Search results for "C3"

showing 10 items of 1295 documents

Protection in a model of liver injury is parallel to energy mobilization capacity under distinct nutritional status

2019

International audience; Objective: Dietary and energetic restrictions are endowed with protection against experimental injuries. However, a drop in cell energetic status under a critical threshold may prevent protection, as previously observed for livers isolated from rat donors undergoing 18-h fasting versus feeding. The aim of this study was to further explore, in the latter model, links between nutritional status, energy availability, and protection through lengthening of rat fasting to 24 h and withdrawal of energy sources from perfusions.Methods: Energy-free perfused ex vivo livers from fed, 18-h-fasted, and 24-h-fasted rats were studied during 135 min for cytolysis (potassium, asparta…

0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and Metabolism[SDV]Life Sciences [q-bio]Nutritional Status030209 endocrinology & metabolismCaspase 303 medical and health scienceschemistry.chemical_compound0302 clinical medicineLactate dehydrogenaseInternal medicineAutophagymedicineAnimalsOrgan protection2. Zero hungerLiver injury030109 nutrition & dieteticsNutrition and DieteticsLiver injury modelGlycogenLiver cytolysisAutophagyEnergy mobilizationFastingSciences bio-médicales et agricolesProtective Factorsmedicine.diseaseLC3IIRats[SDV] Life Sciences [q-bio]PerfusionCytolysisDisease Models AnimalEndocrinologychemistryLiverApoptosisChemical and Drug Induced Liver InjuryEnergy sourceEnergy Metabolism
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Genetic inactivation of the sigma-1 chaperone protein results in decreased expression of the R2 subunit of the GABA-B receptor and increased suscepti…

2021

There is a growing body of evidence demonstrating the significant involvement of the sigma-1 chaperone protein in the modulation of seizures. Several sigma-1 receptor (Sig1R) ligands have been demonstrated to regulate the seizure threshold in acute and chronic seizure models. However, the mechanism by which Sig1R modulates the excitatory and inhibitory pathways in the brain has not been elucidated. The aim of this study was to compare the susceptibility to seizures of wild type (WT) and Sig1R knockout (Sig1R−/−) mice in intravenous pentylenetetrazol (PTZ) and (+)-bicuculline (BIC) infusion-induced acute seizure and Sig1R antagonist NE-100-induced seizure models. To determine pos…

0301 basic medicinemedicine.medical_specialtyKnockoutGene ExpressionNitric Oxide Synthase Type IISigma-1 receptorConvulsantsAnisolesSigma-1 receptor Knockout GABA-B receptor Seizures Medial habenula NE-100BicucullineHippocampuslcsh:RC321-571Mice03 medical and health sciences0302 clinical medicineDownregulation and upregulationSeizuresInternal medicineGene expressionmedicineAnimalsReceptors sigmaGABA-B receptorGenetic Predisposition to DiseasePentylenetetrazolReceptorlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMice KnockoutHabenulaSigma-1 receptorPropylaminesSeizure thresholdChemistryMedial habenulaWild typeAntagonistReceptors GABA-A030104 developmental biologyEndocrinologyReceptors GABA-BNeurologyNE-100Pentylenetetrazole030217 neurology & neurosurgerymedicine.drugNeurobiology of Disease
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Vaccination in multiple sclerosis patients treated with highly effective disease-modifying drugs: an overview with consideration of cladribine tablets

2021

Infectious diseases are an important consideration in autoimmune conditions such as multiple sclerosis. Infective episodes may trigger relapses and significantly deteriorate the course of the disease. Some immunotherapies may cause increased rates of infection-related adverse events. Thus, infection and vaccine-related issues should be included in the individualized patient-specific treatment strategy and counseling before starting therapy and regularly on treatment. Clinical and epidemiological studies as well as pharmacovigilance data repeatedly demonstrated the safety of the great majority of vaccines in multiple sclerosis patients. Moreover, studies have shown that vaccinations with kil…

0301 basic medicinemedicine.medical_specialtyMedizincladribine tabletsDiseaseReviewimmunizationmultiple sclerosis1117 Public Health and Health Services03 medical and health sciences0302 clinical medicineNatalizumabInternal medicinevaccinePharmacovigilancemedicineddc:610CladribineAdverse effectRC346-42911 Medical and Health SciencesPharmacologybusiness.industryMultiple sclerosisCOVID-19Vaccine efficacymedicine.diseaseVaccinationCoronavirus030104 developmental biologyNeurology1107 ImmunologyNeurology (clinical)Neurology. Diseases of the nervous systembusiness030217 neurology & neurosurgerymedicine.drugTherapeutic Advances in Neurological Disorders
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Obesity as a Risk Factor for Alzheimer’s Disease: Implication of Leptin and Glutamate

