Search results for "C25"

showing 10 items of 566 documents

The infinite dihedral group

2022

We describe the infinite dihedral group as automaton group. We collect basic results and give full proofs in details for all statements.

FOS: Mathematics20F65 (Primary) 05C25 20E08 68Q70 13F25 (Secondary)Computer Science::Symbolic ComputationGroup Theory (math.GR)Nonlinear Sciences::Cellular Automata and Lattice GasesMathematics - Group TheoryComputer Science::Formal Languages and Automata Theory
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Estrategias de afrontamiento en padres de niños con cáncer

2012

Se pretende conocer las estrategias de afrontamiento que utilizan los padres de niños con cáncer ante al reto que supone esta enfermedad. Se ha utilizado una metodología de investigación grupal y cualitativa. La muestra la constituyen 14 madres/padres cuyos hijos hace más de dos años que han contraído la enfermedad. Se ha comprobado que los padres utilizan una gran variedad de estrategias. Unas son activas: centrada en el problema (adecuación a la acción y calma, entereza y reflexión); regulación afectiva; búsqueda de información; adquisición de apoyo social, instrumental, informativo y/o emocional; reevaluación o reestructuración cognitiva y pensamiento positivo; y distracción. Otras son p…

Family therapyCàncer en els infantsfamilymedia_common.quotation_subjectDiscourse analysislcsh:BF1-990Pediatric oncologycoping strategieslcsh:RC254-282Developmental psychologySocial supportOptimismhospital pedagogy.Distractionmedicinemedia_commonCourageorientación familiarfamiliafamily counselingpedagogía hospitalaria.Cognitive restructuringlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensestrategias de afrontamientoClinical Psychologylcsh:PsychologyOncologyRuminationmedicine.symptomPsychologyCartographyOncología pediátricaPsicooncologia
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A Rare Case of Docetaxel-Induced Hydrocephalus Presenting with Gait Disturbances Mimicking and Coexisting with Taxane-Associated Polyneuropathy: The …

2017

Docetaxel constitutes a widely used chemotherapeutic agent as a first-line treatment for several neoplastic diseases. One of the most common side effects induced by this drug is polyneuropathy, which among other symptoms can cause gait disbalance. However, in exceptional cases gait disturbances could be related to docetaxel-induced hydrocephalus, a rare event that up to the present has been overseen throughout the medical literature and should be meticulously differentiated from polyneuropathy, since its clinical features, treatment, and prognosis differ drastically. We present the case of a woman with a progressive gait disturbance that started immediately after having been treated with do…

Gait disturbancesmedicine.medical_specialtyCase ReportDocetaxelVentricular systemlcsh:RC254-28203 medical and health sciences0302 clinical medicineBreast cancerNormal pressure hydrocephalusPolyneuropathyMedicinebusiness.industryGait Disturbancelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseHydrocephalusSurgeryOncologyDocetaxel030220 oncology & carcinogenesisDifferential diagnosisbusinessPolyneuropathy030217 neurology & neurosurgeryHydrocephalusmedicine.drugCase Reports in Oncology
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Some notes on a superlinear second order Hamiltonian system

2016

Variational methods are used in order to establish the existence and the multiplicity of nontrivial periodic solutions of a second order dynamical system. The main results are obtained when the potential satisfies different superquadratic conditions at infinity. The particular case of equations with a concave-convex nonlinear term is covered.

General Mathematicsmedia_common.quotation_subject010102 general mathematicsMathematical analysisPrimary 34C25; Secondary 34B15; Mathematics (all)Algebraic geometryDynamical systemInfinity01 natural sciencesHamiltonian systemTerm (time)010101 applied mathematicsNonlinear systemNumber theorySecondary 34B15Order (group theory)Primary 34C250101 mathematicsMathematicsmedia_common
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The Clinical Significance of Unknown Sequence Variants in BRCA Genes.

2010

Abstract: Germline mutations in BRCA1/2 genes are responsible for a large proportion of hereditary breast and/or ovarian cancers. Many highly penetrant predisposition alleles have been identified and include frameshift or nonsense mutations that lead to the translation of a truncated protein. Other alleles contain missense mutations, which result in amino acid substitution and intronic variants with splicing effect. The discovery of variants of uncertain/unclassified significance (VUS) is a result that can complicate rather than improve the risk assessment process. VUSs are mainly missense mutations, but also include a number of intronic variants and in-frame deletions and insertions. Over …

GeneticsCancer ResearchBRCA genesNonsense mutationReviewBiologylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282oncogenetic counselingFrameshift mutationintegrated modelsGermline mutationOncologyvariantRNA splicingMissense mutationClinical significanceAlleleGeneCancers
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Putative Breast Cancer Driver Mutations in TBX3 Cause Impaired Transcriptional Repression

2015

The closely related T-box transcription factors TBX2 and TBX3 are frequently overexpressed in melanoma and various types of human cancers, in particular, breast cancer. The overexpression of TBX2 and TBX3 can have several cellular effects, among them suppression of senescence, promotion of epithelial–mesenchymal transition, and invasive cell motility. In contrast, loss of function of TBX3 and most other human T-box genes causes developmental haploinsufficiency syndromes. Stephens and colleagues (1), by exome sequencing of breast tumor samples, identified five different mutations in TBX3, all affecting the DNA-binding T-domain. One in-frame deletion of a single amino acid, p.N212delN, was ob…

