Search results for "CELL-LINE"

showing 10 items of 11 documents

Facial cutaneo-mucosal venous malformations can develop independently of mutation of TEK gene but may be associated with excessive expression of Src…

2017

International audience; We aimed to search for mutations in the germline and somatic DNA of the TEK gene and to analyze the expression level of Src and phospho- Src (p-Src) in tumor and healthy tissues from patients with facial cutaneo-mucosal venous malformations (VMCM). Eligible patients from twelve families and thirty healthy controls were recruited respectively at the Departments of Stomatology and Oral Surgery, and Transfusion Medicine of Tlemcen University Medical Centre. Immunoblot analyses of Src and p-Src were performed after direct DNA sequencing. No somatic or germline mutations were found in all the 23 exons and their 5' and 3' intronic flanking regions, except for one case in w…

0301 basic medicineMaleSomatic cellVascular MalformationsCutaneo-mucosal venous malformationsTyrosine Kinase Tie2Bioinformaticsmedicine.disease_causeGermlineMetastasisp-SrcExonPharmacology Toxicology and Pharmaceutics(all)General Pharmacology Toxicology and PharmaceuticsPhosphorylationCancerMedicine(all)MutationBrief ReportGeneral MedicineReceptor TIE-2[SDV.BDD.MOR] Life Sciences [q-bio]/Development Biology/Morphogenesis3. Good healthsrc-Family Kinases[SDV.BBM.GTP] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]FemaleProto-oncogene tyrosine-protein kinase SrcReceptorSrc[SDV.MHEP.AHA] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]AdolescentDirect sequencingContext (language use)BiologyVegfGeneral Biochemistry Genetics and Molecular BiologyPermeability03 medical and health sciencesGermline mutationTEK gene[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN][ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologymedicine[SDV.MHEP.AHA]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansAmino Acid SequenceGeneMucous MembraneCell-Lines[ SDV ] Life Sciences [q-bio]Base SequenceBiochemistry Genetics and Molecular Biology(all)[SDV.BDD.MOR]Life Sciences [q-bio]/Development Biology/MorphogenesisGermline and somatic DNA030104 developmental biologyFaceMutationCancer researchSkin AbnormalitiesAngiogenesisPathwayJournal of negative results in biomedicine
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Host-range expansion of Spodoptera exigua multiple nucleopolyhedrovirus to Agrotis segetum larvae when the midgut is bypassed.

2010

Given the high similarity in genome content and organization between Spodoptera exigua multiple nucleopolyhedrovirus (SeMNPV) and Agrotis segetum nucleopolyhedrovirus (AgseNPV), as well as the high percentages of similarity found between their 30 core genes, the specificity of these NPVs was analysed for the respective insect hosts, S. exigua and A. segetum. The LD(50) for AgseNPV in second-instar A. segetum larvae was 83 occlusion bodies per larva and the LT(50) was 8.1 days. AgseNPV was orally infectious for S. exigua, but the LD(50) was 10 000-fold higher than for SeMNPV. SeMNPV was not infectious for A. segetum larvae when administered orally, but an infection was established by injecti…

BaculoviridaeLaboratory of VirologyMothsSpodopterain-vivoheliothis-virescens larvaeLaboratorium voor VirologiebaculovirusBeet armywormVirologyExiguaparasitic diseasescalifornica-m-nucleopolyhedrovirusAnimalsPeritrophic matrixRNA MessengerLarvabiologyReverse Transcriptase Polymerase Chain ReactionfungiNuclear Polyhedrosis VirusMidgutocclusion-derived virusbiology.organism_classificationPE&RCVirologyNucleopolyhedrovirusesperitrophic matrixIntestinesAutographa californicacell-linesbeet armywormautographa-californicanuclear polyhedrosis-virusLarvaThe Journal of general virology
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Intestinal Scavenger Receptors Are Involved in Vitamin K 1 Absorption

2014

International audience; Vitamin K-1 (phylloquinone) intestinal absorption is thought to be mediated by a carrier protein that still remains to be identified. Apical transport of vitamin K-1 was examined using Caco-2 TC-7 cell monolayers as a model of human intestinal epithelium and in transfected HEK cells. Phylloquinone uptake was then measured ex vivo using mouse intestinal explants. Finally, vitamin K-1 absorption was compared between wild-type mice and mice overexpressing scavenger receptor class B type I (SR-BI) in the intestine and mice deficient in cluster determinant 36 (CD36). Phylloquinone uptake by Caco-2 cells was saturable and was significantly impaired by co-incubation with al…

