Search results for "COLOR"

showing 10 items of 2721 documents

EGFR genomic alterations in cancer: prognostic and predictive values.

2011

The role of EGFR in cancer development and progression has been recognized for long time in a variety of human malignancies including lung, head and neck, colon, breast, ovary and glioma. Recently its role as a target of antineoplastic agents has also been identified and a variety of EGFR-targeted drugs is already being used in a clinical setting and others are at present under investigation. Many data involving EGFR protein expression are now available for the choice of anti-EGFR monoclonal antibodies in colorectal cancer and with regard to EGFR gene mutations for the choice of tyrosine kinase inhibitors in lung cancer. Other EGFR-related molecular factors, including the EGFR gene copy num…

General Immunology and MicrobiologySettore MED/06 - Oncologia MedicaColorectal cancerbusiness.industryGene DosageCancerGene mutationmedicine.diseasePrognosisGene dosageGeneral Biochemistry Genetics and Molecular BiologyErbB ReceptorsGliomaMutationmedicineCancer researchHumansCopy-number variationEGFR cancerLung cancerbusinessTyrosine kinaseFrontiers in bioscience (Elite edition)
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Two-Color Radiation Generated in a Seeded Free-Electron Laser with Two Electron Beams.

2015

We present the experimental evidence of the generation of coherent and statistically stable two-color free-electron laser radiation obtained by seeding an electron beam double peaked in energy with a laser pulse single spiked in frequency. The radiation presents two neat spectral lines, with time delay, frequency separation, and relative intensity that can be accurately controlled. The analysis of the emitted radiation shows a temporal coherence and a shot-to-shot regularity in frequency significantly enhanced with respect to the self-amplified spontaneous emission. © 2015 American Physical Society. © 2015 American Physical Society.

General Physics and AstronomyColorElectronsElectronseeding FELRadiation01 natural scienceslaw.inventionPhysics and Astronomy (all)OpticsColor Computer Simulation Image Processing Computer-Assisted Spectrum Analysis Electrons LaserslawFrequency separation0103 physical sciencesElectron beams; free electron lasers; physicsImage Processing Computer-AssistedSpontaneous emissionComputer Simulation010306 general physicsPhysicsFEL010308 nuclear & particles physicsbusiness.industryLasersSpectrum AnalysisSettore FIS/01 - Fisica SperimentaleFree-electron laserElectron beamsSPARCLaserFEL seeding FEL lasersFree Electron Laserfree electron lasersCathode raybusinessSeeded FELphysicsCoherence (physics)Physical review letters
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Cloning and Expression of the mRNA of Human Galectin-4, an S-type Lectin Down-Regulated in Colorectal Cancer

1997

We are interested in the characterization of genes whose expressions in the colon are modified during colorectal carcinogenesis. Our approach was to establish the phenotype of a colon tumor by partial sequencing of a large number of transcripts, then to select mRNAs of potential interest by differential screening with complex probes from normal or cancerous colon. In this paper, we report the cloning and sequencing of a mRNA strongly underexpressed in colorectal cancer. It corresponded to a protein comprising 323 amino acids, that appeared to be human galectin-4 on the basis of 76% and 79% amino acid identity to the rat and pig counterparts, respectively. Tissue distribution analysis showed…

Genetic MarkersDNA ComplementaryColorectal cancerGalectin 4Molecular Sequence DataDown-RegulationRectumBiologyBiochemistryLectinsBiomarkers TumorTumor Cells CulturedmedicineHumansAmino Acid SequenceRNA MessengerRNA NeoplasmCloning MolecularGeneCloningExpressed sequence tagMessenger RNABase SequenceSequence Homology Amino AcidDNA Neoplasmmedicine.diseaseMolecular biologyPhenotypedigestive system diseasesGene Expression Regulation NeoplasticHemagglutininsmedicine.anatomical_structureCell cultureColorectal NeoplasmsEuropean Journal of Biochemistry
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Real-Time Quantification in Plasma of Human Telomerase Reverse Transcriptase (hTERT) mRNA: A Simple Blood Test to Monitor Disease in Cancer Patients

2001

Real-Time Quantification in Plasma of Human Telomerase Reverse Transcriptase (hTERT) mRNA: A Simple Blood Test to Monitor Disease in Cancer Patients

Genetic MarkersTelomeraseDiseaseBiologyPathology and Forensic MedicineNeoplasmsBiomarkers TumormedicineHumansBlood testTelomerase reverse transcriptaseRNA MessengerLymphoma FollicularTelomeraseMolecular BiologyMessenger RNAmedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionCancerCell BiologyPrognosismedicine.diseaseNucleotidyltransferaseMolecular biologyDNA-Binding ProteinsReverse transcription polymerase chain reactionRNAColorectal NeoplasmsLaboratory Investigation
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IL-6 Signaling Promotes Tumor Growth in Colorectal Cancer

