Search results for "C3"

showing 10 items of 1295 documents

Music lessons improve auditory perceptual and cognitive performance in deaf children

2014

Despite advanced technologies in auditory rehabilitation of profound deafness, deaf children often exhibit delayed cognitive and linguistic development and auditory training remains a crucial element of their education. In the present cross-sectional study, we assess whether music would be a relevant tool for deaf children rehabilitation. In normal-hearing children, music lessons have been shown to improve cognitive and linguistic-related abilities, such as phonetic discrimination and reading. We compared auditory perception, auditory cognition, and phonetic discrimination between 14 profoundly deaf children who completed weekly music lessons for a period of 1.5 to 4 years and 14 deaf child…

Auditory perceptionAuditory scene analysisauditory working memorymedia_common.quotation_subjectbehavioral disciplines and activitieslcsh:RC321-571Behavioral NeurosciencePerceptionReading (process)phonetic discriminationotorhinolaryngologic diseasesEffects of sleep deprivation on cognitive performancelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological PsychiatryOriginal Researchmedia_commonWorking memoryCongenitally deaf childrenCognitionPsychiatry and Mental healthNeuropsychology and Physiological PsychologyNeurologyAuditory PerceptionPsychologymusic trainingPeriod (music)NeuroscienceCognitive psychologyFrontiers in Human Neuroscience
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Prior Precision Modulates the Minimization of Auditory Prediction Error

2019

International audience; The predictive coding model of perception proposes that successful representation of the perceptual world depends upon canceling out the discrepancy between prediction and sensory input (i.e., prediction error). Recent studies further suggest a distinction to be made between prediction error triggered by non-predicted stimuli of different prior precision (i.e., inverse variance). However, it is not fully understood how prediction error with different precision levels is minimized in the predictive process. Here, we conducted a magnetoencephalography (MEG) experiment which orthogonally manipulated prime-probe relation (for contextual precision) and stimulus repetition…

Auditory perceptionrepetitionMean squared prediction errorSpeech recognitionmedia_common.quotation_subjectStimulus (physiology)050105 experimental psychologylcsh:RC321-571Cognitive Penetration[SCCO]Cognitive science03 medical and health sciencesBehavioral Neuroscience0302 clinical medicinePerceptual learningPerceptionmedicinemagnetoencephalography (MEG)0501 psychology and cognitive sciencesaivotutkimuspredictive codinglcsh:Neurosciences. Biological psychiatry. Neuropsychiatryennakointita515Biological PsychiatryOriginal ResearchVisual CortexMathematicsmedia_commonPredictive codingprediction errorMEGmedicine.diagnostic_testmagnetoencephalagraphy (MEG)[SCCO.NEUR]Cognitive science/Neuroscience05 social sciencesMagnetoencephalographykuuloauditory perceptionPsychiatry and Mental healthNeuropsychology and Physiological Psychologyhavainnointi ja aistiminenNeurologyMinificationtoistoärsykkeet030217 neurology & neurosurgeryNeuroscienceCoding TheoryFrontiers in Human Neuroscience
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Neuropsychological Profile, Emotional/Behavioral Problems, and Parental Stress in Children with Neurodevelopmental Disorders

2021

Background: The aim of our study was to trace a specific neuropsychological profile, to investigate emotional-behavioral problems and parental stress in children with Autism Spectrum Disorder Level 1/High functioning (ASD-HF), Specific Learning Disorders (SLD) and Attention Deficit/Hyperactivity Disorder (ADHD) disorders and to highlight similarities and differences among the three groups. Methods: We retrospectively collected the data from a total of 62 subjects with ASD-HF (n = 19) ADHD (n = 21), SLD (n = 22) and 20 typical development. All the participants underwent neuropsychological standardized test for the evaluation of cognitive profile (Wechsler Intelligence Scale for Children Four…

Autism Spectrum DisorderCBCLNeurosciences. Biological psychiatry. Neuropsychiatryemotional/behavioral problemparental stressbehavioral disciplines and activitiesArticle03 medical and health sciences0302 clinical medicineemotional/behavioral problemsmental disordersmedicineSpecific Learning DisorderAttention deficit hyperactivity disorder0501 psychology and cognitive sciencesSpecific Learning DisorderChild Behavior ChecklistWechsler Intelligence Scale for ChildrenSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaGeneral Neuroscience05 social sciencesNeuropsychologyCognitionmedicine.diseaseAutism Spectrum Disorder; Specific Learning Disorder; Attention Deficit/Hyperactivity Disorder; cognitive profile; emotional/behavioral problems; parental stressSettore MED/39 - Neuropsichiatria Infantilecognitive profileAutism spectrum disorderParental streAttention Deficit/Hyperactivity DisorderPsychology030217 neurology & neurosurgery050104 developmental & child psychologyClinical psychologyRC321-571Brain Sciences
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Transcriptomic metaanalyses of autistic brains reveals shared gene expression and biological pathway abnormalities with cancer