CD36 Antigens030309 nutrition & dietetics[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionCD36medicine.medical_treatmentBiochemistryIntestinal absorptionchemistry.chemical_compoundMiceVitamin EHUMAN PLASMACAROTENOIDSComputingMilieux_MISCELLANEOUSMicelles0303 health sciencesbiologyCELL-LINESR-BIVitamin K 1Scavenger Receptors Class BCD36 DEFICIENCYPostprandial PeriodIntestinal epitheliumLipidsCholesterolVitaminmedicine.medical_specialtyPHYLLOQUINONE VITAMIN-K-103 medical and health sciencesInternal medicinemedicineB TYPE-I;SR-BI;PHYLLOQUINONE VITAMIN-K-1;MENAQUINONE-4 VITAMIN-K-2;CD36 DEFICIENCY;HUMAN PLASMA;CELL-LINE;TRANSPORT;CACO-2;CAROTENOIDSAnimalsHumansScavenger receptorMolecular BiologyMENAQUINONE-4 VITAMIN-K-2030304 developmental biologyVitamin ECell MembraneCACO-2Cell BiologyTRANSPORT[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologyEnterocytesHEK293 CellschemistryIntestinal AbsorptionCaco-2B TYPE-Ibiology.proteinCaco-2 Cells[SDV.AEN]Life Sciences [q-bio]/Food and NutritionEx vivo
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Effects of BRCA2 cis-regulation in normal breast and cancer risk amongst BRCA2 mutation carriers

2012

Introduction: Cis-acting regulatory single nucleotide polymorphisms (SNPs) at specific loci may modulate penetrance of germline mutations at the same loci by introducing different levels of expression of the wild-type allele. We have previously reported that BRCA2 shows differential allelic expression and we hypothesize that the known variable penetrance of BRCA2 mutations might be associated with this mechanism. Methods: We combined haplotype analysis and differential allelic expression of BRCA2 in breast tissue to identify expression haplotypes and candidate cis-regulatory variants. These candidate variants underwent selection based on in silico predictions for regulatory potential and di…

HeterozygoteColorectal-cancerPredisposition[SDV.CAN]Life Sciences [q-bio]/CancerSingle-nucleotide polymorphismRegulatory Sequences Nucleic AcidBiologyPolymorphism Single NucleotideAssociation03 medical and health sciences0302 clinical medicineBreast cancerGermline mutation[SDV.CAN] Life Sciences [q-bio]/CancerReference ValuesmedicineHumansGenetic Predisposition to DiseaseAllelic imbalanceGene-expressionAllelePromoter Regions Geneticskin and connective tissue diseases030304 developmental biologyMedicine(all)BRCA2 ProteinGenetics0303 health sciencesHuman genomeCarcinomaHaplotypemedicine.diseasePenetranceCommon3. Good healthGene Expression Regulation NeoplasticMinor allele frequencyGene Expression RegulationHaplotypesRegulatory sequence030220 oncology & carcinogenesisBeadarrayCancer researchFemaleCell-lineTranscription FactorsResearch ArticleBreast Cancer Research
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Increased expression of leptin and the leptin receptor as a marker of breast cancer progression: possible role of obesity-related stimuli.

2006

Abstract Purpose: Recent in vitro studies suggested that the autocrine leptin loop might contribute to breast cancer development by enhancing cell growth and survival. To evaluate whether the leptin system could become a target in breast cancer therapy, we examined the expression of leptin and its receptor (ObR) in primary and metastatic breast cancer and noncancer mammary epithelium. We also studied whether the expression of leptin/ObR in breast cancer can be induced by obesity-related stimuli, such as elevated levels of insulin, insulin-like growth factor-I (IGF-I), estradiol, or hypoxic conditions. Experimental Design: The expression of leptin and ObR was examined by immunohistochemistry…

