano-technologyBreast NeoplasmHumanSignal TransductionCancer microenvironmentStromal cellSecretory VesicleSciencePrimary Cell CultureBreast NeoplasmsMicrotubuleGolgi ApparatuSettore MED/08 - Anatomia Patologicaalpha SubunitGeneral Biochemistry Genetics and Molecular BiologyArticleCell Line03 medical and health sciencessymbols.namesakeCell Line TumormedicineAnimalsHumansSettore MED/05 - Patologia ClinicaSecretionTumor microenvironmentNeoplasticAnimalSecretory VesiclesGeneral ChemistryOncogenesGolgi apparatusHDAC6FibroblastsMicroreviewHypoxia-Inducible Factor 1 alpha SubunitmicroenvironmentXenograft Model Antitumor AssaysMicroRNAs030104 developmental biologyGene Expression RegulationMutationlcsh:QTumor Suppressor Protein p53Carcinogenesis
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Comparing Targeted vs. Untargeted MS2 Data-Dependent Acquisition for Peak Annotation in LC-MS Metabolomics

2020

One of the most widely used strategies for metabolite annotation in untargeted LCMS is based on the analysis of MSn spectra acquired using data-dependent acquisition (DDA), where precursor ions are sequentially selected from MS scans based on user-selected criteria. However, the number of MSn spectra that can be acquired during a chromatogram is limited and a trade-off between analytical speed, sensitivity and coverage must be ensured. In this research, we compare four different strategies for automated MS2 DDA, which can be easily implemented in the frame of standard QA/QC workflows for untargeted LC&ndash

0301 basic medicineBioquímicaBiologiaComputer scienceEndocrinology Diabetes and Metabolismlcsh:QR1-50201 natural sciencesBiochemistryliquid chromatography–mass spectrometryArticlelcsh:Microbiology03 medical and health sciencesAnnotationMetabolomicsLiquid chromatography–mass spectrometrypeak annotationMolecular BiologyData dependentliquid chromatography-mass spectrometrydata dependent acquisitionbusiness.industry010401 analytical chemistryhuman milkPattern recognition0104 chemical sciencesWorking range030104 developmental biologyFeature (computer vision)Reference databaseArtificial intelligencebusinessMETABOLIC FEATURES
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Serum Amino Acid Profiles in Childhood Predict Triglyceride Level in Adulthood: A 7-Year Longitudinal Study in Girls.

2016

AbstractContext:Branched-chain and aromatic amino acids are associated with high risk of developing dyslipidemia and type II diabetes in adults.Objective:This study aimed to examine whether serum amino acid profiles associate with triglyceride concentrations during pubertal growth and predict hypertriglyceridemia in early adulthood.Design:This was a 7.5-year longitudinal study.Setting:The study was conducted at the Health Science Laboratory, University of Jyväskylä.Participants:A total of 396 nondiabetic Finnish girls aged 11.2 ± 0.8 years at the baseline participated in the study.Main Outcome Measures:Body composition was assessed by dual-energy x-ray absorptiometry; serum concentrations o…

0301 basic medicineBlood GlucoseLongitudinal studymedicine.medical_specialtyMagnetic Resonance SpectroscopyAdolescentEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryContext (language use)030204 cardiovascular system & hematologyBiochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyAbsorptiometry PhotonInternal medicinemedicineHumansInsulinLongitudinal StudiesAmino AcidsChildTriglyceridesTriglyceridebusiness.industryInsulinBiochemistry (medical)Hypertriglyceridemiamedicine.disease030104 developmental biologyEndocrinologychemistryBody CompositionFemaleIsoleucineLeucinebusinessDyslipidemiaThe Journal of clinical endocrinology and metabolism
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Human Achilles tendon glycation and function in diabetes

2016

Diabetic patients have an increased risk of foot ulcers, and glycation of collagen may increase tissue stiffness. We hypothesized that the level of glycemic control (glycation) may affect Achilles tendon stiffness, which can influence gait pattern. We therefore investigated the relationship between collagen glycation, Achilles tendon stiffness parameters, and plantar pressure in poorly ( n = 22) and well ( n = 22) controlled diabetic patients, including healthy age-matched (45–70 yr) controls ( n = 11). There were no differences in any of the outcome parameters (collagen cross-linking or tendon stiffness) between patients with well-controlled and poorly controlled diabetes. The overall effe…

