Search results for "CALCIUM"

showing 10 items of 1740 documents

Cellular ionic alterations with age: relation to hypertension and diabetes.

2000

BACKGROUND: Cytosolic free calcium (Cai) and magnesium (Mgi) are vital to cellular homeostasis and function. OBJECTIVE: To evaluate cellular divalent cations in normal subjects at different ages and their relationship to ion levels in essential hypertension and diabetes. DESIGN: A cross-sectional study. SETTING: A university hospital in New York. PARTICIPANTS: A total of 103 subjects (32 older, 71.1 ± 1.2 y/o, and 71 young/middle aged subjects, 51.1 ± 2.3 y/o). INTERVENTION: Oral glucose tolerance test. MEASUREMENTS: 19F and 31P NMR spectroscopy were used to measure Cai and Mgi levels in erythrocytes from normal (>65 y/o, n = 11; 65 y/o, n = 9; 65 y/o, n = 12; <65 y/o, n = 15) subjects; the…

Blood GlucoseMalemedicine.medical_specialtyAgingErythrocytesMagnetic Resonance Spectroscopymedicine.medical_treatmentCellular homeostasisEssential hypertensionCytosolInternal medicineDiabetes mellitusmedicineHumansInsulinMagnesiumMagnesium ionAgedAged 80 and overGlucose tolerance testmedicine.diagnostic_testbusiness.industryInsulinAge FactorsGlucose Tolerance TestMiddle Agedmedicine.diseaseCausalityEndocrinologyGlycemic indexBlood pressureCross-Sectional StudiesDiabetes Mellitus Type 2Case-Control StudiesHypertensionCalciumFemaleGeriatrics and GerontologybusinessJournal of the American Geriatrics Society
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Mechanisms involved in the relaxant action of testosterone in the renal artery from male normoglycemic and diabetic rabbits.

2009

Kidney disease is a frequent complication in diabetes, and significant differences have been reported between male and female patients. Our working hypothesis was that diabetes might modify the vascular actions of testosterone in isolated rabbit renal arteries and the mechanisms involved in these actions. Testosterone (10(-8) to 10(-4)M) induced relaxation of precontracted arteries, without significant differences between control and diabetic rabbits. Both in control and diabetic rabbits endothelium removal inhibited testosterone relaxant action. In arteries with endothelium, incubation with indomethacin (10(-5)M), N(G)-nitro-l-arginine (10(-5)M) or tetraethylammonium (10(-5)M) did not modi…

Blood GlucoseMalemedicine.medical_specialtyEndotheliumNitric Oxide Synthase Type IIIThromboxaneBlotting WesternIndomethacinNitric Oxide Synthase Type IIProstacyclinVasodilationNitroarginineMuscle Smooth VascularDiabetes Mellitus ExperimentalImmunoenzyme TechniquesThromboxane A2Renal ArteryEnosInternal medicinemedicine.arteryDiabetes mellitusmedicinePotassium Channel BlockersAnimalsCyclooxygenase InhibitorsProstaglandins ITestosteroneRenal arteryPharmacologybiologyDose-Response Relationship Drugbusiness.industryTetraethylammoniumTestosterone (patch)medicine.diseasebiology.organism_classificationVasodilationEndocrinologymedicine.anatomical_structureCyclooxygenase 2Cyclooxygenase 1PotassiumCalciumEndothelium VascularRabbitsbusinessmedicine.drugSignal TransductionPharmacological research
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Modulation of platelet activation and initial cytokine release by alloplastic bone substitute materials.

2010

Objectives: Platelet-derived cytokines play a crucial role in tissue regeneration. In regenerative dental medicine, bone substitute materials (BSM) are widely used. However, initial interactions of BSM and platelets are still unknown. The aim of this study was to evaluate the potential of platelet activation and subsequent initial cytokine release by different commercial alloplastic BSM. Material and methods: Eight commercial BSM of different origins and chemical compositions (tricalcium phosphate, hydroxyapatite, bioactive glass: SiO2 and mixtures) were incubated with a platelet concentrate (platelet-rich plasma, PRP) of three healthy volunteers at room temperature for 15 min. Platelet cou…

Blood PlateletsCalcium PhosphatesVascular Endothelial Growth Factor APlatelet Aggregationmedicine.medical_treatmentPharmacologyCell Degranulationlaw.inventionlawmedicineHumansPlateletPlatelet activationPlatelet-Derived Growth FactorbiologyChemistryPlatelet CountPlatelet-Rich PlasmaGrowth factorDegranulationFlow CytometryPlatelet ActivationSilicon DioxideP-SelectinCytokineDurapatiteBioactive glassPlatelet-rich plasmaImmunologyBone Substitutesbiology.proteinOral SurgeryPlatelet-derived growth factor receptorClinical oral implants research
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PHOSPHODIESTERASE INHIBITORS PIROXIMONE AND ENOXIMONE INHIBIT PLATELET AGGREGATION IN VIVO AND IN VITRO

