Search results for "CYTOKINE"

showing 10 items of 1787 documents

Interferon-alpha stimulates production of interleukin-10 in activated CD4+ T cells and monocytes

1996

In the present study, we investigated the effect of interferon-alpha (IFN-alpha) on the expression of interleukin-10 (IL-10) mRNA and protein synthesis in human monocytes and CD4+ T cells. In mononuclear cells, IFN-alpha induced expression of IL-10 mRNA and further enhanced lipopolysaccharide (LPS)-stimulated IL-10 expression. In purified monocytes, a strong expression of IL-10 mRNA induced by LPS was not further enhanced by IFN-alpha. In highly purified CD4+ T cells, IFN- alpha upregulated IL-10 mRNA upon activation with phytohemagglutinin and phorbol myristate acetate. In purified monocytes, an effect of IFN- alpha on IL-10 protein synthesis was dependent on costimulation with LPS. Maxima…

LipopolysaccharideMonocytemedicine.medical_treatmentCD3ImmunologyCD28Alpha interferonCell BiologyHematologyBiologyBiochemistryMolecular biologyPeripheral blood mononuclear cellchemistry.chemical_compoundInterleukin 10medicine.anatomical_structureCytokinechemistryImmunologymedicinebiology.proteinBlood
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Persistent inflammation alters the function of the endogenous brain stem cell compartment

2008

Endogenous neural stem/precursor cells (NPCs) are considered a functional reservoir for promoting tissue homeostasis and repair after injury, therefore regenerative strategies that mobilize these cells have recently been proposed. Despite evidence of increased neurogenesis upon acute inflammatory insults (e.g. ischaemic stroke), the plasticity of the endogenous brain stem cell compartment in chronic CNS inflammatory disorders remains poorly characterized. Here we show that persistent brain inflammation, induced by immune cells targeting myelin, extensively alters the proliferative and migratory properties of subventricular zone (SVZ)-resident NPCs in vivo leading to significant accumulation…

Encephalomyelitis Autoimmune Experimentalexperimental autoimmune encephalomyelitisSubventricular zoneInflammationBiologymultiple sclerosisMice03 medical and health sciences0302 clinical medicineNeuroblastCell MovementPrecursor cellischemic strokemedicineAnimalsCells CulturedTissue homeostasisCell Proliferationneural stem cells030304 developmental biology0303 health sciencesStem CellsCell CycleNeurogenesisOriginal Articlesbrain cell stemNeural stem cellClone CellsNerve RegenerationMice Inbred C57BLMicroscopy Electronneurogenesismedicine.anatomical_structureinflammationChronic DiseaseModels AnimalCytokinesFemaleNeurology (clinical)Stem cellmedicine.symptomNeuroscience030217 neurology & neurosurgeryBrain StemBrain
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Immunomodulatory and Hematopoietic Effects of Recombinant Human Interleukin-6 in Patients with Advanced Renal Cell Cancer

1996

Interleukin-6 (IL-6) is a cytokine with pleiotropic biologic activities on B cells, T cells, and hematopoietic progenitors. The present study was undertaken to assess pharmacodynamic effects of subcutaneous administration of IL-6 on blood counts, immunologic parameters, and acute-phase reactants. Blood samples were taken from patients with advanced renal cell cancer participating in a phase II trial of recombinant human IL-6. Multiparameter FACS analyses of peripheral blood mononuclear cells were performed using antibodies against CD3, CD4, CD8, HLA-DR, CD56, CD28, CD38, CD19, sIgM, and sIgG. Serum levels of IL-10, soluble CD23 (sCD23), sCD25, IL-1 receptor antagonist protein (IL-1RA), solu…

medicine.medical_specialtyInjections Subcutaneousmedicine.medical_treatmentImmunologyPeripheral blood mononuclear cellCD19chemistry.chemical_compoundImmunophenotypingAdjuvants ImmunologicVirologyInternal medicinemedicineHumansAcute-Phase ReactionCarcinoma Renal CellbiologyInterleukin-6business.industryCD23NeopterinCell BiologyKidney NeoplasmsRecombinant ProteinsBlood Cell CountHematopoiesisHaematopoiesisCytokineEndocrinologychemistryImmunologybiology.proteinbusinessCD8Journal of Interferon & Cytokine Research
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The pituitary gland prevents shock-associated death by controlling multiple inflammatory mediators

