Search results for "CD86"

showing 10 items of 31 documents

Succinate receptor mediates intestinal inflammation and fibrosis.

2018

Succinate, an intermediate of the tricarboxylic acid cycle, is accumulated in inflamed areas and its signaling through succinate receptor (SUCNR1) regulates immune function. We analyze SUCNR1 expression in the intestine of Crohn's disease patients and its role in murine intestinal inflammation and fibrosis. We show that both serum and intestinal succinate levels and SUCNR1 expression in intestinal surgical resections were higher in CD patients than in controls. SUCNR1 co-localized with CD86, CD206, and alpha-SMA(+) cells in human intestine and we found a positive and significant correlation between SUCNR1 and alpha-SMA expression. In human isolated fibroblasts from CD patients SUCNR1 expres…

0301 basic medicineAdultMaleAdolescentImmunologyMacrophage polarizationSuccinic Acid610 Medicine & healthProinflammatory cytokineReceptors G-Protein-Coupled03 medical and health sciencesMiceYoung Adult0302 clinical medicineImmune systemCrohn DiseaseFibrosismedicineImmunology and AllergyAnimalsHumansIntestinal MucosaFibroblastReceptorCells CulturedCD86InflammationMice Knockout2403 Immunologybusiness.industryMacrophagesmedicine.diseaseColitisFibrosisCitric acid cycleMice Inbred C57BLDisease Models Animal10219 Clinic for Gastroenterology and Hepatology030104 developmental biologymedicine.anatomical_structure2723 Immunology and AllergyCancer researchFemalebusiness030215 immunologyMucosal immunology
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Polymorphisms in genes involved in T-cell co-stimulation are associated with blood pressure in women.

2019

In recent years, conclusive data have emerged on a relationship between immune system, especially the T-cell, and blood pressure (BP). The objective of the present study was to determine the association between BP and four polymorphisms in CD80, CD86, CD28 and CTLA4 genes that code for key proteins in the T-cell co-stimulation process, in a female cohort. To that end, an association study in a cohort of 934 women over 40 years old from two hospitals was done. Raw data showed a significant association between the SNP rs1129055 of CD86 gene and BP. Analyzing this association against inheritance patterns, higher SBP (p  0.000) and DBP (p = 0.005) values were observed in AA than in GG/GA genoty…

0301 basic medicineAdultT cellT-LymphocytesPhysiologychemical and pharmacologic phenomenaBlood PressureBiologyPolymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineImmune systemCD28 AntigensGeneticsmedicineInheritance PatternsSNPHumansCTLA-4 AntigenGenetic Predisposition to DiseaseGeneCD86hemic and immune systemsGeneral MedicineMiddle Aged030104 developmental biologyBlood pressuremedicine.anatomical_structure030220 oncology & carcinogenesisCohortB7-1 AntigenFemaleB7-2 AntigenGene
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Toll like receptor mediated immune stimulation can be visualized in vivo by [ 18 F]FDG-PET

2016

Abstract Introduction High uptake of [ 18 F]-2-fluorodeoxyglucose ([ 18 F]FDG) by inflammatory cells is a frequent cause of false positive results in [ 18 F]FDG-positron-emission tomography (PET) for cancer diagnostics. Similar to cancer cells, immune cells undergo significant increases in glucose utilization following activation, e.g., in infectious diseases or after vaccination during cancer therapy. The aim of this study was to quantify certain immune effects in vitro and in vivo by [ 18 F]FDG-PET after stimulation with TLR ligands and specific antibodies. Methods In vivo [ 18 F]FDG-PET/magnetic resonance imaging (MRI) and biodistribution was performed with C57BL/6 mice immunized with Cp…

0301 basic medicineCD86Cancer ResearchPathologymedicine.medical_specialtyB-cell receptorCD28Biology03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureImmune systemIn vivo030220 oncology & carcinogenesisCancer cellmedicineCancer researchMolecular MedicineRadiology Nuclear Medicine and imagingLymph nodeCD80Nuclear Medicine and Biology
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TCR signalling network organization at the immunological synapses of murine regulatory T cells.

