Search results for "Brain Edema"

showing 10 items of 56 documents

Blood constituents trigger brain swelling, tissue death, and reduction of glucose metabolism early after acute subdural hematoma in rats

2009

Outcome from acute subdural hematoma is often worse than would be expected from the pure increase of intracranial volume by bleeding. The aim was to test whether volume-independent pathomechanisms aggravate damage by comparing the effects of blood infusion with those of an inert fluid, paraffin oil, on intracranial pressure (ICP), cerebral perfusion pressure (CPP), local cerebral blood flow (CBF), edema formation, glucose metabolism ([18F]-deoxyglucose, MicroPET), and histological outcome. Rats were injured by subdural infusion of 300 μL venous blood or paraffin. ICP, CPP, and CBF changes, assessed during the first 30 mins after injury, were not different between the injury groups at most …

MaleIntracranial PressureBlood PressureBrain EdemaCerebral edemaLesionRats Sprague-DawleyFluorodeoxyglucose F18medicineAnimalsHematoma Subdural AcuteCerebral perfusion pressureIntracranial pressureVascular diseasebusiness.industryVenous bloodmedicine.diseaseRatsBlood pressureGlucoseNeurologyCerebral blood flowParaffinAnesthesiaCerebrovascular CirculationPositron-Emission TomographyOriginal ArticleNeurology (clinical)medicine.symptomRadiopharmaceuticalsCardiology and Cardiovascular MedicinebusinessEnergy Metabolism
researchProduct

Effects of a small acute subdural hematoma following traumatic brain injury on neuromonitoring, brain swelling and histology in pigs.

2011

An acute subdural hematoma (ASDH) induces pathomechanisms which worsen outcome after traumatic brain injury, even after a small hemorrhage. Synergistic effects of a small ASDH on brain damage are poorly understood, and were studied here using neuromonitoring for 10 h in an injury model of controlled cortical impact (CCI) and ASDH. Pigs (n = 32) were assigned to 4 groups: sham, CCI (2.5 m/s), ASDH (2 ml) and CCI + ASDH. Intracranial pressure was significantly increased above sham levels by all injuries with no difference between groups. CCI and ASDH reduced ptiO<sub>2</sub> by a maximum of 36 ± 9 and 26 ± 11%, respectively. The combination caused a 31 ± 11% drop. ASDH alone and i…

MaleMicrodialysisanimal structuresIntracranial PressureTraumatic brain injurySus scrofaGlutamic AcidBrain EdemaBrain damageEvoked Potentials SomatosensorymedicineAnimalsHematoma Subdural AcuteLactic AcidIntracranial pressureMonitoring Physiologicbusiness.industryGlutamate receptorHistologymedicine.diseasenervous system diseasesDisease Models AnimalSomatosensory evoked potentialAnesthesiaBrain InjuriesCerebrovascular CirculationSurgerymedicine.symptombusinessAcute subdural hematomapsychological phenomena and processesEuropean surgical research. Europaische chirurgische Forschung. Recherches chirurgicales europeennes
researchProduct

Time course of cerebrovascular autoregulation during extreme Trendelenburg position for robotic-assisted prostatic surgery

2013

Trendelenburg positioning in combination with pneumoperitoneum during robotic-assisted prostatic surgery possibly impairs cerebrovascular autoregulation. If cerebrovascular autoregulation is disturbed, arterial hypertension might induce cerebral hyperaemia and brain oedema, while low arterial blood pressure can induce cerebral ischaemia. The time course of cerebrovascular autoregulation was investigated during use of the Trendelenburg position and a pneumoperitoneum for robotic-assisted prostatic surgery using transcranial Doppler ultrasound. Cerebral blood flow velocity was correlated with arterial blood pressure and the autoregulation index (Mx) was calculated. In 23 male patients, Mx was…

