Search results for "C3"

showing 10 items of 1295 documents

Quantitative mass spectrometry for human melanocortin peptides in vitro and in vivo suggests prominent roles for β-MSH and desacetyl α-MSH in energy …

2018

Objective The lack of pro-opiomelanocortin (POMC)-derived melanocortin peptides results in hypoadrenalism and severe obesity in both humans and rodents that is treatable with synthetic melanocortins. However, there are significant differences in POMC processing between humans and rodents, and little is known about the relative physiological importance of POMC products in the human brain. The aim of this study was to determine which POMC-derived peptides are present in the human brain, to establish their relative concentrations, and to test if their production is dynamically regulated. Methods We analysed both fresh post-mortem human hypothalamic tissue and hypothalamic neurons derived from …

MalePluripotent Stem CellsLeptinlcsh:Internal medicineendocrine systemhPSC human pluripotent stem cellsPro-Opiomelanocortin[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyHypothalamusMass SpectrometryTandem Mass Spectrometry[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]beta-MSHHomeostasisHumansHuman pluripotent stem cellObesitylcsh:RC31-1245MSHNeuronsintegumentary systemReceptors MelanocortinLC-MS/MS liquid chromatography tandem mass spectrometryNeuropeptidesdigestive oral and skin physiologyPOMCPVH the paraventricular nucleus of the hypothalamusCTX cerebral cortexMelanocortinsNeuropeptidealpha-MSHOriginal ArticleFemalehormones hormone substitutes and hormone antagonistsChromatography Liquid
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The chronnectome of musical beat

2020

Keeping time is fundamental for our everyday existence. Various isochronous activities, such as locomotion, require us to use internal timekeeping. This phenomenon comes into play also in other human pursuits such as dance and music. When listening to music, we spontaneously perceive and predict its beat. The process of beat perception comprises both beat inference and beat maintenance, their relative importance depending on the salience of beat in the music. To study functional connectivity associated with these processes in a naturalistic situation, we used functional magnetic resonance imaging to measure brain responses of participants while they were listening to a piece of music contai…

MalePower graph analysisPeriodicityInferencemusiikkipsykologiatoiminnallinen magneettikuvaus0302 clinical medicineCerebellumMusic information retrievalDefault mode networkmedia_commonmedicine.diagnostic_testfMRI05 social sciencesMotor CortexMagnetic Resonance ImagingBeatNeurologyAuditory PerceptionFemalePsychologybeatCognitive psychologyAdultNaturalistic imagingMusic information retrievalCognitive Neurosciencemedia_common.quotation_subjectmusic information retrievaldynamic connectivity050105 experimental psychologylcsh:RC321-571Young Adult03 medical and health sciencesPerceptionConnectomemedicineHumansmusic0501 psychology and cognitive scienceslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryAuditory Cortexnaturalistic imagingrytmiDynamic connectivityAcoustic Stimulationkognitiivinen neurotiedeCentralityFunctional magnetic resonance imagingBeat (music)Music030217 neurology & neurosurgeryNeuroImage
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Two distinct phenotypes, hemiplegic migraine and episodic Ataxia type 2, caused by a novel common CACNA1A variant

2020

Abstract Background To investigate the genetic and environmental factors responsible for phenotype variability in a family carrying a novel CACNA1A missense mutation. Mutations in the CACNA1A gene were identified as responsible for at least three autosomal dominant disorders: FHM1 (Familial Hemiplegic Migraine), EA2 (Episodic Ataxia type 2), and SCA6 (Spinocerebellar Ataxia type 6). Overlapping clinical features within individuals of some families sharing the same CACNA1A mutation are not infrequent. Conversely, reports with distinct phenotypes within the same family associated with a common CACNA1A mutation are very rare. Case presentation A clinical, molecular, neuroradiological, neuropsy…

