Search results for "CB1"

showing 10 items of 146 documents

Cannabinoid receptor 1 and acute resistance exercise – In vivo and in vitro studies in human skeletal muscle

2015

Abstract Aim This study aimed to determine whether Cannabinoid receptor 1 (CB1) is involved in mammalian target of rapamycin (mTOR) signaling and skeletal muscle protein synthesis. Methods This study used human vastus lateralis skeletal muscle biopsies obtained before and after a resistance exercise (RE) bout in young men (n = 18). The signaling mechanisms were studied in vitro in human myotubes. Protein expression was determined by Western blot and confocal microscopy, and gene expression by quantitative PCR. Protein synthesis was measured in vitro using puromycin-based SuNSET technique. Results In human skeletal muscle, an anabolic stimulus in the form of RE down-regulated CB1 expression.…

Malemedicine.medical_specialtyPhysiologyMAP Kinase Signaling SystemMuscle Fibers SkeletalGene ExpressionSkeletal muscleP70-S6 Kinase 1Cell Cycle ProteinsBiochemistryCell LineCellular and Molecular NeuroscienceYoung AdultEndocrinologyPiperidinesReceptor Cannabinoid CB1Internal medicinemedicineCannabinoid receptor type 2HumansCannabinoid receptor 1PhosphorylationMuscle Skeletalta315PI3K/AKT/mTOR pathwayAdaptor Proteins Signal TransducingChemistryMyogenesista1184Eukaryotic initiation factor 4E bindingSkeletal muscleRibosomal Protein S6 Kinases 70-kDaResistance TrainingPhosphoproteinsResistance exerciseCell biologymedicine.anatomical_structureEndocrinologyRibosomal protein s6Protein BiosynthesismTOR signalingPhosphorylationPyrazolesProtein synthesisProtein Processing Post-TranslationalPeptides
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Alterations in the Hippocampal Endocannabinoid System in Diet-Induced Obese Mice

2010

The endocannabinoid (eCB) system plays central roles in the regulation of food intake and energy expenditure. Its alteration in activity contributes to the development and maintenance of obesity. Stimulation of the cannabinoid receptor type 1 (CB1receptor) increases feeding, enhances reward aspects of eating, and promotes lipogenesis, whereas its blockade decreases appetite, sustains weight loss, increases insulin sensitivity, and alleviates dysregulation of lipid metabolism. The hypothesis has been put forward that the eCB system is overactive in obesity. Hippocampal circuits are not directly involved in the neuronal control of food intake and appetite, but they play important roles in hed…

Malemedicine.medical_specialtyPolyunsaturated Alkamidesmedicine.medical_treatmentmedia_common.quotation_subjectArachidonic AcidsBiologyHippocampusArticlegamma-Aminobutyric acidGlyceridesMice03 medical and health sciences0302 clinical medicineReceptor Cannabinoid CB1Internal medicineCannabinoid Receptor ModulatorsCannabinoid receptor type 1medicineAnimalsObesityReceptorgamma-Aminobutyric Acid030304 developmental biologymedia_commonMice KnockoutNeurons0303 health sciencesLong-Term Synaptic DepressionGeneral NeuroscienceAppetiteDietary FatsEndocannabinoid systemMice Inbred C57BLDisease Models AnimalLipoprotein LipaseEndocrinologynervous systemSynapsesSynaptic plasticitylipids (amino acids peptides and proteins)CannabinoidDiet-induced obese030217 neurology & neurosurgeryEndocannabinoidsmedicine.drugThe Journal of Neuroscience
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Neurobiology of cannabinoid receptor signaling
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2020

The endocannabinoid system (ECS) is a highly versatile signaling system within the nervous system. Despite its widespread localization, its functions within the context of distinct neural processes are very well discernable and specific. This is remarkable, and the question remains as to how such specificity is achieved. One key player in the ECS is the cannabinoid type 1 receptor (CBEl sistema endocannabinoide (SEC) apareció como un sistema de señalización muy versátil en el sistema nervioso. A pesar de su existencia amplia y ubicua, sus funciones están integradas en el contexto de distintos procesos neuronales y, en última instancia, son bastante bien discernibles y específicas. Esto es n…