0301 basic medicineBlood GlucoseMaleGlycosylationPhysiologyFoot/physiologyDiabetes Mellitus/physiopathologychemistry.chemical_compound0302 clinical medicineGlycationta315GaitAchilles tendondiabetesBiomechanical Phenomena/physiologyta3141ta3142Middle Agedenzymatic and non-enzymatic collagen cross-linkingAchilles Tendon/physiopathologymusculoskeletal systemTendonBiomechanical Phenomenamedicine.anatomical_structureGait/physiologymusculoskeletal diseasesmedicine.medical_specialtyUrologyConnective tissue030209 endocrinology & metabolismta3111Achilles TendonGlycemic Index/physiology03 medical and health sciencesPhysiology (medical)Diabetes mellitusJoint capsulemedicineDiabetes MellitusHumansPentosidinebusiness.industryFootForefootmedicine.diseasefoot ulcerSurgerybody regionsBlood Glucose/physiology030104 developmental biologyCross-Sectional StudieschemistryGlycemic IndexAchilles tendon mechanicsEnzymatic and nonenzymatic collagen cross-linkingbusiness
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Unsaturated Oral Fat Load Test Improves Glycemia, Insulinemia and Oxidative Stress Status in Nondiabetic Subjects with Abdominal Obesity.

2016

Aims To evaluate the changes in glycemia, insulinemia, and oxidative stress markers during an oral fat load test in nondiabetic subjects with abdominal obesity and to analyze the association between postprandial oxidative stress markers and postprandial glucose and insulin responses. Methods We included 20 subjects with abdominal obesity (waist circumference > 102 cm for men and > 88 cm for women) and 20 healthy lean controls (waist circumference < 102 cm for men and < 88 cm for women). After 12 hours of fasting we performed a standardized fat load test (0–8 hours) with supracal® (50 g/m2). We determined metabolic parameters, oxidized and reduced glutathione, and malondialdehyde. Results In…

0301 basic medicineBlood GlucoseMalePhysiologymedicine.medical_treatmentlcsh:MedicineBiochemistryFatschemistry.chemical_compound0302 clinical medicineEndocrinologyMalondialdehydeMedicine and Health SciencesInsulinlcsh:ScienceAbdominal obesityMultidisciplinaryOrganic CompoundsMonosaccharidesMiddle AgedMalondialdehydePostprandial PeriodGlutathioneLipidsChemistryPostprandialCholesterolPhysiological ParametersObesity AbdominalPhysical SciencesFemalemedicine.symptomResearch ArticleAdultmedicine.medical_specialtyWaistAdolescentLipoproteinsCarbohydrates030209 endocrinology & metabolism03 medical and health sciencesYoung AdultInsulin resistanceInternal medicinemedicineHumansObesityAgedDiabetic EndocrinologyEndocrine Physiologybusiness.industryInsulinUnsaturated fatlcsh:RBody WeightOrganic ChemistryChemical CompoundsBiology and Life SciencesProteinsCell Biologymedicine.diseaseObesityHormonesFats UnsaturatedOxidative Stress030104 developmental biologyEndocrinologyGlucosechemistryCase-Control Studieslcsh:QInsulin ResistancebusinessPloS one
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Baseline metabolic disturbances and the twenty-five years risk of incident cancer in a Mediterranean population.

2016

Abstract Background and aims Obesity is predictive of metabolic syndrome (metS), type 2 diabetes, cardiovascular (CV) disease and cancer. The aim of the study is to assess the risk of incident cancer connected to obesity and metS in a Mediterranean population characterized by a high prevalence of obesity. Methods and results As many as 1133 subjects were enrolled in two phases and followed for 25 years (859 subjects) or 11 years (274 subjects) and incident cancer was registered in the follow-up period. Anthropometric measures and biochemical parameters were filed at baseline and evaluated as predictors of incident cancer by measuring hazards ratios (HR) using multivariate Cox parametric haz…

0301 basic medicineBlood GlucoseMaleSettore MED/09 - Medicina InternaTime FactorsMediterranean dietEpidemiologyEndocrinology Diabetes and MetabolismMedicine (miscellaneous)Type 2 diabetesDiet Mediterranean0302 clinical medicineRisk FactorsNeoplasmsPrevalenceCancerMetabolic Syndromeeducation.field_of_studyNutrition and DieteticsIncidence (epidemiology)IncidenceLipidMiddle AgedLipidsItalyCardiovascular Diseases030220 oncology & carcinogenesisArea Under CurveFemaleDiet HealthyCardiology and Cardiovascular Medicinemedicine.medical_specialtyPopulationRisk AssessmentDisease-Free Survival03 medical and health sciencesInternal medicinemedicineHumansObesityeducationAgedProportional Hazards ModelsRetrospective StudiesChi-Square Distributionbusiness.industryProportional hazards modelCancerProtective Factorsmedicine.diseaseObesity030104 developmental biologyEndocrinologyROC CurveMultivariate AnalysisMetabolic syndromeInsulin ResistancebusinessBiomarkersNutrition, metabolism, and cardiovascular diseases : NMCD
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