2004

Recent investigations support an important role for TGF-beta in the development of colorectal cancer. However, the molecular consequences of TGF-beta signaling in the colon remains incompletely understood. In a recent study in Immunity, we analyzed the role of TGF-beta in a murine model of colon cancer. Using transgenic mice overexpressing TGF-beta or a dominant negative TGF-beta receptor II under control of the CD2 minigene, we show that TGF-beta signaling in tumor infiltrating T lymphocytes regulates the growth of dysplastic colon epithelial cells, as determined by histology and a novel system for high resolution chromoendoscopy in vivo. At the molecular level, TGF-beta signaling in T cel…

Genetically modified mouseCell growthColorectal cancerCell BiologyBiologyEndoglinmedicine.diseaseTumor progressionIn vivoImmunologyCancer researchbiology.proteinmedicineADAM17 ProteinInterleukin 6Molecular BiologyDevelopmental BiologyCell Cycle
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TGF-β Suppresses Tumor Progression in Colon Cancer by Inhibition of IL-6 trans-Signaling

2004

Alterations of TGF-beta signaling have been described in colorectal cancer, although the molecular consequences are largely unknown. By using transgenic mice overexpressing TGF-beta or a dominant-negative TGF-betaRII, we demonstrate that TGF-beta signaling in tumor infiltrating T lymphocytes controls the growth of dysplastic epithelial cells in experimental colorectal cancer, as determined by histology and a novel system for high-resolution chromoendoscopy. At the molecular level, TGF-beta signaling in T cells regulated STAT-3 activation in tumor cells via IL-6. IL-6 signaling required tumor cell-derived soluble IL-6R rather than membrane bound IL-6R and suppression of such TGF-beta-depende…

Genetically modified mouseSTAT3 Transcription FactorColorectal cancerRecombinant Fusion ProteinsT-LymphocytesImmunologyBlotting WesternEnzyme-Linked Immunosorbent AssayMice TransgenicProtein Serine-Threonine KinasesMiceIn vivoTransforming Growth Factor betamedicineImmunology and AllergyAnimalsHumansEndoscopy Digestive SystemIntestinal MucosaInterleukin 6Autocrine signallingMice KnockoutbiologyInterleukin-6Reverse Transcriptase Polymerase Chain ReactionReceptor Transforming Growth Factor-beta Type IIHistologymedicine.diseaseImmunohistochemistryReceptors Interleukin-6DNA-Binding ProteinsDisease Models AnimalInfectious DiseasesTumor progressionImmunologyColonic NeoplasmsCancer researchbiology.proteinDisease ProgressionTrans-ActivatorsReceptors Transforming Growth Factor betaTransforming growth factorSignal TransductionImmunity
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Non-small cell lung cancer (NSCLC), EGFR downstream pathway activation and TKI targeted therapies sensitivity: Effect of the plasma membrane-associat…

2017

Adenocarcinoma of Non-Small Cell Lung Cancer (NSCLC) is a severe disease. Patients carrying EGFR mutations may benefit from EGFR targeted therapies (e.g.: gefitinib). Recently, it has been shown that sialidase NEU3 directly interacts and regulates EGFR. In this work, we investigate the effect of sialidase NEU3 overexpression on EGFR pathways activation and EGFR targeted therapies sensitivity, in a series of lung cancer cell lines. NEU3 overexpression, forced after transfection, does not affect NSCLC cell viability. We demonstrate that NEU3 overexpression stimulates the ERK pathway but this activation is completely abolished by gefitinib treatment. The Akt pathway is also hyper-activated upo…