2018

Abstract Bacterial infections cause a major burden of disease worldwide. Sepsis and septic shock are life-threatening complications of infections. The hypothalamic-pituitary-adrenal (HPA) axis initiates the release of endogenous glucocorticoids that modulate the host stress response and acute inflammation during septic shock. It is an ongoing controversial debate, if therapeutic manipulations of the HPA axis could benefit the clinical situation in the context of shock. Here, we have studied Long Evans rats with hypophysectomy followed by endotoxic shock. The shock-associated lethality was substantially higher in hypophysectomized rats as compared to control mice after cranial sham surgery (…

0301 basic medicineMalePituitary glandmedicine.medical_specialtyLipopolysaccharidemedicine.medical_treatmentBiophysicsInflammationBiochemistryArticleSepsis03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineAnimalsRats Long-EvansMolecular BiologyInflammationSeptic shockbusiness.industryCell Biologymedicine.diseaseShock SepticMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureEndocrinologyCytokinechemistry030220 oncology & carcinogenesisShock (circulatory)Pituitary GlandCytokinesTumor necrosis factor alphamedicine.symptomInflammation Mediatorsbusiness
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Impact of antigen presentation on TCR modulation and cytokine release: implications for detection and sorting of antigen-specific CD8+ T cells using …

2002

Abstract Soluble MHC class I molecules loaded with antigenic peptides are available either to detect and to enumerate or, alternatively, to sort and expand MHC class I-restricted and peptide-reactive T cells. A defined number of MHC class I/peptide complexes can now be implemented to measure T cell responses induced upon Ag-specific stimulation, including CD3/CD8/ζ-chain down-regulation, pattern, and quantity of cytokine secretion. As a paradigm, we analyzed the reactivity of a Melan-A/MART-1-specific and HLA-A2-restricted CD8+ T cell clone to either soluble or solid-phase presented peptides, including the naturally processed and presented Melan-A/MART-1 peptide AAGIGILTV or the peptide ana…

Cytotoxicity ImmunologicT cellCD8 AntigensImmunologyAntigen presentationReceptors Antigen T-CellDown-RegulationEpitopes T-LymphocyteCD8-Positive T-LymphocytesMHC class IHLA-A2 AntigenmedicineImmunology and AllergyCytotoxic T cellHumansAntigen PresentationPeptide analogbiologyAntigen processingMembrane ProteinsMHC restrictionMolecular biologymedicine.anatomical_structureAmino Acid SubstitutionReceptor-CD3 Complex Antigen T-Cellbiology.proteinMutagenesis Site-DirectedCytokinesCD8Journal of immunology (Baltimore, Md. : 1950)
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Indicaxanthin inhibits NADPH oxidase (NOX)-1 activation and NF-κB-dependent release of inflammatory mediators and prevents the increase of epithelial…

2014

Dietary redox-active/antioxidant phytochemicals may help control or mitigate the inflammatory response in chronic inflammatory bowel disease (IBD). In the present study, the anti-inflammatory activity of indicaxanthin (Ind), a pigment from the edible fruit of cactus pear (Opuntia ficus-indica, L.), was shown in an IBD model consisting of a human intestinal epithelial cell line (Caco-2 cells) stimulated by IL-1β, a cytokine known to play a major role in the initiation and amplification of inflammatory activity in IBD. The exposure of Caco-2 cells to IL-1β brought about the activation of NADPH oxidase (NOX-1) and the generation of reactive oxygen species (ROS) to activate intracellular signal…