2019

Este es el artículo que se ha publicado de forma definitiva en: https://molecularautism.biomedcentral.com/articles/10.1186/s13229-019-0262-8 En este artículo también participa Joan Climent, Vera Pancaldi, Lourdes Fañanás, Celso Arango, Mara Parellada, Anaïs Baudot, Daniel Vogt, John L. Rubenstein, Alfonso Valencia y Rafael Tabarés-Seisdedos. Background: Epidemiological and clinical evidence points to cancer as a comorbidity in people with autism spectrum disorders (ASD). A significant overlap of genes and biological processes between both diseases has also been reported. Methods: Here, for the first time, we compared the gene expression profiles of ASD frontal cortex tissues and 22 cancer t…

Autism.AutismComorbidityBioinformaticsAutismo.lcsh:RC346-429Expresión génica.TranscriptomeAfectividad - Trastornos.0302 clinical medicineNeoplasmsGene expression2.1 Biological and endogenous factorsAetiologyCàncerCáncer - Aspectos genéticos.ComputingMilieux_MISCELLANEOUSCancer0303 health sciencesProstate CancerBrainAffective disorders.3. Good healthPsychiatry and Mental healthMental HealthSignal transductionSignal TransductionBiotechnologyUrologic DiseasesIntellectual and Developmental Disabilities (IDD)1.1 Normal biological development and functioningClinical SciencesBiologyASDBiological pathway03 medical and health sciencesDevelopmental NeuroscienceUnderpinning researchmental disordersGeneticsmedicineHumansAutistic DisorderIntellectual and Developmental DisabilitiesGeneMolecular Biologylcsh:Neurology. Diseases of the nervous systemPI3K/AKT/mTOR pathwayCancer - Genetic aspects.030304 developmental biologyResearchNeurosciencesMultimorbidityCancermedicine.diseaseExpressió gènicaHuman geneticsBrain DisordersMeta-analysisGene expression.AutismGene expressionAutisme[INFO.INFO-BI]Computer Science [cs]/Bioinformatics [q-bio.QM]TranscriptomeKidney cancer030217 neurology & neurosurgeryDevelopmental BiologyMolecular Autism
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Impaired circadian heart rate variability in Parkinson’s disease: a time-domain analysis in ambulatory setting

2020

Abstract Background Heart rate variability (HRV) decreases in Parkinson’s disease (PD) and it can be considered a marker for cardiovascular dysautonomia. The purpose of this pilot study is to evaluate long-term time-domain analysis of HRV of PD patients and compare the results with those of matched healthy individuals. Methods Idiopathic PD patients without comorbidity impairing HRV, and age-matched healthy individuals were recruited in a pilot study. A long-term time domain analysis of HRV using 24-h ambulatory ECG was performed. Results Overall, 18 PD patients fulfilling inclusion criteria completed the evaluation (mean age was 55.6 ± 8.8, disease duration: 5.0 ± 4.7). Mean SCOPA-AUT scor…

Autonomic disordersmedicine.medical_specialtyParkinson's diseaseNeurologyAutonomic disorderPopulationPilot ProjectsNon-motor symptomsPrimary DysautonomiasAutonomic disorderlcsh:RC346-429Antiparkinson AgentsLevodopaHeart RateInternal medicinemedicineHumansHeart rate variabilityeducationlcsh:Neurology. Diseases of the nervous systemAgededucation.field_of_studybusiness.industryDysautonomiaParkinson DiseaseGeneral MedicineMiddle Agedmedicine.diseaseComorbidityCircadian RhythmHeart rate variability SCOPA-AUTCardiovascular DiseasesAmbulatoryCardiologyNeurology (clinical)medicine.symptombusinessResearch ArticleBMC Neurology
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A novel function of Huntingtin in the cilium and retinal ciliopathy in Huntington's disease mice

2015

Huntington's disease (HD) is a neurodegenerative disorder caused by the toxic expansion of polyglutamine in the Huntingtin (HTT) protein. The pathomechanism is complex and not fully understood. Increasing evidence indicates that the loss of normal protein function also contributes to the pathogenesis, pointing out the importance of understanding the physiological roles of HTT. We provide evidence for a novel function of HTT in the cilium. HTT localizes in diverse types of cilia — including 9 + 0 non-motile sensory cilia of neurons and 9 + 2 motile multicilia of trachea and ependymal cells — which exert various functions during tissue development and homeostasis. In the photoreceptor cilium,…

AxonemeMalecongenital hereditary and neonatal diseases and abnormalitiesHuntingtinCentrioleMice TransgenicNerve Tissue ProteinsBiologyMicrotubulesPhotoreceptor cellRetinalcsh:RC321-571MiceHuntington's diseaseIntraflagellar transportmental disordersmedicineAnimalsHumansPhotoreceptor CellsHuntingtinCilialcsh:Neurosciences. Biological psychiatry. NeuropsychiatryComputingMilieux_MISCELLANEOUSHuntingtin ProteinPhotoreceptorCiliumNuclear ProteinsHuntington's diseasemedicine.diseaseCell biologyCiliopathyDisease Models Animalmedicine.anatomical_structureHEK293 CellsHuntington DiseaseNeurologyFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]sense organs
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Lipofuscin Hypothesis of Alzheimer’s Disease