Nervous systemCannabinoid receptormedicine.medical_treatment2-ArachidonoylglycerolContext (language use)BiologyReceptor Cannabinoid CB203 medical and health scienceschemistry.chemical_compound0302 clinical medicineNeurobiologyReceptor Cannabinoid CB1Cannabinoid receptor type 2medicineAnimalsHumansanandamideReceptorReceptors Cannabinoidbehaviorcannabinoid receptorEndocannabinoid systemState of the Art030227 psychiatry2-arachidonoylglycerolmedicine.anatomical_structurechemistryneural communicationCannabinoidNeuroscienceEndocannabinoidsSignal TransductionDialogues in clinical neuroscience
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Neuronal populations mediating the effects of endocannabinoids on stress and emotionality

2011

An adequate emotional response to stress is essential for survival and requires the fine-tuned regulation of several distinct neuronal circuits. Therefore, a precise control of these circuits is necessary to prevent behavioral imbalances. During the last decade, numerous investigations have evidenced that the endocannabinoid (eCB) system is able to crucially control stress coping. Its central component, the cannabinoid type 1 receptor (CB1 receptor), is located at the presynapse, where it is able to attenuate neurotransmitter release after its activation by postsynaptically produced and released eCBs. To date, the eCB system has been found to control the neurotransmitter release from severa…

NeuronsHypothalamo-Hypophyseal SystemGeneral NeuroscienceEmotionsGlutamate receptorPituitary-Adrenal SystemContext (language use)Endocannabinoid systemAmygdalaPresynapsemedicine.anatomical_structureReceptor Cannabinoid CB1nervous systemStress PhysiologicalCannabinoid Receptor ModulatorsSynapsesmedicineAnimalsLocus coeruleusPremovement neuronal activityPsychologyPrefrontal cortexNeuroscienceStress PsychologicalEndocannabinoidsNeuroscience
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EFFECT OF ACETALDEHYDE INTOXICATION AND WITHDRAWAL ON NEUROPEPTIDE Y EXPRESSION. FOCUS ON CB1 RECEPTOR ROLE.

2014

Neuropeptide Y (NPY) acts as an endogenous anxiolytic, and, like endocannabinoids, plays an important role in the regulation of neuronal excitability during ethanol withdrawal (Rubio et al., 2011). Since acetaldehyde is considered a mediator of ethanol central effects, this research aims at investigating, following intoxication and during withdrawal, the effects of acetaldehyde on NPY expression in brain areas particularly vulnerable to alcohol, and the influence of cannabinoid system on it. Rats underwent acetaldehyde intoxication (450mg/kg, i.g., 4 times daily for 4 days); AM281, a CB1 selective antagonist (2,5 mg/kg, i.p.) was administered during abstinence. Immunohistochemical analysis …

Neuropeptide YAcetaldehydeCB1 antagonist
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Behavioural and pharmacological characterization of a novel cannabinomimetic adamantane-derived indole, APICA, and considerations on the possible mis…

2015

The novel adamantane derivative APICA (N-(adamantan-1-yl)-1-pentyl-1H-indole-3-carboxamide) was recently identified as a cannabinomimetic indole of abuse. Despite its novel structure, APICA recalls cannabinomimetic indoles, such as representative member JWH-018.In present study, the effects of APICA (1-3 mg/kg, i.p.) were tested in C57BL/6J mice, in the Tetrad task which includes the assessment of: body temperature; locomotor activity and behavioural reactivity; nociception; motor coordination; declarative memory. Furthermore, pre-treatment with the CB1 antagonist AM251 (3 mg/kg, i.p.) or the CB2 antagonist AM630 (3 mg/kg, i.p.) was carried out to characterize APICA activity.Our results sho…

Nociception0301 basic medicineAM251AgonistCB1 agonistIndazolesmedicine.drug_class2734Poison controlAPICAAdamantanePharmacologyOpen fieldDesigner DrugsPathology and Forensic MedicineMice03 medical and health sciences0302 clinical medicineAPICAmedicineAnimalsNew psychotropic substanceDose-Response Relationship DrugCannabinoidsChemistryAntagonistForensic MedicineMotor coordinationMice Inbred C57BL030104 developmental biologyNociceptionFemaleLawSynthetic cannabinoid030217 neurology & neurosurgerymedicine.drug
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The yeast inositol monophosphatase is a lithium- and sodium-sensitive enzyme encoded by a non-essential gene pair