2017

Regulatory T (Treg) cells require T-cell receptor (TCR) signalling to exert their immunosuppressive activity, but the precise organization of the TCR signalling network compared to conventional T (Tconv) cells remains elusive. By using accurate mass spectrometry and multi-epitope ligand cartography (MELC) we characterized TCR signalling and recruitment of TCR signalling components to the immunological synapse (IS) in Treg cells and Tconv cells. With the exception of Themis which we detected in lower amounts in Treg cells, other major TCR signalling components were found equally abundant, however, their phosphorylation-status notably discriminates Treg cells from Tconv cells. Overall, this s…

0301 basic medicineProteomicsImmunological SynapsesProteomeCD3ImmunologyReceptors Antigen T-Cellchemical and pharmacologic phenomenaBiologyT-Lymphocytes RegulatoryArticleImmunological synapse03 medical and health sciencesT-Lymphocyte SubsetsImmunology and AllergyAnimalsPhosphorylationReceptorCells CulturedCD86Mice Inbred BALB CZAP-70 Protein-Tyrosine KinaseZAP70T-cell receptorCD28hemic and immune systemsImmunological SynapsesCell biology030104 developmental biologyMicroscopy Fluorescencebiology.proteinFemaleSignal TransductionEuropean journal of immunology
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Laquinimod dampens IL-1β signaling and Th17-polarizing capacity of monocytes in patients with MS

2020

ObjectiveTo assess the impact of laquinimod treatment on monocytes and to investigate the underlying immunomodulatory mechanisms in MS.MethodsIn this cross-sectional study, we performed in vivo and in vitro analyses of cluster of differentiation (CD14+) monocytes isolated from healthy donors (n = 15), untreated (n = 13), and laquinimod-treated patients with MS (n = 14). Their frequency and the expression of surface activation markers were assessed by flow cytometry and the viability by calcein staining. Cytokine concentrations in the supernatants of lipopolysaccharide (LPS)-stimulated monocytes were determined by flow cytometry. The messenger ribonucleic acid (mRNA) expression level of gene…

AdultMale41Lipopolysaccharide[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyCD14medicine.medical_treatmentInterleukin-1betaQuinolonesLymphocyte ActivationMonocytesArticleFlow cytometrychemistry.chemical_compoundMultiple Sclerosis Relapsing-RemittingDownregulation and upregulationmedicineHumansCD86medicine.diagnostic_testbusiness.industry[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyInterleukin[SDV.IMM.IMM]Life Sciences [q-bio]/Immunology/ImmunotherapyMiddle AgedMolecular biologyCross-Sectional StudiesCytokineNeurologychemistryTh17 CellsFemaleNeurology (clinical)[SDV.IMM.IMM] Life Sciences [q-bio]/Immunology/ImmunotherapybusinessLaquinimodSignal TransductionNeurology - Neuroimmunology Neuroinflammation
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Partial and Ineffective Activation of Vγ9Vδ2 T Cells by Mycobacterium tuberculosis-Infected Dendritic Cells

2010

Abstract γδ T cells and dendritic cells (DCs) participate in early phases of immune response against Mycobacterium tuberculosis. We investigated whether a close functional relationship exists between these two cell populations using an in vitro coculture in a human system. Vγ9Vδ2 T cells induce full maturation of M. tuberculosis-infected immature DCs, as demonstrated by upregulation of the costimulatory CD80, CD86, CD40, and HLA-DR molecules on infected DCs after 24 h of coculture. Reciprocally, infected DCs induced substantial activation of Vγ9Vδ2 T cells upon coculture, which was cell-to-cell contact and TCR dependent, as demonstrated in transwell experiments. However, infected DCs select…

AdultMaleImmunologyAntigen presentationchemical and pharmacologic phenomenaBiologyLymphocyte ActivationImmunophenotypingInterleukin 21T-Lymphocyte SubsetsCell Line TumorHumansImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellgamma delta T cells Mycobacterium tuberculosis dendritic cellsCells CulturedCD86Cell DifferentiationReceptors Antigen T-Cell gamma-deltaDendritic CellsMycobacterium tuberculosisMiddle AgedCytotoxicity Tests ImmunologicNatural killer T cellCoculture TechniquesCell biologyImmunologyFemaleImmunologic MemoryCD80T-Lymphocytes CytotoxicThe Journal of Immunology
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FTY720 (fingolimod) treatment tips the balance towards less immunogenic antigen-presenting cells in patients with multiple sclerosis.

2015

Objective: We aimed to clarify whether fingolimod has direct effects on antigen-presenting cells in multiple sclerosis patients. Methods: Frequency and phenotype of directly ex vivo dendritic cells and monocytes were analyzed in 43 individuals, including fingolimod-treated and untreated multiple sclerosis patients as well as healthy subjects. These cells were further stimulated with lipopolysaccharide to determine functional effects of fingolimod treatment. Results: Absolute numbers of CD1c+ dendritic cells and monocytes were not significantly reduced in fingolimod-treated patients indicating that fingolimod did not block the migration of antigen-presenting cells to peripheral blood. CD86 w…