LeptinCancer ResearchER-BETAmedicine.medical_treatmentHYPOXIA-INDUCIBLE FACTOR-1NeoplasmsTumor Cells CulturedBreastInsulin-Like Growth Factor Iskin and connective tissue diseasesReceptorAged 80 and overEstradiolIGF-I RECEPTORCELL-LINEReverse Transcriptase Polymerase Chain ReactionLeptindigestive oral and skin physiologyCarcinoma Ductal BreastMiddle AgedMetastatic breast cancerINSULINCell HypoxiaESTROGENOncologyDisease ProgressionIGF-I RECEPTOR; HYPOXIA-INDUCIBLE FACTOR-1; OB GENE; GROWTH-FACTOR; CELL-LINE; ER-BETA; ESTROGEN; ALPHA; INSULIN; MCF-7Receptors LeptinFemaleOB GENEhormones hormone substitutes and hormone antagonistsAdultmedicine.medical_specialtyGROWTH-FACTORBreast NeoplasmsReceptors Cell SurfaceBreast cancerInternal medicinemedicineBiomarkers TumorHumansObesityAutocrine signallingAgedLeptin receptorbusiness.industryInsulinmedicine.diseaseALPHAEndocrinologyTumor progressionCase-Control StudiesMCF-7business
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Isoindolo[2,1-a]quinoxaline derivatives, novel potent antitumor agents with dual inhibition of tubulin polymerization and topoisomerase I.

2008

Isoindoloquinoxalines 4 and 5 were obtained by refluxing 2-(2'-aminoaryl)-1-cyanoisoindoles 3a- e in acetic or formic acid. All derivatives were screened by the National Cancer Institute (Bethesda, MD) for the in vitro one dose primary anticancer assay against a 3-cell line panel. Compounds 4a- e, screened against a panel of about 60 human tumor cell lines, showed remarkable antineoplastic activity; they had GI 50 values in the low micromolar or submicromolar range and reached, in the case of 4c, nanomolar concentrations on 88% of the 59 tested cell lines. Flow cytometric analysis of cell cycle after treatment with 4c demonstrated an arrest of the cell cycle in G2/M phase. This effect was a…

Mitotic indexMagnetic Resonance SpectroscopySpectrophotometry InfraredPolymersFLUORESCENT-PROBELIGAND-DNA SYSTEMSMitosisCELL-LINESAntineoplastic AgentsACRIDINE-ORANGETopoisomerase-I InhibitorMITOCHONDRIATubulinCell Line TumorQuinoxalinesDrug DiscoveryHumansCytotoxicitybiologyChemistryTopoisomeraseB-DNACell CycleCell cycleAPOPTOSISCDEnzyme ActivationMICROTUBULESBiochemistryMicroscopy FluorescenceCell cultureApoptosisEnzyme inhibitorLINEAR DICHROISM SPECTROSCOPYCaspasesbiology.proteinMolecular MedicineDrug Screening Assays AntitumorTopoisomerase I InhibitorsReactive Oxygen SpeciesJournal of medicinal chemistry
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Design and Synthesis of 4-Substituted Indolo[3,2-e][1,2,3]triazolo[1,5-a]pyrimidine Derivatives with Antitumor Activity

2008

New derivatives of the indolo[3,2- e][1,2,3]triazolo[1,5- a]pyrimidine system, substituted in the 4 position, were designed as novel antitumor agents because of their theoretical capability to form stable complexes with DNA fragments. The calculated free energies of binding were found in the range -12.76 --> -39.68 Kcal/mol. The docking studies revealed a common binding mode with the chromophore intercalated between GC base pairs, whereas the side chain lies along the minor groove. Compounds, selected on the basis of the docking studies and suitably synthesized, showed antiproliferative activity against each type of tumor cell line investigated, generally in the low micromolar range. The mo…

Models MolecularSEQUENCE SPECIFICITYMolecular modelPyrimidineStereochemistryDNA-BINDINGBIOLOGICAL INTERESTStereoisomerismAntineoplastic AgentsPyrimidinonesChemical synthesisHeterocyclic Compounds 4 or More RingsAUTOMATED DOCKINGchemistry.chemical_compoundStructure-Activity RelationshipCell Line TumorDrug DiscoveryStructure–activity relationshipHumansALGORITHMBinding siteCell ProliferationBinding SitesMolecular StructureChemistryBiological activityStereoisomerismDOMINO REACTIONDNADocking (molecular)Drug DesignNATIONAL-CANCER-INSTITUTEACTINOMYCIN-DMolecular MedicineCOMPLEXESDrug Screening Assays AntitumorTUMOR-CELL-LINES
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Persistence of Human Bocavirus 1 in Tonsillar Germinal Centers and Antibody-Dependent Enhancement of Infection

2021

Human bocavirus 1 (HBoV1), a common pediatric respiratory pathogen, can persist in airway secretions for months hampering diagnosis. It also persists in tonsils, providing potential reservoirs for airway shedding, with the exact location, host cell types, and virus activity unknown.