2011

The primary culprit responsible for Alzheimer’s disease (AD) remains unknown. Aβ protein has been identified as the main component of amyloid of senile plaques, the hallmark lesion of AD, but it is not definitively established whether the formation of extracellular Aβ deposits is the absolute harbinger of the series of pathological events that hit the brain in the course of sporadic AD. The aim of this paper is to draw attention to a relatively overlooked age-related product, lipofuscin, and advance the hypothesis that its release into the extracellular space following the death of neurons may substantially contribute to the formation of senile plaques. The presence of intraneuronal Aβ, sim…

Aβ proteinNeurofibrillary tanglesAmyloidAmyloidCognitive Neurosciencelcsh:Geriatricslcsh:RC346-429LipofuscinLipofuscinLesionExtracellularMedicineOriginal Research ArticleSenile plaquesPathologicallcsh:Neurology. Diseases of the nervous systembusiness.industryMacular degenerationAlzheimer's diseaseMacular degenerationmedicine.diseaseBiochemistry of Alzheimer's diseaselcsh:RC952-954.6Psychiatry and Mental healthmedicine.symptombusinessAlzheimer’s diseaseNeuroscienceDementia and Geriatric Cognitive Disorders Extra
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Differential efferent projections of the anterior, posteroventral, and posterodorsal subdivisions of the medial amygdala in mice

2012

The medial amygdaloid nucleus (Me) is a key structure in the control of sociosexual behaviour in mice. It receives direct projections from the main and accessory olfactory bulbs, as well as an important hormonal input. To better understand its behavioural role, in this work we investigate the structures receiving information from the Me, by analysing the efferent projections from its anterior (MeA), posterodorsal (MePD) and posteroventral (MePV) subdivisions, using anterograde neuronal tracing with biotinylated and tetrametylrhodamine-conjugated dextranamines.The Me is strongly interconnected with the rest of the chemosensory amygdala, but shows only moderate projections to the central nucl…

BiologiaEfferentNeuroscience (miscellaneous)BiologyAmygdalachemical signalslcsh:RC321-571lcsh:QM1-695ventromedial hypothalamusCellular and Molecular Neurosciencesexual behaviorPiriform cortexvomeronasal amygdalamedicinedefensive behaviourdefensive behaviorOriginal Research Articlelcsh:Neurosciences. Biological psychiatry. Neuropsychiatrysexual behaviourlcsh:Human anatomyGranule cellNeuronal tracingStria terminalismedicine.anatomical_structurenervous systemolfactory amygdalaHypothalamusAnatomyNucleusNeuroscienceNeuroscience
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An inherited deficiency of the third component of complement, C3, in guinea pigs

1986

Hereditary deficiency of the third component of complement, C3, is found very seldom in the human. C3 deficiency is associated with severe bacterial infections revealing the central role of C3 in complement activation via the classical or alternative pathway. We describe a new hereditary C3 deficiency in strain 2 guinea pigs. Serum from these animals had a markedly reduced lytic activity in a standard assay for complement-dependent, antibody-mediated cytotoxicity. In functional assays of individual components, the hemolytic activity of the components C4, C2, C5 and of factors B, D and H was in the normal range. The functional C3 titer, and similarly C3 antigenic activity in the serum of the…

Blood Bactericidal ActivityGuinea PigsImmunologyMacrophage-1 Antigenchemical and pharmacologic phenomenaBiologyHemolysisMajor Histocompatibility ComplexGuinea pigInbred strainAntigenIn vivoAnimalsImmunology and AllergyComplement ActivationRecombination GeneticComplement C3Molecular biologyIn vitroPedigreeReceptors ComplementComplement systemImmunologyAlternative complement pathwaybiology.proteinC3a receptorEuropean Journal of Immunology
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Association of obesity with proteasomal gene polymorphisms in children.

2013

The aim of this study was to ascertain possible associations between childhood obesity, its anthropometric and clinical parameters, and three loci of proteasomal genes rs2277460 (PSMA6c.-110C>A), rs1048990 (PSMA6c.-8C>G), and rs2348071 (PSMA3c. 543+138G>A) implicated in obesity-related diseases. Obese subjects included 94 otherwise healthy children in Latvia. Loci were genotyped and then analyzed using polymerase chain reactions, with results compared to those of 191 nonobese controls.PSMA3SNP frequency differences between obese children and controls, while not reaching significance, suggested a trend. These differences, however, proved highly significant (P<0.002) in the subset…

Blood GlucoseMalelcsh:Internal medicinePediatric ObesityProteasome Endopeptidase ComplexArticle SubjectAdolescentGenotypeEndocrinology Diabetes and MetabolismPSMA6Blood PressurePSMA3Polymorphism Single NucleotideChildhood obesityBody Mass IndexmedicineHumansGenetic Predisposition to Diseaselcsh:RC31-1245ChildGeneGeneticsAnalysis of Variancebusiness.industryCholesterol LDLAnthropometrymedicine.diseaseObesityCase-Control StudiesChild PreschoolFemalebusinessResearch ArticleJournal of obesity
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