1999

Inositol monophosphatases (IMPases) are lithium-sensitive enzymes that participate in the inositol cycle of calcium signalling and in inositol biosynthesis. Two open reading frames (YHR046c and YDR287w) with homology to animal and plant IMPases are present in the yeast genome. The two recombinant purified proteins were shown to catalyse inositol-1-phosphate hydrolysis sensitive to lithium and sodium. A double gene disruption had no apparent growth defect and was not auxotroph for inositol. Therefore, lithium effects in yeast cannot be explained by inhibition of IMPases and inositol depletion, as suggested for animal systems. Overexpression of yeast IMPases increased lithium and sodium toler…

PLCB1ATPaseGenes FungalMolecular Sequence DataPLCB2PLCB3Inositol monophosphataseSaccharomyces cerevisiaeLithiumMicrobiologychemistry.chemical_compoundInositolAmino Acid SequenceCloning MolecularMolecular BiologybiologySodiumPhosphoric Monoester HydrolasesRecombinant ProteinsYeastchemistryBiochemistrybiology.proteinCalciumGene DeletionInositolIntracellularPlasmidsMolecular Microbiology
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Presynaptic CB1 Receptors Regulate Synaptic Plasticity at Cerebellar Parallel Fiber Synapses

2011

Endocannabinoids are potent regulators of synaptic strength. They are generally thought to modify neurotransmitter release through retrograde activation of presynaptic type 1 cannabinoid receptors (CB1Rs). In the cerebellar cortex, CB1Rs regulate several forms of synaptic plasticity at synapses onto Purkinje cells, including presynaptically expressed short-term plasticity and, somewhat paradoxically, a postsynaptic form of long-term depression (LTD). Here we have generated mice in which CB1Rs were selectively eliminated from cerebellar granule cells, whose axons form parallel fibers. We find that in these mice, endocannabinoid-dependent short-term plasticity is eliminated at parallel fiber…

PhysiologyPresynaptic TerminalsNeural facilitationNonsynaptic plasticityParallel fiberSynaptic TransmissionMice03 medical and health sciences0302 clinical medicineReceptor Cannabinoid CB1CerebellumMetaplasticitymedicineAnimalsLong-term depression030304 developmental biologyMice Knockout0303 health sciencesNeuronal PlasticitySynaptic scalingHomosynaptic plasticityChemistryLong-Term Synaptic DepressionGeneral NeuroscienceArticlesMice Inbred C57BLmedicine.anatomical_structurenervous systemSynaptic plasticityNeuroscience030217 neurology & neurosurgeryJournal of Neurophysiology
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Central side-effects of therapies based on CB1 cannabinoid receptor agonists and antagonists: focus on anxiety and depression

2009

Both agonists (e.g. Delta(9)-tetrahydrocannabinol, nabilone) and antagonists (e.g. rimonabant, taranabant) of the cannabinoid type-1 (CB(1)) receptor have been explored as therapeutic agents in diverse fields of medicine such as pain management and obesity with associated metabolic dysregulation, respectively. CB(1) receptors are widely distributed in the central nervous system and are involved in the modulation of emotion, stress and habituation responses, behaviours that are thought to be dysregulated in human psychiatric disorders. Accordingly, CB(1) receptor activation may, in some cases, precipitate episodes of psychosis and panic, while its inhibition may lead to behaviours reminiscen…

PsychosisCannabinoid receptorEndocrinology Diabetes and Metabolismmedicine.medical_treatmentPharmacologyEndocrinologyTaranabantPiperidinesReceptor Cannabinoid CB1RimonabantCannabinoid Receptor ModulatorsmedicineAnimalsHumansDronabinolDepressive Disorderbusiness.industryCannabinoid Receptor Agonistsmedicine.diseaseAnxiety DisordersEndocannabinoid systemNabiloneAffectPyrazolesCannabinoidRimonabantbusinessNeurosciencemedicine.drugBest Practice & Research Clinical Endocrinology & Metabolism
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CB1 receptor expression in pancreatic islet of the obese and diabetes Zucher rats

2011

Settore BIO/17 - IstologiaCB1 receptorPancreaObesityZucker ratImmunohistochemistry
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