Search results for "CD4"

showing 10 items of 566 documents

Cross Talk between β1and αVIntegrins: β1Affects β3mRNA Stability

2001

There is increasing evidence that a fine-tuned integrin cross talk can generate a high degree of specificity in cell adhesion, suggesting that spatially and temporally coordinated expression and activation of integrins are more important for regulated cell adhesive functions than the intrinsic specificity of individual receptors. However, little is known concerning the molecular mechanisms of integrin cross talk. With the use of β1-null GD25 cells ectopically expressing the β1A integrin subunit, we provide evidence for the existence of a cross talk between β1and αVintegrins that affects the ratio of αVβ3and αVβ5integrin cell surface levels. In particular, we demonstrate that a down-regulati…

biologyIntegrinAlpha (ethology)Cell BiologyCD49cMolecular biologyCell biologyCollagen receptorIntegrin alpha MIntegrin alphaVbiology.proteinIntegrin beta 6Beta (finance)Molecular BiologyMolecular Biology of the Cell
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Defective expression of CD95 (FAS/APO-1) molecule suggests apoptosis impairment of T and B cells in HLA-B8, DR3-positive individuals.

1997

Activation-induced apoptosis is one of the primary control mechanisms for the negative selection of an immune response, leading to maintenance of immune homeostasis and selective T cell deletion. The interaction between the surface molecule Fas and its ligand (FasL) has been proposed as a primary mechanism initiating T cell apoptosis. The T cell receptor modulates the expression and function of these molecules. Defects in the Fas/FasL apoptosis pathway have been shown to result in autoimmune disease in humans and in murine models. Because subjects carrying the HLA-B8, DR3 haplotype show a number of immune dysfunctions, including membrano-proliferative glomerulonephritis, systemic lupus eryt…

CD3 ComplexT cellCD8 AntigensT-LymphocytesImmunologyAntigens CD19Lipopolysaccharide ReceptorsApoptosisBiologyFas ligandHLA-B8 AntigenImmune systemHLA-DR3 AntigenmedicineImmunology and AllergyCytotoxic T cellHumansfas ReceptorAutoimmune diseaseB-LymphocytesHistocompatibility TestingT-cell receptorGeneral Medicinemedicine.diseaseFas receptorFlow Cytometrymedicine.anatomical_structureApoptosisImmunologyCD4 AntigensCancer researchHuman immunology
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Blue light irradiation suppresses dendritic cells activationin vitro

2013

Blue light is a UV-free irradiation suitable for treating chronic skin inflammation, for example, atopic dermatitis, psoriasis, and hand- and foot eczema. However, a better understanding of the mode of action is still missing. For this reason, we investigated whether dendritic cells (DC) are directly affected by blue light irradiation in vitro. Here, we report that irradiation neither induced apoptosis nor maturation of monocyte-derived and myeloid DC. However, subsequent DC maturation upon LPS/IFNγ stimulation was impaired in a dose-dependent manner as assessed by maturation markers and cytokine release. Moreover, the potential of this DC to induce cytokine secretion from allogeneic CD4 T …

CD4-Positive T-LymphocytesLipopolysaccharidesLightUltraviolet Raysmedicine.medical_treatmentStimulationInflammationCell SeparationDermatologyLymphocyte ActivationBiochemistryInterferon-gammaPsoriasismedicineHumansIrradiationMolecular BiologyImmunosuppression TherapyInflammationChemistryDendritic Cellsmedicine.diseaseCoculture TechniquesIn vitroCell biologyCytokineApoptosisImmunologyCytokinesCytokine secretionmedicine.symptomExperimental Dermatology
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Activated glycoprotein A repetitions predominant (GARP)-expressing regulatory T cells inhibit allergen-induced intestinal inflammation in humanized m…

2015

Background Recently, we developed a humanized mouse model of allergen-induced IgE-dependent gut inflammation in PBMC-engrafted immunodeficient mice. Objective In the present study, we wanted to investigate the role of regulatory T (Treg) cells and their activation status in this model. Methods Nonobese diabetic-severe combined immunodeficiency-γc −/− mice were injected intraperitoneally with human PBMCs from allergic donors together with the respective allergen or NaCl as control in the presence or absence of different concentrations of CD4 + CD25 + Treg cells of the same donor. After an additional allergen boost 1 week later, mice were challenged with the allergen rectally on day 21 and gu…