2019

Obesity is known to induce leptin and insulin resistance. Leptin is a peptide hormone synthesized in adipose tissue that mainly regulates food intake. It has been shown that insulin stimulates the production of leptin when adipocytes are exposed to glucose to encourage satiety; while leptin, via a negative feedback, decreases the insulin release and enhances tissue sensitivity to it, leading to glucose uptake for energy utilization or storage. Therefore, resistance to insulin is closely related to leptin resistance. Obesity in middle age has also been related to Alzheimer’s disease (AD). In recent years, the relation between impaired leptin signaling pathway and the onset of AD has been stu…

0301 basic medicinemedicine.medical_specialtyMini Reviewmedicine.medical_treatmentGlucose uptakeExcitotoxicityAdipose tissuemedicine.disease_causelcsh:RC321-57103 medical and health sciences0302 clinical medicineInsulin resistanceInternal medicinemedicineoverweightleptin-resistanceReceptorlcsh:Neurosciences. Biological psychiatry. Neuropsychiatrybusiness.industryGeneral NeuroscienceInsulinLeptindigestive oral and skin physiologyGlutamate receptormedicine.disease030104 developmental biologyEndocrinologyLTPbusinessexcitotoxicity030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsNeurosciencedementiaFrontiers in Neuroscience
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The Assessment of Serum Concentrations of AGEs and Their Soluble Receptor (sRAGE) in Multiple Sclerosis Patients

2021

Background: Advanced glycation end products (AGEs) are involved in the pathogenesis of many diseases, including neurodegenerative diseases such as multiple sclerosis (MS). The aim of the study was to determine serum concentrations of AGEs and their soluble receptor (sRAGE) in MS patients and healthy controls and to investigate their possible influence on disease activity. Methods: Serum concentrations of AGE and sRAGE in patients with MS and healthy controls were determined by enzyme-linked immunosorbent assay (ELISA). Results: The mean serum AGE concentration in patients with MS was higher than in healthy controls, whereas the mean serum sRAGE concentration was lower than in the control gr…

0301 basic medicinemedicine.medical_specialtyNeurosciences. Biological psychiatry. NeuropsychiatryDiseasemultiple sclerosisGastroenterologyArticlePathogenesis03 medical and health sciencesAGE0302 clinical medicineGlycationInternal medicineMedicineIn patientReceptoradvanced glycation end products; AGE; RAGE; sRAGE; multiple sclerosis; ELISAExpanded Disability Status Scalebusiness.industryadvanced glycation end productsGeneral NeuroscienceMultiple sclerosisSerum concentrationmedicine.diseaseRAGE030104 developmental biologyELISAbusiness030217 neurology & neurosurgeryRC321-571sRAGEBrain Sciences
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Diagnostic Criteria for Parkinson's Disease: From James Parkinson to the Concept of Prodromal Disease.

2018

The diagnosis of Parkinson’s disease (PD) is based on clinical features and differently to the common opinion that detecting this condition is easy, seminal clinico-pathological studies have shown that up one-fourth of patients diagnosed as PD during life has an alternative diagnosis at post-mortem. The misdiagnosis is even higher when only the initial diagnosis is considered, since the diagnostic accuracy improves by time, during follow-up visits. Given that the confirmation of the diagnosis of PD can be only obtained through neuropathology, to improve and facilitate the diagnostic-therapeutic workup in PD, a number of criteria and guidelines have been introduced in the last three decades.…

0301 basic medicinemedicine.medical_specialtyParkinson's disease diagnostic criteriapreclinical Parkinson’s diseaseParkinson's diseaseParkinson’s disease guidelinesNeuropathologyDiseaseReviewlcsh:RC346-429prodromal Parkinson’s disease03 medical and health sciences0302 clinical medicinePreclinical Parkinson's diseasemedicineProdromal Parkinson's diseaseIntensive care medicinelcsh:Neurology. Diseases of the nervous systemJames Parkinsonbusiness.industryTask forceNoveltymedicine.diseaseClinical trial030104 developmental biologyNeurologyClinical diagnosisParkinson's disease guidelineParkinson’s disease diagnostic criteriaNeurology (clinical)Early phasebusiness030217 neurology & neurosurgeryNeuroscienceFrontiers in neurology
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Somatosensory Training Improves Proprioception and Untrained Motor Function in Parkinsons Disease

2018

Background: Proprioceptive impairment is a common feature of Parkinson's disease (PD). Proprioceptive function is only partially restored with anti-parkinsonian medication or deep brain stimulation. Behavioral exercises focusing on somatosensation have been promoted to overcome this therapeutic gap. However, conclusive evidence on the effectiveness of such somatosensory-focused behavioral training for improving somatosensory function is lacking. Moreover, it is unclear, if such training has any effect on motor performance in PD.Objective: To investigate, whether proprioception improves with a somatosensory focused, robot-aided training in people with PD (PWPs), and whether enhanced proprioc…