1997

The phosphodiesterase type III inhibitors piroximone (PIR) and enoximone (ENO) exert positive inotropic and vasodilating effects in patients with severe heart failure. PIR and ENO raise cyclic AMP levels in cardiac and vascular smooth muscle cells. Platelet activity is also regulated by intracellular levels of cyclic AMP. In this study we have investigated the effects of PIR and ENO on platelet activity in vivo and in vitro. PIR and ENO inhibited ADP induced platelet aggregation in a time- and concentration-dependent manner with IC50-values of 67 +/- 14 mumol/l and 129 +/- 6 mumol/l, respectively. Coincubation of PIR with the adenylate cyclase activator iloprost resulted in a synergistic po…

Blood PlateletsMalemedicine.medical_specialtyCardiotonic AgentsVascular smooth musclePlatelet AggregationPhosphodiesterase InhibitorsVasodilationIn vivoInternal medicineCyclic AMPmedicineAnimalsHumansEnoximonePlateletPlatelet activationRats WistarEnoximonebiologyChemistryImidazolesPhosphodiesteraseHematologyRatsEndocrinologyEnzyme inhibitorbiology.proteinCalciumPlatelet Aggregation Inhibitorsmedicine.drugThrombosis Research
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Platelet-endothelial interaction in tumor angiogenesis and microcirculation

2003

Activated platelets release angiogenic growth factors and have therefore been proposed to contribute to tumor angiogenesis within a potentially prothrombotic tumor microcirculation. The aim of the study was to investigate interactions of platelets with the angiogenic microvascular endothelium of highly vascularized solid tumors during growth and in response to endothelial stimulation in comparison with normal subcutaneous tissue. Experiments were performed in the dorsal skinfold chamber preparation of C57BL/6J mice bearing the Lewis lung carcinoma (LLC-1) or methylcholanthrene-induced fibrosarcoma (BFS-1). Fluorescently labeled rolling and adherent platelets, red blood cell velocity, and ve…

Blood PlateletsMalemedicine.medical_specialtyEndotheliumAngiogenesisFibrosarcomaImmunologyBiologyBiochemistrySkin Window TechniqueNeovascularizationCarcinoma Lewis LungMicePlatelet AdhesivenessCell MovementPlatelet adhesivenessInternal medicinemedicineAnimalsPlateletPlatelet activationCalcimycinIonophoresNeovascularization PathologicMicrocirculationVideotape RecordingLewis lung carcinomaCell BiologyHematologyPlatelet ActivationMice Inbred C57BLEndothelial stem cellmedicine.anatomical_structureEndocrinologyImmunologyCalciumEndothelium Vascularmedicine.symptomBlood Flow VelocityMethylcholanthreneBlood
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Membrane fluidity, membrane lipid pattern, and cytosolic Ca2+ content in platelets from a group of type II diabetic patients with macrovascular compl…

1995

OBJECTIVE To evaluate platelet membrane fluidity and some platelet metabolic parameters in type II diabetic patients with macrovascular complications. RESEARCH DESIGN AND METHODS In a group of 21 type II diabetic patients with macrovascular complications, we evaluated platelet membrane fluidity [marking intact resting platelets with the fluorescent probe 1,4-(trimethylamino)-phenyl-4-phenylhexatriene (TMA-DPH)], platelet membrane lipid pattern (cholesterol :phospholipid [C:PL] ratio and individual phospholipids), and platelet cytosolic Ca2+ content (marking intact resting platelets with the fluorescent probe Fura 2AM). RESULTS Platelet membrane fluidity is decreased in type II diabetic pat…

Blood PlateletsMalemedicine.medical_specialtyMembrane FluidityEndocrinology Diabetes and MetabolismPhospholipidchemistry.chemical_elementCalciumchemistry.chemical_compoundMembrane LipidsCytosolInternal medicineDiabetes mellitusInternal MedicineMembrane fluiditymedicineHumansPlateletAgedAdvanced and Specialized NursingCholesterolbusiness.industryPhosphatidylserineMiddle Agedmedicine.diseaseCytosolEndocrinologySpectrometry FluorescencechemistryDiabetes Mellitus Type 2CalciumFemalebusinessDiabetic Angiopathies
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Preparation and Application of an Innovative Thrombocyte/Leukocyte-Enriched Plasma to Promote Tissue Repair in Chelonians

2015

Platelet concentrates are widely used in mammalian regenerative medicine to improve tissue healing. Chelonians (Testudines) would benefit from the application of thrombocyte preparations to regenerate damaged tissues, since traumatic injuries are leading causes of morbidity and mortality for both wild-living and domesticated animals. The aim of this study was to establish a protocol that optimized the recovery of the thrombocytes from blood samples and to show the efficacy of thrombocyte-enriched plasma in chelonians. Peripheral blood samples were obtained from Testudo spp. (n = 12) and Trachemys scripta elegans (n = 10). Blood cells were fractionated by sodium diatrizoate-sodium polysucros…

