Search results for "CHEMOKINE"

showing 10 items of 372 documents

Subendothelial infiltration of neutrophil granulocytes and liberation of matrix-destabilizing enzymes in an experimental model of human neo-intima.

2008

SummaryIt was the objective of this study to examine the role of human neutrophil granulocytes (PMN) in an in-vitro model of human neo-intima developed for the study of atherosclerosis. Human granulocytes were subjected to a co-culture model of human endothelial and smooth muscle cells. Subendothelial lipid accumulation was achieved by addition of native LDL to the culture medium. Tissue samples were analyzed by immunohistochemistry and scanning/transmission electron microscopy, and culture supernatants were examined for the presence of interleukin- 8 (IL-8), MCP-1, GRO-α, elastase and matrixmetalloproteinase-8 (MMP-8). Following addition of 2 mg/ml LDL, adherence, transmigration and infilt…

Pathologymedicine.medical_specialtyTime FactorsEndotheliumNeutrophilsChemokine CXCL1Myocytes Smooth MuscleApoptosisBiologyGranulocyteMuscle Smooth VascularmedicineMyocyteHumansSecretionLeukocyte RollingCells CulturedChemokine CCL2ElastaseInterleukin-8InterleukinEndothelial CellsHematologymedicine.diseaseAtherosclerosisMolecular biologyCoculture TechniquesCulture MediaExtracellular MatrixLipoproteins LDLmedicine.anatomical_structureMatrix Metalloproteinase 8Neutrophil InfiltrationApoptosisLeukocyte ElastaseTunica IntimaInfiltration (medical)Signal TransductionThrombosis and haemostasis
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Spatial and temporal heterogeneity of ventilator-associated lung injury after surfactant depletion.

2008

Volutrauma and atelectrauma have been proposed as mechanisms of ventilator-associated lung injury, but few studies have compared their relative importance in mediating lung injury. The objective of our study was to compare the injury produced by stretch (volutrauma) vs. cyclical recruitment (atelectrauma) after surfactant depletion. In saline-lavaged rabbits, we used high tidal volume, low respiratory rate, and low positive end-expiratory pressure to produce stretch injury in nondependent lung regions and cyclical recruitment in dependent lung regions. Tidal changes in shunt fraction were assessed by measuring arterial Po2 oscillations. After ventilating for times ranging from 0 to 6 h, lu…

Pathologymedicine.medical_specialtyVentilator-associated lung injuryPhysiologyNitric Oxide Synthase Type IIInflammationEnzyme-Linked Immunosorbent AssayPulmonary EdemaRespiratory physiologyLung injuryPhysiology (medical)medicineAnimalsInterleukin 8LungChemokine CCL2PeroxidaseLungVentilators Mechanicalbusiness.industryRespiratory diseaseInterleukin-8Pulmonary SurfactantsLung Injuryrespiratory systemmedicine.diseasePulmonary edemarespiratory tract diseasesOxygenmedicine.anatomical_structureNeutrophil InfiltrationCalibrationRespiratory MechanicsCytokinesFluid TherapyFemaleRabbitsmedicine.symptomBlood Gas AnalysisChemokinesbusinessJournal of applied physiology (Bethesda, Md. : 1985)
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OP0081 Tissue Deficiency of The Atypical Chemokine Receptor D6 Is Associated with The Selective Increase of Gut-Derived Pro-Inflammatory CXCR1HIGHLY6…

2016

Background Gut derived innate lymphoid cells of type 3 (ILC)3 are increased in number in the circulation and inflamed tissues of AS patients. Factors influencing the maintainace of ILC3 in an activated status are not clear. The atypical chemokine receptor D6 is a decoy and scavenger receptor for most inflammatory CC chemokines and acts preventing exacerbated inflammatory reactions. Mice lacking D6 expression in the non-hematopoietic compartment display a significant increase of pro-inflammatory monocytes in the peripheral blood and in secondary lymphoid tissues. The role of D6 in human inflammatory disorders has not been inverstigated. Objectives To evaluate whether modulation of D6 express…

Pathologymedicine.medical_specialtymedicine.diagnostic_testbusiness.industryMonocyteImmunologyInnate lymphoid cellC-C chemokine receptor type 7IleumGeneral Biochemistry Genetics and Molecular BiologyFlow cytometrymedicine.anatomical_structureRheumatologyImmunologymedicineImmunology and AllergyMacrophageBone marrowScavenger receptorbusinessAnnals of the Rheumatic Diseases
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Plasma profile of pro-inflammatory cytokines and chemokines in cocaine users under outpatient treatment: influence of cocaine symptom severity and ps…