MaleProbandmedicine.medical_specialtyNeurologyMigraine with AuraFamilial hemiplegic migraine type 1Mutation MissenseneuropsychologyCase Reportmedicine.disease_causeNystagmus Pathologiclcsh:RC346-42903 medical and health sciences0302 clinical medicinemedicineHumansSpinocerebellar ataxia type 6Missense mutationFamilyChildFamilial hemiplegic migrainelcsh:Neurology. Diseases of the nervous system030304 developmental biologyEpisodic ataxiaGenetics0303 health sciencesMutationbusiness.industryCACNA1A geneEpisodic ataxia type2Cognitive affective syndromeGeneral Medicinemedicine.diseasePhenotypePhenotypeAtaxiaCalcium ChannelsNeurology (clinical)businessCognitive affective syndrome neuropsychology.030217 neurology & neurosurgeryBMC Neurology
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In toxic demyelination oligodendroglial cell death occurs early and is FAS independent

2010

Oligodendroglial cell death is a frequent phenomenon of many neurological diseases, e.g. in demyelinating diseases such as multiple sclerosis (MS). The underlying mechanisms are largely unknown. Here, we demonstrate that in the toxic demyelination cuprizone model, oligodendroglial cell death and downregulation of myelin genes start days after initiation of the cuprizone diet and weeks before demyelination is obvious. In early – but not in later – stages, dying oligodendrocytes express activated caspase 3, suggesting a switch from classical apoptotic pathways to caspase 3-independent mechanisms during the course of the cuprizone diet. The expression level of FAS in the corpus callosum, a cel…

MaleProgrammed cell deathDown-RegulationMice TransgenicCaspase 3ApoptosisNerve Fibers MyelinatedArticleCorpus Callosumlcsh:RC321-571Mice03 medical and health sciencesMyelinCuprizone0302 clinical medicineDownregulation and upregulationmedicineAnimalsRNA Messengerfas Receptorlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCaspase030304 developmental biology0303 health sciencesCell DeathbiologyCaspase 3CytotoxinsMultiple sclerosisExperimental autoimmune encephalomyelitisFASmedicine.disease3. Good healthMice Inbred C57BLDisease Models AnimalOligodendrogliamedicine.anatomical_structureGene Expression RegulationNeurologyApoptosisMyelinImmunologybiology.proteinFemaleMyelin Proteins030217 neurology & neurosurgeryDemyelinating DiseasesSignal TransductionNeurobiology of Disease
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Proteomic signature of the Dravet syndrome in the genetic Scn1a-A1783V mouse model.

2021

Abstract Background Dravet syndrome is a rare, severe pediatric epileptic encephalopathy associated with intellectual and motor disabilities. Proteomic profiling in a mouse model of Dravet syndrome can provide information about the molecular consequences of the genetic deficiency and about pathophysiological mechanisms developing during the disease course. Methods A knock-in mouse model of Dravet syndrome with Scn1a haploinsufficiency was used for whole proteome, seizure, and behavioral analysis. Hippocampal tissue was dissected from two- (prior to epilepsy manifestation) and four- (following epilepsy manifestation) week-old male mice and analyzed using LC-MS/MS with label-free quantificati…

MaleProteomics0301 basic medicineProteomeHippocampusEpilepsies MyoclonicHaploinsufficiencyScn1aHippocampusSynaptic TransmissionElevated Plus Maze TestEpilepsyMice0302 clinical medicineTandem Mass Spectrometry11-beta-Hydroxysteroid Dehydrogenase Type 1Genetic epilepsyCarbon-Nitrogen LigasesGene Knock-In TechniquesGliosisNeuronal PlasticityBehavior AnimalEpileptic encephalopathyImmunohistochemistryAstrogliosisNeurologyProteomeDisease ProgressionFemaleHaploinsufficiencySignal TransductionRC321-571Dopamine and cAMP-Regulated Phosphoprotein 32Neovascularization PhysiologicNeurosciences. Biological psychiatry. NeuropsychiatryBiologyNitric Oxide03 medical and health sciencesDravet syndromemedicineAnimalsHyperthermiaSocial Behaviorras-GRF1Proteomic Profilingmedicine.diseaseVascular Endothelial Growth Factor Receptor-2NAV1.1 Voltage-Gated Sodium ChannelDisease Models Animal030104 developmental biologyRotarod Performance TestSynaptic plasticityEpileptic Encephalopathy ; Genetic Epilepsy ; Mice ; Proteome ; Scn1aCalcium-Calmodulin-Dependent Protein Kinase Type 2Open Field TestNeuroscience030217 neurology & neurosurgeryChromatography Liquid
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Beneficial effects of C1 esterase inhibitor in ST-elevation myocardial infarction in patients who underwentsurgical reperfusion: a randomized double-…

