Search results for "Bow"

showing 10 items of 1006 documents

Mast cells crosstalk with B cells in the gut and sustain IgA response in the inflamed intestine.

2021

B lymphocytes are among the cell types whose effector functions are modulated by mast cells (MCs). The B/MC crosstalk emerged in several pathological settings, notably the colon of inflammatory bowel disease (IBD) patients is a privileged site in which MCs and IgA+ cells physically interact. Herein, by inducing conditional depletion of MCs in red MC and basophil (RMB) mice, we show that MCs control B cell distribution in the gut and IgA serum levels. Moreover, in dextran sulfate sodium (DSS)-treated RMB mice, the presence of MCs is fundamental for the enlargement of the IgA+ population in the bowel and the increase of systemic IgA production. Since both conventional B-2 and peritoneal-deriv…

0301 basic medicineCell typeColon[SDV]Life Sciences [q-bio]ImmunologyPopulationInflammationBasophilBiologySettore MED/08 - Anatomia Patologicabehavioral disciplines and activitiesInflammatory bowel diseasecell-to-cell interplay colitis IgAinnate-like B cells mast cells03 medical and health sciencesMice0302 clinical medicinemedicineImmunology and AllergyAnimalsMast CellsColitisIntestinal MucosaeducationB cellComputingMilieux_MISCELLANEOUSInflammationeducation.field_of_studyB-LymphocytesTumor Necrosis Factor-alphaDextran Sulfatemedicine.diseaseColitisInflammatory Bowel DiseaseshumanitiesInnate-like B cellsGastrointestinal MicrobiomeImmunoglobulin AMice Inbred C57BLCrosstalk (biology)030104 developmental biologymedicine.anatomical_structureCell-to-cell interplayCell-to-cell interplay; Colitis; IgA; Innate-like B cells; Mast cellsImmunologymedicine.symptomIgA030215 immunologyEuropean journal of immunologyReferences
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The activation of Wnt signaling by a STAT6-dependent macrophage phenotype promotes mucosal repair in murine IBD

2016

The complete repair of the mucosa constitutes a key goal in inflammatory bowel disease (IBD) treatment. The Wnt signaling pathway mediates mucosal repair and M2 macrophages that coordinate efficient healing have been related to Wnt ligand expression. Signal transducer and activator of transcription 6 (STAT6) mediates M2 polarization in vitro and we hypothesize that a STAT6-dependent macrophage phenotype mediates mucosal repair in acute murine colitis by activating the Wnt signaling pathway. Our results reveal an impaired mucosal expression of M2 macrophage-associated genes and delayed wound healing in STAT6(-/-) mice treated with 2,4,6-trinitrobenzenesulfonic acid (TNBS). These mice also ex…

0301 basic medicineCellular differentiationImmunologyBiology03 medical and health sciencesMice0302 clinical medicineImmunology and AllergyAnimalsHumansIntestinal MucosaCells CulturedSTAT6Mice KnockoutMice Inbred BALB CWound HealingWnt signaling pathwayLGR5LRP5Cell DifferentiationColitisInflammatory Bowel DiseasesCell biologyWnt Proteins030104 developmental biologyPhenotypeTrinitrobenzenesulfonic AcidImmunologySTAT proteinMacrophages PeritonealSignal transductionWound healingSTAT6 Transcription Factor030215 immunologySignal Transduction
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Progressive derivation of serially homologous neuroblast lineages in the gnathal CNS of Drosophila

2018

Along the anterior-posterior axis the central nervous system is subdivided into segmental units (neuromeres) the composition of which is adapted to their region-specific functional requirements. In Drosophila melanogaster each neuromere is formed by a specific set of identified neural stem cells (neuroblasts, NBs). In the thoracic and anterior abdominal region of the embryonic ventral nerve cord segmental sets of NBs resemble the ground state (2nd thoracic segment, which does not require input of homeotic genes), and serial (segmental) homologs generate similar types of lineages. The three gnathal head segments form a transitional zone between the brain and the ventral nerve cord. It has be…