Cell Membrane PermeabilityPyridinesPyridinemedicine.medical_treatmentInterleukin-1betaMedicine (miscellaneous)Nitric Oxide Synthase Type IIIndicaxanthinNADPH OxidaseInflammatory bowel diseaseIntestinal absorptionAntioxidantschemistry.chemical_compoundSettore BIO/10 - BiochimicaInflammation MediatorCaco-2 CellNutrition and DieteticsNADPH oxidasebiologyNF-kappa BNADPH Oxidase 1OpuntiaCell biologyBetaxanthinsCytokineNADPH Oxidase 1EnterocyteAntioxidantmedicine.symptomInflammation MediatorsReactive Oxygen SpecieIndicaxanthinHumanRedox-active phytochemicalInflammationIn vitro modelmedicineHumansIndicaxanthin Betalain pigments Inflammatory bowel disease Redox-active phytochemicalsInterleukin 8Inflammationbusiness.industryInterleukin-6Interleukin-8NADPH OxidasesInflammatory Bowel DiseasesEnzyme ActivationEnterocyteschemistryIntestinal AbsorptionCaco-2Cyclooxygenase 2BetaxanthinFruitImmunologybiology.proteinCaco-2 CellsbusinessReactive Oxygen SpeciesThe British journal of nutrition
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Macrophage migration inhibitory factor is critically involved in basal and fluoxetine-stimulated adult hippocampal cell proliferation and in anxiety,…

2011

Intensive research is devoted to unravel the neurobiological mechanisms mediating adult hippocampal neurogenesis, its regulation by antidepressants, and its behavioral consequences. Macrophage migration inhibitory factor (MIF) is a pro-inflammatory cytokine that is expressed in the CNS, where its function is unknown. Here, we show, for the first time, the relevance of MIF expression for adult hippocampal neurogenesis. We identify MIF expression in neurogenic cells (in stem cells, cells undergoing proliferation, and in newly proliferated cells undergoing maturation) in the subgranular zone of the rodent dentate gyrus. A causal function for MIF in cell proliferation was shown using genetic (M…

Receptors SteroidStem-Cellsanimal diseasesmedicine.medical_treatmentHippocampusExpressionHippocampal formationHippocampusSubgranular zonememoryMice0302 clinical medicineConditioning PsychologicalCyclin D2Rat Dentate GyrusMice KnockoutNeurons0303 health sciencesMicroscopy ConfocalChronic StressMifNeurogenesisBrainFearrespiratory systemanxietyPsychiatry and Mental healthC-Reactive ProteinCytokinemedicine.anatomical_structuredepressionAntidepressive Agents Second-GenerationStem cellPsychologyAnimal-ModelNeurogenesisSpatial BehaviorNerve Tissue Proteinschemical and pharmacologic phenomena03 medical and health sciencesCellular and Molecular Neurosciencemedicineotorhinolaryngologic diseasesAnimalsRats WistarMaze LearningMacrophage Migration-Inhibitory FactorsMolecular BiologyCell Proliferation030304 developmental biologyMemory DisordersDentate gyrusfluoxetineFactor Mifbiological factorsRatsDisease Models AnimalAcoustic StimulationBromodeoxyuridineMacrophage migration inhibitory factorCorticosteroneNeuroscience030217 neurology & neurosurgery
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IL-10 down-regulates T cell activation by antigen-presenting liver sinusoidal endothelial cells through decreased antigen uptake via the mannose rece…

1998

SUMMARYOur study demonstrates that antigen-presenting liver sinusoidal endothelial cells (LSEC) induce production of interferon-gamma (IFN-γ) from cloned Th1 CD4+ T cells. We show that LSEC used the mannose receptor for antigen uptake, which further strengthened the role of LSEC as antigen-presenting cell (APC) population in the liver. The ability of LSEC to activate cloned CD4+ T cells antigen-specifically was down-regulated by exogenous prostaglandin E2 (PGE2) and by IL-10. We identify two separate mechanisms by which IL-10 down-regulated T cell activation through LSEC. IL-10 decreased the constitutive surface expression of MHC class II as well as of the accessory molecules CD80 and CD86 …