2014

The treatment for cocaine use constitutes a clinical challenge because of the lack of appropriate therapies and the high rate of relapse. Recent evidence indicates that the immune system might be involved in the pathogenesis of cocaine addiction and its co-morbid psychiatric disorders. This work examined the plasma pro-inflammatory cytokine and chemokine profile in abstinent cocaine users (n = 82) who sought outpatient cocaine treatment and age/sex/body mass-matched controls (n = 65). Participants were assessed with the diagnostic interview Psychiatric Research Interview for Substance and Mental Diseases according to the Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition,…

Pharmacologymedicine.medical_specialtyChemokinebiologybusiness.industrymedicine.medical_treatmentAddictionmedia_common.quotation_subjectPsychological interventionMedicine (miscellaneous)Psychiatry and Mental healthMoodCytokineEtiologymedicinebiology.proteinAnxietymedicine.symptomCX3CL1Psychiatrybusinessmedia_commonAddiction Biology
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Zur Immunpathogenese des Asthma bronchiale

2000

Allergic asthma is a chronic pulmonary disease associated with bronchoconstriction and inflammation. Recent studies have shown that mediator substances and proinflammatory cytokines produced by mast cells, eosinophils and T-lymphocytes appear to be important for the pathogenesis of asthma. These substances contribute both to the initiation and perpetuation of the disease. In particular, it has been shown that allergic asthma is associated with increased TH2 (IL-4, IL-5, IL-13) cytokine production that causes activation of eosinophils and T-cells and production of chemokines (e.g. eotaxin) by pulmonary fibroblasts. Based on recent advances in our understanding of the immunopathogenesis of as…

Pulmonary and Respiratory MedicineEotaxinChemokinebiologybusiness.industrymedicine.medical_treatmentInflammationImmunoglobulin Emedicine.diseaseProinflammatory cytokineCytokineImmunologybiology.proteinMedicineBronchoconstrictionmedicine.symptombusinessAsthmaPneumologie
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Indoor air pollutants stimulate interleukin-8-specific mRNA expression and protein secretion of alveolar macrophages.

1998

Indoor air pollutants may cause inflammatory changes of the airways and adjacent pulmonary tissue. After phagocytosis of inhaled particles alveolar macrophages (AM) release chemotactic mediators capable of attracting inflammatory cells into the lung tissue. To evaluate these mechanisms further peripheral blood mononuclear cells (PBMNC) and human AM (freshly recovered from the lower respiratory tract) were exposed to the indoor particles Soot FR 101 and Printex 90, the asbestos fiber Chrysotile B, and titanium dioxide (TiO2) at concentrations of 10 or 50 microg/10(6) cells for up to 8 h. The migration of granulocytes into the conditioned supernatants of AM and PBMNC was quantified by chemota…

Pulmonary and Respiratory MedicineGranulocyte migrationAdultMaleChemokineBiologyGranulocytePeripheral blood mononuclear cellPolymerase Chain ReactionMicrobiologyMacrophages AlveolarmedicineHumansInterleukin 8RNA MessengerAgedAir PollutantsLungInterleukin-8InterleukinMolecular biologyChemotaxis Leukocytemedicine.anatomical_structureAir Pollution Indoorbiology.proteinLeukocytes MononuclearFemaleBronchoalveolar Lavage FluidChemotaxis assayLung
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TLR4 Up-regulation and Reduced Foxp3 Expression in Mechanically Ventilated Smokers with Obstructive Chronic Bronchitis

2013

Background: Chronic bronchitis (CB) is a risk factor in chronic obstructive pulmonary disease (COPD) for accelerated lung function decline and increased mortality. The lung and systemic inflammatory and immunological profile of COPD patients with CB which acutely experience respiratory failure upon a disease exacerbation is unknown. Methods: In this study, we explored the expression of Foxp3 by western blot analysis, TLR4 by immunocytochemistry and the concentrations of IP-10 and IL-8 by ELISA in the mini-bronchoalveolar lavages (mini-BAL) and in the peripheral blood of patients with respiratory failure requiring intubation and mechanical ventilation. The recruited subjects were separated i…

Pulmonary and Respiratory MedicineMaleChronic bronchitismedicine.medical_specialtyPathologyNeutrophilsmedicine.medical_treatmentSettore MED/41 - AnestesiologiaGastroenterologyStatistics NonparametricLeukocyte CountPulmonary Disease Chronic ObstructiveAcute Lung InjuryToll-like receptors Foxp3 Chemokines Smokers Respiratory failureInternal medicineMedicineHumansRisk factorAgedMechanical ventilationAged 80 and overCOPDLungbusiness.industryInterleukin-8SmokingFOXP3Forkhead Transcription Factorsmedicine.diseaseRespiration Artificialrespiratory tract diseasesUp-RegulationBronchitis ChronicChemokine CXCL10Toll-Like Receptor 4medicine.anatomical_structureRespiratory failureBronchitisFemalebusinessBronchoalveolar Lavage Fluid
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Critical role of fractalkine (CX3CL1) in cigarette smoke-induced mononuclear cell adhesion to the arterial endothelium.