MaleMiddle Cerebral ArteryTime FactorsUltrasonography Doppler Transcranialmedicine.medical_treatmentTrendelenburg positionBlood PressureBrain EdemaPatient PositioningHead-Down TiltPneumoperitoneumMonitoring IntraoperativeHomeostasisHumansMedicineGeneral anaesthesiaAutoregulationProspective StudiesProspective cohort studyAgedProstatectomybusiness.industryRoboticsMiddle Agedmedicine.diseaseTranscranial DopplerAnesthesiology and Pain MedicineBlood pressureCerebral blood flowCerebrovascular CirculationAnesthesiabusinessPneumoperitoneum ArtificialBlood Flow VelocityAnaesthesia
researchProduct

Volatile Anesthetics Influence Blood-Brain Barrier Integrity by Modulation of Tight Junction Protein Expression in Traumatic Brain Injury

2012

Disruption of the blood-brain barrier (BBB) results in cerebral edema formation, which is a major cause for high mortality after traumatic brain injury (TBI). As anesthetic care is mandatory in patients suffering from severe TBI it may be important to elucidate the effect of different anesthetics on cerebral edema formation. Tight junction proteins (TJ) such as zonula occludens-1 (ZO-1) and claudin-5 (cl5) play a central role for BBB stability. First, the influence of the volatile anesthetics sevoflurane and isoflurane on in-vitro BBB integrity was investigated by quantification of the electrical resistance (TEER) in murine brain endothelial monolayers and neurovascular co-cultures of the B…

MaleMouse610 MedizinBrain EdemaPharmacologyCardiovascularMiceAnesthesiology610 Medical sciencesEdemaMolecular Cell BiologyClaudin-5MultidisciplinaryIsofluraneQRAnimal ModelsHead Injurymedicine.anatomical_structureNeurologyBlood-Brain BarrierAnesthesiaAnesthetics InhalationMedicineCellular Typesmedicine.symptomResearch Articlemedicine.drugMethyl EthersTraumatic brain injuryCerebrovascular DiseasesScienceBrain damageBlood–brain barrierSevofluraneCell LineTight JunctionsCerebral edemaSevofluraneModel OrganismsVascular BiologymedicineAnimalsBiologybusiness.industryEndothelial Cellsmedicine.diseaseCoculture TechniquesIsofluraneBrain InjuriesAnestheticZonula Occludens-1 ProteinMolecular NeurosciencebusinessNeurosciencePLoS ONE
researchProduct

Inhibition of myosin light chain kinase reduces brain edema formation after traumatic brain injury.

2010

The role of the endothelial contractile apparatus in the process of brain edema formation after brain trauma is not characterized. Phosphorylation of myosin light chains by myosin light chain kinases (MLCK) activates endothelial contractile elements and results in a rearrangement of the cytoskeleton. This may enhance post-traumatic blood-brain barrier dysfunction. In order to investigate the role of the MLCK on brain edema formation and blood-brain barrier permeability after brain injury, mice were anesthetized and subjected to a controlled cortical impact (CCI). MLCK expression is significantly up-regulated after CCI with a maximum 12 h post-injury. Specific inhibition of MLCK by ML-7 resu…

MaleMyosin light-chain kinaseMyosin Light ChainsTime FactorsEndotheliumIntracranial PressureTraumatic brain injuryCentral nervous systemBrain Edemamacromolecular substancesBrain damageNaphthalenesBlood–brain barrierBiochemistryNeuroprotectionDrug Administration ScheduleFunctional LateralityStatistics NonparametricCerebral edemaCellular and Molecular NeuroscienceMicemedicineAnimalsEnzyme InhibitorsMyosin-Light-Chain KinaseNeurologic Examinationbusiness.industryAzepinesmedicine.diseaseConstrictionCell biologyMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureGene Expression RegulationBlood-Brain BarrierBrain Injuriesmedicine.symptombusinessNeuroscienceEvans BlueJournal of neurochemistry
researchProduct