0301 basic medicinemedicine.medical_specialtyParkinson's diseaseDeep brain stimulationMovement disordersmedicine.medical_treatmenteducationWristSomatosensory systemlcsh:RC346-42903 medical and health sciences0302 clinical medicinePhysical medicine and rehabilitationmedicinerobotic rehabilitationNeurorehabilitationlcsh:Neurology. Diseases of the nervous systemproprioceptive learningOriginal ResearchneurorehabilitationProprioceptionsensorimotor learningbusiness.industrymedicine.diseaseProprioceptive function030104 developmental biologymedicine.anatomical_structureNeurologysomatosensory learningmovement disordersNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryFrontiers in Neurology
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Vancomycin resistant Enterococcus faecium (VRE) vertebral osteomyelitis after uneventful spinal surgery: A case report and literature review

2017

Abstract Objective Case report and literature review. Background Enterococcus faecium is an emerging pathogen responsible for post procedural infections in patients who have undergone spinal decompression surgery. In this case report, the authors discuss and review recent literature on approaches to post-operative spinal infection. Case report We herein report the case of a 55-year-old HIV-negative Caucasian Italian woman who showed vertebral osteomyelitis with abscesses around the interbody cage caused by an Enterococcus faecium vancomycin resistant gen-Van A, following a Transforaminal Lumbar Interbody Fusion (TLIF). The same strain was detected in disc biopsy, urine culture and rectal sw…

0301 basic medicinemedicine.medical_specialtySettore MED/07 - Microbiologia E Microbiologia ClinicaEnterococcus faecium; Spinal surgery; Transforaminal Lumbar Interbody Fusion (TLIF); Vertebral osteomyelitis; Surgery; Neurology (clinical)Settore MED/17 - Malattie Infettive030106 microbiologyEnterococcus faeciumlcsh:Surgerylcsh:RC346-42903 medical and health sciencesEmerging pathogen0302 clinical medicineAntibiotic resistanceVertebral osteomyelitisBiopsymedicineVertebral osteomyelitislcsh:Neurology. Diseases of the nervous systemVancomycin resistant Enterococcus faeciumTransforaminal Lumbar Interbody Fusion (TLIF)medicine.diagnostic_testbiologybusiness.industrySettore MED/27 - Neurochirurgialcsh:RD1-811biology.organism_classificationmedicine.diseaseSpinal surgerySurgerySurgeryImplantSpinal surgeryNeurology (clinical)Vertebral osteomyelitibusiness030217 neurology & neurosurgeryEnterococcus faeciumInterdisciplinary Neurosurgery
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Successful management of a cerebral abscess secondary to chronic cholesteatoma caused by Prevotella melaninogenica and Peptococcus anaerobius – A cas…

2016

Abstract Cerebral abscess following cholesteatomatous otomastoiditis is a life-threatening complication and poses diagnostic and therapeutic challenges. We report a case of life-threatening cerebritis and cerebral abscess due to a collection of pus from an aerobic super infections occurring months after the apparent resolution of an otogenic brain abscess in a 67-year-old immunocompetent Italian female. Two gram-positive anaerobic pathogens were isolated during secondary neurosurgical procedures and antibiotic treatment was adopted to resolve the complications. Another objective of this study was to review the literature on gram-positive anaerobic pathogens and brain abscess complications i…

0301 basic medicinemedicine.medical_specialtySettore MED/17 - Malattie InfettiveFistula030106 microbiologylcsh:Surgerylcsh:RC346-429Surgery; Neurology (clinical)Prevotella melaninogenica03 medical and health sciences0302 clinical medicineMedicineAbscessBrain abscesslcsh:Neurology. Diseases of the nervous systembusiness.industrySettore MED/27 - NeurochirurgiaCholesteatomalcsh:RD1-811medicine.diseasebacterial infections and mycosesSurgeryInfectious disease (medical specialty)CerebritisSurgeryNeurology (clinical)medicine.symptombusinessComplication030217 neurology & neurosurgeryInterdisciplinary Neurosurgery
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Reducing Peripheral Inflammation with Infliximab Reduces Neuroinflammation and Improves Cognition in Rats with Hepatic Encephalopathy

2016

Inflammation contributes to cognitive impairment in patients with hepatic encephalopathy (HE). However, the process by which peripheral inflammation results in cognitive impairment remains unclear. In animal models, neuroinflammation and altered neurotransmission mediate cognitive impairment. Taking into account these data, we hypothesized that in rats with HE: (1) peripheral inflammation is a main contributor to neuroinflammation; (2) neuroinflammation in hippocampus impairs spatial learning by altering AMPA and/or NMDA receptors membrane expression; (3) reducing peripheral inflammation with infliximab (anti-TNF-a) would improve spatial learning; (4) this would be associated with reduced n…

0301 basic medicinemedicine.medical_specialtyTNF-ahepatic encephalopathyHippocampusMorris water navigation taskInflammationAMPA receptorNeurotransmissionlcsh:RC321-571neuroinflammation03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineInternal medicinemedicineneurotransmissionlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular BiologyNeuroinflammationOriginal Researchcognitive impairmentbusiness.industryGlutamate receptor030104 developmental biologyEndocrinologynervous systemNMDA receptormedicine.symptombusinessNeuroscience030217 neurology & neurosurgeryNeuroscience
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