GeneticsCancer Researchp21frameshift mutationin-frame deletionMelanomadriver mutationTBX3Biologylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaselcsh:RC254-282Frameshift mutationbreast cancerBreast cancerOncologymedicinesomatic mutationsHaploinsufficiencyGeneTranscription factorLoss functionExome sequencingOriginal ResearchFrontiers in Oncology
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Simultaneous Aurora-A/STK15 overexpression and centrosome amplification induce chromosomal instability in tumour cells with a MIN phenotype

2007

Abstract Background Genetic instability is a hallmark of tumours and preneoplastic lesions. The predominant form of genome instability in human cancer is chromosome instability (CIN). CIN is characterized by chromosomal aberrations, gains or losses of whole chromosomes (aneuploidy), and it is often associated with centrosome amplification. Centrosomes control cell division by forming a bipolar mitotic spindle and play an essential role in the maintenance of chromosomal stability. However, whether centrosome amplification could directly cause aneuploidy is not fully established. Also, alterations in genes required for mitotic progression could be involved in CIN. A major candidate is represe…

Genome instabilityCancer ResearchCellular differentiationAneuploidyApoptosisCell CommunicationSpindle ApparatusBiologyProtein Serine-Threonine Kinaseslcsh:RC254-282Aurora KinasesChromosome instabilityChromosomal InstabilitymedicineTumor Cells CulturedGeneticsHumansRNA Small InterferingMitosisIn Situ Hybridization FluorescenceAurora Kinase ACentrosomePloidiesReverse Transcriptase Polymerase Chain ReactionAurora-A centrosomes amplification aneuploidyCell Differentiationlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseAneuploidyCell biologySpindle apparatusUp-RegulationSettore BIO/18 - GeneticaCell Transformation NeoplasticPhenotypeMicroscopy FluorescenceOncologyCentrosomeColonic NeoplasmsEctopic expressionMicrosatellite InstabilityResearch ArticleBMC Cancer
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CENPA overexpression promotes genome instability in pRb-depleted human cells

2009

Abstract Background Aneuploidy is a hallmark of most human cancers that arises as a consequence of chromosomal instability and it is frequently associated with centrosome amplification. Functional inactivation of the Retinoblastoma protein (pRb) has been indicated as a cause promoting chromosomal instability as well centrosome amplification. However, the underlying molecular mechanism still remains to be clarified. Results Here we show that pRb depletion both in wild type and p53 knockout HCT116 cells was associated with the presence of multipolar spindles, anaphase bridges, lagging chromosomes and micronuclei harbouring whole chromosomes. In addition aneuploidy caused by pRb acute loss was…

Genome instabilityCancer ResearchChromosomal Proteins Non-HistoneBlotting WesternBiologyAutoantigensRetinoblastoma Proteinlcsh:RC254-282Genomic InstabilityRNA interferenceChromosome instabilityCentromere Protein ACell Line TumorHumansRNA Processing Post-TranscriptionalDNA PrimersCENPABase SequenceReverse Transcriptase Polymerase Chain ReactionResearchRetinoblastoma proteincentromere protein aneuploidy pRBlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensMolecular biologyCell biologySettore BIO/18 - GeneticaSpindle checkpointOncologyMicroscopy FluorescenceCentrosomebiology.proteinMolecular MedicineRNA Interferencebiological phenomena cell phenomena and immunityCentromere Protein AMolecular Cancer
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TCR Clonality and Genomic Instability Signatures as Prognostic Biomarkers in High Grade Serous Ovarian Cancer.

2021

Simple Summary High-grade serous ovarian carcinoma (HGSC) could be analyzed with a molecular stratification defined by different genomic instability signatures associated with specific mutational process and prognostic biomarkers. Immune infiltrate is known to be a robust biomarker in HGSC. We aimed to investigate immune parameters according to genomic instability signatures. We observed that homologous recombination deficiency positive, copy cumber variant signature 7 and TCR (T cells receptor) clonality are good prognostic biomarkers in HGSC. Combining TCR clonality and genomic instability signature or T cell infiltration improved the prognostic value compared to each variable taken alone…

Genome instabilityCancer ResearchTumor microenvironmentmedicine.medical_treatmentT cellT-cell receptorTCR clonalityNeoplasms. Tumors. Oncology. Including cancer and carcinogensbiomarkersImmunotherapyBiologyHGSCArticleSerous fluidImmune systemmedicine.anatomical_structureOncologyHRDmedicineCancer researchCopy-number variationprognosticRC254-282Cancers
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Bypass of cell cycle arrest induced by transient DNMT1 post-transcriptional silencing triggers aneuploidy in human cells

2012

Abstract Background Aneuploidy has been acknowledged as a major source of genomic instability in cancer, and it is often considered the result of chromosome segregation errors including those caused by defects in genes controlling the mitotic spindle assembly, centrosome duplication and cell-cycle checkpoints. Aneuploidy and chromosomal instability has been also correlated with epigenetic alteration, however the molecular basis of this correlation is poorly understood. Results To address the functional connection existing between epigenetic changes and aneuploidy, we used RNA-interference to silence the DNMT1 gene, encoding for a highly conserved member of the DNA methyl-transferases. DNMT1…

Genome instabilityCell cycle checkpointDNA damageAneuploidyBiologylcsh:RC254-282BiochemistryChromosome instabilitymedicineCentrosome duplicationEpigeneticsaneuploidylcsh:QH573-671Molecular BiologyGeneticsDNA methylationG1 arrestlcsh:CytologyResearchDNMT1Cell Biologylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseCell biologySettore BIO/18 - GeneticaDNMT1 Aneuploidy epigenetic p14/ARF siRNADNA methylation
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