CD4-Positive T-LymphocytesMalemedicine.medical_treatmentImmunologyInflammationNodMice SCIDBiologyImmunoglobulin ET-Lymphocytes RegulatoryMicemedicineHypersensitivityImmunology and AllergyAnimalsHumansIL-2 receptorAntibodies BlockingCells CulturedCell ProliferationImmunosuppression TherapyInflammationSevere combined immunodeficiencyInterleukin-2 Receptor alpha SubunitMembrane Proteinshemic and immune systemsForkhead Transcription FactorsDendritic cellAllergensImmunoglobulin Emedicine.diseaseIntestinesDisease Models AnimalCytokineImmunologyHumanized mouseAntibody FormationCD4 Antigensbiology.proteinLeukocytes MononuclearFemalemedicine.symptomThe Journal of allergy and clinical immunology
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Protection from graft-versus-host disease by HIV-1 envelope protein gp120-mediated activation of human CD4+CD25+ regulatory T cells.

2009

AbstractNaturally occurring CD4+CD25+ regulatory T cells (Tregs) represent a unique T-cell lineage that is endowed with the ability to actively suppress immune responses. Therefore, approaches to modulate Treg function in vivo could provide ways to enhance or reduce immune responses and lead to novel therapies. Here we show that the CD4 binding human immunodeficiency virus-1 envelope glycoprotein gp120 is a useful and potent tool for functional activation of human Tregs in vitro and in vivo. Gp120 activates human Tregs by binding and signaling through CD4. Upon stimulation with gp120, human Tregs accumulate cyclic adenosine monophosphate (cAMP) in their cytosol. Inhibition of endogeneous cA…

ImmunologyTransplantation HeterologousGraft vs Host Diseasechemical and pharmacologic phenomenaCHO CellsMice SCIDBiologyHIV Envelope Protein gp120Lymphocyte ActivationBiochemistryT-Lymphocytes RegulatoryImmune tolerancechemistry.chemical_compoundMiceImmune systemCricetulusIn vivoMice Inbred NODCricetinaeCyclic AMPImmune ToleranceAnimalsHumansCyclic adenosine monophosphateIL-2 receptorhemic and immune systemsCell BiologyHematologyEnvelope glycoprotein GP120Cell biologyTransplantationchemistryImmunologyCD4 Antigensbiology.proteinHIV-1Signal transductionSignal TransductionBlood
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Modulation of CD4 T Cell Response According to Tumor Cytokine Microenvironment

2021

Simple Summary It is now accepted that CD4 T lymphocytes play an essential role in the anti-tumor response. CD4 T lymphocytes can activate and regulate several aspects of innate and adaptive immunity and participate in the rejection of tumors. Understanding the impact of the tumor, through cytokines present in the microenvironment, but also the effect of anti-cancer therapies are critical aspects of immunotherapy research aiming at improving the anti-tumor response dependent on CD4 T lymphocytes. Abstract The advancement of knowledge on tumor biology over the past decades has demonstrated a close link between tumor cells and cells of the immune system. In this context, cytokines have a majo…

0301 basic medicineCancer Researchmedicine.medical_treatmentContext (language use)ReviewBiologylcsh:RC254-28203 medical and health sciences0302 clinical medicineImmune systemmedicinecancerTumor microenvironmentImmunotherapyAcquired immune systemlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensPhenotypeCD4cytokines030104 developmental biologyCytokineOncology030220 oncology & carcinogenesisCancer researchimmunotherapyHomeostasisCancers
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Both IL-12p70 and IL-23 are synthesized during active Crohnʼs disease and are down-regulated by treatment with anti-IL-12 p40 monoclonal antibody

2005

Background: Interleukin (IL)-12p70 and IL-23 are key T helper-1 (TH1) cytokines that drive the inflammation seen in numerous models of intestinal inflammation. These molecules contain an identical p40 chain that is bound to a p35 chain in IL-12 and a p19 chain in IL-23, making both potentially susceptible to modulation by an anti-IL-12p40 monoclonal antibody (mAb). Methods: In the present study, we sought to determine whether active inflammation in Crohn's disease (CD) is associated with the increased synthesis of both of these cytokines and whether patients treated with an anti-IL-12p40 mAb down-regulate IL-23 as well as IL-12p70 as previous reported. Results: To this end we initially dete…