0301 basic medicineCentral Nervous SystemEmbryologylcsh:MedicineSerial homologyGene ExpressionNervous SystemAnimal CellsMedicine and Health SciencesBrainbow Labelinglcsh:ScienceNeuronsBrain MappingMultidisciplinarybiologyAnatomyNeuromereNeural stem cellChemistryPhysical SciencesDrosophilaDrosophila melanogasterAnatomyCellular TypesHomeotic geneResearch ArticleLineage (genetic)Imaging TechniquesNeuroimagingResearch and Analysis MethodsComposite Images03 medical and health sciencesNeuroblastInterneuronsGeneticsAnimalsCell LineageMolecular Biology TechniquesMolecular BiologyGround Statelcsh:REmbryosBiology and Life SciencesCell BiologyQuantum Chemistrybiology.organism_classification030104 developmental biologyVentral nerve cordCellular Neurosciencelcsh:QCloningNeuroscienceDevelopmental BiologyPLoS ONE
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Gut-CNS-Axis as Possibility to Modulate Inflammatory Disease Activity-Implications for Multiple Sclerosis.

2017

In the last decade the role of environmental factors as modulators of disease activity and progression has received increasing attention. In contrast to classical environmental modulators such as exposure to sun-light or fine dust pollution, nutrition is an ideal tool for a personalized human intervention. Various studies demonstrate a key role of dietary factors in autoimmune diseases including Inflammatory Bowel Disease (IBD), rheumatoid arthritis or inflammatory central nervous system (CNS) diseases such as Multiple Sclerosis (MS). In this review we discuss the connection between diet and inflammatory processes via the gut–CNS-axis. This axis describes a bi-directional communication syst…

0301 basic medicineCentral Nervous SystemMultiple SclerosisCentral nervous systemInflammationReviewBiologyInflammatory bowel diseaseModels BiologicalCatalysisInorganic ChemistryDisease activitylcsh:Chemistry03 medical and health sciencesImmune systemmedicinemicrobiotaAnimalsHumansPhysical and Theoretical ChemistryMolecular Biologylcsh:QH301-705.5SpectroscopyInflammationMultiple sclerosisOrganic ChemistryGeneral Medicinemedicine.diseaseComputer Science ApplicationsGastrointestinal Tractgut–CNS-axisimmune system030104 developmental biologymedicine.anatomical_structurenutritionlcsh:Biology (General)lcsh:QD1-999Rheumatoid arthritisAdjunctive treatmentImmunologymedicine.symptomInternational journal of molecular sciences
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Hydrodynamic IL10 Gene Transfer in Human Colon: Results from an "EX VIVO" Study with Potential Clinical Application in Crohn's Disease.

2017

Background: The aim of this work is to evaluate the efficacy of hydrodynamic venous IL10 gene delivery to "ex vivo" human colon segments and to determine its potential interest in Crohn's disease treatment. Methods: Twenty human colon segments were obtained from surgical resections. Hydrodynamic transfection through the main vein of the pedicle with 50 mL of hIL10 plasmid (20 mu g/mL) solution was performed on 13 of them. Tissue sections were cultured and DNA, RNA, and protein copies were determined after 1, 2, and 4 days. Data obtained were compared with 6 nontransfected specimens. Finally, 1 specimen was injected with gold nanoparticles, and their distribution was examined under electron …

0301 basic medicineColoninterleukin-10Metal NanoparticlesGene deliverylocoregionalTranslational Research Biomedical03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCrohn Diseaseinflammatory bowel diseaseSubmucosamedicineImmunology and AllergyHumansGenehydrodynamicChemistryGastroenterologyGene Transfer TechniquesRNATransfectionGenetic Therapygene therapyMolecular biologyInterleukin-10Interleukin 10030104 developmental biologymedicine.anatomical_structureHydrodynamics030211 gastroenterology & hepatologyGoldDNAEx vivoInflammatory bowel diseases
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INFLAMMATORY BOWEL DISEASE, COLORECTAL CANCER AND TYPE 2 DIABETES MELLITUS: THE LINKS