Blood PlateletsPathologymedicine.medical_specialtySodiumchemistry.chemical_elementlcsh:MedicineCentrifugationCalciumBiologyRegenerative MedicineAndrologyPlasmaBlood plasmamedicineLeukocytesAnimalsCentrifugationPlateletlcsh:ScienceWhole bloodCryopreservationMultidisciplinarylcsh:RTissue repairTurtleschemistryPlatelet-rich plasmalcsh:QResearch ArticlePLoS ONE
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The histone deacetylase sirtuin 2 is a new player in the regulation of platelet function

2015

SummaryBackground Histone deacetylases (HDACs) play a key role in signaling in many cell types. However, little is known about the participation of HDACs, particularly sirtuins (SIRTs), in platelet reactivity. Objective To investigate the role of HDACs in platelets, we examined the effects of SIRT inhibition on platelet function and protein acetylation in human platelets. Methods We used washed platelets obtained from healthy subjects. Cambinol (SIRT1 and SIRT2 inhibitor), AGK2 (specific SIRT2 inhibitor) and EX527 (specific SIRT1 inhibitor) were used as SIRT inhibitors. Platelets were stimulated with collagen, thrombin, or U46619, and platelet responses were determined according to optical …

Blood PlateletsPlatelet AggregationCytoplasmic GranulesSIRT2Glycogen Synthase Kinase 3Akt3 protein kinaseSirtuin 2sirtuinsHumansPlateletRNA MessengerPhosphorylationProtein kinase Bacetylationblood plateletGlycogen Synthase Kinase 3 betabiologySecretory VesiclesAcetylationHematologyCell biologyHistone Deacetylase InhibitorsBiochemistryAcetylationSirtuinbiology.proteinPhosphorylationPlatelet aggregation inhibitorCalciumHistone deacetylaseProtein Processing Post-TranslationalProto-Oncogene Proteins c-aktPlatelet Aggregation Inhibitorssignal transductionSignal TransductionJournal of Thrombosis and Haemostasis
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Phosphorylation of CalDAG-GEFI by protein kinase A prevents Rap1b activation.

2013

Summary Background Signaling via protein kinase A (PKA) and protein kinase G (PKG) is critical for maintaining platelets in the resting state. Both kinases down-regulate the activity of the small GTPase Rap1b, a critical signaling switch for integrin activation and platelet aggregation. However, the mechanism of Rap1b regulation by PKA and PKG is largely unknown. Objective To identify the PKA phosphorylation sites in calcium and diacylglycerol-regulated guanine nucleotide exchange factor I (CalDAG-GEFI), the main GEF for Rap1b in platelets, and the effect of CalDAG-GEFI phosphorylation in Rap1b activation. Methods The phosphorylation sites in CalDAG-GEFI were identified by radio-active phos…

Blood PlateletsPlatelet AggregationMolecular Sequence DataBiologyMass SpectrometryPhosphorylation cascadeCyclic AMPGuanine Nucleotide Exchange FactorsHumansImmunoprecipitationProtein phosphorylationAmino Acid SequenceCalcium SignalingPhosphorylationProtein kinase ACalcium signalingAlanineSequence Homology Amino AcidKinaseHematologyCyclic AMP-Dependent Protein KinasesEnzyme Activationrab1 GTP-Binding ProteinsHEK293 CellsBiochemistryMutationPhosphorylationGuanine nucleotide exchange factorGuanosine TriphosphatecGMP-dependent protein kinasePlasmidsSignal TransductionJournal of thrombosis and haemostasis : JTH
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Flow Cytometric Analysis of Calcium Mobilization in Whole‐Blood Platelets

2003

Flow cytometry provides a convenient method to evaluate platelet activation by following the kinetics of intracellular free Ca2+, using sensitive fluorescent indicators that can be loaded into intact cells. Moreover, in the clinical setting, whole-blood techniques have obvious advantages to avoid artifactual platelet activation and allow the maintenance of near-physiological conditions. This unit describes a fast and sensitive flow cytometric procedure using the Ca2+-sensitive dye fluo-3 AM and the platelet-specific antibody CD41-PE to determine the kinetics of intracellular Ca2+ mobilization in whole-blood platelets with minimal manipulation of the samples. The technique may be applied to …

Blood PlateletsPlatelet Membrane Glycoprotein IIbHistologyStimulation030204 cardiovascular system & hematologyBiochemistryFlow cytometry03 medical and health sciences0302 clinical medicineThrombinMethodsmedicineAnimalsHumansPlateletCalcium SignalingPlatelet activationFluorescent Dyes030304 developmental biologyWhole blood0303 health sciencesAniline Compoundsmedicine.diagnostic_testChemistryAntibodies MonoclonalGeneral MedicineFlow CytometryIn vitro3. Good healthKineticsMedical Laboratory TechnologyXanthenesBiochemistryBiophysicsCalciumIntracellularmedicine.drugCurrent Protocols in Cytometry
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