Role of GABAergic antagonism in the neuroprotective effects of bilobalide

2006

Bilobalide, a constituent of Ginkgo biloba, has neuroprotective properties. Its mechanism of action is unknown but it was recently found to block GABA(A) receptors. The goal of this study was to test the potential role of a GABAergic mechanism for the neuroprotective activity of bilobalide. In rat hippocampal slices exposed to NMDA, release of choline indicates breakdown of membrane phospholipids. NMDA-induced choline release was almost completely blocked in the presence of bilobalide (10 microM) and under low-chloride conditions. Bicuculline (100 microM), a competitive antagonist at GABA(A) receptors, reduced NMDA-induced choline release to a small extent (-23%). GABA (100 microM) partiall…

MaleN-MethylaspartateBrain EdemaCyclopentanesIn Vitro TechniquesPharmacologyBicucullineInhibitory postsynaptic potentialHippocampusArticlegamma-Aminobutyric acidCholineGABA AntagonistsRats Sprague-Dawleychemistry.chemical_compoundBilobalideExcitatory Amino Acid AgonistsmedicineAnimalsPicrotoxinDrug InteractionsFuransMolecular Biologygamma-Aminobutyric AcidChemistryGABAA receptorGeneral NeuroscienceBicucullineGABA receptor antagonistBridged Bicyclo Compounds HeterocyclicRatsGinkgolidesNeuroprotective Agentsnervous systemNonlinear DynamicsMechanism of actionArea Under CurveGABAergicNeurology (clinical)medicine.symptomSynaptosomesDevelopmental Biologymedicine.drugBrain Research
researchProduct

Glutamate-containing parenteral nutrition doubles plasma glutamate: A risk factor in neurosurgical patients with blood-brain barrier damage?

1999

OBJECTIVES: Animal studies have shown that the elevation of plasma glutamate levels increase cerebral edema formation whenever the blood-brain barrier is disturbed. Therefore, changes in plasma glutamate levels as influenced by the administration of a glutamate-containing amino acid solution were investigated in neurosurgical patients. DESIGN: Prospective, descriptive study. SETTING: Eight-bed neurosurgical intensive care unit in a university hospital. PATIENTS: Twenty-three neurosurgical patients requiring parenteral nutrition. INTERVENTIONS: Parenteral nutrition was begun 24 hrs after craniotomy. Patients receiving a glutamate-containing amino acid solution (3.75 g/L glutamate) were compa…

MaleParenteral Nutritionmedicine.medical_specialtyGlutamineGlutamic AcidBrain EdemaCritical Care and Intensive Care MedicineBlood–brain barrierCerebral edemaHospitals UniversityRisk FactorsIntensive careInternal medicineBlood plasmamedicineHumansProspective StudiesChromatography High Pressure Liquidchemistry.chemical_classificationAspartic Acidbusiness.industryGlutamate receptorMiddle Agedmedicine.diseaseAmino acidIntensive Care UnitsTreatment OutcomeEndocrinologyParenteral nutritionmedicine.anatomical_structurechemistryBlood-Brain BarrierAnesthesiaFemaleRenal thresholdAsparaginebusinessCraniotomyCritical Care Medicine
researchProduct

Higher vascularity at infiltrated peripheral edema differentiates proneural glioblastoma subtype

2020

[EN] Background and purpose Genetic classifications are crucial for understanding the heterogeneity of glioblastoma. Recently, perfusion MRI techniques have demonstrated associations molecular alterations. In this work, we investigated whether perfusion markers within infiltrated peripheral edema were associated with proneural, mesenchymal, classical and neural subtypes. Materials and methods ONCOhabitats open web services were used to obtain the cerebral blood volume at the infiltrated peripheral edema for MRI studies of 50 glioblastoma patients from The Cancer Imaging Archive: TCGA-GBM. ANOVA and Kruskal-Wallis tests were carried out in order to assess the association between vascular fea…