Genetics and Molecular Biology (all)0301 basic medicineOncologyMAPK/ERK pathwayLung NeoplasmsColorectal cancerCell Membraneslcsh:Medicinenon-small cell lung cancer (NSCLC)BiochemistryLung and Intrathoracic TumorsAntineoplastic Agent0302 clinical medicineProtein-Tyrosine KinaseCarcinoma Non-Small-Cell LungMedicine and Health SciencesPost-Translational ModificationPhosphorylationNon-Small-Cell Lunglcsh:ScienceTumorMultidisciplinaryBlottingGefitinibTransfectionProtein-Tyrosine KinasesBIO/10 - BIOCHIMICAErbB ReceptorsOncology030220 oncology & carcinogenesisAdenocarcinomaPhosphorylationHyperexpression TechniquesElectrophoresis Polyacrylamide GelCellular Structures and OrganellesWesternReceptorHumanmedicine.drugSignal TransductionResearch ArticleElectrophoresismedicine.medical_specialtyBlotting WesternNeuraminidaseAntineoplastic AgentsReal-Time Polymerase Chain ReactionTransfectionResearch and Analysis MethodsCell Line03 medical and health sciencesGefitinibInternal medicineCell Line TumormedicineGeneticsGene Expression and Vector TechniquesHumansPoint MutationMolecular Biology TechniquesMolecular BiologyPI3K/AKT/mTOR pathwayColorectal CancerMolecular Biology Assays and Analysis TechniquesPolyacrylamide GelBiochemistry Genetics and Molecular Biology (all)Epidermal Growth Factorbusiness.industryCarcinomalcsh:RCell MembraneQuinazolineCancers and NeoplasmsBiology and Life SciencesProteinsCell Biologymedicine.diseaserespiratory tract diseasesNon-Small Cell Lung CancerLung Neoplasm030104 developmental biologyAgricultural and Biological Sciences (all)MutationQuinazolineslcsh:QReceptor Epidermal Growth FactorAntineoplastic Agents; Blotting Western; Carcinoma Non-Small-Cell Lung; Cell Line Tumor; Cell Membrane; Electrophoresis Polyacrylamide Gel; Humans; Lung Neoplasms; Neuraminidase; Protein-Tyrosine Kinases; Quinazolines; Real-Time Polymerase Chain Reaction; Receptor Epidermal Growth Factor; Signal Transduction; Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)businessPloS one
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Infectious transfer of a fertility factor inStreptomyces coelicolor

1973

SUMMARYInitial Fertility (IF) strains ofStreptomyces coelicolorare able to convert recipient strains (UF) to the IF condition by contact, without concomitant transfer of chromosomal markers. The conversion is prevented by the presence of acridine orange in the medium of the mixed culture. Acridine orange is also moderately effective in inducing the formation of UF variants from IF-treated strains. No effect of the drug is observed on UF variant formation from Normal Fertility (NF) strains nor on the behaviour of the fertility factor in NF × UF mixed cultures. The hypothesis is put forward that the fertility factor works as an episome inS. coelicolor, fixed to the chromosome in the NF strain…

Genetics MicrobialGeneticsFertility factor (bacteria)biologymedia_common.quotation_subjectStreptomyces coelicolorAcridine orangeChromosomeFertilityGeneral MedicineNormal fertilitybiology.organism_classificationStreptomyceschemistry.chemical_compoundFertilitychemistryMixed cultureGeneticsCrossing Over GeneticAllelesmedia_commonGenetical Research
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An operon for histidine biosynthesis in Streptomyces coelicolor

1973

On the assumption that a cluster of five his genes (eight cistrons) in S. coelicolor corresponds to an operon, a genetic analysis of a constitutive mutant was carried out. This strain has a multi-site mutation localized at the (conventional) right end of the his cluster and is derepressed for at least two enzymes coded by genes of the cluster. The study of suitable heterozygous clones (heteroclones), showed the mutation to be cis-dominant, suggesting that the operator region is affected. Most likely the strain has a deletion connecting the his operon to an adjacent amm (ammonium requirement) operon as demonstrated by its inability to utilize nitrate as nitrogen source and to complement or r…

Genetics MicrobialHeterozygoteOperator (biology)Genetic LinkageOperonBiologyGenetic analysisOperonGeneticsHistidineAminesMolecular BiologyGeneAllelesCrosses GeneticGenes Dominantchemistry.chemical_classificationGeneticsNitratesStrain (chemistry)Streptomyces coelicolorChromosome MappingDrug Resistance Microbialbiology.organism_classificationStreptomycesQuaternary Ammonium CompoundsButyratesEnzymechemistryMutation (genetic algorithm)Molecular and General Genetics MGG
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Two distinct amplification events of the c-myc locus in a colorectal tumour.

2008

Southern hybridisation of genomic DNA extracted from a human primary colorectal carcinoma revealed amplification of a fragment containing the wild-type c-myc locus. Two additional rearranged DNA fragments, lying upstream of c-myc, fused to distant non-contiguous sequences from the same chromosome, with an opposite configuration (head to head vs. head to tail), were also found to be amplified. Sequences analysis suggested that these rearrangements resulted from illegitimate recombination at two distinct points within the DNA sequence just upstream of the c-myc ORF and further that these events triggered two different amplification mechanisms, only one of which, involving a strand invasion ev…

GeneticsBase SequencePhysiologyMolecular Sequence DataClinical BiochemistryGene AmplificationGenes mycColorectal tumourLocus (genetics)Cell BiologyBiologyMolecular biologyDNA sequencingBlotting Southernchemistry.chemical_compoundgenomic DNAchemistryGene duplicationHumansStrand invasionColorectal Neoplasmsgene amplification c-myc CRCDNARecombination
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