2007

Background: The inflammatory cascade has been hypothesized to be an important mechanism of post-ischaemic myocardial reperfusion injury and several studies demonstrated that C1 esterase inhibitor (C1 -INH) is effective in post-ischaemia myocardial protection. Therefore, we aimed to investigate prospectively in a randomised double-blind study the cardioprotective effects of C1-INH in ST segment elevation myocardial infarction (STEMI) in patients who underwent emergent reperfusion with coronary artery bypass grafting (CABG). Methods: In this study, we enrolled 80 patients affected with STEMI who underwent emergent CABG. Patients were assigned in two groups (C1-INH group: receive 1000 Ul of C1…

MalePulmonary and Respiratory MedicineCardiac function curvemedicine.medical_specialtyMean arterial pressureCardiotonic AgentsMyocardial InfarctionCardiac indexMyocardial ReperfusionComplement C1 Inactivator ProteinsCoronary artery bypass surgeryReperfusion therapyDouble-Blind MethodInternal medicinemedicineHumansProspective StudiesMyocardial infarctionCoronary Artery BypassInfusions IntravenousSTEMI patients CABG C1 esterase inhibitor Reperfusion injury Complement cascade Myocardial function recoverybusiness.industryST elevationTroponin IComplement C4aGeneral MedicineMiddle Agedmedicine.diseaseMyocardial ContractionComplement Inactivating AgentsTreatment OutcomeComplement C3aCardiologyFemaleSurgeryCardiology and Cardiovascular MedicinebusinessReperfusion injury
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Evidence that C1q, a Subcomponent of the First Component of Complement, is an Fc Receptor of Peritoneal and Alveolar Macrophages

1980

Abstract Guinea pig peritoneal macrophages were cultured for 24 h in the presence of two inhibitors of the biosynthesis of collagen-like molecules such as C1q : 10 -3 M 3,4-dehydroproline or 10 -4 M 2,2′-dipyridyl. Their Fc-receptor activity was measured by rosette formation, using sheep erythrocytes (E) coated with rabbit anti-sheep IgG (EA IgG ). The Fc-receptor activity was decreased by 40 to 70% of control cultures depending on the amount of IgG on the E. The activity of a second receptor on the macrophages, mediating the binding of C3b coated E, was not altered by this treatment. Rat alveolar macrophages were depleted of their Fc-receptor activity by pronase treatment (1.5 mg/ml) in th…

MaleRosette FormationProlineGuinea PigsImmunologyFc receptorReceptors FcPronaseGuinea pigchemistry.chemical_compound22'-DipyridylBiosynthesisComplement C1AnimalsAscitic FluidImmunology and AllergySecretionReceptorIncubationbiologyMacrophagesComplement C3HematologyMolecular biologyRatsReceptors ComplementPulmonary AlveoliMembraneBiochemistrychemistryPronasebiology.proteinFemaleImmunobiology
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Increase of Substance P Concentration in Saliva after Pharyngeal Electrical Stimulation in Severely Dysphagic Stroke Patients – an Indicator of Decan…

2017

Background/Aims: Substance P (SP) is a neuropeptide, likely acting as a neurotransmitter in the pharyngeal mucosa enhancing the swallow and cough reflex. Pharyngeal Electrical Stimulation (PES) induces a temporary increase of salivary SP levels in healthy adults. Previous evidence suggests that post-stroke dysphagia is related to reduced SP levels. Here, we investigated the effects of PES on SP levels in severely dysphagic stroke patients and a possible link between increase of SP and treatment success. Methods: 23 tracheotomized stroke patients who could not be decannulated due to severe and persisting dysphagia according to endoscopic evaluation received PES for 10 minutes a day over thre…