2016

The co-occurrence of the three disease entities, inflammatory bowel disease (IBD), colorectal cancer (CRC), type 2diabetes mellitus (T2DM) along with inflammation and dismicrobism has been frequently reported. Some authors have even suggested that dysbiosis could be the link through a molecular crosstalk of multiple inflammatory loops including TGFβ, NFKB, TNFα and ROS among others. This review focuses on the inflammatory process along with the role of microbiota in the pathophysiology of the three diseases. The etiology of IBD is multifactorial, and like CRC and T2DM, it is associated with a widespread and sustained GI inflammation and dismicrobism, whereby an array of pro-inflammatory med…

0301 basic medicineColorectal cancerIBDT2DMInflammationDiseaseReviewSystemic inflammationProbioticInflammatory bowel diseasePathology and Forensic MedicinePathogenesis03 medical and health sciences0302 clinical medicinePhysiology (medical)medicineInflammationbusiness.industrymedicine.diseasedigestive system diseasesCRC030104 developmental biology030220 oncology & carcinogenesisImmunologyInflammatory Bowel diseases IBD colorectal cancer diabetes mellitusMolecular MedicineDysbiosisTumor necrosis factor alphamedicine.symptombusinessDysbiosis
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Hsp60 as a Novel Target in IBD Management: A Prospect

2019

Inflammatory bowel disease (IBD) encompasses various pathological conditions similar but distinct that share a multifactorial etiology, including involvement of the intestinal barrier function, the immune system, and intestinal microorganisms. Hsp60 is a chaperonin component of the chaperoning system, present in all cells and tissues, including the intestine. It plays important roles in cell physiology outside and inside mitochondria, its canonical place of residence. However, Hsp60 can also be pathogenic in many conditions, the Hsp60 chaperonopathies, possibly including IBD. The various clinico-pathological types of IBD have a complicated mix of causative factors, among which Hsp60 can be …

0301 basic medicineColorectal cancerMini Reviewchaperoning systemDiseaseBioinformaticsInflammatory bowel diseasePathogenesis03 medical and health sciences0302 clinical medicineImmune systemintestinal wallinflammatory bowel diseasemedicinemicrobiotaPharmacology (medical)PathologicalchaperonotherapyPharmacologybusiness.industrylcsh:RM1-950fungimedicine.diseaseHsp60Biomarkerimmune systemlcsh:Therapeutics. Pharmacology030104 developmental biology030220 oncology & carcinogenesisEtiologychaperonopathybusinessFrontiers in Pharmacology
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Gut microbiota imbalance and colorectal cancer

2016

International audience; The gut microbiota acts as a real organ. The symbiotic interactions between resident micro-organisms and the digestive tract highly contribute to maintain the gut homeostasis. However, alterations to the microbiome caused by environmental changes (e.g., infection, diet and/or lifestyle) can disturb this symbiotic relationship and promote disease, such as inflammatory bowel diseases and cancer. Colorectal cancer is a complex association of tumoral cells, non-neoplastic cells and a large amount of micro-organisms, and the involvement of the microbiota in colorectal carcinogenesis is becoming increasingly clear. Indeed, many changes in the bacterial composition of the g…