AdultMaleAdolescentBiopsyT-Lymphocytesmedicine.medical_treatmentT cellDown-RegulationInterleukin-23Crohn DiseaseInterleukin 23medicineHumansImmunology and AllergyCells CulturedAgedLamina propriaMucous MembraneCD40biologyInterleukin-12 Subunit p40Interleukin-6InterleukinsMacrophagesGastroenterologyAntibodies MonoclonalInterleukinMiddle AgedInterleukin-12Protein Subunitsmedicine.anatomical_structureCytokineImmunologyInterleukin-23 Subunit p19biology.proteinInterleukin 12FemaleTumor necrosis factor alphaInterleukin-1Inflammatory Bowel Diseases
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Antigen-presenting function and B7 expression of murine sinusoidal endothelial cells and Kupffer cells.

1996

Abstract BACKGROUND & AIMS: Inflammatory liver disease as well as rejection of liver allografts are thought to be mediated by resident antigen- presenting cells in the liver. At the same time, in vivo antigen presentation in the liver appears to be a more tolerogenic than systemic antigen challenge. The aim of this study was to show and characterize the antigen-presenting capability of sinusoidal endothelial cells and Kupffer cells. METHODS: Purified murine sinusoidal endothelial cells and Kupffer cells were studied for their ability to serve as accessory cells and antigen-presenting cells by proliferation assays. They were also studied for their expression of interleukin 1 and the B7 costi…

Liver cytologyKupffer CellsAntigen presentationMolecular Sequence DataAntigen-Presenting CellsBiologyLymphocyte ActivationPolymerase Chain ReactionMicemedicineAnimalsRNA MessengerAntigen-presenting cellInterleukin 3Antigen PresentationMice Inbred BALB CCD40HepatologyBase SequenceKupffer cellGastroenterologyBlotting NorthernCell biologyInterleukin-10RatsInterleukin 33medicine.anatomical_structureLiverImmunologybiology.proteinInterleukin 12B7-1 AntigenEndothelium VascularInterleukin-1Gastroenterology
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Modulation of dendritic cell properties by laquinimod as a mechanism for modulating multiple sclerosis.

2013

Laquinimod is an orally administered compound that is under investigation in relapsing-remitting multiple sclerosis. To understand the mechanism by which laquinimod exerts its clinical effects, we have performed human and murine studies assessing its immunomodulatory properties. In experimental autoimmune encephalomyelitis, the therapeutic administration of laquinimod beginning during the recovery of SJL mice, prevented further relapses as expected and strongly reduced infiltration of CD4+ and CD8+ T cells in the central nervous system. We hypothesized that this beneficial effect was mediated by dendritic cells, since we and others found a modulation of different dendritic cell subsets unde…

CD4-Positive T-LymphocytesChemokineEncephalomyelitis Autoimmune ExperimentalT cellQuinoloneschemistry.chemical_compoundMiceMultiple Sclerosis Relapsing-RemittingmedicineAnimalsHumansbiologyMonocyteExperimental autoimmune encephalomyelitisNF-kappa BDendritic cellDendritic Cellsmedicine.diseaseMice Inbred C57BLmedicine.anatomical_structurechemistryImmunologybiology.proteinCytokine secretionFemaleNeurology (clinical)LaquinimodCD8Brain : a journal of neurology
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Phenotype and function of monocyte derived dendritic cells in chronic hepatitis B virus infection.

2004

The antiviral T cell failure of patients with chronic hepatitis B virus (HBV) infection was suggested to be caused by a T cell stimulation defect of dendritic cells (DC). To address this hypothesis, monocyte derived DC (MDDC) of patients with chronic or resolved acute HBV infection and healthy controls were studied phenotypically by FACS analyses and functionally by mixed lymphocyte reaction, ELISA, ELISpot and proliferation assays of MDDC cultures or co-cultures with an allogeneic HBc-specific Th cell clone. HBV infection of MDDC was studied by quantitative PCR. MDDC from HBV patients seemed to be infected by the HBV, showed a reduced surface expression of HLA DR and CD40 and exhibited a r…

T cellHLA-DR7 Antigenmedicine.disease_causeMonocytesHepatitis B ChronicVirologymedicineHumansCD40 AntigensHepatitis B virusCD40biologyMonocyteELISPOTvirus diseasesDendritic cellDendritic CellsMixed lymphocyte reactionVirologydigestive system diseasesHBcAgmedicine.anatomical_structurePhenotypeImmunologyDNA Viralbiology.proteinCytokinesThe Journal of general virology
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