MalePathologyMolecular compositionPhysiologyPeripheral edemaBrain Edema030218 nuclear medicine & medical imagingDiagnostic Radiology0302 clinical medicineVascularityMedicine and Health SciencesMedicine03.- Garantizar una vida saludable y promover el bienestar para todos y todas en todas las edadesEdemaCerebral Blood VolumeNeurological TumorsProneural GlioblastomaAged 80 and overMultidisciplinaryBlood VolumeBrain NeoplasmsRadiology and ImagingQRMiddle AgedPrognosisMagnetic Resonance Imaging3. Good healthBody FluidsBloodOncologyNeurology030220 oncology & carcinogenesisMedicineFemaleAnalysis of variancemedicine.symptomAnatomyPerfusionResearch ArticleAdultmedicine.medical_specialtyAdolescentImaging TechniquesBlastomaScienceResearch and Analysis Methods03 medical and health sciencesYoung AdultText miningSigns and SymptomsMalignant TumorsDiagnostic MedicineGeneticsHumansAgedRetrospective StudiesMri techniquesCancer och onkologibusiness.industryProportional hazards modelMesenchymal stem cellCancers and NeoplasmsBiology and Life Sciencesmedicine.diseaseSurvival AnalysisCancer and OncologyFISICA APLICADAClinical MedicinebusinessGlioblastoma030217 neurology & neurosurgeryGlioblastoma MultiformeMagnetic Resonance AngiographyGlioblastomaPLoS ONE
researchProduct

Brain Edema and Intracerebral Necrosis Caused by Transcranial Low-Frequency 20-kHz Ultrasound

2006

Background and Purpose— Ultrasound-accelerated thrombolysis is a promising approach toward acute stroke treatment. In previous in vitro studies, we demonstrated enhanced thrombus destruction induced by 20-kHz ultrasound. However, little is known about biological interactions of low-frequency ultrasound with brain tissue. The aim of this in vivo MRI study was to assess safety aspects of transcranial low-frequency ultrasound in rats. Methods— The cranium of 33 male Wistar rats was sonificated for 20 minutes (20-kHz continuous wave). Power output was varied between 0 and 2.6 W/cm 2 . Tympanal and rectal temperature was monitored. Diffusion-weighted imaging and T2-weighted imaging was performe…

MalePathologymedicine.medical_specialtyBrain EdemaBrain IschemiaCerebral edemaBrain ischemiaCentral nervous system diseaseNecrosisIn vivomedicineAnimalsThrombolytic TherapyUltrasonicsRats WistarThrombusStrokeAdvanced and Specialized Nursingmedicine.diagnostic_testbusiness.industryUltrasoundDose-Response Relationship RadiationMagnetic resonance imagingmedicine.diseaseMagnetic Resonance ImagingRatsRadiographyNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessStroke
researchProduct

Evolution of brain abscess in cats formation of capsule and resolution of brain edema

1980

Brain abscess evolution was studied in an experimental model in the cat correlating the computed tomographic scan appearance with intracranial pressure, brain edema and histopathological findings. Brain inflammation was produced by direct inoculation of Staphylococcus aureus into the white matter. Abscesses developed in all animals. The ring enhancement around the necrotic focus seen at an early stage after contrast-medium injection cannot be equated with capsule formation as long as the abscess diameter increased. Parallel to the acute stage of abscess, the intraventricular pressure increased due to the rising mass effect and the spreading edma. The morphological investigations revealed on…

MalePathologymedicine.medical_specialtyIntracranial PressureBrain AbscessBrain EdemaWhite matterEdemaAnimalsMedicineAbscessBrain abscessIntracranial pressureCATSbusiness.industryCapsuleGeneral MedicineStaphylococcal Infectionsmedicine.diseaseDisease Models AnimalMicroscopy Electronmedicine.anatomical_structureCatsFemaleSurgeryNeurology (clinical)Neurosurgerymedicine.symptomTomography X-Ray ComputedbusinessNeurosurgical Review
researchProduct