2012

Background Cigarette smoking is an important risk factor for the development of cardiovascular disease, yet the pathways through which this may operate are poorly understood. Therefore, the mechanism underlying cigarette smoke (CS)-induced arterial endothelial dysfunction and the potential link with fractalkine/CX3CL1 upregulation were investigated. Methods and results Stimulation of human arterial umbilical endothelial cells (HUAECs) with pathophysiological concentrations of CS extract (1% CSE) increased CX3CL1 expression. Neutralisation of CX3CL1 activity under dynamic flow conditions significantly inhibited CSE-induced mononuclear cell adhesion to HUAECs (67%). The use of small interferi…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyEndotheliumPeripheral blood mononuclear cellMiceInternal medicineCX3CR1Cell AdhesionMedicineAnimalsHumansEndothelial dysfunctionRNA Small InterferingCell adhesionNADPH oxidasebiologybusiness.industryChemokine CX3CL1MicrocirculationSmokingNOX4Membrane ProteinsNADPH Oxidasesmedicine.diseaseUp-RegulationEndothelial stem cellmedicine.anatomical_structureEndocrinologyNADPH Oxidase 5Immunologybiology.proteinEndothelium VascularbusinessThorax
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Second-generation Langerhans cells originating from epidermal precursors are essential for CD8+ T cell priming.

2014

Abstract In vivo studies questioned the ability of Langerhans cells (LCs) to mediate CD8+ T cell priming. To address this issue, we used intradermal immunization with plasmid DNA, a system in which activation of CD8+ T cells depends on delayed kinetics of Ag presentation. We found that dendritic cells (DCs) located in the skin at the time of immunization have limited ability to activate CD8+ T cells. This activity was mediated by a second generation of DCs that differentiated in the skin several days after immunization, as well as by lymph node–resident DCs. Intriguingly, CD8+ T cell responses were not affected following treatment with clodronate liposomes, immunization of CCR2−/− mice, or …

Receptors CCR2T cellImmunologyPriming (immunology)CD11cchemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationMiceImmune systemGiant Cells LanghansmedicineImmunology and AllergyCytotoxic T cellAnimalsSkinMice KnockoutChemokine CCL20integumentary systemhemic and immune systemsCell DifferentiationDendritic CellsMolecular biologyCD11c AntigenCCL20Mice Inbred C57BLmedicine.anatomical_structureImmunologyIntercellular Signaling Peptides and ProteinsClodronic AcidCD8Ex vivoHeparin-binding EGF-like Growth FactorPlasmidsJournal of immunology (Baltimore, Md. : 1950)
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Amazing IL-9: revealing a new function for an “old” cytokine

2012

Th9 cells are a subset of CD4+ Th cells that produce the pleiotropic cytokine IL-9. IL-9/Th9 can function as both positive and negative regulators of immune response, but the role of IL-9/Th9 in tumor immunity is unknown. We examined the role of IL-9/Th9 in a model of pulmonary melanoma in mice. Lack of IL-9 enhanced tumor growth, while tumor-specific Th9 cell treatment promoted stronger antitumor responses in both prophylactic and therapeutic models. Th9 cells also elicited strong host antitumor CD8+ CTL responses by promoting Ccl20/Ccr6-dependent recruitment of DCs to the tumor tissues. Subsequent tumor antigen delivery to the draining LN resulted in CD8+ T cell priming. In agreement with…

Receptors CCR6Lung Neoplasmsmedicine.medical_treatmentBiologyCD8-Positive T-LymphocytesMiceImmunityCell Line TumormedicineAnimalsLack of knowledgeInterleukin 9MelanomaMice KnockoutAntigen PresentationImmunity CellularChemokine CCL20Antitumor immunityMelanomaInterleukin-9General MedicineDendritic CellsT-Lymphocytes Helper-Inducermedicine.diseaseCytokineImmunologyCommentaryImmunotherapySkin cancerFunction (biology)
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