MaleSalivaStroke patientCough reflexStimulationSubstance PSubstance Plcsh:RC346-42903 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compoundTracheostomy0302 clinical medicinePharyngeal electrical stimulationDevelopmental NeurosciencemedicineHumansProspective Studies030212 general & internal medicineSalivaStrokelcsh:Neurology. Diseases of the nervous systemAgedAged 80 and overbusiness.industrylcsh:QP351-495Middle Agedmedicine.diseaseDysphagiaElectric StimulationStrokePESlcsh:Neurophysiology and neuropsychologyNeurologychemistryAnesthesiaTracheal decannulationPharynxBiomarker (medicine)Femalemedicine.symptomDeglutition Disordersbusiness030217 neurology & neurosurgeryNeurosignals
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Effect of intermittent exposure to ethanol and MDMA during adolescence on learning and memory in adult mice

2012

Abstract Background Heavy binge drinking is increasingly frequent among adolescents, and consumption of 3,4-methylenedioxymethamphetamine (MDMA) is often combined with ethanol (EtOH). The long-lasting effects of intermittent exposure to EtOH and MDMA during adolescence on learning and memory were evaluated in adult mice using the Hebb-Williams maze. Methods Adolescent OF1 mice were exposed to EtOH (1.25 g/kg) on two consecutive days at 48-h intervals over a 14-day period (from PD 29 to 42). MDMA (10 or 20 mg/kg) was injected twice daily at 4-h intervals over two consecutive days, and this schedule was repeated six days later (PD 33, 34, 41 and 42), resulting in a total of eight injections. …

MaleSerotoninmedicine.medical_specialtyMDMA34-Dihydroxyphenylacetic acidDopamineN-Methyl-34-methylenedioxyamphetamineCognitive NeuroscienceBinge drinkingStriatumHippocampuslcsh:RC346-429MiceBehavioral Neurosciencechemistry.chemical_compoundSerotonin AgentsMemoryDopamineSerotonin AgentsInternal medicinemental disordersmedicineAnimalsLearningHippocampus (mythology)Maze Learninglcsh:Neurology. Diseases of the nervous systemBiological PsychiatryBehavior AnimalEthanolResearchMDMAGeneral MedicineHydroxyindoleacetic AcidCorpus StriatumEndocrinologychemistryAnesthesia34-Dihydroxyphenylacetic AcidHebb Williams mazeSerotoninPsychologypsychological phenomena and processesmedicine.drugBehavioral and Brain Functions
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TLR4 response mediates ethanol-induced neurodevelopment alterations in a model of fetal alcohol spectrum disorders

2017

Background Inflammation during brain development participates in the pathogenesis of early brain injury and cognitive dysfunctions. Prenatal ethanol exposure affects the developing brain and causes neural impairment, cognitive and behavioral effects, collectively known as fetal alcohol spectrum disorders (FASD). Our previous studies demonstrate that ethanol activates the innate immune response and TLR4 receptor and causes neuroinflammation, brain damage, and cognitive defects in the developmental brain stage of adolescents. We hypothesize that by activating the TLR4 response, maternal alcohol consumption during pregnancy triggers the release of cytokines and chemokines in both the maternal …

MaleSerum0301 basic medicineChemokineDevelopmental Disabilitiesmedicine.medical_treatmentlcsh:RC346-429MiceMyelin0302 clinical medicineNeuroinflammationPregnancyTLR4Maternal BehaviorFetal alcohol spectrum disordersMice KnockoutMicrogliabiologyGeneral NeuroscienceAge FactorsBrainCerebral cortexBehavior impairmentsmedicine.anatomical_structureCytokineNeurologyPrenatal Exposure Delayed EffectsCytokinesFemalemedicine.symptomMyelin ProteinsAmniotic fluidmedicine.medical_specialtyOffspringImmunologyNerve Tissue ProteinsBrain damage03 medical and health sciencesCellular and Molecular NeuroscienceInternal medicineAvoidance LearningmedicineAnimalsMaze Learninglcsh:Neurology. Diseases of the nervous systemNeuroinflammationEthanolbusiness.industryResearchBody WeightCentral Nervous System DepressantsMice Inbred C57BLToll-Like Receptor 4Disease Models AnimalMicroscopy Electron030104 developmental biologyEndocrinologyAnimals NewbornPrenatal ethanol exposureImmunologybiology.proteinTLR4business030217 neurology & neurosurgeryJournal of Neuroinflammation
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