0301 basic medicineColorectal cancer[SDV]Life Sciences [q-bio]enterotoxigenic bacteroides-fragilisGut floraCyclomodulin[ SDV.CAN ] Life Sciences [q-bio]/CancerTopic Highlightstreptococcus-gallolyticus infectionbiologyGastrointestinal MicrobiomeGastroenterologyGeneral Medicinecytolethal-distending toxin3. Good healthlactobacillus-acidophilus deficientIntestinesCell Transformation NeoplasticHost-Pathogen InteractionsInflammation MediatorsColorectal NeoplasmsVirulence Factorspolymerase-chain-reaction[SDV.CAN]Life Sciences [q-bio]/CancerGut microbiotaoxidative dna-damageMicrobiologyescherichia-coli strains03 medical and health scienceshelicobacter-pylori infectionmedicineAnimalsHumansMicrobiomeBacteria[ SDV ] Life Sciences [q-bio]inflammatory-bowel-diseaseCancerHelicobacter pyloribiology.organism_classificationmedicine.diseaseStreptococcus bovisColorectal cancerGastrointestinal MicrobiomeHépatologie et Gastroentérologie030104 developmental biologytoll-like receptorsOxidative stressImmunologyHépatology and GastroenterologyDysbiosiscolorectal cancer;gut microbiota;dysbiosis;cyclomodulin;oxidative;stress;enterotoxigenic bacteroides-fragilis;oxidative dna-damage;cytolethal-distending toxin;inflammatory-bowel-disease;streptococcus-gallolyticus infection;lactobacillus-acidophilus;deficient;helicobacter-pylori infection;polymerase-chain-reaction;escherichia-coli strains;toll-like receptorsDysbiosisDNA Damage
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Characterization of myofibroblasts isolated from the intestine of patients with inflammatory bowel disease

2019

Background: Intestinal fibrosis represents a serious complication of inflammatory bowel diseases (IBD), often necessitating surgical resections. Myofibroblasts are primarily responsible for interstitial matrix accumulation in fibrotic diseases. However intestinal myofibroblasts (IMF) remain inadequately characterized.  The aim was to examine fibroblast markers and fibrosis-associated gene expression in IMF isolated from resected intestine from IBD and control patients. As well as determining the effect of the fibrogenic cytokine TGFβ. Methods: Intestinal resections were obtained (n =35) from consenting patients undergoing elective surgery (2014-16). Primary cultures of IMF were isolated usi…

0301 basic medicineCrohn's diseaseGeneral Immunology and Microbiologybusiness.industrymedicine.medical_treatmentGeneral Medicinemedicine.diseaseInflammatory bowel diseaseGeneral Biochemistry Genetics and Molecular BiologyCTGF03 medical and health sciences030104 developmental biology0302 clinical medicineCytokineInterstitial matrixFibrosismedicineCancer research030211 gastroenterology & hepatologyTumor necrosis factor alphaGeneral Pharmacology Toxicology and PharmaceuticsbusinessTIMP1F1000Research
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Specific norovirus interaction with Lewis x and Lewis a on human intestinal inflammatory mucosa during refractory inflammatory bowel disease

2021

Inflammatory bowel disease (IBD), including Crohn’s disease (CD) and ulcerative colitis (UC), are progressive diseases affecting millions of people each year. Flare-ups during IBD result in severe mucosal alterations of the small intestine (in CD) and in the colon and rectum (in CD and UC).

0301 basic medicineCrohn’s diseaseMaleSeverity of Illness IndexInflammatory bowel diseasechemistry.chemical_compound0302 clinical medicineMedicineIntestinal MucosaCrohn's disease0303 health sciencesMiddle AgedImmunohistochemistryUlcerative colitisQR1-502HBGA3. Good healthmedicine.anatomical_structure030220 oncology & carcinogenesisImmunohistochemistry030211 gastroenterology & hepatologyFemalegut inflammationResearch ArticleAdultCA-19-9 Antigenmedicine.drug_classLewis X AntigenRectumMonoclonal antibodyMicrobiologydigestive systemVirusHost-Microbe BiologyYoung Adult03 medical and health sciencesAntigenHumansMolecular Biologyulcerative colitis030304 developmental biologybusiness.industryNorovirus[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and GastroenterologySialyl-Lewis AInflammatory Bowel Diseasesmedicine.diseasedigestive system diseasesSmall intestineGastrointestinal Tract030104 developmental biologySialyl-Lewis XchemistryinflammationImmunologybusiness[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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