NASOPHARYNXviruksetPalatine TonsilFc receptorCHILDRENvirus persistenceMonocytesHuman bocavirusCONGENITAL INSENSITIVITYBokavirusChildviruspersistenssi11832 Microbiology and virology0303 health sciencesB-LymphocytesbiologyHuman bocavirusvasta-aineetDENGUE-VIRUS-INFECTIONrespiratory systemMiddle AgedQR1-5023. Good healthLymphatic systemB-CELLSChild PreschoolAntibodyCELL-LINE U937HUMAN PARVOVIRUSResearch ArticleAdultAdolescentEndosomesMicrobiologyinfektiotVirusHost-Microbe BiologyParvoviridae Infections03 medical and health sciencesYoung AdultImmune systemnielurisaVirologytonsilsHumansAntibody-dependent enhancementRESPIRATORY VIRUSESparvovirukset030304 developmental biologyAgedRECEPTOR030306 microbiologyparvovirusInfant NewbornGerminal centerInfantbiology.organism_classificationGerminal CenterAntibody-Dependent Enhancementrespiratory tract diseasesgerminal centerImmunologyDNA Viralbiology.protein1182 Biochemistry cell and molecular biology3111 Biomedicinein situ hybridizationADEB-soluTRACTmBio
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CASP8 SNP D302H (rs1045485) is associated with worse survival in MYCN-amplified neuroblastoma patients

2014

Background Neuroblastoma is a pediatric cancer that exhibits a wide clinical spectrum ranging from spontaneous regression in low-risk patients to fatal disease in high-risk patients. The identification of single nucleotide polymorphisms (SNPs) may help explain the heterogeneity of neuroblastoma and assist in identifying patients at higher risk for poor survival. SNPs in the TP53 pathway are of special importance, as several studies have reported associations between TP53 pathway SNPs and cancer. Of note, less than 2% of neuroblastoma tumors have a TP53 mutation at diagnosis. Patients and Methods We selected 21 of the most frequently studied SNPs in the TP53 pathway and evaluated their assoc…

OncologyGenotyping TechniquesMedizinlcsh:MedicineGenome-wide association studyPROGRESSIONSUSCEPTIBILITYBioinformaticsNeuroblastomaCHEMOSENSITIVITYMedicine and Health SciencesMissense mutationlcsh:ScienceOncogene ProteinsCaspase 8N-Myc Proto-Oncogene ProteinMultidisciplinaryCELL-LINENuclear ProteinsCANCERAPOPTOSISGENOTYPEGene Expression Regulation NeoplasticResearch Articlemedicine.medical_specialtySingle-nucleotide polymorphismBiologyN-Myc Proto-Oncogene ProteinPolymorphism Single NucleotideDisease-Free SurvivalMDM2 SNP309Molecular GeneticsNeuroblastomaInternal medicineCASPASE-8medicineGeneticsCancer GeneticsSNPHumansneoplasmsNeoplasm StagingClinical GeneticsP53lcsh:RGene AmplificationCancerInfantBiology and Life Sciencesmedicine.diseasePediatric cancerGeriatricsGenetics of Diseaselcsh:Q
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Second report on chicken genes and chromosomes 2005.

2005

International audience

medicine.medical_specialtyChickens/genetics[SDV]Life Sciences [q-bio]Single-nucleotide polymorphismAnimal Breeding and Genomicsin-situ hybridizationMajor histocompatibility complexChromosomes5S ribosomal RNAMolecular geneticssingle-nucleotide polymorphismsMHC class IGeneticsmedicineAnimalsmhc class-itranslation initiation factor-4aFokkerij en GenomicaCYTOGENETIC MAPSMolecular BiologyGeneexpressed sequence tagsGenetics (clinical)ComputingMilieux_MISCELLANEOUSnucleolar-size polymorphismsGeneticsExpressed sequence tagCHICKENSModels GeneticbiologyChromosomes/geneticsdt40 cell-linetelomerase rna genemajor histocompatibility complexHuman genetics[SDV] Life Sciences [q-bio]GENETIC MAPS5s ribosomal-rnaWIASbiology.protein
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