Liver cytologyT cellT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsDown-RegulationReceptors Cell SurfaceBiologyLymphocyte ActivationDinoprostoneMiceAntigenAntigens CDmedicineImmunology and AllergyAnimalsLectins C-TypeCD86Antigen PresentationMice Inbred BALB CMembrane GlycoproteinsHistocompatibility Antigens Class IIOriginal ArticlesInterleukin-10Interleukin 10medicine.anatomical_structureMannose-Binding LectinsLiverImmunologyB7-1 AntigenCytokinesFemaleB7-2 AntigenEndothelium VascularMannoseCD80Mannose receptorMannose ReceptorClinical and experimental immunology
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In Vitro Cytokine Production by HLA-B8, DR3 Positive Subjects

1994

It is well known that healthy subjects carrying the HLA-B8,DR3 haplotype may show an impairment of immune system, the T cells being the most affected. To gain insight into the mechanism(s) of the impairment displayed by these subjects, efforts have been centered on the study of in vitro cytokine production because of the pivotal role played by these mediators in the activation and control of several immune functions. The available results indicate that the ability to several immune functions. The available results indicate that the ability to produce interleukin-1 (IL-1), IL-2 and the soluble form of its receptor (sIL-2R) is impaired in HLA-B8,DR3 positive healthy subjects. To better charac…

AdultMalemusculoskeletal diseasesInterleukin 2Cellular immunitymedicine.medical_treatmentImmunologyAutoimmunityPeripheral blood mononuclear cellHLA-B8 AntigenInterferon-gammaHLA-DR3 AntigenImmune systemimmune system diseasesmedicineHumansImmunology and AllergyInterferon gammaPhytohemagglutininsInterleukin 6Cells CulturedInterleukin 4biologyInterleukin-6Receptors Interleukin-2Middle AgedRecombinant ProteinsCytokineAntibody FormationImmunologyLeukocytes Mononuclearbiology.proteinCytokinesInterleukin-2FemaleInterleukin-4medicine.drugAutoimmunity
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CD14+CD16+ monocytes in coronary artery disease and their relationship to serum TNF-α levels

2004

SummaryMonocytes play a central role in the inflammatory disease atherosclerosis. CD14+CD16+ monocytes are considered proinflammatory monocytes, as they have an increased capacity to produce proinflammatory cytokines, such as TNF-α, and are elevated in various inflammatory diseases. We hypothesized that patients with coronary artery disease (CAD) have increased levels of CD14+CD16+ monocytes, and that CD14+CD16+ monocytes are associated with inflammation markers. We investigated CD14+CD16+ monocytes in 247 patients with CAD and 61 control subjects using flow cytometry. In addition serum concentrations of TNF-α, IL-6, and Hs-CRP were assessed. Patients with CAD had higher levels of CD14+CD16…

MaleArteriosclerosismedicine.medical_treatmentCD14Lipopolysaccharide ReceptorsInflammationCell SeparationCoronary Artery DiseaseCD16MonocytesBody Mass IndexProinflammatory cytokineCoronary artery diseaseRisk FactorsOdds RatioHumansMedicineAgedInflammationAnalysis of VarianceInterleukin-6Tumor Necrosis Factor-alphabusiness.industryMonocyteReceptors IgGAntibodies MonoclonalHematologyMiddle AgedFlow Cytometrymedicine.diseaseLogistic ModelsCytokinemedicine.anatomical_structureCase-Control StudiesImmunologyFemaleTumor necrosis factor alphamedicine.symptombusinessThrombosis and Haemostasis
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