AdultMaleMultiple Sclerosismedicine.medical_treatmentMonocytesYoung AdultMedicineHumansAntigen-presenting cellCD86business.industryFingolimod HydrochlorideMonocyteDendritic cellImmunotherapyDendritic CellsMiddle AgedFingolimodCytokinemedicine.anatomical_structureNeurologyImmunologyCytokinesFemaleNeurology (clinical)businessEx vivoImmunosuppressive Agentsmedicine.drugMultiple sclerosis (Houndmills, Basingstoke, England)
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Induction of cytokine production in naive CD4+ T cells by antigen-presenting murine liver sinusoidal endothelial cells but failure to induce differen…

1999

Abstract Background & Aims: Murine liver sinusoidal endothelial cells (LSECs) constitutively express accessory molecules and can present antigen to memory Th1 CD4+ T cells. Using a T-cell receptor transgenic mouse line, we addressed the question whether LSECs can prime naive CD4+ T cells. Methods: Purified LSECs were investigated for their ability to induce activation and differentiation of naive CD4+ T cells in comparison with bone marrow–derived antigen-presenting cells and macrovascular endothelial cells. Activation of T cells was determined by cytokine production. LSECs were further studied for expression of interleukin (IL)-12 by reverse-transcription polymerase chain reaction, and the…

CD4-Positive T-LymphocytesCellular differentiationAntigen presentationAntigen-Presenting CellsGene ExpressionPriming (immunology)BiologyMonocytesCell LineInterferon-gammaMiceInterleukin 21AnimalsEndotheliumAntigen-presenting cellCells CulturedCD86Mice Inbred BALB CHepatologyGastroenterologyCell DifferentiationTh1 CellsInterleukin-12Cell biologyEndothelial stem cellPhenotypeLiverImmunologyCytokinesFemaleBiomarkersCD80Gastroenterology
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Inhibition of human allergic T-cell responses by IL-10–treated dendritic cells: Differences from hydrocortisone-treated dendritic cells

2001

Abstract Background: Dendritic cells (DCs) are able to induce human allergic T H 1 responses as well as T H 2 responses. Objective: In this study, we examined the effect of antiinflammatory agents such as IL-10 and hydrocortisone (HC) on the accessory function of DCs and the resulting T-cell response, especially that of T H 2 cells. Methods: Naive and memory CD4 + T cells from atopic donors were stimulated with autologous allergen-pulsed DCs generated from CD14 + monocytes by culture with GM-CSF/IL-4 and fully matured with IL-1β, TNF-α, and PGE 2 in the presence or absence of IL-10 or HC. Results: IL-10–treated DCs and, to a lesser extent, HC-treated DCs showed a decreased expression of MHC…

CD4-Positive T-LymphocytesHypersensitivity ImmediateHydrocortisoneT-LymphocytesCD14T cellImmunologyAntigen presentationAnti-Inflammatory Agentschemical and pharmacologic phenomenaBiologyInterferon-gammaTh2 CellsmedicineHumansImmunology and AllergyAntigen-presenting cellCD86Antigen PresentationModels Immunologicalhemic and immune systemsDendritic CellsDendritic cellT lymphocyteAllergensInterleukin-10Interleukin 10medicine.anatomical_structureImmunologyCytokinesInterleukin-4Interleukin-5Immunologic MemoryJournal of Allergy and Clinical Immunology
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Inhibitors of β-catenin affect the immuno-phenotype and functions of dendritic cells in an inhibitor-specific manner

2015

Many tumors are characterized by mutation-induced constitutive activation of β-catenin which promotes tumor growth and survival. Consequently, the development of specific β-catenin inhibitors for tumor therapy has come into the focus of drug development. β-Catenin was also shown to contribute to the tolerance-promoting function of unstimulated dendritic cells (DCs). In response to activation, DCs acquire potent T cell stimulatory capacity and induce profound tumor antigen-specific immune responses. Here we asked for effects of pre-clinically established β-catenin inhibitors (CCT-031374, iCRT-5, PNU-75654) on mouse bone marrow-derived (BM)DCs. All three inhibitors moderately increased surfac…

CD4-Positive T-LymphocytesLipopolysaccharides0301 basic medicineCell SurvivalOvalbuminT cellImmunologyPopulationAntineoplastic AgentsBone Marrow CellsMice Transgenicchemical and pharmacologic phenomena03 medical and health sciencesImmune systemmedicineAnimalsImmunology and AllergyeducationCells Culturedbeta CateninCell ProliferationPharmacologyCD86education.field_of_studyCD40biologyFollicular dendritic cellsCell growthhemic and immune systemsDendritic CellsCell biologyMice Inbred C57BLPhenotype030104 developmental biologymedicine.anatomical_structurebiology.proteinCytokinesCD80International Immunopharmacology
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